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1 ncogenic retroviruses are common in animals, human T-lymphotropic virus 1 (HTLV-1) is the only transm
2 Recent studies suggest that infection with human T-lymphotropic virus 1 (HTLV-1) might be associate
3 lly oncogenic activation of NF-kappaB by the human T-lymphotropic virus 1 (HTLV-1) oncoprotein Tax im
4 chronic progressive myelopathies, including human T-lymphotropic virus 1 (HTLV-1)-associated myelopa
5 ence of all known viral genomes and discover human T-lymphotropic virus 1 integrations in six samples
10 tin-3 is abundantly expressed in a number of human T lymphotropic virus (HTLV)-I-infected human T cel
11 and CD86 on T cells following infection with human T lymphotropic virus (HTLV)-I/II and its functiona
12 ional studies support sexual transmission of human T lymphotropic virus (HTLV)-I/II; however, prospec
13 ripheral blood mononuclear cells (PBMC) from human T lymphotropic virus (HTLV)-II-infected persons we
16 CV), human immunodeficiency virus (HIV), and human T-lymphotropic virus (HTLV) among tissue donors.
19 risk people from Central Africa identified 2 human T-lymphotropic virus (HTLV)-4-infected individuals
27 eukemia Virus (BLV) and humans infected with Human T Lymphotropic Virus type 1 (HTLV-1) which togethe
28 munologic interaction between infection with human T lymphotropic virus type 1 (HTLV-1), a retrovirus
29 o-cell transmission of retroviruses, such as human T lymphotropic virus type 1 (HTLV-1), is well docu
30 eukemia virus type 1 (HTLV-1), also known as human T lymphotropic virus type 1, was the first exogeno
35 clinical analysis of 308 Jamaican children, human T lymphotropic virus type I (HTLV-I) infection was
36 ic profile associated with susceptibility to human T lymphotropic virus type I (HTLV-I) infection, we
37 versity of the T cell repertoire involved in human T lymphotropic virus type I (HTLV-I) infections, p
45 on of autoimmune disease, from patients with human T lymphotropic virus type I (HTLV-I)-associated my
49 l lines that can be productively infected by human T-lymphotropic virus type 1 (HTLV-1) and can sprea
55 ty to interact with a TG-rich element in the human T-lymphotropic virus type 1 (HTLV-1) enhancer thou
57 Over the past 25 years, animal models of human T-lymphotropic virus type 1 (HTLV-1) infection and
61 Studies that analyzed the effect of A3G on human T-lymphotropic virus type 1 (HTLV-1) infectivity r
79 information on the biochemical properties of human T-lymphotropic virus type 1 (HTLV-1) reverse trans
82 ervation that transgenic mice expressing the human T-lymphotropic virus type 1 (HTLV-1) tax gene unde
85 his study, we analyzed the effect of Brd4 on human T-lymphotropic virus type 1 (HTLV-1) transcription
88 46 with aggressive subtype) associated with human T-lymphotropic virus type 1 (HTLV-1) were analyzed
91 ncoded by the pX open reading frame I of the human T-lymphotropic virus type 1 (HTLV-1), is a hydroph
93 no recombinant strain had been observed for human T-lymphotropic virus type 1 (HTLV-1), the first is
94 molecular mimicry, we studied patients with human T-lymphotropic virus type 1 (HTLV-1)-associated my
96 There are currently 5 million to 10 million human T-lymphotropic virus type 1 (HTLV-1)-infected peop
100 BV), Kaposi's sarcoma herpesvirus (KSHV) and human T-lymphotropic virus type 1 are major contributors
101 , completely abrogated for activation of the human T-lymphotropic virus type 1 long terminal repeat a
103 ral oncoproteins, adenovirus type 9 E4-ORF1, human T-lymphotropic virus type 1 Tax, and high-risk hum
104 HTLV-1-associated myelopathy (HAM; HTLV-1 is human T-lymphotropic virus type 1) is a chronic debilita
105 embrane binding for the Gag matrix domain of human T-lymphotropic virus type 1, which provides insigh
108 iral populations from subjects infected with human T-lymphotropic virus type 1; (ii) T cell antigen r
110 nce of antibody to Tax regulatory protein of human T-lymphotropic virus type I (HTLV-I) are character
116 well as Tax-mediated transactivation of the human T-lymphotropic virus type I (HTLV-I) promoter.
120 ficantly reduced HIV-1 plasma viral load and human T-lymphotropic virus type I proviral load in infec
122 o the diagnosis include infections with HIV, human T-lymphotropic virus type I, or hepatitis C virus.
123 hese cells compared with Jurkat cells or the human T-lymphotropic virus type I-infected C81 and MT2 c
124 nd human immunodeficiency virus (HIV) and/or human T-lymphotropic virus type II (HTLV-II) is common a
125 some of whom may be coinfected with HIV and human T-lymphotropic virus type II (HTLV-II), are at hig
126 The molecular and genetic factors induced by human T-lymphotropic virus type-1 (HTLV-1) that initiate
129 e demographic and geographic determinants of human T lymphotropic virus types I and II (HTLV-I and -I
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