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1 h live-cell imaging (in Escherichia coli and human embryonic kidney 293 cells).
2 or endocytosis] in the same cell background (human embryonic kidney 293S cells).
3 eta) and phosphoinositide-3-kinase (PI3K)-in human embryonic kidney 293 cells.
4 with a full-length protein in co-transfected human embryonic kidney 293 cells.
5 of CaV2.3 through NK1 receptors expressed in human embryonic kidney 293 cells.
6 pendent inactivation of recombinant I(Ca) in human embryonic kidney 293 cells.
7 ng mutated subunits transiently expressed in human embryonic kidney 293 cells.
8 when it is over-expressed on the surface of human embryonic kidney 293 cells.
9 3gamma2 receptors recombinantly expressed in human embryonic kidney 293 cells.
10 alternative coreceptor for AAV2 infection in human embryonic kidney 293 cells.
11 iotensin II type 1(A) receptor, expressed in human embryonic kidney 293 cells.
12 tation of alpha4beta2 receptors assembled in human embryonic kidney 293 cells.
13 ne glomerular epithelial cells as well as in human embryonic kidney 293 cells.
14 nd vice versa and expressed the receptors in human embryonic kidney 293 cells.
15 SR141716A, and was absent in nontransfected human embryonic kidney 293 cells.
16 sponse was restored in TLR4-MD-2-transfected human embryonic kidney 293 cells.
17 MTMR2 and MTMR13 proteins are associated in human embryonic kidney 293 cells.
18 urrent at 1, 3, and 10 muM, respectively, in human embryonic kidney 293 cells.
19 d mutated cDNAs in Chinese hamster ovary and human embryonic kidney 293 cells.
20 ased mouse 71 (M71) OR surface expression in human embryonic kidney 293 cells.
21 beta1AR expressed in baby hamster kidney and human embryonic kidney 293 cells.
22 ith MEK1 in vascular smooth-muscle cells and human embryonic kidney 293 cells.
23 pha3beta4 nAChRs heterologously expressed in human embryonic kidney 293 cells.
24 soforms, were expressed in varying ratios in human embryonic kidney 293 cells.
25 neurons and patch-clamp electrophysiology in human embryonic kidney 293 cells.
26 ed with Kv4.3 and its beta-subunit KChIP2 in human embryonic kidney 293 cells.
27 nduced NF-kappaB activation and apoptosis in human embryonic kidney 293 cells.
28 st-induced endocytosis in stably transfected human embryonic kidney 293 cells.
29 and fluorescence imaging after expression in human embryonic kidney 293 cells.
30 wild-type DGK zeta, but not DGK zeta S/D, in human embryonic kidney 293 cells.
31 (nAChR) subtypes heterologously expressed in human embryonic kidney 293 cells.
32 at dopaminergic cell line (N27 cells) and in human embryonic kidney 293 cells.
33 accumulation in calcitonin receptor-bearing human embryonic kidney 293 cells.
34 lpha1beta receptors transiently expressed in human embryonic kidney 293 cells.
35 oexpression of constitutively active PI3K in human embryonic kidney 293 cells.
36 CV and ACII, respectively, were expressed in human embryonic kidney 293 cells.
37 orly expressed on the surface of transfected human embryonic kidney 293 cells.
38 ds also inhibit CYP27A1 reporter activity in human embryonic kidney 293 cells.
39 full complement of L-type CaV1.2 subunits in human embryonic kidney 293 cells.
40 the mutant were characterized in transfected human embryonic kidney 293 cells.
41 ressed with the alpha2 and beta1 subunits in human embryonic kidney 293 cells.
42 oth the wild-type and the mutant receptor in human embryonic kidney 293 cells.
43 hemagglutinin epitopes and transfected into human embryonic kidney 293 cells.
44 ation, desensitization, and sequestration in human embryonic kidney 293 cells.
45 a and beta(3) subunits were transfected into human embryonic kidney 293 cells.
46 unctional selectivity of the AT1 receptor in human embryonic kidney 293 cells.
47 both inward currents and calcium influx into human embryonic kidney 293 cells.
48 trated after expression of these elements in human embryonic kidney 293 cells.
49 ta4274-4535) were generated and expressed in human embryonic kidney 293 cells.
50 outside-out patches excised from transfected human embryonic kidney 293 cells.
51 hrin tyrosine phosphorylation in transfected human embryonic kidney 293 cells.
52 sK(ATP) channels expressed heterologously in human embryonic kidney 293 cells.
53 to dynamin when the receptor is expressed in human embryonic kidney 293 cells.
54 nally evaluated by patch-clamp studies using human embryonic kidney 293 cells.
55 was carried out by patch-clamp technique in human embryonic kidney 293 cells.
56 nescence resonance energy transfer (BRET) in human embryonic kidney 293 cells.
57 ed as TRPM1 by labeling of TRPM1-transfected human embryonic kidney 293 cells.
58 f muscle acetylcholine receptor expressed in human embryonic kidney 293 cells.
59 w fluorescent protein (YFP) and expressed in human embryonic kidney 293 cells.
60 a signaling and intracellular trafficking in human embryonic kidney 293 cells.
61 ressed with the alpha3 and beta4 subunits in human embryonic kidney 293 cells.
62 has no effect on PC1 surface localization in human embryonic kidney 293 cells.
63 ot Rac1, RhoQ, RhoD, or RhoV, in transfected human embryonic kidney 293 cells.
64 ogenously expressed H1 histamine receptor in human embryonic kidney 293 cells.
65 l promoter-directed luciferase expression in human embryonic kidney 293 cells.
66 agonist-induced receptor internalization in human embryonic kidney 293 cells.
67 tion, and agonist-induced desensitization in human embryonic kidney 293 cells.
68 nsiveness to reporter gene in both HepG2 and human embryonic kidney 293 cells.
69 ,N188Q, and N181Q,N188Q,N205Q), expressed in human embryonic kidney-293 cells.
70 s toward store-operated Ca2+ entry (SOCE) in human embryonic kidney-293 cells.
71 thetic LXR-dependent promoter in transfected human embryonic kidney-293 cells.
72 erentiated THP-1 cells, and FPR1-transfected human embryonic kidney-293 cells.
73 also able to promote rapamycin resistance in human embryonic kidney-293 cells.
74 imental system using adenovirus infection of human embryonic kidney (293) cells.
75 ys with membranes of human Kv11.1-expressing human embryonic kidney 293 cells, 2 existing compounds (
76 ine delta opioid receptor (DOR) expressed in human embryonic kidney 293 cells, a well characterized m
77 bserved that Rheb does not activate TORC2 in human embryonic kidney 293 cells, although it potently s
78 beta4 recombinant nAChRs expressed stably in human embryonic kidney 293 cells and accelerates its act
79 nesis and a miR-107 inhibitor in transfected human embryonic kidney 293 cells and Be(2)C cells that e
80 as investigated by voltage-clamp analysis in human embryonic kidney 293 cells and by current-clamp an
81 s study, we stably transfected HJV cDNA into human embryonic kidney 293 cells and characterized the p
82 produced soluble rHLA-G1 (rsG1) and rsG2 in human embryonic kidney 293 cells and characterized the p
83 fibronectin through alpha5beta1 integrin in human embryonic kidney 293 cells and depended on determi
84 e mutant constructs were stably expressed in human embryonic kidney 293 cells and exhibited similar e
86 receptor subunit, GluR1, and expressed it in human embryonic kidney 293 cells and hippocampal neurons
87 esponsible for store-operated Ca2+ influx in human embryonic kidney 293 cells and human T cells and f
89 gulation of the microOR was examined in both human embryonic kidney 293 cells and in betaarr2-KO mice
90 tably transfected cells induces apoptosis in human embryonic kidney 293 cells and in the human breast
91 ed a beta-catenin/TCF-responsive reporter in human embryonic kidney 293 cells and in two human colore
92 RyR1 function, following their expression in human embryonic kidney 293 cells and incorporation in li
93 or kappa-ORs with human Cav2.3 or Cav2.2 in human embryonic kidney 293 cells and measured depolariza
95 pha1beta2 and alpha1beta2-R207C receptors in human embryonic kidney 293 cells and studied receptor ki
96 expressed human Ca(v)3.1 T-type channels in human embryonic kidney 293 cells and studied the state-d
97 (beta2AR) were characterized in both intact human embryonic kidney 293 cells and subcellular fractio
98 educes store-operated Ca(2+) entry (SOCE) in human embryonic kidney 293 cells and the Ca(2+) release-
99 r alpha2beta1epsilon subunit combinations in human embryonic kidney 293 cells and used rapid solution
100 protocol for diolistic labeling of cultured human embryonic kidney 293 cells and whole brain using a
101 t cloned kappa opioid receptors expressed in human embryonic kidney-293 cells and at native kappa opi
102 17N significantly attenuated I(Na) by 27% in human embryonic kidney-293 cells and by 32% in neonatal
103 and ZASP1-D117N on Na(v)1.5 were studied in human embryonic kidney-293 cells and neonatal rat cardio
104 atically reduced the expression of HSV-TK in human embryonic kidney 293 cells, and it subsequently in
105 expressed wild-type and mutated receptors in human embryonic kidney 293 cells, and recorded resulting
106 ells and independently expressed proteins in human embryonic kidney 293 cells, and using guanosine 5'
107 Mutant forms of SCN5A were expressed in human embryonic kidney-293 cells, and functions were ass
109 mic vacuoles in the PIKfyveK1831E-expressing human embryonic kidney 293 cells as enlarged multivesicu
110 toring K(ATP) single-channel activities from human embryonic kidney 293 cell-attached patches express
112 oexpression of PSD-95 with NMDA receptors in human embryonic kidney 293 cells blocked cleavage of NR2
114 overexpression increased ENaC endocytosis in human embryonic kidney 293 cells but had no effect in Fi
115 was retained in the ER of stably transfected human embryonic kidney 293 cells but rapidly reached the
116 Hsp, alphaA-crystallin, was overexpressed in human embryonic kidney 293 cells, but wild-type CFTR bio
117 MD-2 were used to investigate activation of human embryonic kidney 293 cells by different TLR4 agoni
119 ns that coimmunoprecipitated with IRS-4 from human embryonic kidney 293 cells by microsequencing thro
120 a(v)3.2 channels heterologously expressed in human embryonic kidney 293 cells, by initiating the meta
121 erologously expressed in Xenopus oocytes and human embryonic kidney 293 cells, causing a specific dec
122 sed cysteinyl leukotriene type 1 receptor in human embryonic kidney 293 cells, clearly demonstrate th
125 Gi activation and arrestin-3 recruitment in human embryonic kidney 293 cells confirmed that norbupre
128 of FPRL1, but not by untransfected parental human embryonic kidney 293 cells, confirming the use of
129 h TrpC channels was reconstituted in HEK293 (human embryonic kidney 293) cells cotransfected with mGl
131 e fraction, as well as when overexpressed in human embryonic kidney 293 cells, demonstrating that Glu
132 (DAT), which is heterologously expressed in human embryonic kidney 293 cells, either with copper phe
134 ly inhibited SK export, and SK-1-transfected human embryonic kidney 293 cells exhibited enhanced rele
135 ressed receptor; however, we determined that human embryonic kidney 293 cells express an endogenous V
139 Galpha(q)/Gbetagamma heterotrimer in living human embryonic kidney 293 cells expressing fluorescent-
141 d human umbilical vein endothelial cells and human embryonic kidney 293 cells expressing green fluore
142 single site in brush border membranes and to human embryonic kidney 293 cells expressing NPC1L1.
143 lation of the innate immune system, cultured human embryonic kidney 293 cells expressing one of seven
148 lar concentration of Ca(2+) ([Ca(2+)](e)) in human embryonic kidney 293 cells expressing the Ca(2+)-s
150 ctions were obtained from double-recombinant human embryonic kidney 293 cells expressing the human A3
152 ss of agonist binding was observed on intact human embryonic kidney 293 cells expressing the W203A re
154 The CB2/Hsp90 interaction was confirmed in human embryonic kidney 293 cells expressing transfected
155 n TRPV5 activity using biochemical assays in Human Embryonic Kidney 293 cells expressing TRPV5, isoel
157 rly and late phases of recycling of AT1-R in human embryonic kidney 293 cells expressing wild-type or
158 experiments combined with calcium imaging in Human Embryonic Kidney-293 cells expressing DMI1 (the wi
163 antial promoter activity in mouse NIH 3T3 or human embryonic kidney 293 cells grown in the presence o
164 ansfer (BRET) assay platform, our studies in human embryonic kidney 293 cells have identified norepin
166 was demonstrated in selectivity study using human embryonic kidney 293 cells (HEK 293) as normal cel
167 ong human epithelial carcinoma cells (HeLa), human embryonic kidney 293 cells (HEK-293), and mouse my
169 e ventricular cardiac myocytes (24+/-8%) and human embryonic kidney 293 cells heterologously expressi
172 ) receptor also precipitates FLAG-S100B from human embryonic kidney 293 cell homogenates and endogeno
173 lating cAMP accumulation in receptor-bearing human embryonic kidney 293 cells in a concentration-depe
174 3A)) expressed in Xenopus laevis oocytes and human embryonic kidney 293 cells in a manner that was de
175 Induced expression of wild-type MAB21L2 in human embryonic kidney 293 cells increased phospho-ERK (
177 In addition, single-channel recordings using human embryonic kidney 293 cells indicated profoundly al
179 Expression of recombinant ABCA3 in non-lung human embryonic kidney 293 cells induced formation of la
180 pression of FZD4 and Galpha12 or Galpha13 in human embryonic kidney 293 cells induces WNT-dependent m
182 ced increase in intracellular iron levels in human embryonic kidney 293 cells is dependent on the pre
183 and pharmacology in vivo, in slices, and in human embryonic kidney 293 cells, it was shown that the
184 bestrophins were heterologously expressed in human embryonic kidney-293 cells, large Ca2+-activated C
186 Human recombinant PEDF was expressed in the human embryonic kidney 293 cell line and purified by amm
189 stion, we established tetracycline-inducible human embryonic kidney 293 cell lines for overexpression
190 endocytic, and recycling pathways in stable human embryonic kidney 293 cell lines inducibly expressi
193 on in an antibody-independent manner, stable human embryonic kidney-293 cell lines were produced that
195 rom cardiac myocytes and Na(V)1.5-expressing human embryonic kidney 293 cells, negative pressure incr
196 ith a ChoRE-containing reporter plasmid into human embryonic kidney 293 cells, no increase in promote
200 copper with cDDP (and several analogs) into human embryonic kidney 293 cells overexpressing wild-typ
203 a2 subunit with alphabeta-tandem subunits in human embryonic kidney 293 cells produced currents that
204 r to recombinant wild type human Na(v)1.5 in human embryonic kidney 293 cells produces a significant
205 receptor density in transiently transfected human embryonic kidney 293 cells provided parameters of
207 ter and mobility shift assays in transfected human embryonic kidney 293 cells, quantitative polymeras
208 so inhibited Ras-induced Raf-1 activation in human embryonic kidney 293 cells, Raf-1 and mitogen-acti
210 re, transient expression of TLR1 and TLR2 in human embryonic kidney 293 cells resulted in activation
211 ditionally, expression of alpha(M)beta(2) on human embryonic kidney 293 cells resulted in enhanced ce
212 nd that siRNA-mediated knockdown of EXTL2 in human embryonic kidney 293 cells resulted in increased c
214 nctional expression of the R672S channels in human embryonic kidney 293 cells revealed a small but si
218 In vitro studies in beta2AR-overexpressing human embryonic kidney 293 cells showed significant beta
220 and whole-cell recording after expression in human embryonic kidney 293 cells showed that P2X2[T339S]
221 ern blot analysis and confocal microscopy of human embryonic kidney 293 cells showed that the majorit
222 ceptors, which are endogenously expressed in human embryonic kidney 293 cells, showed little change i
223 d atypical BrS mutations were coexpressed in Human Embryonic Kidney-293 cells, showing a reduction in
227 ular calcium by several hundred nanomolar in human embryonic kidney 293 cells stably expressing CB(1)
228 forming whole-cell patch-clamp recordings of human embryonic kidney 293 cells stably expressing human
229 cs of agonist-induced currents recorded from human embryonic kidney 293 cells stably expressing the r
231 iguration was used on Na(+) and DA-preloaded human embryonic kidney 293 cells stably transfected with
234 Co-expression of GSK3beta and Rap1GAP in human embryonic kidney 293 cells stimulated proteasome-d
235 K1-dependent phosphorylation targets in HEK (human embryonic kidney) 293 cells stimulated by mitochon
237 In response to phagocytosis of latex beads, human embryonic kidney 293 cells synthesized cholesterol
239 (2)) by the Ca(2+)-sensing receptor (CaR) in human embryonic kidney 293 cells that express either the
240 ic siRNA library using an autophagy assay in human embryonic kidney 293 cells that measures lipidatio
241 by introduction of human alpha(1A) cDNAs in human embryonic kidney 293 cells that stably coexpressed
242 wo proteins produced a fluorescent signal in human embryonic kidney 293 cells that was not obtained w
243 CRF-induced cAMP response was 8.6 nmol/L in human embryonic kidney-293 cells that express only CRF(1
248 When expressed via an inducible promoter in human embryonic kidney 293 cells, the rat Mas-related ge
249 a inhibits Ca(2+) uptake in TRPV5-expressing human embryonic kidney 293 cells through the activation
250 bimolecular fluorescence complementation in human embryonic kidney 293 cells to compare the abilitie
251 humans, and the mouse gene was expressed in human embryonic kidney 293 cells to confirm its identity
252 and beta2 subunit cDNA were transfected into human embryonic kidney 293 cells to create a novel model
256 Furthermore, CRAMP induced the chemotaxis of human embryonic kidney 293 cells transfected with either
257 f MRP7 using membrane vesicles prepared from human embryonic kidney 293 cells transfected with MRP7 e
258 h TLR4 for both proteins was demonstrated in human embryonic kidney 293 cells transfected with TLR4 a
259 ese functional effects were also observed in human embryonic kidney 293 cells transfected with variou
260 adenosine monophosphate (cAMP) production in human embryonic kidney-293 cells transfected with CRF(1)
261 increased inositol triphosphate formation in human embryonic kidney-293 cells transfected with human
262 paB activation induced by B. pseudomallei in human embryonic kidney-293 cells transfected with TLR5 a
263 tivated current in Chinese hamster ovary and human embryonic kidney 293 cells transiently expressing
264 methyl-d-aspartate (NMDA)-evoked currents in human embryonic kidney 293 cells transiently expressing
265 by the whole-cell patch clamp technique from human embryonic kidney 293 cells transiently transfected
266 phosphatase 1B, as well as intact and lysed human embryonic kidney 293 cells treated with S-oxidizin
267 ta1gamma2 GABARs in outside-out patches from human embryonic kidney 293 cells triggered by rapidly ap
268 phosphorylation of endogenous human CTPS1 in human embryonic kidney 293 cells under the conditions te
269 NADH fluorescence properties in vitro and in human embryonic kidney 293 cells upon aS aggregation.
270 lities of the homodimer were investigated in human embryonic kidney 293 cells using agonists that bin
271 residues Ser202/Thr205 and Ser396/Ser404 in human embryonic kidney 293 cells using immunodetection a
272 e measured from chimeric pendrin-transfected human embryonic kidney 293 cells using the voltage clamp
273 N1/GluN2A receptors transiently expressed in human embryonic kidney 293 cells using whole-cell and si
274 yed for functional activity by expression in human embryonic kidney 293 cells, using as probes both t
275 K(ATP)) channels (Kir6.2/SUR2A) expressed in human embryonic kidney 293 cells, using inside-out patch
276 L GABA(A) receptors transiently expressed in human embryonic kidney 293 cells, using the cell-attache
277 or Y1593C mutants functionally expressed in human embryonic kidney 293 cells, using the patch-clamp
279 to I(AC), the current density of bTREK-1 in human embryonic kidney-293 cells was not increased by ra
280 d in Chinese hamster ovary cells, but not in human embryonic kidney 293 cells, was arrested in the ER
281 m the readthrough efficacy of the 2 drugs in Human Embryonic Kidney 293 cells, we did not observe res
282 and dominant-negative forms of RhoA in HEK (human embryonic kidney) 293 cells, we investigated the r
286 t human CB(1) receptors, stably expressed in human embryonic kidney 293 cells, were used to investiga
287 ar calcium mobilization and DMR responses in human embryonic kidney 293 cells, when stimulated with o
288 his is a distinct role from that in HeLa and human embryonic kidney 293 cells where HFE has been show
289 on levels of mGlu(5) receptor expression in human embryonic kidney 293 cells, whereas PAM potency is
292 pandins have inhibitory activity for SOCE in human embryonic kidney-293 cells while having no effect
293 es robust activation of recombinant TRPV3 in human embryonic kidney 293 cells with an EC50 of 28 micr
294 e, we treated bovine eNOS stably transfected human embryonic kidney 293 cells with Hsp90 inhibitors a
296 of the identified mutations, we transfected human embryonic kidney 293 cells with plasmids encoding
300 paB in transfected Chinese hamster ovary and human embryonic kidney 293 cells, with enhancement of th
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