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1 pression mediated by RosE is required during host pathogen interaction.
2 expression of bacterial genes important for host pathogen interaction.
3 r the extracellular matrix in regulating the host-pathogen interaction.
4 the battle for survival has shaped a complex host-pathogen interaction.
5 cted link between cellular reprogramming and host-pathogen interaction.
6 strategy generally applicable to studies of host-pathogen interaction.
7 he need for better understanding this unique host-pathogen interaction.
8 and collagen-alginate matrix to dissect the host-pathogen interaction.
9 ignificantly affect bacterial physiology and host-pathogen interaction.
10 TI and renal scarring based on research into host-pathogen interaction.
11 from host serum and the incorporation of the host-pathogen interaction.
12 PGLs play key roles in pathogenicity and host-pathogen interaction.
13 or pathologic inflammation in asthma and for host-pathogen interaction.
14 of the host's metabolic resources is a core host-pathogen interaction.
15 ociated molecular patterns, which govern the host-pathogen interaction.
16 4E phosphorylation is a key component of the host-pathogen interaction.
17 ns where in genes previously associated with host pathogen interactions.
18 of C. jejuni cell surface glycoconjugates in host pathogen interactions.
19 recognition, pathogenesis inflammation, and host pathogen interactions.
20 our understanding of infectious diseases and host pathogen interactions.
21 sents a new and unique biological process of host pathogen interactions.
22 ential to provide deeper insights into other host-pathogen interactions.
23 ction for AGMO-dependent lipid metabolism in host-pathogen interactions.
24 e mechanisms of viral immune suppression and host-pathogen interactions.
25 izable for further examination of additional host-pathogen interactions.
26 studies of alphavirus molecular biology and host-pathogen interactions.
27 y arms race, as observed for genes governing host-pathogen interactions.
28 peroxynitrite, both of which are involved in host-pathogen interactions.
29 A (vsRNA) analysis for getting insights into host-pathogen interactions.
30 obust model systems that enable the study of host-pathogen interactions.
31 relevant route of infection when addressing host-pathogen interactions.
32 lcineurin programs different outcomes during host-pathogen interactions.
33 de all the harder by the added complexity of host-pathogen interactions.
34 Competition for iron influences host-pathogen interactions.
35 afish an attractive model host to understand host-pathogen interactions.
36 ming and infection opens up a new premise in host-pathogen interactions.
37 ogrammed cell death is an important facet of host-pathogen interactions.
38 elegans have provided detailed insight into host-pathogen interactions.
39 ing a mechanistic understanding of migratory host-pathogen interactions.
40 of coding capacity and profoundly influences host-pathogen interactions.
41 g adhesive or immune-modulatory functions in host-pathogen interactions.
42 lanarians as a powerful system for analyzing host-pathogen interactions.
43 ons including 23 genes that are essential to host-pathogen interactions.
44 which results in functional consequences for host-pathogen interactions.
45 s whose populations are strongly affected by host-pathogen interactions.
46 antimicrobial resistance but may also impact host-pathogen interactions.
47 cell adhesion, cell-cell communication, and host-pathogen interactions.
48 n unsuspected role for the nervous system in host-pathogen interactions.
49 oups, this means that they experience strong host-pathogen interactions.
50 7 cells as a small intestinal model to study host-pathogen interactions.
51 phospholipases play many important roles in host-pathogen interactions.
52 ignal molecules involved in reproduction and host-pathogen interactions.
53 g of V. para. infections and more generally, host-pathogen interactions.
54 enic infection and quantify their impacts on host-pathogen interactions.
55 els are simple experimental systems to study host-pathogen interactions.
56 ignificantly affect bacterial physiology and host-pathogen interactions.
57 adds to understanding of complex network of host-pathogen interactions.
58 cted component in determining the outcome of host-pathogen interactions.
59 for new pathogen discovery and research into host-pathogen interactions.
60 he postulated roles of this metalloenzyme in host-pathogen interactions.
61 upregulated has additional implications for host-pathogen interactions.
62 ication of virulence determinants or explore host-pathogen interactions.
63 entric access, analysis and visualization of host-pathogen interactions.
64 rphisms alter RopB function and modulate GAS host-pathogen interactions.
65 and improved integration and presentation of host-pathogen interactions.
66 toskeletal remodeling, sugar metabolism, and host-pathogen interactions.
67 agronomically important model of biotrophic host-pathogen interactions.
68 uent and the potential impact it may have on host-pathogen interactions.
69 or cell-cell recognition and adhesion and in host-pathogen interactions.
70 ystems biology approach toward understanding host-pathogen interactions.
71 odel a unique tool for understanding mucosal host-pathogen interactions.
72 ng the community ecology and epidemiology of host-pathogen interactions.
73 the evolutionary and population dynamics of host-pathogen interactions.
74 ions and impact significantly the outcome of host-pathogen interactions.
75 l but also important molecules implicated in host-pathogen interactions.
76 ted xylose residues are important for normal host-pathogen interactions.
77 ost immune responses and are associated with host-pathogen interactions.
78 understanding the dynamics of sex ratios and host-pathogen interactions.
79 n of iron homeostasis is a major strategy in host-pathogen interactions.
80 stems are infamous for their contribution to host-pathogen interactions.
81 nt when choosing a macrophage model to study host-pathogen interactions.
82 MCV is very poorly characterized in terms of host-pathogen interactions.
83 hematophagy, pathogen transmission, and tick-host-pathogen interactions.
84 nation of TB is our lack of understanding of host-pathogen interactions.
85 as9 screens to discover processes underlying host-pathogen interactions.
86 of m(6)A in the immune system or its role in host-pathogen interactions.
87 ther highlighting the pivotal role of S1P in host-pathogen interactions.
88 pathogenicity, antimicrobial resistance and host-pathogen interactions.
89 ations and new understandings of biology and host-pathogen interactions.
90 the importance of cell-cycle control during host-pathogen interactions.
91 lular architecture and mechanisms underlying host-pathogen interactions.
92 the potential for in-depth visualization of host-pathogen interactions.
93 ggestive of an involvement of this enzyme in host/pathogen interactions.
94 globulin related gene and may be involved in host-pathogen interaction, a well-known target of balanc
95 biochemistry of gene regulation involved in host-pathogen interactions, a quantitative understanding
97 osure of the antigen and a potential role in host-pathogen interaction, active immunization of mice w
99 resultant disease is the outcome of complex host-pathogen interactions, an integrated, quantitative
100 ance when evaluating other animal models for host-pathogen interaction and by taking greater advantag
102 n host cells that unveil new features of the host-pathogen interaction and may also be exploited as a
104 l provide the best opportunity to understand host-pathogen interaction and the most powerful approach
105 has begun to outline the complexity of this host-pathogen interaction and to reveal how the homeosta
107 d with regard to the evolution of virulence, host-pathogen interactions and antifungal drug therapies
108 ese finding provide targets for the study of host-pathogen interactions and applied research into alt
110 host proteins and can be applied to examine host-pathogen interactions and develop host-based therap
111 ses, including translation, gene regulation, host-pathogen interactions and environmental sensing.
112 a better understanding of the complexity of host-pathogen interactions and have identified some of t
113 est the involvement of HbN in modulating the host-pathogen interactions and immune system of the host
115 used on protein structure-function analyses, host-pathogen interactions and in developing vaccines.
116 ese responses is important for understanding host-pathogen interactions and informing vaccine design.
117 e an active cell wall glycoconjugate driving host-pathogen interactions and participating in the immu
119 important resource for the investigation of host-pathogen interactions and reductive evolution in on
120 r, these data introduce a novel regulator of host-pathogen interactions and secreted-protein producti
121 e complex and contextual role of anorexia in host-pathogen interactions and suggest that microbes hav
122 he natural route of infection for studies of host-pathogen interactions and suggest that the detrimen
123 ediated signaling represents a mechanism for host-pathogen interactions and that Epac1 is a potential
124 be important for increased understanding of host-pathogen interactions and the development of future
125 it through these pathways affects downstream host-pathogen interactions and the generation of an immu
126 romising model could facilitate the study of host-pathogen interactions and the investigation of the
127 ra has revolutionized approaches to defining host-pathogen interactions and the investigation of the
128 ssue systems to advance our understanding of host-pathogen interactions and the role of human genetic
129 To better understand the role of LPS in host-pathogen interactions and to elucidate the antigeni
130 development that has led to new insights in host-pathogen interactions and, as well, led to insight
131 m pathogen biology, evolutionary dynamics of host--pathogen interactions, and the ecology of infectio
133 anism of exNef secretion, identifies a novel host-pathogen interaction, and introduces an HIV-derived
134 cluding liver development, disease modeling, host-pathogen interactions, and drug metabolism and toxi
135 is of GBPs, which mediate mammalian biology, host-pathogen interactions, and immune recognition of gl
136 ide is an immunodominant antigen, can define host-pathogen interactions, and is under consideration a
137 C platform can be used to uncover aspects of host-pathogen interactions, and it has the potential to
138 henotype may apply to artificial selections, host-pathogen interactions, and other biomedically relev
139 urrent use in understanding diarrhea-causing host-pathogen interactions, and potential future applica
140 system (i.e., the more trophic interactions, host-pathogen interactions, and the like there are [i.e.
141 is a receptor for PLA of Y. pestis, and this host-pathogen interaction appears to play a key role in
148 y, some outer membrane proteins that mediate host-pathogen interactions are strongly regulated by con
151 sed as a model to offer unique insights into host-pathogen interactions as well as a platform to effi
152 ng a highly informative means to investigate host-pathogen interactions as well as assess potential n
153 n the context of bacterial virulence and the host-pathogen interaction, as small molecule inhibitors
158 nce of these in vivo results and explore the host-pathogen interaction between DCs and cell-free HTLV
160 obiota plays an important role in modulating host-pathogen interactions, but little is known about th
161 Mammalian LBs display an important role in host-pathogen interactions, but the role of parasite LBs
162 -seq approach was deployed to understand the host-pathogen interaction by identifying differentially
163 down-regulated protein 9 (NEDD9)-govern the host-pathogen interaction by regulating the uptake of C.
166 , we explore how better understanding of the host-pathogen interaction can substantially enhance, and
167 disease, focusing on infectious diseases and host-pathogen interactions, cancer, and rare genetic dis
170 ssues challenge an overly simplistic view of host-pathogen interaction during mucosal infection, but
171 s transmitted via the respiratory route, the host-pathogen interactions during acute VZV infection in
172 l systems-level datasets designed to dissect host-pathogen interactions during influenza A infection
175 grated use of these approaches can elucidate host-pathogen interactions during this infection and can
176 of host hybridization and clines in altering host-pathogen interactions, dynamics that can have impor
177 resolve sepsis heterogeneity by considering host-pathogen interactions, enabling progress towards a
179 should find its wide application to studying host-pathogen interactions for any other interconnected
180 s for use in diagnosis and interference with host-pathogen interactions for the purpose of disease co
181 tion for TDP2 and suggest an unusual role in host-pathogen interactions for this cellular enzyme.
182 galectins play multiple roles in regulating host-pathogen interactions; for example, galectins can p
185 alivary glands and other tissues involved in host-pathogen interactions has revealed a wide range of
186 malian GTPase defines further how intimately host pathogen interactions have coevolved through simila
187 m repeat-containing proteins associated with host-pathogen interactions have been identified in Ehrli
188 er, the highly human-specific nature of most host-pathogen interactions hinders the in vivo establish
189 proteins (ECMp) plays important roles during host-pathogen interaction, however its genetic regulatio
192 nd pathophysiological cell functions such as host-pathogen interactions, immune regulation, and tumor
193 omics approaches have provided insights into host-pathogen interactions, immunogenetics, and viral ev
194 long been recognized as mediators of initial host-pathogen interactions important for the progression
197 Understanding the complex nature of the host-pathogen interaction in each of these contexts is e
198 studies provide additional insights into the host-pathogen interaction in pneumonic Burkholderia infe
201 in a model of periodontitis and reveal a new host-pathogen interaction in which P. gingivalis activat
202 f the mechanisms of astrovirus infection and host-pathogen interactions in a genetically manipulable
204 portant for helical shape, colonization, and host-pathogen interactions in C. jejuni Therefore, chang
205 selection pressure as a result of long-term host-pathogen interactions in certain regions or populat
206 e organism to gain a better understanding of host-pathogen interactions in chronic respiratory infect
208 that environmental heterogeneity can mediate host-pathogen interactions in fragmented landscapes, and
209 strates the value of proteomics in unveiling host-pathogen interactions in genetically challenging mi
212 portance of the human fibrinolytic system in host-pathogen interactions in invasive GAS infections.
214 ul investigations of the immune response and host-pathogen interactions in Mycobacterium tuberculosis
215 otein processing can be an important part of host-pathogen interactions in other systems, suggesting
216 e the knowledge from experimental studies of host-pathogen interactions in several diseases to build
218 t into the inflammatory mediators regulating host-pathogen interactions in the female genital tract.
219 eraged this animal model to characterize the host-pathogen interactions in the ganglia during both ac
221 These studies provide novel insights into host-pathogen interactions in the sensory ganglia during
226 ovide insights into the factors that mediate host-pathogen interactions in zygomycosis and establish
227 alpha-glucan degradation contributes to GAS host-pathogen interaction, including how GAS uses human
228 lycans are involved in processes that affect host-pathogen interactions, including adhesion, recognit
232 s MicroReview, the contribution of Mn to the host-pathogen interaction is reviewed, and key questions
233 cus on epidemiology, ecological modeling and host-pathogen interactions is expected to yield a better
235 ogenicity derived from these analyses of Y2H host-pathogen interactions is the identification of euka
237 Despite the high evolutionary pressure in host-pathogen interactions, it is still widely assumed t
238 utionary change is likely to destabilize the host-pathogen interaction leading to population cycles.
239 l biomarker discovery requires understanding host-pathogen interactions leading to biomarker expressi
240 n extended to the most essential outcomes of host-pathogen interactions: namely, infection and diseas
241 and abiotic factors that potentially affect host-pathogen interactions: natural vegetation, canopy d
242 lpB will facilitate future investigations of host-pathogen interactions occurring during H. pylori in
245 and used it to explore similarities between host-pathogen interactions of B. mallei, Yersinia pestis
248 llular functions that participate in complex host-pathogen interactions or processes related to disea
251 pathogenic bacteria play important roles in host-pathogen interactions ranging from interfering with
252 ase, the distinct mechanisms that govern the host-pathogen interaction remain enigmatic, particularly
256 but the molecular interface involved in this host-pathogen interaction remains poorly characterized.
258 significance of pathogenic noncoding RNAs in host-pathogen interactions remains largely unclear.
259 cation of bio-threat agents and the study of host-pathogen interactions require a high-resolution det
261 es (ORFs) involved have functions related to host-pathogen interaction, so genotype-specific evolutio
264 l, but have not been extended to incorporate host-pathogen interactions, such as immunosuppression or
265 anism has already been demonstrated in other host-pathogen interaction systems, this is the first rep
266 , for questions about specific mechanisms of host-pathogen interaction that cannot be answered feasib
267 rotocol to identify important aspects of the host-pathogen interaction that determine the outcome of
268 uggest that this phenomenon reflects a novel host-pathogen interaction that is central to infection w
269 o Pneumocystis stimulation, but the upstream host-pathogen interactions that activate AEC signaling c
270 ndings support a role for YapE in modulating host-pathogen interactions that are important for coloni
272 till unraveling the molecular details of the host-pathogen interactions that enable this virus to esc
273 l studies have allowed us to reconstruct the host-pathogen interactions that have shaped Tetherin and
274 ancing our mechanistic knowledge base of the host-pathogen interactions that influence disease outcom
275 The focus of this review is to consider the host-pathogen interactions that influence neurotropic in
276 h disease pathogenesis and immunity, but the host-pathogen interactions that mediate these effects ar
277 tic markers and may provide insight into the host-pathogen interactions that occur during T. pallidum
281 S6(Pro) This work provides new insights into host-pathogen interactions that regulate stress pathways
282 cs as a platform to identify and study novel host-pathogen interactions that shape the outcome of an
283 ndings provide new insights into features of host-pathogen interactions that shape viral evolution, a
284 ment plays a critical role in regulating the host-pathogen interaction through modulation of the fung
285 to its ability to form biofilms and modulate host-pathogen interactions through the secretion of viru
287 ide a platform for in silico manipulation of host-pathogen interactions to gain insight into this inf
288 e adapted to assess other host-commensal and host-pathogen interactions using genomic and transcripto
291 ze the effect of titan cell formation on the host-pathogen interaction, we utilized a previously desc
292 icry and diversifying selection-hallmarks of host-pathogen interactions-were identified within host a
293 l contribute greatly to our understanding of host-pathogen interactions while reducing the number of
295 he population and evolutionary dynamics of a host-pathogen interaction with free-living stages are ex
297 P will affect the population dynamics of the host-pathogen interaction, with previous theoretical wor
299 s a potentially attractive resource to study host-pathogen interactions without the need for animal e
300 cally leads to resistance in an incompatible host-pathogen interaction, yet no gene-for-gene resistan
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