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1 g cancers caused by EBV, hepatitis B, C, and human T cell lymphotropic virus.
2 ncluding hepatitis B and C viruses, HIV, and human T-cell lymphotropic virus.
3 A was found in several cell types, including human T-cell lymphotropic virus 1 (HTLV-1)-infected cell
4 urrent study we examined the response of the human T cell lymphotropic virus-1 (HTLV-1) Tax-specific
5 B and C viruses (HBV and HCV, respectively), human T cell lymphotropic virus-1 (HTLV-1), and Kaposi's
7 ing protein (CREB) activity is stimulated by human T-cell lymphotropic virus-1 (HTLV-1) Tax through t
8 y block transcription from DNA constructs of human T-cell lymphotropic virus-1 (HTLV-1), which use th
10 in-Barr virus, human immunodeficiency virus, human T-cell lymphotropic virus-1, Helicobacter pylori,
11 es such as the human immunodeficiency virus, human T-cell lymphotropic virus and murine leukaemia vir
12 titis B and C, human immunodeficiency virus, human T-cell lymphotropic virus, and bacterial contamina
13 rizes the current pathophysiologic basis for human T cell lymphotropic virus-associated leukemia/lymp
14 on by CD4(+) cells in patients infected with human T cell lymphotropic virus (HTLV) is associated wit
15 s to understanding the immunopathogenesis of human T cell lymphotropic virus (HTLV) type I-associated
16 EIA) kits for the measurement of antibody to human T-cell lymphotropic virus (HTLV) type 1, one from
17 the human Immuno-deficiency virus (HIV), the human T-cell lymphotropic virus (HTLV), the hepatitis C
18 ed from peripheral B cells of a patient with human T-cell lymphotropic virus (HTLV)-associated myelop
19 C virus (HCV), human papilloma virus (HPV), human T-cell lymphotropic virus (HTLV-1) and Kaposi's as
21 ther IL-15 contributes to the persistence of human T cell lymphotropic virus I viral-specific CD8 cel
22 rsion of Mikbeta1 in groups of patients with human T cell lymphotropic virus I-associated myelopathy/
23 of IL-15 in the neurological disease termed human T cell lymphotropic virus I-associated myelopathy/
24 donors are being screened for infection with human T cell lymphotropic viruses I and II (HTLV-I/II) b
25 Adult T-cell leukemia (ATL) is caused by human T-cell lymphotropic virus I (HTLV-1) and is an agg
26 We found that transcription directed by the human T-cell lymphotropic virus I activator protein Tax
27 egulated transcription of human retroviruses human-T cell lymphotropic virus I and human immunodefici
29 f this study show that DUB-2 is expressed in human T-cell lymphotropic virus-I (HTLV-1)-transformed T
30 mycosis fungoides (MF), are seronegative for human T-cell lymphotropic virus-I or -II (HTLV-I/II) whe
32 v), are being evaluated for the treatment of human T cell lymphotropic virus type 1 (HTLV-1)-associat
34 r exclusive to delta-retroviruses, including human T cell lymphotropic virus type 1 and 2 (HTLV-1 and
35 ted T cell clone, a T cell clone infected by human T cell lymphotropic virus type I (HTLV-I) displays
36 sited by Yamano et al., who demonstrate that human T cell lymphotropic virus type I (HTLV-I) infectio
39 that practically all patients with MF harbor human T cell lymphotropic virus type I (HTLV-I) proviral
40 measured from total CD8(high) cells and from human T cell lymphotropic virus type I (HTLV-I) Tax11-19
41 ined the association between mother-to-child human T cell lymphotropic virus type I (HTLV-I) transmis
42 re, its expression is 3- to 4-fold higher in human T cell lymphotropic virus type I (HTLV-I)-infected
46 +)CD25(+) T cell population in patients with human T cell lymphotropic virus type I-associated (HTLV-
48 ysiological mechanisms that are operative in human T cell lymphotropic virus type I-associated myelop
49 , natural killer-like YT leukemia cells, and human T cell lymphotropic virus type I-transformed MT-2
52 (ATLL) is an aggressive malignancy caused by human T cell lymphotropic virus type-I (HTLV-I) without
53 pha mRNA and protein levels are increased in human T cell lymphotropic virus type-I (HTLV-I)-associat
55 t present in individual integration sites of human T-cell lymphotropic virus type 1 (HTLV-1) and huma
61 the long-standing puzzle on progression from Human T-cell Lymphotropic Virus Type 1 (HTLV-1) infectio
62 e susceptibility of dendritic cells (DCs) to human T-cell lymphotropic virus type 1 (HTLV-1) infectio
71 lted in increased Tax transactivation of the human T-cell lymphotropic virus type 1 (HTLV-1) long ter
72 orescence spectroscopy was used to study the human T-cell lymphotropic virus type 1 (HTLV-1) nucleoca
74 sly determined that amino acids 64 to 120 of human T-cell lymphotropic virus type 1 (HTLV-1) Rex can
78 nd utilized to determine the distribution of human T-cell lymphotropic virus type 1 (HTLV-1) tax prov
79 cells (PBMC) with the ACH molecular clone of human T-cell lymphotropic virus type 1 (HTLV-1) to study
80 ing protein (CBP)/p300-binding domain of the human T-cell lymphotropic virus type 1 (HTLV-1) transact
84 iral expression in individuals infected with human T-cell lymphotropic virus type 1 (HTLV-1), a real-
85 play a critical role in the pathogenesis of human T-cell lymphotropic virus type 1 (HTLV-1)-associat
86 onic progressive neurologic diseases such as human T-cell lymphotropic virus type 1 (HTLV-1)-associat
88 exposure events over a 10-year period, in a human T-cell lymphotropic virus type 1 (HTLV-1)-endemic
89 ew monoclonal antibodies (MAbs) that inhibit human T-cell lymphotropic virus type 1 (HTLV-1)-induced
90 irus (HIV), we found that VCAM-1 can mediate human T-cell lymphotropic virus type 1 (HTLV-1)-induced
99 uction of NF-kappa B DNA binding activity in human T-cell lymphotropic virus type 1-transformed C81 c
102 existence of at least two major subtypes of human T-cell lymphotropic virus type 2 (HTLV-2), designa
103 onal activity of the p53 tumor suppressor in human T-cell lymphotropic virus type 2 (HTLV-2)-transfor
113 transcription factors Ets1 and Sp1 and that human T-cell lymphotropic virus type I (HTLV-I) Tax coop
117 te to central nervous system inflammation in human T-cell lymphotropic virus type I (HTLV-I)-associat
118 mononuclear cells (PBMCs) from patients with human T-cell lymphotropic virus type I (HTLV-I)-associat
119 nd CD28RE-TRE is the exclusive target of the human T-cell lymphotropic virus type I oncoprotein Tax.
122 blood mononuclear cells, and a high ratio of human T-cell lymphotropic virus type I proviral load in
125 ormation of multinucleated cells, similar to human T-cell lymphotropic virus type I Tax-expressing ce
127 , the picornavirus group, influenza A virus, human T-cell lymphotropic virus type I, and St Louis enc
128 Bolivian arenaviruses), the human retrovirus human T-cell lymphotropic virus type I, Lyme disease and
129 fluid may play a role in the pathogenesis of human T-cell lymphotropic virus type I-associated myelop
130 virus type I proviral load in patients with human T-cell lymphotropic virus type I-associated myelop
131 thought to be related to the pathogenesis of human T-cell lymphotropic virus type I-associated myelop
132 viral load in cerebrospinal fluid cells from human T-cell lymphotropic virus type I-associated myelop
133 servations suggest that accumulation of both human T-cell lymphotropic virus type I-infected cells an
134 infected cells and preferential expansion of human T-cell lymphotropic virus type I-specific CD8+ cel
135 65/RelA at Ser-536 in the presence of Tax in human T-cell lymphotropic virus type I-transformed cells
137 low virion-associated polymerase activity of human T-cell lymphotropic virus type-1 (HTLV-1) are due
138 -cell leukemia/lymphoma (ATL) resulting from human T-cell lymphotropic virus type-1 (HTLV-1) infectio
139 initiation site (DIS) of the deltaretrovirus human T-cell lymphotropic virus type-I (HTLV-I) has been
140 moter is up-regulated during transmission of human T-cell lymphotropic virus type-I (HTLV-I) to prima
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