ライフサイエンスコーパス: homoplasy

1 level of convergent and parallel evolution (homoplasy).

2 ared ancestry (homology) versus convergence (homoplasy).

3 ons at the same site and of reversion (i.e., homoplasy).

4 confirmed that these shared STs were due to homoplasy.

5 s problematic due to high mutation rates and homoplasy.

6 dex (CI) provide relatively good measures of homoplasy.

7 of these behaviors suggests a high degree of homoplasy.

8 accounting for copy-number polymorphism and homoplasy.

9 group of monkeys argued to display extensive homoplasy.

10 of 5-HT-ir neurons exhibits a great deal of homoplasy.

11 , at short evolutionary distances, pervasive homoplasy.

12 e) is characterized by extreme morphological homoplasy.

13 ssed as well as the concept of developmental homoplasy.

14 ustly than expected from stochastic sequence homoplasies.

15 Such characters are called homoplasies.

16 taxa that are later correctly identified as homoplasy after more taxa are added.

17 llites with respect to mutational models and homoplasy among alleles.

18 lineages to iteratively evolve developmental homoplasies and convergent ecological specializations, p

19 at fail to discriminate between homology and homoplasy and further implies virus-like rates of nucleo

20 lear evidence of interspecific allele length homoplasy and microsatellite mutational saturation was o

21 onsiderable amounts of cryptic variation and homoplasy and significantly aid in our understanding of

22 etic analyses reveal that unsuspected dental homoplasy and the detrimental effects of missing data co

23 merged pathogens, but they are less prone to homoplasy and thus extremely valuable for phylogenetic a

24 annotation of trees with synapomorphies and homoplasies, and motif detection.

25 cause retroposon insertions show very little homoplasy, and because the ancestral state (absence of t

26 d by factors (such as a choice of sequences, homoplasy, and different mutation rates) resulting in in

27 Patterns of linkage disequilibrium, homoplasy, and incompatibility are difficult to interpre

28 ta show less schismogenesis, higher rates of homoplasy, and more bursts of contact-induced change tha

29 more informative sites, were subject to less homoplasy, and provided better support for well-accepted

30 As homoplasies are infrequent in patterns of nucleotide sub

31 netic approaches to deal with high levels of homoplasy are discussed.

32 ulate relationships such as recombination or homoplasy are displayed as cycles.

33 goettingensis complex by analyzing the extra homoplasy arising in hybrid genomes from the simultaneou

34 re is a reduction in the known occurrence of homoplasy (as reported previously).

35 e best supported gene trees, 58% of apparent homoplasy at amino sites in the species tree is due to g

36 sis of the variable repeats shows ubiquitous homoplasy at the level of divergence represented by the

37 ecialized morphology of Lineatriton displays homoplasy at two hierarchical levels: (i) with respect t

38 How can homoplasy be recognized and factored into attempts to re

39 x mtDNA sequence data sets show an excess of homoplasy, but this could be due to either recombination

40 In such cases, inferred homoplasy can be entirely misleading with regard to tree

41 Size homoplasy can be inferred for alleles within populations

42 As a result, inferred trends in homoplasy can differ markedly from the underlying trend

43 Both of these problems, secondary loss and homoplasy, confound the interpretation of evolutionary r

44 Homoplasy (convergence in the size of different alleles)

45 tionary changes, suggesting a high degree of homoplasy (convergences and reversals).

46 his is not always reflected in the levels of homoplasy detected in a parsimony analysis, because high

47 of genotypes admit a phylogeny with a single homoplasy event.

48 eny with q homoplasy events, so that all the homoplasy events occur in a single site.

49 en set of genotypes admit a phylogeny with q homoplasy events, so that all the homoplasy events occur

50 ions, which biases the results to minimizing homoplasy events.

51 on from perfect phylogeny is entirely due to homoplasy events.

52 that microinversions provide a source of low-homoplasy evolutionary characters.

53 ed among randomised character data using the homoplasy excess ratio (HER).

54 h multiple alleles were sequenced, extensive homoplasy for size was uncovered both within and between

55 The lack of homoplasy found in this study supports the robustness of

56 use their mode of evolution is predominantly homoplasy-free and unidirectional.

57 Such families of repetitive DNA are homoplasy-free characters that can be used for cost-effi

58 s can continue to be regarded as essentially homoplasy-free genetic characters.

59 We used these homoplasy-free markers to construct a mobile element ins

60 copies has been deemed to be an essentially homoplasy-free phylogenetic character because the probab

61 n L1 (LINE-1) retrotransposon are apparently homoplasy-free phylogenetic characters.

62 e locus may be rare, SINE insertions are not homoplasy-free phylogenetic markers.

63 mong 162,000 preselected cases 102 virtually homoplasy-free, phylogenetically informative retroelemen

64 persed elements, which are considered nearly homoplasy-free, to elucidate the phylogeny of hominids.

65 Effects on homoplasy from the number of character states are furthe

66 The problem of homoplasy, however, can diminish the value of LINEs as p

67 analysis of tRNAs can help to recognize this homoplasy, improving gene-order-based phylogenetic hypot

68 e phylogenetic tree that are attributable to homoplasies in the data set.

69 eral gene transfer appears to have generated homoplasy in our character dataset.

70 ed to dentognathic (that is, jaws and teeth) homoplasy in Plio-Pleistocene hominins, and shows that s

71 ic systems may also exhibit a high degree of homoplasy in some groups of organisms.

72 It is likely that homoplasy in the coding region will confound evolutionar

73 roblem is posed by convergent evolution (or 'homoplasy' in genetic terminology): a feature or a molec

74 es, testing each gene fusion for evidence of homoplasy, including gene fission, convergence, and hori

75 ecause it creates patterns of substitutions (homoplasies) inconsistent with the hypothesis of a singl

76 egard to tree quality (with higher levels of homoplasy inferred for better quality trees).

77 ion using molecular data is often subject to homoplasy, leading to inaccurate conclusions about phylo

78 cter incompatibility (as well as the maximum homoplasy measurable with some indices).

79 dex (RI), and particularly by accounting for homoplasy measured among randomised character data using

80 The homoplasies observed caution against relying too heavily

81 scendants, and strengthens the hypothesis of homoplasy of 'tribosphenic-like' molars among mammals.

82 is new eutriconodont adds to the evidence of homoplasy of vertebral characters in the thoraco-lumbar

83 history of a set of samples contain frequent homoplasies or recombination events quasi-median network

84 titution changes (e.g., those not exhibiting homoplasy or reversals) from the unambiguous branches of

85 there are more trunk vertebrae, a widespread homoplasy (parallelism) in salamanders, the genus Lineat

86 Such examples of homoplasy present opportunities to discover the foundati

87 Over megabases, homoplasy rates fluctuate 1.9-fold, peaking towards the

88 ivisions with less homologous recombination (homoplasy ratio, 0.2).

89 by split decomposition and homoplasy tests (homoplasy ratio, 0.56).

90 bB showing low mean compatibility scores and homoplasy ratios of 0.71 and 0.67, respectively, recombi

91 state space affects both the known levels of homoplasy (recorded during simulated evolution) and thos

92 Analysis of the mechanisms generating such homoplasy requires an independent molecular phylogeny.

93 ion, as indicated by split decomposition and homoplasy tests (homoplasy ratio, 0.56).

94 ids, and illuminates previously-unrecognized homoplasy that complicates dromaeosaurid phylogeny and s

95 estimation of pivotal parameters, can allow homoplasy to bias these estimates and ultimately the PB-

96 Size homoplasy was apparent, caused by base substitutions wit

97 Homoplasy was high, with multiple changes per site and n

98 Accounting for these considerable sources of homoplasy, we identified fusion characters that provide