ライフサイエンスコーパス: house dust mite

1 ivate GPCRs such as Alternaria alternata and house dust mite.

2 and respiratory challenge with an extract of house dust mite.

3 zation with two different allergens, OVA and house dust mite.

4 hallenge relative to the common aeroallergen house dust mite.

5 onths was associated with all endpoints, but house dust mite.

6 asthma via regulation of immune response to house dust mite.

7 at the baseline and after sensitization with house dust mite.

8 t of a commonly studied airway allergen, the house dust mite.

9 irected against staple food antigens but not house dust mites.

10 eatment for local allergic rhinitis (LAR) to house dust mites.

11 tradermal SIT in children/adults allergic to house dust mite (10 trials), grass pollen or other inhal

12 The prevalence of sensitization to house dust mite (-4.3%; 95% CI, -6.0% to -2.6%) and cat

13 rse, (2) timothy grass/birch, (3) molds, (4) house dust mites, (5) peanut/wheat flour/mugwort, (6) pe

14 firmed in a chronic model of asthma by using house dust mite, a human allergen.

15 y disease induced by either acute or chronic house dust mite Ag exposure.

16 Skin prick test (SPT) sensitivity to house dust mite allergen (HDM) and current wheeze were a

17 d decreased airway resistance in response to house dust mite allergen (HDM).

18 ild-type mice with Aspergillus fumigatus and house dust mite allergen and compared the effects on air

19 through 6 months, to filtered air (FA) with house dust mite allergen and ozone using a protocol that

20 NC/Nga mice were sensitized with house dust mite allergen and treated topically with HOCl

21 on of immunodominant epitopes from the major house dust mite allergen Der p 1.

22 The major house dust mite allergen Der p 2 is a structural and fun

23 otects against epicutaneous sensitization to house dust mite allergen Der p 2.

24 In addition, we discuss of house dust mite allergen extracts as a prototypical comp

25 ing repeated exposures of 3 hours per day to house dust mite allergen in an allergen challenge chambe

26 lized a mechanistic IL-13-driven model and a house dust mite allergen mucosal sensitization model of

27 -type (WT) mice following sensitization with house dust mite (allergen) (HDM).

28 Diverse factors contribute to house dust mite allergenicity through the activation of

29 against enterotoxins, grass pollen (GP), and house dust mite allergens and total IgE levels were meas

30 d late airway responses after challenge with house dust mite allergens in 15 patients with mild aller

31 levels of IgE specific for staple foods and house dust mite allergens in DOCK8-deficient patients an

32 y hyper-responsiveness following exposure to house dust mite allergens, even though their lungs harbo

33 sensitization after cutaneous challenge with house dust mite allergens.

34 junction downregulation in a mouse model of house dust mite allergic airway inflammation.

35 NA networks operate to regulate Th2 cells in house dust mite-allergic or helminth-infected animals an

36 More than 90% of house dust mite-allergic patients are sensitized to the

37 ll tolerated and reduces the CPT reaction in house dust mite-allergic patients.

38 nd IgA2 contribute to the clinical status of house dust mite-allergic patients.

39 Specifically, for patients with house dust mite allergies, which are often underestimate

40 th tablets containing carbamylated monomeric house dust mite allergoids was to determine the most eff

41 (TSSs) were recorded by 21 participants with house dust mite allergy (M+) in the natural setting and

42 of common airborne allergens, including the house dust mite, Alternaria, and Aspergillus, for up to

43 Chronic exposure to house dust mite and A alternata were compared in a neona

44 Age-specific prevalence of sensitization to house dust mite and cat did not differ between year-of-b

45 lower levels of sensitization, especially to house dust mite and cat, after the age of 20 years.

46 of ImmunoCAP-identified IgE directed against house dust mite and cockroach, but not against timothy g

47 initial recognition and uptake of the major house dust mite and dog allergens Der p 1 and Can f 1, r

48 ta allergen, Aspergillus fumigatus antigens, house dust mite and endotoxin antigens increase the risk

49 sthma and rhinitis, focusing on responses to house dust mite and grass.

50 ysteine protease and major allergen from the house dust mite and is associated with allergic rhinitis

51 xt of acute allergic lung inflammation using house dust mite and OVA murine models.

52 m airway hyper-responsiveness in response to house dust mite and ovalbumin sensitization and challeng

53 al allergens including insect allergens from house dust mites and cockroaches contribute to allergic

54 and fruits, certain inhalant allergens from house dust mites and cockroaches, and lipocalins.

55 We assess whether exposure to fungi, house dust mites and endotoxin increases the risk of ecz

56 nalyses for a reduction in SPT reactivity to house dust mites and perennial allergens.

57 d with lower odds of sensitization to grass, house dust mite, and cat allergens, but rodent ownership

58 y to the age of 7 with asthma, atopy (grass, house dust mite, and cat skin prick test) and atopic vs.

59 hibited inflammatory responses to ovalbumin, house dust mite, and IL-13 overexpression.

60 acute challenge with Alternaria alternata or house dust mite, and secretion of IL-33 and activation o

61 otein D (rfhSP-D) has been shown to suppress house dust mite- and Aspergillus fumigatus-induced aller

62 As a consequence house dust mite- and IL-33-driven lung inflammation, lat

63 enuated allergic airway responses to fungi-, house dust mite-, and cockroach-associated allergens in

64 sthmatic) 4 hours after instillation of LPS, house dust mite antigen, and saline in three distinct su

65 After exposure to house dust mite antigen, Zbtb46-negative CD64(+) inflamm

66 oup of mice was also intranasally exposed to house dust mite antigen.

67 severity of allergic lung disease caused by house dust mite antigens or IL-13.

68 House dust mites are the major source of indoor allergen

69 ecent publications were identified by using "house dust mite" as a key search term to evaluate the cu

70 Cell culture and ovalbumin (OVA)- or house dust mite-based murine asthma models were used in

71 stablished whereby mice were sensitized with house dust mite before subsequently being challenged wit

72 that lacked the IL-33 receptor given inhaled house dust mite, cat dander, or Alternaria, and the effe

73 serum specific IgE to common aeroallergens (house dust mite, cat, and grass) and total IgE levels we

74 lative Moldiness Index (ERMI) and endotoxin, house dust mite, cat, dog, and cockroach allergens.

75 articipants underwent skin prick testing for house dust mite, cat, grasses and moulds.

76 eriostin were also decreased in the lungs of house dust mite-challenged ERp57-deleted mice.

77 duced by 6.2-fold in pulmonary epithelium of house dust mite-challenged mice.

78 hyperreactivity and mucus hypersecretion in house dust mite-challenged mice.

79 Purified group 2 major allergen from house dust mite chemically conjugated to 4-(6-amino-9-be

80 House dust mite concentrations varied across cohorts.

81 he airways upon intranasal immunization with house dust mite, confirming the ability of IL-9-producin

82 eceived sublingual immunotherapy with either house dust mite (D. farinae) or a mixture of up to five

83 timulation with lipid A, peptidoglycan, PHA, house dust mite (Der p 1), or Der p 1 plus lipid A. mRNA

84 es the presence of respiratory allergen from house dust mite, Der p 1, in human breast milk.

85 House dust mite-derived proteases contribute to allergic

86 tion of Th2 and Th17 immune responses to the house dust mite Dermatophagoides farinae through the gen

87 eration when challenged with an extract from house dust mite Dermatophagoides farinae, compared with

88 es (cys-LTs) can mediate Th2 immunity to the house dust mite, Dermatophagoides farinae, via the type

89 Der p 1 is a major allergen from the house dust mite, Dermatophagoides pteronyssinus, that be

90 s, chromatin immunoprecipitation assays, and house dust mite-driven allergic airway inflammation.

91 Likewise, in house dust mite-driven asthma, CD2-Gata3 Tg mice were si

92 ergen 1 (Der p 1), the major allergen of the house dust mite, efficiently activates various facets of

93 House dust mite exposure caused significant gene-by-envi

94 velopment and TH2 polarization, as seen in a house dust mite exposure model.

95 In the tropics, house dust mite exposure, a known risk factor for asthma

96 sponsiveness (AHR) compared with those after house dust mite exposure.

97 roup, dust samples were collected to measure house dust mite exposure.

98 d for gene-by-environment interactions using house dust mite exposure.

99 hyperoxia promoted allergic TH responses to house dust mite exposure.

100 prolonged ovalbumin challenge or continuous house-dust mite exposure to induce chronic inflammation

101 ukin 13 (IL-13) when stimulated by papain or house dust mite extract (HDM) and induce eosinophilic in

102 mice administering either ovalbumin (OVA) or house dust mite extract (HDM) for sensitization.

103 ing dimaprit in both the ovalbumin (OVA) and house dust mite extract (HDM) murine models of respirato

104 hich repeated epicutaneous applications of a house dust mite extract and Staphylococcal enterotoxin B

105 sal priming and challenge of these mice with house dust mite extract or ovalbumin as allergens led to

106 Here we report that mice challenged with house dust mite extract or papain in the absence of TSLP

107 model was exposed to allergens (ovalbumin or house dust mite extract) to decipher in vivo the implica

108 -type and RAGE knockout mice by using IL-33, house dust mite extract, or Alternaria alternata extract

109 rimental asthma induced by repeat intranasal house dust mite extract.

110 controls when challenged intranasally with a house dust mite extract.

111 ation in mice following acute challenge with house dust mite extract.

112 rway inflammation in response to Der p 1 and house dust mite extracts in a murine model of asthma.

113 e show that continual exposure (for 8 wk) of house dust mite extracts induced a severe phenotype of c

114 Culprit allergens responsible include house dust mite, grass and olive pollens, and many other

115 House dust mites have been implicated in the etiology an

116 Some allergens such as derived from house dust mites have proteolytic activity which can ind

117 House dust mite (HDM) acts on the airway epithelium to i

118 itization phase of either ovalbumin (OVA) or house dust mite (HDM) acute asthma models.

119 House dust mite (HDM) affects the immunological and phys

120 line) was extracted and analyzed for cat and house dust mite (HDM) allergen content using ELISA, all

121 The house dust mite (HDM) allergen Der p 13 could be a lipid

122 a natural field study, sublingual tablets of house dust mite (HDM) allergen extracts (STG320) were ef

123 how endogenous CD4(+) T cells specific for a house dust mite (HDM) allergen form and function.

124 concept for oral immunotherapy to high-dose house dust mite (HDM) allergen in infancy in the prevent

125 t the safety and efficacy of challenges with house dust mite (HDM) allergen in the Fraunhofer allerge

126 events preceding sensitization to the common house dust mite (HDM) allergen remains to be elucidated.

127 In the present study, the task force for house dust mite (HDM) allergen standardization of the Co

128 Der p 23, a new, major house dust mite (HDM) allergen that is recognized by >70

129 s a mixture of many components, of which the house dust mite (HDM) allergen, Der p 1, is the most all

130 House dust mite (HDM) allergens are a common cause of al

131 Upon inhalation, house dust mite (HDM) allergens are deposited at the nas

132 Mice were sensitized with house dust mite (HDM) allergens from days 3, 15, or 60 a

133 tion was almost exclusively directed against house dust mite (HDM) allergens.

134 alyses revealed that IgE from crustaceans or House dust mite (HDM) allergic patients showed cross-rea

135 rent standards are limited for the effect of house dust mite (HDM) allergy immunotherapy in asthmatic

136 immunotherapy (AIT) lack recommendations for house dust mite (HDM) allergy.

137 kout and wild-type mice were challenged with house dust mite (HDM) and infected with RV1B to determin

138 or WT mice were challenged over 3 weeks with house dust mite (HDM) antigen.

139 eeks to DEP at 1.2 or 6.0 mg/kg body weight, house dust mite (HDM) at 0.8, 1.2 or 6.0 mg/kg of DEP in

140 emonstrate that Streptococcus pneumoniae and house dust mite (HDM) bear similar phosphorylcholine (PC

141 Standard ovalbumin (OVA)-alum and house dust mite (HDM) bone marrow-derived DC (BMDC)-depe

142 istosoma egg antigen (SEA) immunization, and house dust mite (HDM) challenge without affecting cytoto

143 The house dust mite (HDM) Dermatophagoides pteronyssinus is

144 inhalation of airborne allergens such as the house dust mite (HDM) effectively activates both innate

145 protective effects of gamma-tocotrienol in a house dust mite (HDM) experimental asthma model.

146 rgen-specific IgG immune complexes (ICs) and house dust mite (HDM) extract both induced dendritic cel

147 immunized through the intranasal route with house dust mite (HDM) extract derived from Dermatophagoi

148 In the current study, we used an intranasal house dust mite (HDM) extract exposure regimen time cour

149 We used alphaT-catenin knockout mice and a house dust mite (HDM) extract model of atopic asthma, wi

150 examined constitutive and TNFalpha/IL-1beta, house dust mite (HDM) extract or lipopolysaccharide (LPS

151 ntrol mice were intranasally challenged with house dust mite (HDM) extract or PBS five days per week

152 nd pulmonary Th2 inflammation in response to house dust mite (HDM) extract, as both were decreased in

153 in mice with prolonged i.n exposure to crude house dust mite (HDM) extract.

154 ce were challenged with DEPs with or without house dust mite (HDM) extract.

155 ced by means of intranasal administration of house dust mite (HDM) extract.

156 gested the efficacy of sublingual tablets of house dust mite (HDM) extracts in adults with allergic r

157 16HBE cells were stimulated with house dust mite (HDM) extracts.

158 (EPIT) on further sensitization to peanut or house dust mite (HDM) in a murine model of sensitization

159 (ECP)] induced by bronchial instillation of house dust mite (HDM) in patients with asthma on mainten

160 A damage and DNA damage responses induced by house dust mite (HDM) in vivo and in vitro.

161 Our data shows that intranasal exposure to house dust mite (HDM) increases the expression of HIF-1a

162 The house dust mite (HDM) is a major perennial allergen sour

163 Sensitization to house dust mite (HDM) is a risk factor for the developme

164 House dust mite (HDM) is one of the most common allergen

165 The initial immune response to house dust mite (HDM) is orchestrated by an interplay be

166 -/-) ) mice were used in ovalbumin (OVA) and house dust mite (HDM) models of AAI.

167 Utilizing the ovalbumin (OVA) and house dust mite (HDM) models of asthma in C57BL/6 mice,

168 These studies use a house dust mite (HDM) mouse model of asthma/allergic air

169 ty were evaluated in the ovalbumin (OVA) and house dust mite (HDM) murine models.

170 Mice were sensitized to house dust mite (HDM) or cockroach at day 0, treated wit

171 ) mice were challenged with saline, DEPs, or house dust mite (HDM) or DEP+HDM.

172 of allergic disease to the common allergens house dust mite (HDM) or peanut.

173 n D-replete diet, and exposure to intranasal house dust mite (HDM) or saline was commenced from day 3

174 iously showed that exposure to a low dose of house dust mite (HDM) resulted in enhanced HDM-mediated

175 Atopic status remained stable; however, house dust mite (HDM) sensitisation decreased by 5.6% (1

176 We employed acute ovalbumin (OVA) and house dust mite (HDM) sensitization and challenge models

177 As house dust mite (HDM) sensitization is dependent on TLR4

178 airway inflammation was induced following a house dust mite (HDM) sensitization protocol.

179 nd during the last 8 weeks of treatment in 2 house dust mite (HDM) SLIT-tablet trials (n = 1768).

180 acebo-controlled, randomized clinical trial, house dust mite (HDM) sublingual AIT was found to be eff

181 The house dust mite (HDM) sublingual allergen immunotherapy

182 The house dust mite (HDM) sublingual immunotherapy (SLIT) ta

183 ent mice, challenged with ovalbumin (OVA) or house dust mite (HDM), and accessed for TH2 inflammation

184 re quantified in neonatal mice given inhaled house dust mite (HDM), and the effect of blocking IL-13

185 l exposure to common human allergens such as house dust mite (HDM), in the absence of additional adju

186 Exposure to environmental antigens, such as house dust mite (HDM), often leads to T helper 2 (Th2) c

187 Exposure to allergens, such as house dust mite (HDM), through the skin often precedes a

188 ng 24 (Trim24) was predicted to be active in house dust mite (HDM)- and helminth-elicited Il4(gfp+)al

189 r D. farinae were assessed in sera from 1302 house dust mite (HDM)-allergic patients living in variou

190 tch signaling induced by DCs is critical for house dust mite (HDM)-driven allergic airway inflammatio

191 igate the therapeutic efficacy of SAHM1 in a house dust mite (HDM)-driven asthma model.

192 ing the Cd11c promotor) to acute and chronic house dust mite (HDM)-driven asthma models.

193 e sought to address the role of B cells in a house dust mite (HDM)-driven TH2-high asthma mouse model

194 e) control mice were evaluated in a model of house dust mite (HDM)-induced AAI.

195 expansion and cytokine production to prevent house dust mite (HDM)-induced airway inflammation and re

196 important role for the VLDLR in attenuating house dust mite (HDM)-induced airway inflammation in exp

197 s adaptive immune responses in patients with house dust mite (HDM)-induced airways disease.

198 sure to DEPs and in response to DEP-enhanced house dust mite (HDM)-induced allergic airway inflammati

199 Mice with house dust mite (HDM)-induced allergic airway inflammati

200 inhibition of mTOR with rapamycin prevented house dust mite (HDM)-induced allergic asthma in mice.

201 de of effect of sublingual immunotherapy for house dust mite (HDM)-induced allergic rhinitis with or

202 evaluating several dosages in patients with house dust mite (HDM)-induced allergic rhinoconjunctivit

203 TJs in the nasal epithelium of patients with house dust mite (HDM)-induced AR and in an HDM-induced m

204 leukocyte infiltration, protecting mice from house dust mite (HDM)-induced asthma or Leishmania major

205 OVA- and house dust mite (HDM)-induced murine asthma models were

206 s and the stimulatory effects of IL-1beta on house dust mite (HDM)-induced release of thymic stromal

207 et (ALK) has been developed for treatment of house dust mite (HDM)-induced respiratory allergic disea

208 e sought to investigate the role of TPL-2 in house dust mite (HDM)-mediated allergic airway inflammat

209 pulmonary allergy by adoptively transferring house dust mite (HDM)-pulsed bone marrow-derived DCs (BM

210 The effect of DEPs on house dust mite (HDM)-specific memory responses was dete

211 e investigated the importance of NK cells in house dust mite (HDM)-triggered allergic pulmonary infla

212 exoskeleton of many organisms including the house dust mite (HDM).

213 mice exposed to local airway challenge with house dust mite (HDM).

214 ory responses to a physiologic aeroallergen, house dust mite (HDM).

215 pecific transgenic mice were challenged with house dust mite (HDM).

216 helminth Schistosoma mansoni or the allergen house dust mite (HDM).

217 Allergens from house dust mites (HDM) are a common cause of asthma.

218 Group 1 allergens of house dust mites (HDM) are meaningful targets in this qu

219 the genome, transcriptome and microbiome of house dust mites (HDM) has shown that Staphylococcus aur

220 House dust mites (HDM) may serve as carriers of bacteria

221 ithelial cells, BEAS-2B, directly exposed to house dust mites (HDM) resulted in enhanced DNA damage,

222 Sensitizations to house dust mites (HDM) trigger strong exacerbated allerg

223 nd compare the allergen content of different house dust mites (HDM)' sublingual treatments and to rev

224 The most frequent allergens are house dust mites (HDM), which act in vivo on the bronchi

225 nes were confirmed in reporter assays and in house-dust-mite (HDM) induced AAI and primary human bron

226 House dust mite/HDM atopy patch test/APT elicits positiv

227 response is dominated by a single allergen (house dust mite; HDM).

228 Twenty-three adults allergic to house dust mites (HDMs) (M+) and 15 nonsensitive, nonall

229 Exposure to house dust mites (HDMs) aggravates the course of atopic

230 House dust mites (HDMs) represent one of the most import

231 2 responses triggered by allergens, such as house dust mites (HDMs).

232 tis (AR) and allergic asthma (AA) induced by house dust mites (HDMs).

233 veloped countries demonstrate sensitivity to house dust mites (HDMs).

234 The presence of GP or house dust mite IgE antibodies was not associated with e

235 istence among grass pollen, tree pollen, and house dust mite immunotherapy users in real life and to

236 House dust mite impermeable bedding as an isolated inter

237 To evaluate the use of house dust mite-impermeable bedding and its impact on se

238 to any allergen was present in 17.2% and to house dust mite in 8.7%.

239 life and as allergen levels of dog, cat, and house dust mite in bed dust samples at 1 year.

240 ical features of allergic asthma provoked by house dust mite in vivo.

241 House dust mite -induced inflammation facilitated neosen

242 Experimental acute canine house dust mite-induced AD lesions exhibit an activation

243 During house dust mite-induced airway allergy, rEos features re

244 ed similar responses in acute ovalbumin- and house dust mite-induced allergic airway disease.

245 ate controls were examined in ovalbumin- and house dust mite-induced allergic airway disease.

246 hat both DNA-HSP65 and CpG/CFP downregulated house dust mite-induced allergic airway inflammation via

247 rresponsiveness, and mucus production during house dust mite-induced allergic asthma.

248 The house dust mite-induced allergic inflammation model was

249 ltration into the airways of mice undergoing house dust mite-induced allergic response.

250 rolled dose-finding study, 131 patients with house dust mite-induced allergic rhinoconjunctivitis wer

251 y delivery of NP-CpG could prevent and treat house dust mite-induced allergy by modulating immunity d

252 aptive immune responses were dispensable for house dust mite-induced endoplasmic reticulum stress and

253 nce recovered, subjected to an ovalbumin- or house dust mite-induced experimental asthma protocol.

254 Blockade of Runx2 inhibited the house dust mite-induced goblet cell differentiation with

255 nflammation, and P2Y12 antagonism attenuates house dust mite-induced pulmonary eosinophilia in mice.

256 ema3e(-/-) mice into WT recipients increases house dust mite-induced Th2/Th17 inflammation in the air

257 ceptors, substantially reduced ovalbumin- or house-dust-mite-induced airway inflammation and bronchia

258 Here we show that house dust mite induces DNA-PK phosphorylation, which is

259 Controls were house dust mite-instilled animals receiving intravenous

260 n, prespecified allergen clusters found that house dust mite is the only allergen cluster for which s

261 challenge with different allergens (OVA and house dust mite) led to exacerbated eosinophilic inflamm

262 Our data indicate that exposure to house dust mite markedly reduces Sema3E expression in mo

263 House dust) mite markedly increased CCL20 levels in both

264 or the induction of T(H)2 responses, causing house dust mite-mediated allergic airway inflammation.

265 DCs were tested for their capacity to induce house dust mite-mediated allergic reactions.

266 models of experimental asthma (ovalbumin and house dust mite); miRNAs deregulated in both models were

267 ted animals were challenged in ovalbumin and house dust mite models of AAI.

268 Asthma was induced in mice using intranasal house dust mite or aerosol ova-albumin challenge, and ch

269 or systemically to the TH2-inducing antigens house dust mite or ovalbumin in a model of allergic airw

270 increased after treatment with the allergen house dust mite or the bacteria Escherichia coli and bac

271 llergic rhinitis (with or without asthma) to house dust mites or grass pollens.

272 volved include seasonal or perennial such as house dusts mites, pollens, animal epithelia, moulds (al

273 tracheal delivery of 1 x 10(6) OVA-, but not house dust mite- presenting, DC10s to OVA-asthmatic mice

274 House dust mites produce potent allergens, Der p 1 and D

275 rounds sensitized and challenged with OVA or house dust mite protein extract.

276 and allergic airway inflammation induced by house dust mites, pulmonary function and cytokine profil

277 h optimal doses of grass, birch, recombinant house dust mite (rDer p2) allergen or anti-IgE (n = 10).

278 allergens from Aspergillus fumigatus and the house dust mite, resulting in an asthma-like pathology c

279 Twenty-nine house dust mite-sensitised adults were skin prick and al

280 ly, depletion of alveolar macrophages during house dust mite sensitization or established disease res

281 man atopic dermatitis skin lesions with high house dust mite sensitization.

282 Ara h 2, tree nut, and house dust mite sensitization; coexisting food allergies

283 onic allergen-induced inflammation, OVA- and house dust mite-sensitized mice.

284 difference in wheal size between the diluted house dust mite solution and the bedding dust in spite o

285 bedding dust solutions or comparable diluted house dust mite solutions.

286 tory allergens (ie, grass, olive/ash pollen, house dust mites), specific IgE did not show marked diff

287 cross all populations and at different ages, house dust mite-specific IgG/IgE ratios (but not IgG4/Ig

288 nd difficult to diagnose, the efficacy of SQ house dust mite sublingual immunotherapy tablets has bee

289 Treatment with SQ (standardised quality) house dust mite sublingual tablet for 1 year resulted in

290 acerbation day (from 11% [placebo] to 5% [SQ house dust mite sublingual tablet]) and an increased pro

291 a mild AR day (from 16% [placebo] to 34% [SQ house dust mite sublingual tablet]).

292 ects challenged with ragweed, cat dander, or house dust mite, there were no differences in safety ind

293 model of allergic airways disease induced by house dust mite to determine T(H)9 cell function in vivo

294 ice were sensitized and then challenged with house dust mite to induce AAD while receiving treatment

295 nd classical Th2 allergens such as pollen or house dust mites to microbial products as well as self-a

296 eous application of Dermatophagoides farinae house dust mites to sensitized atopic dogs.

297 OVA- and house dust mite-treated Adam8(-/-) mice had higher lung

298 Allergic sensitization to cat, dog, and house dust mites was diagnosed longitudinally using skin

299 ainst components of grass or tree pollen, or house dust mite, was observed in 74% of the patients.

300 redominately caused by sensitization against house dust mites with a nearly complete penetrance of th