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1 PAR-delta), which is implicated in bile acid homoeostasis.
2 ion and a modulator of intestinal epithelial homoeostasis.
3 s body fat redistribution and alters glucose homoeostasis.
4 cell biology, embryo patterning, and tissue homoeostasis.
5 ion history of a tissue SC during its normal homoeostasis.
6 ce of STAT5 tetramers in maintaining NK cell homoeostasis.
7 ions and mutations can interfere with tissue homoeostasis.
8 eted hormone that plays a key part in energy homoeostasis.
9 bone turnover, and abnormalities in calcium homoeostasis.
10 nd support teeth in situ and preserve tissue homoeostasis.
11 e to a meal, is essential for normal glucose homoeostasis.
12 ) could be important determinants of glucose homoeostasis.
13 on of iron absorption and of whole-body iron homoeostasis.
14 highlight a role of melanocortins in weight homoeostasis.
15 atment, which by itself could affect glucose homoeostasis.
16 proliferate is critical to tissue repair and homoeostasis.
17 educed fat mass and improved glucose/insulin homoeostasis.
18 catenin signalling is crucial for intestinal homoeostasis.
19 ain development requires a fine-tuned copper homoeostasis.
20 trols food consumption to maintain metabolic homoeostasis.
21 reatic cancer cells and was essential for ER homoeostasis.
22 s necessary to dampen activation and restore homoeostasis.
23 ransduction involved in regulating cell wall homoeostasis.
24 of energy balance, thermogenesis and glucose homoeostasis.
25 P1 and WT1) in connective tissue maintenance/homoeostasis.
26 nges during embryonic development and tissue homoeostasis.
27 olic process critical for cell viability and homoeostasis.
28 des and is known to influence systemic lipid homoeostasis.
29 replenished by stem cells to maintain tissue homoeostasis.
30 p1), a GTPase is essential for mitochondrial homoeostasis.
31 a role for HTT on dynamin 1 function and ER homoeostasis.
32 f CNP/NPR-C signaling in preserving vascular homoeostasis.
33 ammalian retina, a process required for disc homoeostasis.
34 atory resolution that re-establish pulmonary homoeostasis.
35 inflammatory responses and improve metabolic homoeostasis.
36 rtant role for regulation of telomere length homoeostasis.
37 y pathway is a major determinant of cellular homoeostasis.
38 role in the regulation of arousal and stress homoeostasis.
39 their complex functions and effects on body homoeostasis.
40 ion, a key cytokine in normal colonic tissue homoeostasis.
41 essary for maintaining normal blood pressure homoeostasis.
42 state of vulnerability to poor resolution of homoeostasis after a stressor event and is a consequence
45 mportant hormone regulator of cardiovascular homoeostasis and an important biomarker for heart failur
50 This may be required to maintain protein homoeostasis and deliver metabolite intermediates for bi
51 veral new factors that promote mitochondrial homoeostasis and demonstrate that the UPR(mt), as curren
53 ribes how Breg cells are critical in humoral homoeostasis and may have implications for the regulatio
54 nown as programmed cell death, is central to homoeostasis and normal development and physiology in al
56 ediated SUMOylation of SHP in maintaining BA homoeostasis and protecting from the BA hepatotoxicity.
57 1 could be important determinants of glucose homoeostasis and provide further evidence for the possib
58 tes IgA production to maintain gut microbial homoeostasis and restrain IL-1alpha-dependent colitis an
59 ein as a regulator of cellular immunological homoeostasis and suggest cellular prion protein as a nov
60 show that LTA is needed for divalent cation homoeostasis and that its absence has severe effects on
61 ramifications for normal tissue development, homoeostasis and the physiological functions of various
63 importance of the microbiota for intestinal homoeostasis, and discuss the similarity between inflamm
64 racellular proteolytic activity in capillary homoeostasis, and identify ADAMTS1 as a marker of activa
65 Hippo pathway plays a central role in tissue homoeostasis, and its dysregulation contributes to tumor
66 utamate are necessary to maintain epithelial homoeostasis, and provision of glutamate drives disrupti
67 ic development, cell differentiation, tissue homoeostasis, and removal of damaged and harmful cells f
68 ttention to aspects of vascular development, homoeostasis, and response to environmental effects.
69 se cytokines function to maintain intestinal homoeostasis, and under what circumstances they contribu
71 s in membrane trafficking and altered Ca(2+) homoeostasis are common features in many lysosomal stora
72 ple, variation in genes involved in synaptic homoeostasis are implicated in autism spectrum disorder
74 auxin oxidation is more important for auxin homoeostasis at lower hormone concentrations, whereas au
76 alance is vital for cell survival and tissue homoeostasis because imbalanced production of reactive o
77 stitutive process required for proper tissue homoeostasis but can be rapidly regulated by a variety o
78 n important function in maintaining cellular homoeostasis by assisting the folding of many proteins,
79 factor ETS-related gene (ERG) promotes liver homoeostasis by controlling canonical TGFbeta-SMAD signa
80 sphatases in mammalian cells, maintains cell homoeostasis by counteracting most of the kinase-driven
81 /GH3.6 and DFL2/GH3.10, which regulate auxin homoeostasis, by binding directly to the TA box in each
83 s, which is critically important to cellular homoeostasis, can be achieved at the level of gene expre
84 s as a source of Wnt in bone development and homoeostasis, complementing their known function as targ
87 egulate cardiac myocyte hypertrophy, calcium homoeostasis, energetics, and cell survival, and process
89 itches the mechanism for maintaining protein homoeostasis from a chaperone-based approach to an appro
93 inflammatory molecules, disrupted glutamate homoeostasis, impaired action of antipsychotics, and dev
94 , we find that EGF signalling alters protein homoeostasis in adults by increasing UPS activity and po
95 pports sodium/potassium pump-dependent ionic homoeostasis in areas of central nervous system injury.
98 l insights into the complex role of dopamine homoeostasis in human disease, and understanding of the
101 inal fat relates to abnormalities in glucose homoeostasis in obese adolescents with prediabetes.
103 n of PE may contribute to the improved lipid homoeostasis in rats on diets high in cholesterol and li
105 ation from HSCs by maintaining immunological homoeostasis in the bone marrow microenvironment, both i
107 poparathyroidism results in impaired mineral homoeostasis, including hypocalcaemia and hyperphosphata
108 rt-term and long-term departures from immune homoeostasis, inhibition of appropriate pathogen recogni
109 cell (SC) proliferation is central to tissue homoeostasis, injury repair, and cancer development.
110 expression of factors crucial for colorectal homoeostasis is affected by physiologic differences in S
113 ophagy to the maintenance of normal cellular homoeostasis, its changes in neurodegenerative disorders
114 adipose tissue dysfunction, insulin-glucose homoeostasis, lipid disturbances, and cardiovascular dis
115 r, and that interfering with telomere length homoeostasis may be one of the mechanism(s) by which oxi
116 failure of dopamine receptor and transporter homoeostasis might underlie the pathophysiology of this
118 ctories of fasting and 2-h postload glucose, homoeostasis model assessment (HOMA) insulin sensitivity
119 ed fasting blood samples (for calculation of homoeostasis model assessment of insulin resistance [HOM
120 all macroalgae and that is important for ion homoeostasis, nutrient uptake and O2/CO2 exchange throug
121 ed below-ground allocation, and the apparent homoeostasis of radial growth, as ca increases today.
122 e channel that has a key role in maintaining homoeostasis of the airway surface liquid layer in the l
124 e cells (PSCs) can perturb the biomechanical homoeostasis of the tumour microenvironment to favour ca
126 t checkpoint for maintaining oligodendrocyte homoeostasis, pointing to a previously uncharacterized E
127 ase and Parkinson's disease, changes in iron homoeostasis result in altered cellular iron distributio
128 thways include dysfunction in global protein homoeostasis resulting from abnormal protein aggregation
129 o chronic cell stress and imbalance of ionic homoeostasis, resulting in axonal and neuronal death.
130 st cells (MCs) play a central role in tissue homoeostasis, sensing the local environment through nume
131 tissues beyond those involved in mineral ion homoeostasis should remain an important focus of researc
132 extrinsic abnormal change affecting calcium homoeostasis stimulating production of parathyroid hormo
133 sms responsible for abnormalities in calcium homoeostasis, the differential diagnosis of hypercalcaem
134 chemical process that is critical for tissue homoeostasis, these results improve our fundamental unde
135 ) regulatory T cells (Tregs) maintain immune homoeostasis through mechanisms that remain incompletely
136 uggests that BAT activation improves glucose homoeostasis through several mechanisms, which could poi
138 p is critical in maintaining telomere length homoeostasis through telomere guanine damage repair, and
139 values, an indicator of neuronal cholesterol homoeostasis, were significantly higher than post-saline
140 gy homeostasis, which acts to restore energy homoeostasis whenever cellular energy charge is depleted
141 e in maintaining whole-body calcium (Ca(2+)) homoeostasis, which is primarily mediated by altering th
142 microbiome maintains a state of basal immune homoeostasis, which modulates immune responses to microb
143 complex regulatory networks, which maintain homoeostasis while accurately distinguishing pathogenic
144 iption at puberty, alters luminal epithelial homoeostasis, yet remains deficient in homologous recomb
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