ライフサイエンスコーパス: hive

1 human brain tissue with HIV-1 encephalitis (HIVE).

2 t, neurological damage and HIV encephalitis (HIVE).

3 nt (SCID) mouse model of HIV-1 encephalitis (HIVE).

4 topathologic features of HIV-1 encephalitis (HIVE).

5 n human immunodeficiency virus encephalitis (HIVE).

6 an animal model of HIV-induced encephalitis (HIVE).

7 d human immunodeficiency virus encephalitis (HIVE).

8 mmatory condition known as HIV encephalitis (HIVE).

9 human immunodeficiency virus-1 encephalitis (HIVE).

10 tiviral T-cell response in HIV encephalitis (HIVE).

11 human brain tissues with HIV-1 encephalitis (HIVE).

12 led monocyte migration into the brain during HIVE.

13 an underlying cause of BBB impairment during HIVE.

14 il than did age-matched bees confined to the hive.

15 Honey bees begin life working in the hive.

16 ed the effects of regular HAART treatment on HIVE.

17 bees with 2D barcodes in a small observation hive.

18 values similar to those observed in cases of HIVE.

19 caudate and putamen of SCID mice, generating HIVE.

20 cking and homeostasis in the pathogenesis of HIVE.

21 nonobese diabetic (NOD)-SCID mouse model of HIVE.

22 lly restricted to a narrow sector around the hive.

23 d in brain tissue from an adult patient with HIVE.

24 both neurons and microglia in patients with HIVE.

25 rtical gray matter in patients who died with HIVE.

26 lesions in the brain of children with severe HIVE.

27 ving only brood, food, and few adults in the hive.

28 (HIVE); D) Infected with substantial NCI and HIVE.

29 vae spores can be distributed throughout the hive.

30 uropsychologically impaired subjects without HIVE.

31 te the cognitive impairments associated with HIVE.

32 on in the mechanisms of neurodegeneration in HIVE.

33 spectively, where we placed 120 experimental hives.

34 the social environments of small observation hives.

35 xidant derived from the propolis of honeybee hives.

36 "bioethics," they break out in intellectual hives.

37 cterized by relapsing appearance of pruritic hives.

38 ion of bees' behaviours in small observation hives.

39 behaviour were determined using observation hives.

40 ver, reported nausea, vomiting, and systemic hives 20 to 30 minutes after ingestion of antipasto made

41 ut metabolism of coumaphos, a widely used in-hive acaricide, by approximately 60%.

42 Analyses of brain tissues from HIVE, AD and FTD patients showed that PINCH is increased

43 Young bees perform nursing duties within the hive and have high Vg and low JH; as older bees transiti

44 ain endothelial cells in the mouse model for HIVE and human HIVE brains featuring mononuclear cell in

45 fferentiating between pesticide exposures in-hive and in agricultural fields.

46 ulted in decreased survivorship, both in the hive and in laboratory experiments in which bees were ex

47 that can be transported by honey bees to the hive and incorporated into the honey.

48 bees with repeated opportunities to view the hive and landscape features from different viewpoints, s

49 ethism, in which young workers remain in the hive and perform tasks there, whereas old workers perfor

50 es hippocampal synaptic impairment in murine HIVE and provide a rationale for its use in infected hum

51 eled IDV-NP was readily observed in areas of HIVE and specifically in brain subregions with active as

52 changes in cell cycle proteins occur in both HIVE and the simian model and that these changes have fu

53 ignificant increase in E2F1 and ppRb in both HIVE and the simian model.

54 py/allergies, asthma, eczema, hay fever, and hives and childhood/adolescent leukemia, acute lymphobla

55 ic honey samples collected directly from the hives and find that a large proportion (37%) of Manuka h

56 dominant vibratory urticaria have localized hives and systemic manifestations in response to dermal

57 HIV-1 encephalitis (HIVE) and its associated dementia can occur in up to 20%

58 untered by foraging honeybees and within the hive, and are additive with combined application.

59 Efficacy (itch severity, hive, and urticaria activity scores) was evaluated at we

60 ch to survey microflora in CCD hives, normal hives, and imported royal jelly.

61 More than 1 million hives are transported to California each year just to po

62 human immunodeficiency virus-1 encephalitis, HIVE) are associated with oxidative stress and inflammat

63 In contrast, Li restored HIVE-associated loss of microtubule-associated protein-2

64 that the increase in JH that influences the hive bee-forager transition may cause many of these chan

65 f stores of food that are consumed by within-hive bees that convert stored pollen and honey into roya

66 exposome by analyzing residues from live in-hive bees, stored pollen, and wax in migratory colonies

67 P is freely available for download at http://hive.biochemistry.gwu.edu/dna.cgi?cmd=phylosnp.

68 The database is available at hive.biochemistry.gwu.edu/review/codon .

69 IDV levels and reduced HIV-1 replication in HIVE brain regions.

70 findings was evaluated in HIV-encephalitis (HIVE) brain samples in which decreased levels of MCP-2 a

71 cells in the mouse model for HIVE and human HIVE brains featuring mononuclear cell infiltration acro

72 rty-nine plant taxa were recorded from three hives but only ten at greater than 1%.

73 human immunodeficiency virus encephalopathy (HIVE) by immunohistochemistry in an effort to gain insig

74 y of pesticide distribution on a comb in the hive can be driven by worker behaviors.

75 t direct experience of the world outside the hive causes mushroom body neuropil growth in bees.

76 A large proportion of CD16+ MPs in HIVE CNS tissue were PCNA+, but do not appear to be prol

77 ome endothelial cells and ependymal cells in HIVE CNS.

78 rved in cerebellar tissue from patients with HIVE compared to HIV-seronegative patients or HIV-1-infe

79 of children with severe HIV-1 encephalitis (HIVE) compared with children with mild HIVE or non-AIDS

80 pediatric patients with HIV-1 encephalitis (HIVE) compared with those without HIVE, or that were HIV

81 equency with which these pesticides occur as hive contaminants and suggests that they present a great

82 nces on various outdoor routes from the same hive could be considerably different.

83 ated Virtual Environment-Codon Usage Tables (HIVE-CUTs), to present and analyse codon usage tables fo

84 th substantial NCI without HIV encephalitis (HIVE); D) Infected with substantial NCI and HIVE.

85 delaying the transition from working in the hive (e.g., brood care, or "nursing") to foraging.

86 A nurturing hive environment and a mutualistic relationship with pla

87 encephalitis (SHIVE) with several classical HIVE features that include astrocyte infection.

88 , and beekeeping in skep, log, box, and tree hives flourished to meet the demand.

89 A worker honeybee performs tasks within the hive for approximately the first 3 weeks of adult life.

90 ion, because foragers bring food back to the hive for storage rather than eating it themselves.

91 ng repeatedly to collect food outside of the hive for the remainder of its 5-6 week life.

92 he rate of resolution (defined as absence of hives for at least 1 year with no treatment) and the ass

93 These can then be introduced into queenless hives for natural mating or insemination, both of which

94 in two millennia, beekeeping with horizontal hives had spread throughout the Mediterranean.

95 ean+/-SE: 6.61 +/- 0.88 ppb clothianidin per hive) had seven times greater concentrations than nectar

96 Traditional hive has no negative effect on quality factors of honey

97 l agriculture, but dramatic losses of entire hives have been reported in numerous countries since 200

98 formed using brain tissue from patients with HIVE, HIV-1 infection without encephalitis, and seronega

99 g human immunodeficiency virus encephalitis (HIVE); however, the basis of these permeability changes

100 lpha has a major role in the pathogenesis of HIVE in mice and is likely important in the development

101 two decades it has encountered pesticides in-hive in the form of acaricides to control Varroa destruc

102 otinoids brought back in pollen to honey bee hives in arable landscapes was from wildflowers, not cro

103 ated neuronal pathways strongly dominated in HIVE, including GABA receptors, glutamate signaling, syn

104 In the frontal cortex of patients with HIVE, increased levels of CDK5 and p35 expression were a

105 co of 121 pesticide contaminants of American hives into the active pocket of CYP9Q1, a broadly substr

106 n active component of propolis from honeybee hives, is known to have antimitogenic, anticarcinogenic,

107 t with chronic urticaria that presented with hives lasting at least 6 weeks between 2013 and 2015 at

108 r relationship to SIV/HIV encephalitis (SIVE/HIVE) lesions and SIV-infected cells.

109 , and the mutant biofilms appeared loose and hive-like, whereas the biofilms of the wild type were sm

110 in the start of foraging of 3.3 h, and whole-hive locomotor-activity rhythms were delayed by an avera

111 le the contribution of pesticides to current hive loss rates is debated, remarkably little is known r

112 We examined how hive mates interpret these dances.

113 eybees, employed foragers recruit unemployed hive mates to food sources by dances from which a human

114 f NP(EO)3-13 and OP(EO)3-13 oligomers in bee hive matrices.

115 ulation of immunosuppressive IDO activity in HIVE may enhance the generation of HIV-1-specific CTLs,

116 ation might improve NCI, whereas NCI without HIVE may not respond in kind; array results suggest that

117 Thus focal inflammation in brain tissue with HIVE may up-regulate neuronal FKN levels, which in turn

118 ent of astrocyte and microglia activation in HIVE mice (10-fold and 16-fold, respectively, compared w

119 ion were reduced by 14.1, 29.5, and 45.3% in HIVE mice compared with sham-injected or control animals

120 d with untreated or control antibody-treated HIVE mice during water radial arm maze behavioral testin

121 By day 15, the CA2 region of HIVE mice expressed 3.8- and 2.6-fold less NF and SP tha

122 Here, we show that the neural damage in HIVE mice extends beyond the basal ganglia and is associ

123 Here, CEP-1347 treatment of HIVE mice showed a dose-dependent reduction in microglio

124 Memantine-treated HIVE mice showed significant improvements in synaptic fu

125 g-term potentiation in hippocampal slices of HIVE mice that were restored by Li.

126 ficantly in the CA2 hippocampal subregion of HIVE mice with limited neuronal apoptosis.

127 itive MDMs were unaltered by Li treatment of HIVE mice.

128 he CA1 region of hippocampal brain slices of HIVE mice.

129 s of dendritic arborization in the brains of HIVE mice.

130 1(+) MDMs) and enhanced neuroinflammation in HIVE mice.

131 HIV-1 encephalitis (HIVE) mice, where human virus-infected monocyte-derived

132 For human HIVE, microglial activation and virus infection correlat

133 ammatory and neuroprotective responses in an HIVE model of human disease and as such warrants further

134 munodeficiency virus-1 (HIV-1) encephalitis (HIVE) models.

135 The human PBL-NOD-SCID HIVE mouse provides a new tool for studies of cellular i

136 e human PBL to generate a human PBL-NOD-SCID HIVE mouse.

137 dy, blocking IFNalpha in a HIV encephalitis (HIVE) mouse model with intraperitoneal injections of IFN

138 e CD40/CD40L dyad plays an important role in HIVE neuroinflammation.

139 genomic approach to survey microflora in CCD hives, normal hives, and imported royal jelly.

140 escribes the direction and distance from the hive of a new food source, and this message is displaced

141 The hive of the honey bee is a suitable habitat for diverse

142 centration of all pesticide brought into the hive on that particular day, it is likely representative

143 Honeybees are known to fly a feeder-to-hive or hive-to-feeder vector according to whether or no

144 itis (HIVE) compared with children with mild HIVE or non-AIDS controls, whereas the frequency of CXCR

145 ounds (GH) and reared them in normal (within hives) or stressed (protein-deficient, asocial) conditio

146 ephalitis (HIVE) compared with those without HIVE, or that were HIV-1 seronegative.

147 This study measured part of the in-hive pesticide exposome by analyzing residues from live

148 d outputs publication-quality Sashimi plots, hive plots and structure plots, enabling better investig

149 and pollen carried by foragers returning to hives, preplanting and in-season soil samples, and wild

150 as observed in CNS tissue from patients with HIVE, relative to seronegative controls and patients wit

151 ropathological features of HIV encephalitis (HIVE) remains unclear.

152 To test this idea, an HIV-1 encephalitis (HIVE) rodent model was used where HIV-1-infected human m

153 In the HIVE SCID mice, a marked accumulation of murine MDM was

154 asures included Skindex-29, current itch and hives scores, total leukocyte histamine content (an indi

155 The type of hives significantly affected the moisture (p<0.01), redu

156 s overall experienced increased time delays, hive stay durations and a decreased number of transfer p

157 donors experienced reduced transfer delays, hive stay durations and an increased number of simultane

158 of patients affected by HIV-1 encephalitis (HIVE), suggesting an important role for the CD40/CD40L d

159 Expression in HIVE suggests that lowering brain HIV-1 replication migh

160 hives; the balanced allocation of workers to hive tasks and foraging; the recovery of a colony from d

161 n activity rhythms; young adult bees perform hive tasks with no daily rhythms, whereas older bees for

162 ight junction disruption is a key feature of HIVE that occurs in regions of histopathological alterat

163 -performance Integrated Virtual Environment (HIVE) that encapsulates Curated Short Read archive (CSR)

164 With HIVE the HIV-1 RNA load in brain tissue was three log(10

165 In HIVE, the transcripts for TLR3, IFN-beta, IDO, and viper

166 first forages and how this age varies among hives; the balanced allocation of workers to hive tasks

167 Third, immunohistochemical analyses of human HIVE tissue defined the relationships between astroglios

168 of pesticide location/deposition within the hive to compare with exposure levels estimated by averag

169 lly regulated transition from working in the hive to foraging that has been previously associated wit

170 lly regulated transition from working in the hive to foraging, which is associated with changes in th

171 asuring exposure of individual bees within a hive to pesticide is at least as difficult as assessing

172 neybees are known to fly a feeder-to-hive or hive-to-feeder vector according to whether or not they h

173 etic xenobiotics that frequently contaminate hives-two herbicides (atrazine and glyphosate) and three

174 and acceptability tests not observed due to hive types and locations.

175 tributes to the neurodegenerative process in HIVE via abnormal tau phosphorylation; thus, reducing CD

176 d, a SCID mouse model of HIV-1 encephalitis (HIVE) was used to determine in vivo monocyte blood-to-br

177 mbined immunodeficient (SCID) mouse model of HIVE, we determined the effects of regular HAART treatme

178 Using a murine model of HIVE, we investigated the effects of alcohol abuse on th

179 Using a model of HIVE, we investigated whether IDO inhibitor 1-methyl-d-t

180 dicators of honey from traditional and frame hives were within the criteria set by Codex Alimentarus

181 The transition from hive work to foraging has been shown to be socially regu

182 he age-related transition by honey bees from hive work to foraging is associated with an increase in

183 -related transition by adult honey bees from hive work to foraging is associated with changes in mess

184 lore how increased mortalities of larvae, in-hive workers, and foragers, as well as reduced egg-layin