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1 galovirus, EBV, adenovirus, BK virus, and/or human herpesvirus 6.
2 sivity factors of herpes simplex virus 1 and human herpesvirus 6.
3 reported types of clinical disease caused by human herpesvirus-6.
4 infection with both human herpesvirus-7 and human herpesvirus-6.
5 virus; cytomegalovirus; Epstein-Barr virus; human herpesviruses 6, 7, and 8; or adeno-associated vir
9 ilation and ICU type, only coreactivation of human herpesvirus 6 and cytomegalovirus was significantl
10 with the detection of cytomegalovirus (CMV), human herpesvirus 6 and human herpesvirus 7 and BK virus
11 -N also inhibited fusion and/or infection by human herpesvirus 6 and measles virus but not by vaccini
14 s of the epidemiology and natural history of human herpesviruses 6 and 7 in infants, a practical meth
16 virus, cytomegalovirus, Epstein-Barr virus, human herpesviruses 6 and 8, gonorrhea, or chlamydia.
17 infections, such as measles, parvovirus B19, human herpesvirus 6, and enteroviruses in developed coun
20 hain reaction to detect human herpesvirus-7, human herpesvirus-6, and cytomegalovirus DNA in lesional
23 inherits a chromosomally integrated copy of human herpesvirus 6 (CI-HHV-6), but the consequences of
24 esence of inherited chromosomally integrated human herpesvirus 6 (ciHHV-6) in hematopoietic cell tran
25 inical relevance of chromosomally integrated human herpesvirus-6 (CIHHV-6) after transplantation is n
28 lls (83%), and serum (100%) samples, whereas human herpesvirus-6 DNA was detected in lesional skin (8
31 ding measles virus, Neisseria gonorrhea, and human herpesvirus 6, have been described, the region of
34 ral reports have suggested an association of human herpesvirus 6 (HHV-6) and multiple sclerosis (MS)
36 test was developed and used with an existing human herpesvirus 6 (HHV-6) antibody avidity test to det
45 esignated DR7, within the SalI-L fragment of human herpesvirus 6 (HHV-6) exhibited transactivation of
48 mory subsets of CD4(+) and CD8(+) T cells to human herpesvirus 6 (HHV-6) have not been previously inv
61 ugh first described as a lymphotropic virus, human herpesvirus 6 (HHV-6) is highly neuropathogenic.
69 the presence of herpes simplex virus (HSV), human herpesvirus 6 (HHV-6), Epstein-Barr virus (EBV), c
70 megalovirus (CMV), Epstein-Barr virus (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types
71 er herpesviruses, including cytomegalovirus, human herpesvirus 6 (HHV-6), HHV-7, HHV-8, and Epstein-B
72 uses such as Marek's disease virus (MDV) and human herpesvirus 6 (HHV-6), integrate their DNA into ho
74 r a candidate multiple sclerosis (MS) virus, human herpesvirus 6 (HHV-6), was sought in biopsy specim
75 ntly reactivated in HIV-infected patients is human herpesvirus 6 (HHV-6), which has been proposed as
76 have performed a screen aimed at identifying human herpesvirus 6 (HHV-6)-encoded proteins that modula
82 96-102), a candidate autoantigen for MS, and human herpesvirus-6 (HHV-6 U24, residues 4-10) that is a
93 ZV); beta includes cytomegalovirus (CMV) and human herpesvirus-6 (HHV-6); and gamma includes Epstein-
95 (EBV), BK virus (BKV), adenovirus (ADV), and human herpesvirus 6 (HHV6) were evaluated to determine o
96 omes of interest were VR of adenovirus, EBV, human herpesvirus 6 (HHV6), cytomegalovirus (CMV), and B
97 , enterovirus, herpes simplex virus 1 and 2, human herpesvirus 6, human parechovirus, varicella-zoste
100 enabled detection of human herpesvirus-7 and human herpesvirus-6 in skin and other tissues isolated f
102 and efficacy of prophylactic strategies for human herpesvirus-6 infection in transplant recipients.
104 miology, clinical features, and treatment of human herpesvirus-6 infection were manually abstracted f
107 rompt recognition of disease associated with human herpesvirus-6 is important because this virus is s
108 ncluding adenovirus, bocavirus, coronavirus, human herpesvirus-6, lymphocytic choriomeningitis virus,
109 cause it can depress cell-mediated immunity, human herpesvirus-6 may facilitate superinfection by oth
110 ear cells expressing human herpesvirus-7 and human herpesvirus-6 mRNA were identified in perivascular
111 9; p = 0.005) compared to patients with only human herpesvirus 6, only cytomegalovirus, or no viral r
112 30 febrile children positive for adenovirus, human herpesvirus 6, or enterovirus infection or with ac
113 ohistochemical staining for the P41 and P101 human herpesvirus 6 protein antigens showed numerous imm
116 ical ventilation, burn ICU, major infection, human herpesvirus 6 reactivation, and cytomegalovirus re
117 y and comprehensively assessed the impact of human herpesvirus 6 reactivation, and its interaction wi
119 pinal fluid studies revealed the presence of human herpesvirus 6, variant B, DNA in all of 3 patients
123 ied through a MEDLINE search from 1986 (when human herpesvirus-6 was discovered) to the present, bibl
124 stein-Barr virus (EBV), cytomegalovirus, and human herpesvirus 6 were assessed by semiquantitative po
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