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1 d ex vivo human tonsillar tissue system) and human herpes viruses.
3 s (HCMV and MCMV), Epstein-Barr virus (EBV), human herpes virus 6 (HHV-6), varicella zoster virus (VZ
5 samples comprised of genomic lambda-DNA and human herpes virus 6 type B (HHV-6B) DNA, we have used o
7 is increasing evidence that the neurotropic human herpes viruses 6 and 7 (HHV-6, HHV-7) comprise a s
10 -1 and HSV-2), varicella-zoster virus (VZV), human herpes virus-6, 7, and 8 (HHV-6, HHV-7, and HHV-8)
11 puzzle as they relate to viral factors-both human herpes virus 8 (HHV-8) and human immunodeficiency
16 osi's sarcoma-associated herpes virus (KSHV)/human herpes virus 8 (HHV8) DNA sequences have been demo
20 sociated herpesvirus, which is also known as human herpes virus 8 (KSHV/ HHV8), has not been reported
23 osi's sarcoma (KS)-associated herpesvirus or human herpes virus 8 is considered the etiological agent
24 osi's sarcoma-associated herpes virus (KSHV)/human herpes virus 8 is oncogenic and stimulates angioge
26 -SP on Kaposi sarcoma-associated herpesvirus/human herpes virus 8 replication and uptake of the ORF45
27 ion are at increased risk for development of human herpes virus 8(HHV-8)-associated Kaposi's sarcoma
28 virus remains uncertain, a new herpes virus, human herpes virus 8, has been identified in the rare su
34 might happen following reciprocal actions of human herpes virus-8 infection, immunosuppression, and c
38 mucosa-associated lymphoid tissue lymphoma, human herpes virus (HHV)-6, HHV-7, chlamydia, Epstein-Ba
39 screened possible pathogen(s) for detecting human herpes virus (HHV1-HHV7), HEV, or Mycoplasma pneum
41 MV, herpes simplex virus type 1 (HSV-1), and human herpes virus six (HHV-6) and for cytotoxicity.
43 virus (HCV), human papillomavirus (HPV), and human herpes viruses, this paradigm is not applicable.
44 e applied to a clinically relevant real-time human herpes virus type 6 (HHV6) PCR assay that used a m
46 ron alpha (IFN-alpha) to induce apoptosis in Human Herpes Virus Type 8 (HHV-8) positive Primary Effus
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