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1 Mobilization of TRPC6 was also evoked by L-homocysteic acid.
2 nea in response to the glutamate agonist D,L-homocysteic acid.
5 proteasome, in cortical neurons treated with homocysteic acid, and in the striatum of R6/2 transgenic
7 ites in the PVN with a microinjection of d,l-homocysteic acid (DLH) elicited either pressor or depres
8 eral PAG at sites where microinjection of DL-homocysteic acid (DLH) evoked increases or decreases res
9 phoretic application of a high current of dl-homocysteic acid (DLH) induced a vagally mediated bradyc
10 scharge (PND) and AP] caused by injecting dl-homocysteic acid (DLH) into the ventral respiratory grou
11 ctive alpha1-agonist) on spontaneous and D,L-homocysteic acid (DLH)-evoked unit activity were examine
12 ithout a change in blood pressure, using D,L homocysteic acid (DLH, 0.2 M) microinjections (50 nl) fr
13 g injections of the excitatory amino acid DL-homocysteic acid (DLH; 5-20 mM, 2-6 nl) from single- or
14 mA, 100 Hz, 0.2 ms) and chemically (10 mM DL-homocysteic acid, DLH), while the spontaneous activity o
15 amino acid (EAA) neurotransmitters, such as homocysteic acid (HCA) and cysteine sulfinic acid (CSA),
16 stral infraradiata cortex, injections of D,L-homocysteic acid in the dorsal subdivision of rostral in
17 ons (50-200 nl) of the glutamate agonist D,L-homocysteic acid into area infraradiata evoked both incr
19 pharmacological stimulation (bicuculline/D,L-homocysteic acid mixture) of the main olfactory bulb (MO
20 ad no effect on the excitations evoked by DL-homocysteic acid (n = 4), NMDA (n = 4) or (AMPA; n = 2).
21 were activated either by microinjecting D,L-homocysteic acid or by disinhibition by microinjecting b
23 ses in this study, similar injections of D,L-homocysteic acid were made into area infraradiata of unr
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