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1 ous cycle of varicella-zoster virus (VZV), a human herpesvirus.
2 nse to infection with this common pathogenic human herpesvirus.
3 The Epstein-Barr virus (EBV) is an oncogenic human herpesvirus.
4 ach of the alpha, beta, and gamma classes of human herpesviruses.
5 gG do not have orthologs in HSV-2 or 8 other human herpesviruses.
6 and gG) do not have orthologs in all 40 non-human herpesviruses.
7 d that have undetectable levels of the eight human herpesviruses.
8 he nucleoside kinase activity of coinfecting human herpesviruses.
9 of novel entry inhibitors of EBV and related human herpesviruses.
10 ause of the species-specific tropism of many human herpesviruses.
11 rticularly murine cytomegalovirus (MCMV) and human herpesviruses.
12 n compared to T-cell responses against other human herpesviruses.
13 to a small genomic region, as seen in other human herpesviruses.
14 the circulating diversity of all classes of human herpesviruses.
16 coma herpesvirus (KSHV), formally designated human herpesvirus 4 (HHV-4) and 8 (HHV-8), respectively,
18 receptor encoded by CMV (also referred to as human herpesvirus 5), a highly prevalent human virus tha
19 inherits a chromosomally integrated copy of human herpesvirus 6 (CI-HHV-6), but the consequences of
20 esence of inherited chromosomally integrated human herpesvirus 6 (ciHHV-6) in hematopoietic cell tran
27 mory subsets of CD4(+) and CD8(+) T cells to human herpesvirus 6 (HHV-6) have not been previously inv
35 megalovirus (CMV), Epstein-Barr virus (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types
36 uses such as Marek's disease virus (MDV) and human herpesvirus 6 (HHV-6), integrate their DNA into ho
37 have performed a screen aimed at identifying human herpesvirus 6 (HHV-6)-encoded proteins that modula
40 (EBV), BK virus (BKV), adenovirus (ADV), and human herpesvirus 6 (HHV6) were evaluated to determine o
41 omes of interest were VR of adenovirus, EBV, human herpesvirus 6 (HHV6), cytomegalovirus (CMV), and B
44 ilation and ICU type, only coreactivation of human herpesvirus 6 and cytomegalovirus was significantl
48 ical ventilation, burn ICU, major infection, human herpesvirus 6 reactivation, and cytomegalovirus re
49 y and comprehensively assessed the impact of human herpesvirus 6 reactivation, and its interaction wi
53 , enterovirus, herpes simplex virus 1 and 2, human herpesvirus 6, human parechovirus, varicella-zoste
54 9; p = 0.005) compared to patients with only human herpesvirus 6, only cytomegalovirus, or no viral r
55 30 febrile children positive for adenovirus, human herpesvirus 6, or enterovirus infection or with ac
57 inical relevance of chromosomally integrated human herpesvirus-6 (CIHHV-6) after transplantation is n
66 ncluding adenovirus, bocavirus, coronavirus, human herpesvirus-6, lymphocytic choriomeningitis virus,
69 ous human diseases have been correlated with human herpesvirus 6A (HHV-6A) and HHV-6B, the lack of an
70 ity of the human population is infected with human herpesvirus 6A (HHV-6A), a betaherpesvirus family
79 ier of an inherited-chromosomally-integrated human herpesvirus-6A (iciHHV-6A) genome in one 19q telom
84 o test for cytomegalovirus, BK polyomavirus, human herpesvirus 6B, HHV-6A, adenovirus, and Epstein-Ba
85 including the closely related human viruses human herpesvirus 7 (HHV-7) and human cytomegalovirus (H
92 py number of human herpesvirus-6 (HHV-6) and human herpesvirus-7 (HHV-7) were measured in both saliva
94 ypothesized that they could be infected with human herpesvirus 8 (HHV-8) (Kaposi's sarcoma [KS]-assoc
96 posi's sarcoma-associated herpesvirus (KSHV)/human herpesvirus 8 (HHV-8) and causes KSHV-like disease
97 o investigate a possible association between human herpesvirus 8 (HHV-8) and prostate cancer, we eval
100 oma (cKS) is an inflammatory tumor caused by human herpesvirus 8 (HHV-8) commonly observed in elderly
101 posi's sarcoma-associated herpesvirus (KSHV)/human herpesvirus 8 (HHV-8) displays two distinct life s
104 children, but there is a paucity of data on human herpesvirus 8 (HHV-8) incidence and routes of infe
119 epithelial cells persistently infected with human herpesvirus 8 (HHV-8), an oncogenic herpesvirus th
120 infected patients, Epstein-Barr virus (EBV), human herpesvirus 8 (HHV-8), cytomegalovirus (CMV), and
121 Other viruses, such as hepatitis B virus or human herpesvirus 8 (HHV-8), establish persistent infect
122 associated herpesvirus (KSHV), also known as human herpesvirus 8 (HHV-8), is a cancer-related human v
124 0-CD200R pathway during infection, including human herpesvirus 8 (HHV-8), the causative agent of Kapo
125 erent efficiencies and with the exception of human herpesvirus 8 (HHV-8), these chimeric variants res
131 aque (RM) monkeys that is closely related to human herpesvirus 8 (HHV-8)/Kaposi's Sarcoma-associated
134 central Italy and a surveillance program for human herpesvirus 8 (HHV8) infection after transplant, a
135 Kaposi's sarcoma-associated herpesvirus/human herpesvirus 8 (HHV8) ORF50 encodes a transactivato
137 The lytic origins of DNA replication for human herpesvirus 8 (HHV8), oriLyt-L and oriLyt-R, are l
139 nsfection replication assay identified eight human herpesvirus 8 (HHV8)-encoded proteins required for
140 utively active chemokine receptor encoded by human herpesvirus 8 (HHV8, also known as Kaposi sarcoma
141 Kaposi's sarcoma-associated herpesvirus/human herpesvirus 8 (KSHV/HHV-8) interacts with cell sur
142 Kaposi's sarcoma-associated herpesvirus/human herpesvirus 8 (KSHV/HHV-8) RTA is an important pro
143 irus Kaposi's sarcoma-associated herpesvirus/human herpesvirus 8 (KSHV/HHV8), exploit microtubule (MT
144 osi's sarcoma-associated herpesvirus (KSHV) (human herpesvirus 8 [HHV-8]) induces the host cell's pre
145 -associated herpesvirus (KSHV; also known as human herpesvirus 8 [HHV-8]) is the etiologic agent of K
146 -associated herpesvirus (KSHV; also known as human herpesvirus 8 [HHV-8]), Epstein-Barr virus (EBV),
147 aposi's sarcoma-associated herpesvirus (KSHV/human herpesvirus 8 [HHV8]) and Epstein-Barr virus (EBV/
148 i's sarcoma-associated herpesvirus (KSHV; or human herpesvirus 8 [HHV8]) is implicated in the pathoge
149 ma-associated herpesvirus (KSHV; also called human herpesvirus 8 [HHV8]), the switch from latency to
153 s sarcoma (KS)-associated herpesvirus (KSHV)/human herpesvirus 8 functions as the key regulator to in
155 6 homologue (viral interleukin-6 [vIL-6]) of human herpesvirus 8 is implicated in viral pathogenesis
157 Viral interleukin-6 (vIL-6) specified by human herpesvirus 8 is, unlike its cellular counterpart,
159 CMV, herpes simplex virus types 1 and 2, and human herpesvirus 8 was 99.4%, 74.9%, 26.2%, 8.0%, and 1
160 osi's sarcoma-associated herpesvirus (KSHV) (human herpesvirus 8) binds to adherent target cell surfa
161 i's sarcoma-associated herpesvirus (KSHV, or human herpesvirus 8) infection in individuals remains a
162 s sarcoma-associated herpesvirus (also named human herpesvirus 8) is a gamma-herpesvirus that undergo
163 persistent gamma-herpesvirus infection (EBV, human herpesvirus 8) is a significant problem in AIDS pa
164 i's sarcoma-associated herpesvirus (KSHV, or human herpesvirus 8) is etiologically linked to KS, and
165 ma-associated herpesvirus (KSHV, also called human herpesvirus 8) is linked to the development of Kap
166 oma-associated herpesvirus (KSHV; also named human herpesvirus 8) is necessary but not sufficient for
167 posi's sarcoma-associated herpesvirus (KSHV, human herpesvirus 8) is the causative agent of Kaposi's
168 s (CMV), herpes simplex virus types 1 and 2, human herpesvirus 8) using multiplex serology on blood s
169 -associated herpesvirus (KSHV, also known as human herpesvirus 8) with the development of the disease
171 a-associated herpesvirus, also identified as human herpesvirus 8, contains genes producing proteins t
172 associated herpesvirus (KSHV), also known as human herpesvirus 8, is the etiologic agent of Kaposi sa
173 associated herpesvirus (KSHV), also known as human herpesvirus 8, is the etiologic agent underlying K
174 The rhadinovirus 1 (RV1) lineage consists of human herpesvirus 8, Kaposi's sarcoma-associated herpesv
179 Epstein-Barr virus-infected lymphocytes, and human herpesvirus 8-positive primary effusion lymphoma,
182 gammaherpesviruses, Epstein-Barr virus, and human herpesvirus 8/Kaposi's sarcoma-associated herpesvi
183 posi's sarcoma-associated herpesvirus (KSHV [human herpesvirus 8; HHV-8]) open reading frame 57 (ORF5
184 onsequence of dysregulated expression of the human herpesvirus-8 (HHV-8 or KSHV)-encoded G protein-co
185 effusion lymphomas (PEL) are associated with human herpesvirus-8 (HHV-8) and usually occur in immunoc
186 ranean Basin, despite the high prevalence of human herpesvirus-8 (HHV-8) infection in this region.
188 s led to a dramatic rise in the incidence of human herpesvirus-8 (HHV-8)-associated Kaposi's sarcoma
189 S), a multifocal vascular neoplasm linked to human herpesvirus-8 (HHV-8/KS-associated herpesvirus [KS
191 i's sarcoma-associated herpesvirus (KSHV, or human herpesvirus-8 [HHV-8]) has another, alternative em
193 eries regarding Kaposi's sarcoma herpesvirus/human herpesvirus-8 infection and its activation of sign
194 t findings show Kaposi's sarcoma herpesvirus/human herpesvirus-8 specific signaling pathways and pira
195 si's sarcoma-associated herpesvirus (KSHV or human herpesvirus-8) is frequently tumorigenic in immuno
196 osi sarcoma-associated herpesvirus (KSHV; or human herpesvirus-8)-encoded protein called K-bZIP (also
197 mely Kaposi's sarcoma-associated herpesvirus/human herpesvirus-8, has been confirmed to be a direct c
198 nflammatory protein-II (vMIP-II), encoded by human herpesvirus-8, has garnered interest because of it
199 rize major recent findings on the biology of human herpesvirus-8, i.e. Kaposi's sarcoma-associated he
200 on with HIV and Kaposi's sarcoma herpesvirus/human herpesvirus-8, which leads to the development of a
202 ma is driven by Kaposi's sarcoma herpesvirus/human herpesvirus-8-specific pathways, which include vir
208 ) and HSV-2, two closely related neurotropic human herpesviruses, achieve neurotropism through ICP34.
210 the structure of region IX is present in all human herpesviruses and thus represents a potential stru
213 e against Epstein-Barr virus (EBV) and other human herpesviruses are limited to those targeting viral
216 Epstein-Barr virus (EBV) is a ubiquitous human herpesvirus associated with many malignant and non
217 Epstein-Barr virus (EBV) is a ubiquitous human herpesvirus associated with the development of bot
220 de analog, is thought to be specific for the human herpesviruses because it requires a virally encode
221 This study examined the association of 4 human herpesviruses (CMV, herpes simplex virus type 1, h
222 -regulating master circuits in HIV-1 and the human herpesvirus cytomegalovirus along with potential c
223 more differentiated T cells specific for the human herpesviruses cytomegalovirus and Epstein-Barr vir
225 Currently, there are approximately 150 known human herpesvirus-encoded miRNAs, although an miRNA(s) e
228 a model our current understanding of how the human herpesvirus Epstein-Barr virus establishes and mai
230 sized that HHV-6A, like other members of the human herpesvirus family, encodes miRNAs, which function
231 l mechanism by which HHV-6A, a member of the human herpesvirus family, may contribute to inadequate m
232 g 3 HSV-1 genomes, one HSV-2 genome, 8 other human herpesvirus genomes, and 40 non-human herpesvirus
238 nd that certain infectious pathogens such as human herpesvirus (HHV) 6, HHV-7, and adenovirus, which
239 immunological tools available to detect both human herpesvirus (HHV) and immune control of replicatio
240 d men have at least one actively replicating human herpesvirus (HHV) in their mucosal secretions at a
242 Both human immunodeficiency virus (HIV) and human herpesvirus (HHV) infections persist lifelong, and
243 plant recipients have reactivation of latent human herpesvirus (HHV)-6 2-4 weeks after transplant.
245 Both intrauterine and sexual transmission of human herpesvirus (HHV)-6 and HHV-7 have been suggested,
246 tem cell donor with chromosomally integrated human herpesvirus (HHV)-6 and monitored the recipient fo
247 ced hypersensitivity syndrome (DIHS), latent human herpesvirus (HHV)-6 is frequently reactivated in a
248 identification of suitable animal models for human herpesvirus (HHV)-6A and HHV-6B infections has bee
249 for a single virus: Epstein-Barr virus (23), human herpesvirus (HHV)-6B (10), human parainfluenza vir
251 the clinical and virologic manifestations of human herpesvirus (HHV)-8 infection in immunocompetent p
252 Kaposi sarcoma (KS) is primarily caused by human herpesvirus (HHV)-8 infection, and the risk is inc
253 nt studies have found evidence of occasional human herpesvirus (HHV)-8 transmission via blood transfu
258 were as follows: cytomegalovirus (CMV), 44%; human herpesvirus [HHV] 6, 18%; HHV8, 6%; Epstein-Barr v
259 ion is associated with an increased risk for human herpesviruses (HHVs) and their related diseases.
261 We investigated the hypothetical role of human herpesviruses (HHVs) in tumour formation of the ce
263 , acts as a DNA chain terminator for several human herpesviruses (HHVs), including HHV-2 (HSV-2), a c
265 lly occurring genetic variants of persistent human herpesviruses imprints on the evolution of the ant
266 us (EBV), known as an oncovirus, is the only human herpesvirus in the genus Lymphocryptovirus (LCV).
268 we found that HDAC inhibiting proteins from human herpesviruses induce human NKG2D ligand ULBP-1.
271 s sarcoma-associated herpesvirus (KSHV) is a human herpesvirus involved in the causation of several h
276 llular protein and has homologs in all other human herpesviruses, it has potential importance as a th
277 the data suggest that HHV-6 is unique among human herpesviruses: it specifically and efficiently int
278 ecently, 11 miRNAs encoded by the pathogenic human herpesvirus Kaposi's sarcoma-associated herpesviru
279 Kaposi sarcoma (KS) to infection by a novel human herpesvirus (Kaposi sarcoma-associated herpesvirus
282 yzed the incidence of infection of the eight human herpesviruses simultaneously in 1 045 peripheral b
283 Human cytomegalovirus (HCMV) is a ubiquitous human herpesvirus that can cause life-threatening diseas
284 Epstein-Barr virus (EBV) is an oncogenic human herpesvirus that dramatically reorganizes host gen
287 Epstein-Barr virus is an orally transmitted human herpesvirus that infects epithelial cells and esta
289 Epstein-Barr virus (EBV) is a ubiquitous human herpesvirus that is causally implicated in the dev
290 , the replication of herpes simplex virus, a human herpesvirus that is closely related to HCMV, was n
291 genomes, combined with the inability of most human herpesviruses to replicate in animals, has until r
294 esviruses (CMV, herpes simplex virus type 1, human herpesvirus type 6, and Epstein-Barr virus) with c
295 afamilial dynamics of endemic infection with human herpesvirus type 8 (HHV-8) in Amerindian populatio
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