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1 m group versus 22.8 hours (95% CI, 13.3-34.0 hr) in the heparin and protamine group (log rank p = 0.0
2 ch patient was 39.2 hours (95% CI, 32.1-48.0 hr) in the citrate and calcium group versus 22.8 hours (
3  was 4.2 hours (interquartile range, 2.7-8.0 hr), divided into a median prehospital delay of 0.52 hou
4 ially expressed genes between TN and TM at 0 hr, 4 hr and 24 hr after stimulation, respectively, with
5 erage of one every 3.6 days to one every 2.1 hr.
6  6.9-8.5 hr, with sleep durations of 5.7-7.1 hr, amounts near the low end of those industrial societi
7 was similar for both groups (150 mL/min: 9.1 hr [5.5-26 hr] vs 10 hr [4.2-17 hr]; p = 0.37).
8         Notably, we demonstrate that acute 1 hr-suppression of PI3K signaling despite the ongoing pre
9 (913 unique compounds) decreased MMP after 1 hr of treatment without affecting cell viability (ATP co
10  induced initially by colistin (15 min and 1 hr) and later by doripenem (4 hr).
11          Colistin caused early (15 min and 1 hr) disruption of the bacterial outer membrane and cell
12 the-art batch methods currently available (1 hr vs. several days).
13 al prostates despite undetectable free BPA 1 hr postexposure.
14                                     Brief (1 hr) periods of postsynaptic bursting selectively depress
15 in sera of individual PND 3 pups collected 1 hr postexposure utilizing ultra-high-pressure tandem mas
16 aintained by inhalation of sevoflurane for 1 hr.
17                    Among those who spent > 1 hr/day outdoors or who had lived 5 or more years at thei
18 t enrollment address and those who spent > 1 hr/day outdoors.
19            Here, we report the long-term (>1 hr) imaging of single mRNAs undergoing hundreds of round
20            Longer duration of exercise (>/=1 hr/day), but not intensity or frequency, was associated
21 r after severe sepsis/shock recognition (< 1 hr reference) had a pooled odds ratio for mortality of 1
22 on-associated proteins in serum obtained: <1 hr (within 1-hour), 4-12 hr and 48-72 hr post injury.
23 ble to discriminate between TBI and HV at <1 hr.
24 L) alone, and their combination at 15 min, 1 hr and 4 hr (n = 4).
25 trolled fashion, 600 mg of intravenous NAC 1 hr before and 2 hr after cerebral angiography performed
26           There was a difference of nearly 1 hr between summer and winter sleep.
27 male was given access to the female within 1 hr.
28  oils (IFO), ranged from 0.4 to 2.2 g kW(-1) hr(-1) and 4.7 to 10.3 g kW(-1) hr(-1), respectively, an
29 hese tests were 12.7, 0.45, and 618 g kW(-1) hr(-1), respectively and were generally below benchmark
30 2.2 g kW(-1) hr(-1) and 4.7 to 10.3 g kW(-1) hr(-1), respectively, and were mainly dependent on the c
31 io for death, 1.03 [95% CI, 1.00-1.05] per 1-hr delay; p < 0.01), as was emergency department delay (
32 otein Bub3 at the kinetochores even after 10 hr extended culture.
33 groups (150 mL/min: 9.1 hr [5.5-26 hr] vs 10 hr [4.2-17 hr]; p = 0.37).
34  serum obtained: <1 hr (within 1-hour), 4-12 hr and 48-72 hr post injury.
35  the evening (LNd) group by approximately 12 hr; PDF alone delays the phase of the DN3 group by appro
36 n primary respiratory epithelial cells at 12 hr p.i.
37  mortality (area under the curve, 0.77 at 12 hr up to 0.93 at 5-10 d after cardiogenic shock onset).
38                                        At 12 hr, the creatinine reduction ratio independently predict
39 ngth significantly predicted major delay (12 hr: odds ratio, 6.98; CI, 2.33-20.89; 8 hr: odds ratio,
40 .5 hr/d) to an extended visitation model (12 hr/d).
41   Our results demonstrate that prolonged (12 hr) mechanical ventilation results in a significant decr
42 e was 0.74, whereas for two samples taken 12 hr apart, R(2) was 0.90.
43 rogeneous cell-cycle times that emerge as 12-hr synchronized cell division cycles.
44  (21 men, 4 women) provided two evaluable 12-hr PK profiles.
45 ned 318 functional direct target genes at 14 hr post inoculation.
46  exposure to a natural winter 9 hr 20 min:14 hr 40 min light-dark cycle as compared to the modern ele
47 er spending a weekend camping in a summer 14 hr 39 min:9 hr 21 min natural light-dark cycle compared
48 y shown that exposure to a natural summer 14 hr 40 min:9 hr 20 min light-dark cycle entrains the huma
49 were often only realized after extended (168 hr) incubations.
50  mL/min: 9.1 hr [5.5-26 hr] vs 10 hr [4.2-17 hr]; p = 0.37).
51 e phase of the DN3 group by approximately 17 hr.
52   We measured SBP at a mean age of 30 +/- 18 hr with an automated device.
53 trol, esmolol, norepinephrine (started at 18 hr after the surgery), and esmolol (started at 4 hr afte
54 the surgery) + norepinephrine (started at 18 hr after the surgery).
55  600 mg of intravenous NAC 1 hr before and 2 hr after cerebral angiography performed to confirm brain
56 r study to 0.3 ppm ozone and clean air for 2 hr at moderate (22 degrees C) temperature and again at a
57 ed for ROCK-mediated cell contraction from 2 hr post infection.
58 etected with beta-tubulin immunoreactivity 2 hr after stimulation.
59 evels in post-shift samples (collected </= 2 hr after completing a shift) were compared with pre-shif
60 rgeted analysis of unfractionated lysates (2 hr) accurately reproduces the quantification of fraction
61 3 sequential nuclear cleavages within only 2 hr of fertilization.
62 fPG) >/= 7.0 mmol/L (>/= 126 mg/dL) and/or 2 hr post-challenge plasma glucose >/= 11.1 mmol/L (>/= 20
63                                     Within 2 hr of immature INP birth, downregulation of repressor ac
64 d enhance the permeability (71 +/- 5 l m(-2) hr(-1) bar(-1) for 150 +/- 15 nm thick membranes).
65 ts (naive) for 4 weeks prior to undergoing 2-hr exposures to filtered air and concentrated ambient pa
66              Before their resolution at 3-20 hr after anaphase, the chromatin bridges induced nuclear
67  (11.6%; 95% CI: 0.8, 22.2; p = 0.04) and 20 hr after CAP exposure in the OO group.
68 reexposure, immediately postexposure, and 20 hr postexposure.
69 thesia and extracorporeal circulation for 20 hr with artificial PaO2 oscillations [3 cycles min(-)(1)
70 thesia and extracorporeal circulation for 20 hr with constant PaO2, n = 10) or an oscillation group (
71 lin-1 levels were significantly increased 20 hr after CAP exposure in the naive group only (17.1%; 95
72  by routine histology) in the OM, at 2 or 20 hr after termination of NA exposure, in humanized mice.
73   The cell death was largely delayed by 5-23 hr and was accompanied by a 5-fold activation of caspase
74 nial ultrasound was performed on day 1 (0-24 hr) and day 3 (48-72 hr).
75 ng, with delayed and sustained kinetics 6-24 hr after TNF stimulation.
76 a model, we investigated the effects of a 24 hr gluing manipulation in which the right index and righ
77 re compared to the starting values (after 24 hr).
78 ides involved in growth and feeding after 24 hr.
79 ssociated with remembered new-words after 24 hr.
80 genes between TN and TM at 0 hr, 4 hr and 24 hr after stimulation, respectively, with more genes expr
81                                  Four and 24 hr post-damage initiation by KBrO3, with BPA-only sample
82 mplex reach a steady-state between 18 and 24 hr.
83 rcadian clock that promotes approximately 24 hr rhythms in many behavioral and physiological processe
84                 Circadian ( approximately 24 hr) clocks regulate daily rhythms in physiology, metabol
85 mulation produced lasting ( approximately 24 hr) enhancement of recollection precision, without effec
86             Its circadian ( approximately 24 hr) oscillations of gene expression and electrical activ
87 reater than 100S) became present while at 24 hr primarily only mid-sized aggregates (20S to 80S) exis
88                                        By 24 hr following inoculation, a proinflammatory signature th
89 d lung cells harvested for flow cytometry 24 hr later.
90 hat exhibited of 4,500- fold expression, 24 hr following exposure to the toxic compound 5-hydroxymet
91 al Organ Failure Assessment (during first 24 hr of admission) score of at least 6.5 presented the bes
92 al Organ Failure Assessment (during first 24 hr of admission), easier to calculate than Acute Physiol
93 iliation pattern emerged within the first 24 hr of induced lineage specification, and these changes h
94 nts, the peak serum lactate (in the first 24 hr) observed in patients admitted to ICU following surge
95 /FIO2 (change in PaO2/FIO2 over the first 24 hr) were associated with mortality, whereas pressure var
96 sed in memory accuracy over the following 24 hr.
97 on >/= 90%) and "hyperoxia" (FIO2 1.0 for 24 hr) groups.
98 umol/L) or bovine serum albumin (BSA) for 24 hr.
99 w for average PAH exposure increased from 24 hr to 1 year, suggesting more of a chronic response.
100   Using a conventional dosing of 16 g/2 g/24 hr continuous infusion, obese patients were more likely
101 ex, < 30 kg/m) were treated with 16 g/2 g/24 hr continuous piperacillin-tazobactam infusion.
102 as inadequate in both groups and 16 g/2 g/24 hr was adequate only in nonobese patients.
103  Carlo simulations showed that 12 g/1.5 g/24 hr was inadequate in both groups and 16 g/2 g/24 hr was
104  longer different from the control groups 24 hr later.
105 rds evacuated their breeding territories >24 hr before the arrival of the storm and atmospheric varia
106 m) and the extensive sample preparation (>24 hr) needed for traditional exosome measurements.
107             Serum creatinine peaked late (24 hr), when clinical recovery was imminent.
108 lescent female mice from littermates for <24 hr increased first spike latency (FSL) and decreased exc
109 mal effect, and are resistant to multiple 24 hr freeze-thaws.
110 intraoperative and perioperative serum Na/24 hr variations compared to controls (16.69 +/- 5.17 vs. 9
111 ostratified epithelium over the course of 24 hr.
112     Consumption of turkey during the past 24 hr was not associated with total arsenic or DMA during t
113  xenon-treated groups in the early phase (24 hr) and up to 4 days after injury.
114 oxidative burden exposure in the previous 24 hr (6.0% increase per interquartile range change in glut
115 itatory synapses, whereas more prolonged (24 hr) firing depressed both AMPAR and NMDAR EPSCs and elim
116 eproductive traits: both the short-term (~24 hr) rate of offspring production after a female's first
117  ON or OFF medication, with memory tested 24 hr later ON or OFF medication (4 conditions, within-subj
118 e, Bdellovibrio can persist for more than 24 hr in vivo yet exert no pathogenic effects on zebrafish
119  further experiments were run without the 24 hr delay, but they too failed to reproduce effects of do
120 nged from -29 to + 40% in relation to the 24 hr mean of Rs, especially in trenched plots.
121  was tested in a semiarid shrubland using 24 hr campaigns using two treatments: trenched plots and pl
122 rk is able to control HD responses within 24 hr of eye opening.
123 se bats will exchange fat reserves within 24 hr, meaning that they must survive on the food of the da
124 orming a blastema at the oral pole within 24 hr.
125         Because of oil droplet binding, a 24-hr exposure was sufficient to create severe cardiac and
126 ariate method is based on collection of a 24-hr time series of plasma melatonin, a suprachiasmatic nu
127                    Although approximately 24-hr periodicity is observed across aerobic eukaryotes, th
128  Drosophila brain confer an approximately 24-hr rhythm onto locomotor activity are unclear, but invol
129  non-dsRNA expressing plants, recorded at 24-hr intervals post-ingestion for three days, was >90% and
130 idase (PO) response and bacterial load at 24-hr postinfection were ascertained.
131 m was to evaluate the association between 24-hr dietary recall of poultry consumption and arsenic exp
132 , cadmium, lead, and mercury by combining 24-hr dietary intake recall data from the 2011-2012 Nationa
133               Circadian clocks coordinate 24-hr rhythms of behavior and physiology.
134                   A shift from solar-day (24-hr period) to lunar-day (24.8-hr period) vertical migrat
135 RNA levels and mRNA decay rates following 24-hr exposure to noncytotoxic concentrations of sodium ars
136   Circadian neural circuits generate near 24-hr physiological rhythms that can be entrained by light
137         Daily ZIP code-level estimates of 24-hr average PM2.5 were calculated by averaging concentrat
138 y urea excretion was measured in repeated 24-hr urine collections between 6 and 18 months after trans
139                            Using a single 24-hr episode of MD and whole-cell patch clamp recording in
140 rom region CA1 neurons in rats across the 24-hr cycle, and we found that their firing increased upon
141                                     Using 24-hr dietary recall data, we quantified: a) fast food inta
142  for both groups (150 mL/min: 9.1 hr [5.5-26 hr] vs 10 hr [4.2-17 hr]; p = 0.37).
143        Thirty perfusion CT scans (median, 27 hr [interquartile range, 20-45] after traumatic brain in
144 2 mRNA rhythms were delayed by 0.97 +/- 0.29 hr (p < 0.01), indicating that human molecular clocks ma
145 lucose rhythms were delayed by 5.69 +/- 1.29 hr (p < 0.001), and average glucose concentration decrea
146 achieve the particulate matter target (< 1-3 hr/week), and substantially limited to meet the carbon m
147 e antibiotic therapy was 6.7 hours (0.0-23.3 hr).
148 ear sunset, onset occurring, on average, 3.3 hr after sunset.
149 P(Sc) reached maximal levels by 30 min and 3 hr, respectively, and blood levels were constantly highe
150 ration robust trace, lasting approximately 3 hr, and a parallel long-duration weak one, lasting up to
151  ST2, and then exposed to ozone (2 ppm for 3 hr).
152 hours after emergency department triage (< 3 hr reference) had a pooled odds ratio for mortality of 1
153 incubation time is extended from 10 min to 3 hr.
154 pecific transcriptional responses emerged 30 hr after injury, involving context-dependent patterning
155 ; P = 0.001) and kidneys with longer than 30 hr of cold ischemia time (CIT) (OR, 0.95; per 5% increme
156                                           32 hr after first sight, she visually recognized an object
157 Rewarming took a median of 21.5 hours (16-35 hr) and was performed without compromise in the cerebral
158 nts' circadian rhythms were measured in a 37-hr constant routine that removes sleep and environmental
159 an mechanical ventilation time (25.2 vs 19.4 hr; p < 0.01), cardiac surgical ICU readmissions (5.3% v
160 ans, the HSR declines precipitously over a 4 hr period in early adulthood coincident with the onset o
161  with opposite valence of plasticity after 4 hr of dark and visual stimulation.
162 ntified transcripts decayed by > 25% after 4 hr.
163 45 min (7.772 vs. 9.459 mM, P = 0.006) and 4 hr (8.202 vs. 10.235 mM, P = 0.003).
164  and their combination at 15 min, 1 hr and 4 hr (n = 4).
165 rom HMP kidneys were sampled at 45 min and 4 hr of preservation with the LifePort Kidney Transporter
166 y different between DGF and IGF kidneys at 4 hr (49.099 vs. 59.513 mM, P = 0.009).
167 fter the surgery), and esmolol (started at 4 hr after the surgery) + norepinephrine (started at 18 hr
168 ycan biosynthesis metabolites decreased at 4 hr by doripenem alone, reflecting its mechanism of actio
169 ry clusterin was measured serially between 4 hr and 7 days after transplantation.
170  (15 min and 1 hr) and later by doripenem (4 hr).
171 teoglycan components of PNNs were enhanced 4 hr after fear conditioning but were no longer different
172          We found that performing exercise 4 hr, but not immediately, after encoding improved the ret
173 n occupationally relevant dose (10 ppm for 4 hr).
174 expressed genes between TN and TM at 0 hr, 4 hr and 24 hr after stimulation, respectively, with more
175 s resulted in rapid defervescence (median, 4 hr) and clinical improvement.
176  group performed exercise immediately, one 4 hr later, and the third did not perform any exercise.
177 UVEC retention within the engraftment site 4 hr post-administration was 24.5 +/- 3.2%.
178                              We found that 4 hr post-damage initiation, BPA exposure and co-exposure
179 to ectopic Erk signaling, except from 1 to 4 hr post-fertilization, when perturbing the spatial exten
180 f promoter-anchored chromatin loops within 4 hr after inducing differentiation of 3T3-L1 preadipocyte
181 allow triaging of patients with DGF within 4 hr of transplantation.
182 (6 hr) cell viability test and long-term (40 hr) cell tracking confirmed that the proposed approach h
183 tion was 15 hours (interquartile range, 2-45 hr).
184 crosis factor-alpha (TNFalpha) protein by 45 hr, with earlier induction of TNFalpha mRNA in OE and OB
185                 Transfusions in the first 48 hr after CHLT were not substantial in most patients.
186 vity drops precipitously during the first 48 hr of differentiation, corresponding to a release of C/E
187 ried in their ratio of protein to fat for 48 hr and then injected with P. luminescens.
188 re survivors (mechanically ventilated for 48 hr or more).
189 y discrete foci of SIV (mac239) infection 48 hr after vaginal inoculation.
190 ere, we show that resveratrol (10 microM, 48 hr) induces both a cell growth arrest and a metabolic re
191 triplicate, analyzing 180 samples in only 48 hr (the equivalent of 16 min/sample).
192                  Postshock resuscitation (48 hr) comprised retransfusion of shed blood, crystalloids
193  tshawytscha), and find that in less than 48 hr, males can upregulate sperm velocity when faced with
194     The analysis, which was completed in 1.5 hr, included loading data, annotation, principal compone
195 velopment of organ dysfunction (median, 10.5 hr).
196  KCNQ1 surface-density declined with a 3.5 hr t1/2 by impaired forward trafficking.
197 licy from a restricted visitation model (4.5 hr/d) to an extended visitation model (12 hr/d).
198 y of 3.6 hours (interquartile range, 2.1-7.5 hr).
199 times from onset to offset, averaged 6.9-8.5 hr, with sleep durations of 5.7-7.1 hr, amounts near the
200 me on ventilator (difference of median, 98.5 hr; p = 0.003) and length of ICU stay (difference of med
201 e assembled a novel MRI dataset containing 5 hr of RSFC data, 6 hr of task fMRI, multiple structural
202            We investigated the effect of a 5-hr delay in meals on markers of the human master clock a
203 s (breakfast, lunch, dinner) were given at 5-hr intervals, beginning either 0.5 (early) or 5.5 (late)
204 ll, we find a simulated 5-day work week of 5-hr-per-night sleep opportunities and ad libitum food int
205 lent to exposure durations from 7.8 to 1,500 hr.
206 ys for 24-hour intervals (median time, 18.52 hr); 31 (26%) for 12-hour intervals (median, 10.58 hr);
207 llected from initiation of exposure until 54 hr later.
208 113 (72%) for 6-hour intervals (median, 9.55 hr).
209 1 (26%) for 12-hour intervals (median, 10.58 hr); 117 (47%) for 8-hour intervals (median, 9.60 hr); a
210 es eject male ejaculates from the uterus 1-6 hr after mating with a stereotypic behavior regulated by
211 ; 8 hr: odds ratio, 23.70; CI, 8.13-69.11; 6 hr: odds ratio, 71.95; CI, 25.13-206.0).
212  the ward prior to readmission (16.6 vs 23.6 hr; p = 0.05).
213 ch showed sustained increases peaking at 3-6 hr, respectively.
214                                         At 6 hr mid-sized aggregates (33S to 84S) and large aggregate
215 which the suite of mid-sized aggregates at 6 hr were most correlative with cytotoxicity.
216  MRI dataset containing 5 hr of RSFC data, 6 hr of task fMRI, multiple structural MRIs, and neuropsyc
217 xposure group) via inhalation for 28 days (6 hr/day for 5 days/week).
218 se in circulating WBCs, augment the early (6 hr postcecal ligation and puncture) recruitment of infla
219 ate to appetite hormone levels with early (6 hr) rises in plasma insulin and leptin followed by persi
220 s, six human participants were exposed for 6 hr in a chamber containing deliberately elevated air con
221  respectively) or citrate buffer alone for 6 hr.
222 ning induced more persistent insertion (>/=6 hr).
223 ne conditioning produced brief insertion (<6 hr after conditioning) of CP-AMPARs, whereas 2.8 kHz ton
224 PARs at LA synapses) when it was performed 6 hr after conditioning.
225 inistered later after the onset of sepsis (6 hr), and yet maintain their therapeutic effect with incr
226                                Short-term (6 hr) cell viability test and long-term (40 hr) cell track
227 half-life from 1.3 (+/- 0.3) to 26 (+/- 2.6) hr.
228 s study show that after rats experienced a 6-hr inhalation exposure to 20- and 110-nm AgNP at a singl
229 117 (47%) for 8-hour intervals (median, 9.60 hr); and 113 (72%) for 6-hour intervals (median, 9.55 hr
230 f 0.52 hours (interquartile range, 0.40-0.66 hr) and a median emergency department delay of 3.6 hours
231 predefined time points (3, 6, 12, 24, 48, 72 hr), animals (>/= 6 per group) underwent echocardiograph
232 ed: <1 hr (within 1-hour), 4-12 hr and 48-72 hr post injury.
233 erformed on day 1 (0-24 hr) and day 3 (48-72 hr).
234  to ozone (0.3 ppm) or filtered air for 6-72 hr.
235 S promoter, which remained elevated after 72 hr, contrary to global H3K9 hypoacetylation.
236         Global hypoacetylation of H3K9 at 72 hr was observed in UROtsa cells following tolerated and
237 cogenes FOS and JUN in both cell lines at 72 hr.
238 est concentration (200 muM) and duration (72 hr) tested.
239 cluded patients who died within the first 72 hr after transplantation, were undergoing simultaneous t
240  T cell/DC interactions into stage 3 (>72 hr) of activation.
241 e quantification of fractionated lysates (72 hr analysis) while obviating the need for peptide detect
242 ter (PM2.5) and carbon monoxide (CO) over 72 hr among a cohort of Tanzanian pregnant women.
243  the rostral ventrolateral medulla (rVLM) 72 hr after EA was increased (n = 9), compared to the sham-
244 ode oscillator cells oscillated for up to 72 hr.
245 rom 0.4 to 200 muM) on HepG2 cells over a 72-hr exposure period.
246 50,000 simulated exomes and it required 1.75 hr for the analysis of a quantitative trait using severa
247 ane administration was 3.33 hours (2.33-5.75 hr), range 1-19 hours with a mean consumption of 7.89 mL
248  (12 hr: odds ratio, 6.98; CI, 2.33-20.89; 8 hr: odds ratio, 23.70; CI, 8.13-69.11; 6 hr: odds ratio,
249 xposure durations at the consumer level of 8 hr or more.
250 llel long-duration weak one, lasting up to 8 hr, and dependent on the strong trace for its formation.
251  solar-day (24-hr period) to lunar-day (24.8-hr period) vertical migration takes place in winter when
252 egulatory metric (annual 4th highest daily 8-hr maximum) and were located only in urban core areas.
253 to meet the carbon monoxide guideline (< 7-9 hr/week).
254  exposure to a natural summer 14 hr 40 min:9 hr 20 min light-dark cycle entrains the human circadian
255 a weekend camping in a summer 14 hr 39 min:9 hr 21 min natural light-dark cycle compared to a typical
256 tored for spontaneous, convulsive seizures 9 hr/day every day for 24-36 days.
257 fter a week's exposure to a natural winter 9 hr 20 min:14 hr 40 min light-dark cycle as compared to t
258 g from the imposed short sleep schedule to 9-hr sleep opportunities for 3 days restored oral insulin
259 n sensitivity to baseline, but 5 days with 9-hr sleep opportunities was insufficient to restore intra
260 ventilation (weighted mean difference, -2.98 hr [95% CI, -5.08 to -0.89]; I = 34%; n = 334, five tria
261  minutes (average cooling rate, 14 degrees C/hr).
262    We estimate that 60 +/- 11 x 10(3) kg CH4 hr(-1) (95% CI) are emitted by natural gas and oil opera
263  performed an associative memory task during hr-fMRI in which they encoded and later retrieved face-n
264 , 8.1 +/- 2.2 years, AHI:11.1 +/- 5.9 events/hr), and 200 control subjects (84 male, 8.2 +/- 2.0 year
265  recessive ppd mutants on a spring-flowering hr mutant background show early, photoperiod-insensitive
266                 Approximately 6.2 +/- 1.1 kg hr(-1) was emitted from off-road diesel activities withi
267 nds facilities, and an additional 116-186 kg hr(-1) formed from the photochemical oxidation of diesel
268    Summed emissions totaled 7022 +/- 2000 kg hr(-1), roughly 9% of the entire basin-wide CH4 emission
269 ion are estimated to be 76 +/- 13 x 10(3) kg hr(-1) (equivalent to 0.66 +/- 0.11 Tg CH4 yr(-1); 95% c
270 the Barnett Shale was 6.6 +/- 0.2 x 10(3) kg hr(-1) and consistent across six days in spring and fall
271 ated hemoglobin polyoxyethylene (20 mg Hb/kg/hr) or an equal volume of placebo, infused for up to 150
272 , 0.15 mg/kg/hr (3.7 mg/kg/d) and 0.11 mg/kg/hr (2.8 mg/kg/d), increased by 36% and 58%, respectively
273  doses on the day of cannulation, 0.15 mg/kg/hr (3.7 mg/kg/d) and 0.11 mg/kg/hr (2.8 mg/kg/d), increa
274 g/kg/hr opioids (9.7 mg/kg/d) and 0.39 mg/kg/hr benzodiazepines (9.4 mg/kg/d) at decannulation, an in
275 followed by a continuous infusion of 1 mg/kg/hr for 5 hours (pFX06; n = 6) or the control group (pCON
276 ischarge, patients were receiving 0.40 mg/kg/hr opioids (9.7 mg/kg/d) and 0.39 mg/kg/hr benzodiazepin
277 onists ELA or Apelin-13 (39 and 15 microg/kg/hr, respectively) versus normal saline.
278 ol: 3 mL/kg during 1 hour before and 1 mL/kg/hr during 6 hours after contrast medium exposure.
279 tients were randomized to receive 0.25 mL/kg/hr pyridoxalated hemoglobin polyoxyethylene (20 mg Hb/kg
280  Sheep were treated with clonidine (1 mug/kg/hr) or saline-vehicle from 24 to 32 hours of sepsis.
281 ted clearance rates ranged from 1.2 to 7.4 L hr(-1) bivalve(-1).
282  cleared very quickly at a rate of 320mmol/L/hr, mostly by liver metabolism and re-conversion of lact
283  and peripheral compartments (V2) to be 35 L/hr (95% CI, 32-39 L/hr), 55 L/hr (95% CI, 42-68 L/hr), 2
284 artments (V2) to be 35 L/hr (95% CI, 32-39 L/hr), 55 L/hr (95% CI, 42-68 L/hr), 203 L (95% CI, 140-26
285 V2) to be 35 L/hr (95% CI, 32-39 L/hr), 55 L/hr (95% CI, 42-68 L/hr), 203 L (95% CI, 140-266 L), and
286 5% CI, 32-39 L/hr), 55 L/hr (95% CI, 42-68 L/hr), 203 L (95% CI, 140-266 L), and 523 L (95% CI, 428-6
287 ared to those without (+0.2 vs -0.006 mmol/L/hr; p < 0.001).
288 , beginning either 0.5 (early) or 5.5 (late) hr after wake.
289 boli (adjusted risk ratio, 1.03/microembolus/hr; 95% CI, 1.01-1.05) and with persistently positive tr
290 exhibited a hydraulic permeability of 130 ml/hr/m(2)/mmHg, which is more than 3 fold greater than exi
291 -19 hours with a mean consumption of 7.89 mL/hr (SD, 2.99).
292 ), an erythrocyte sedimentation rate of 2 mm/hr (normal range, 0-40 mm/hr), and a C-reactive protein
293 ation rate of 2 mm/hr (normal range, 0-40 mm/hr), and a C-reactive protein value of 0.4 mg/L (3.8 nmo
294 l MRI and high-resolution 3T functional MRI (hr-fMRI), we evaluated MTL subfield thickness and functi
295 ion was 58 hours at a median rate of 100 mug/hr.
296 ommunities were 2.8, 3.2, 17.8, and 24.3 muM hr(-1) for SEA, AS, SOIL, and SEW, respectively, and wer
297  mineralizes BACs at a rate up to 2.40 mumol hr(-1) 10(-11) cells.
298 ion ICU length of stay (0.095-d increase per hr of time to deliver appropriate antibiotic therapy; 95
299 ospital length of stay (0.134-d increase per hr of time to deliver appropriate antibiotic therapy; 95
300        Subsequently, all cases of high-risk (hr) HPV-associated high-grade lesions and carcinomas in

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