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1 r outflow reconstruction was achieved with a homograft.
2 also grafted to the left carotid artery as a homograft.
3 egarding evolving pathology in the pulmonary homograft.
4 ss-clamp times, and insertion of a pulmonary homograft.
5 ncludes augmentation of the aortic arch with homograft.
6 fts and in the recanalized lumen of occluded homografts.
7 monstrated in 10 of 10 allografts and 5 of 6 homografts.
8 owth and tumor weight of murine liver cancer homografts.
10 24 patients aged 40+/-11 years) or an aortic homograft (23 patients aged 37+/-11 years) for rereplace
11 astolic diameter was similar in both groups (homografts, 5.0+/-0.9 cm; autografts, 5.2+/-0.6 cm; P=NS
12 roke volumes were maintained in both groups (homografts 67+/-15 mL, autografts 67+/-16 mL; P=NS).
13 ypass graft and aortic root replacement with homograft and 6 weeks of intravenous ampicillin and gent
14 site valved conduits made from cryopreserved homograft and polytetrafluoroethylene (PTFE) in 66 cases
15 ter MVR is higher than after AVR; after AVR, homografts and bioprosthesis have similar rates of SVD.
18 ne-to-one ratio to receive an autograft or a homograft aortic root replacement in one centre in the U
21 fied as O. turbata (four from the blood of a homograft aortic valve-associated endocarditis patient a
27 l AVR, 10.9% a bioprosthesis AVR, and 3.5% a homograft AVR, with Ross patients being significantly yo
31 ars in patients >5 years), with younger age, homograft conduit, conduit diameter < or =10 mm, diagnos
32 ilure (valve explant or late death), and (3) homograft dysfunction (homograft insufficiency or homogr
35 ncreased to a similar amount in both groups (homografts: end-diastolic volume 145+/-34 mL, end-systol
37 nd points included (1) patient survival, (2) homograft failure (valve explant or late death), and (3)
38 donor warm ischemic time as risk factors for homograft failure and dysfunction, whereas, Z: value <2
42 reedom from patient death or reoperation for homograft failure was 82% +/- 7% at 1000 days and 77% +/
46 an evolution away from use of cryopreserved homografts for LVOT reconstructions except when needed f
47 Among 205 patients receiving cryopreserved homografts for reconstruction of the RVOT between Novemb
49 Freedom from reoperation was 93+/-4% in the homograft group versus 100+/-0% in the freestyle group a
54 For groups with FGS, long-surviving renal homografts had a significantly smaller mean glomerular d
58 septal hypertrophy in 2, overlying pulmonary homograft in 1, and overlying ventricular septal defect
59 We also compared all procedures except the homograft in a matched population of young adults, where
60 time, smaller homograft size, use of aortic homograft in the older patient, and extracardiac operati
64 , one surgeon implanted cryopreserved valved homografts into 149 patients--65 since December 1988.
65 th either a pulmonary autograft or an aortic homograft leads to a degree of persistent RV longitudina
66 r, there is concern that over time pulmonary homografts may develop stenosis secondary to low-grade i
68 ergo aortic valve replacement with an aortic homograft or a pulmonary autograft were followed up echo
69 there was evidence of narrowing of the whole homograft or distal suture line in 14 of 15 patients, wi
70 een patients who had undergone either aortic homograft or pulmonary autograft surgery were studied be
71 A total of 20 patients received a stentless (homograft or Toronto) valve, and 13, a stented valve.
72 (PTFE) in 66 cases (54 pulmonary, 12 aortic homografts), other valved conduits in 14, and unvalved P
75 erior after the Ross procedure, the need for homograft reinterventions is an issue to take into accou
76 ng surgical replacement for a putative total homograft-related structural failures rate of 11% at 5 1
77 ivariate analysis of patient-, surgery-, and homograft-related variables did not reveal any significa
78 nts undergoing either pulmonary autograft or homograft replacement of the aortic valve as part of a p
81 trate significantly higher calcium scores in homograft roots than freestyle at 1.5 years (P=0.02), 2
82 vely) were reduced postoperatively (P<0.05) (homografts: SE 1.5+/-0.4 versus 2.3+/-0.6 cm, SR 6.8+/-2
83 ge, longer donor warm ischemic time, smaller homograft size, use of aortic homograft in the older pat
87 eoperation for rapidly progressive pulmonary homograft stenosis; in all 4, there was macroscopic and
88 ted: mechanical valves, cryopreserved aortic homograft, stented heterograft, and pulmonary autograft
89 response to the long-term results of aortic homografts, stentless porcine valves were introduced as
91 ative inflammatory reaction to the pulmonary homograft that leads to extrinsic compression and/or shr
92 iography protocol: in surviving patients and homografts, three valved conduits were judged to have se
96 efficacy of angioplasty of obstructed RV-PA homografts using ultra-noncompliant (UNC) or ultrahigh-p
99 olate from the second patient and one of the homograft valve isolates differ from O. turbata and C. h
100 olates from the first patient and six of the homograft valve isolates represent a single clone of O.
101 from cerebrospinal fluid, and one each from homograft valve, lip wound, and pilonidal cyst) were com
103 ored the long-term function of cryopreserved homograft valves used for reconstruction of the right ve
105 carditis patient and seven from contaminated homograft valves) and one CDC group A-3 strain from the
106 nts were prospectively randomized to undergo homograft versus freestyle total aortic root replacement
107 ong-term degree and rate of calcification of homografts versus Medtronic freestyle aortic roots to de
108 hocardiographic examination of the pulmonary homograft was performed immediately after surgery, then
110 al comparing pulmonary autografts and aortic homografts who have had previous aortic valve replacemen
112 ute hemodynamic changes after angioplasty of homografts with UNC balloons included significantly redu
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