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1 17 men [56.0%]; 187 [89.5%] positive for the human immunodeficiency virus).
2 PID), including an association with risk for human immunodeficiency virus.
3 , aged >/=40 years, white, and infected with human immunodeficiency virus.
4 sis, rubella, cytomegalovirus, syphilis, and human immunodeficiency virus.
5 and 19, herpes simplex virus (HSV) 1 and 2, human immunodeficiency virus 1, hepatitis C virus, enter
8 diseases such as tuberculosis, malaria, and human immunodeficiency virus/acquired immunodeficiency s
9 in immunocompromised individuals, typically human immunodeficiency virus/AIDS patients from developi
11 epatitis B surface antigen (HBsAg) positive, human immunodeficiency virus and hepatitis D virus-negat
12 facilitates transmission of less infectious human immunodeficiency virus, but highly infectious viru
14 ed patients with pulmonary tuberculosis (65% human immunodeficiency virus coinfected) were intensivel
15 istal extremities (ie, neuropathy related to human immunodeficiency virus, diabetes, or Fabry disease
17 istinct from prototypic SIV encephalitis and human immunodeficiency virus encephalitis were identifie
18 s, but there are no data to guide its use in human immunodeficiency virus/HCV coinfected kidney trans
20 ted on PrOD or LDV/SOF, excluding those with human immunodeficiency virus, hepatitis B surface antige
21 cluding rubella, congenital cytomegalovirus, human immunodeficiency virus, hepatitis B virus, and neo
22 avirus (HPV) types among women infected with human immunodeficiency virus (HIV) (WHIV) are needed to
23 SVAX B/E boost afforded 60% efficacy against human immunodeficiency virus (HIV) acquisition at 1 year
25 le of sexual networks in the epidemiology of human immunodeficiency virus (HIV) among black men who h
26 ealthcare quality and access for people with human immunodeficiency virus (HIV) and acquired immune d
27 ide ART availability, the burden of advanced human immunodeficiency virus (HIV) and associated opport
28 virologic control in children infected with human immunodeficiency virus (HIV) and exposed to nevira
29 longitudinal cohort of women coinfected with human immunodeficiency virus (HIV) and hepatitis C virus
32 ed threonine near the C terminus of gp120 of human immunodeficiency virus (HIV) and simian immunodefi
33 ween an exposure resulting in infection with human immunodeficiency virus (HIV) and when a test can r
36 co-infected with hepatitis C virus (HCV) and human immunodeficiency virus (HIV) are at high risk of l
37 d to ensuring that 90% of people living with human immunodeficiency virus (HIV) are diagnosed, 90% st
38 Youths aged 13 to 24 years old living with human immunodeficiency virus (HIV) are less likely than
39 umbers of children infected perinatally with human immunodeficiency virus (HIV) are surviving to adol
40 n 6 US cities, had negative test results for human immunodeficiency virus (HIV) but were at high risk
44 ve been similar in patients with and without human immunodeficiency virus (HIV) coinfection; however,
45 the successful design and implementation of human immunodeficiency virus (HIV) curative strategies i
47 Pain has always been an important part of human immunodeficiency virus (HIV) disease and its exper
48 egnancy is important for control of maternal human immunodeficiency virus (HIV) disease and the preve
49 Facial lipoatrophy (FLA) is associated with human immunodeficiency virus (HIV) disease and the use o
50 , strongly predicts mortality during treated human immunodeficiency virus (HIV) disease in Ugandans a
51 cult to assess the role of thymic failure in human immunodeficiency virus (HIV) disease progression.
53 se, which represents a target for novel anti-human immunodeficiency virus (HIV) drug development.IMPO
54 e a driving factor behind the development of human immunodeficiency virus (HIV) drug resistance (HIVD
58 s are associated with clinically significant human immunodeficiency virus (HIV) drug resistance, assa
59 mic distribution of cell subsets that harbor human immunodeficiency virus (HIV) during antiretroviral
60 iven the well-documented viral archeology of human immunodeficiency virus (HIV) emergence following h
61 enders and populations at risk toward global human immunodeficiency virus (HIV) epidemic control.
66 neutralizing antibodies directed against the human immunodeficiency virus (HIV) has received consider
67 n of mother-to-child transmission (PMTCT) of human immunodeficiency virus (HIV) have been scaled up i
68 as implemented in response to an outbreak of human immunodeficiency virus (HIV) infection among perso
71 eveloped countries has changed the course of Human Immunodeficiency Virus (HIV) infection from an alm
72 ociation of coronary heart disease (CHD) and human immunodeficiency virus (HIV) infection has been we
75 wer potential to induce hepatic steatosis in human immunodeficiency virus (HIV) infection need to be
77 m in 102 asymptomatic Air Force members with human immunodeficiency virus (HIV) infection revealed 19
78 t gains in the treatment of tuberculosis and human immunodeficiency virus (HIV) infection worldwide.
79 is (PrEP) is highly effective for preventing human immunodeficiency virus (HIV) infection, but risk c
80 for adverse birth outcomes among women with human immunodeficiency virus (HIV) infection, but whethe
82 uent cause of meningitis in individuals with human immunodeficiency virus (HIV) infection, resulting
83 Saharan Africa, among patients with advanced human immunodeficiency virus (HIV) infection, the rate o
89 In January 2015, an outbreak of undiagnosed human immunodeficiency virus (HIV) infections among pers
90 individuals with hepatitis C virus (HCV) and human immunodeficiency virus (HIV) infections, but the i
92 Epithelium-Derived Growth Factor (LEDGF) and human immunodeficiency virus (HIV) integrase is an impor
94 g the quality, and proactively using data of human immunodeficiency virus (HIV) load testing are pivo
96 of a combination strategy for prevention of human immunodeficiency virus (HIV) on the incidence of H
99 4657412 polymorphism on the adaptive WMT, in human immunodeficiency virus (HIV) participants compared
100 ch other's replication and perhaps affecting human immunodeficiency virus (HIV) pathogenesis and dise
101 nt-of-care (POC) diagnostic technologies for human immunodeficiency virus (HIV) patients in resource-
103 rom 34,446 respondents to a tuberculosis and human immunodeficiency virus (HIV) prevalence survey tha
104 losis (TB) risk in populations infected with human immunodeficiency virus (HIV) remain understudied,
106 TARBP2) is known to play important roles in human immunodeficiency virus (HIV) replication and micro
109 s regarding the time course and magnitude of human immunodeficiency virus (HIV) RNA decay in semen.
110 ne activation is suspected to modulate local human immunodeficiency virus (HIV) RNA levels and the ri
113 adly neutralizing antibodies (bNAbs) against human immunodeficiency virus (HIV) show great promise in
114 itude), moderate malnutrition, or an unknown human immunodeficiency virus (HIV) status in an HIV-ende
115 cause substantial disease and death despite human immunodeficiency virus (HIV) suppression with anti
117 irus (HR-HPV) is higher in women living with human immunodeficiency virus (HIV) than in the general p
119 Genital inflammation is a key determinant of human immunodeficiency virus (HIV) transmission, and may
124 lattice formed by the capsid protein (CA) of human immunodeficiency virus (HIV) was self-assembled as
127 diseases (NCDs) among individuals aging with human immunodeficiency virus (HIV), but few studies have
128 use is prevalent among persons infected with human immunodeficiency virus (HIV), but its long-term ef
129 HCV-positive persons and excluded those with human immunodeficiency virus (HIV), hepatitis B surface
130 n recipients at increased risk for acquiring human immunodeficiency virus (HIV), hepatitis B virus (H
131 lobally 1.8 million children are living with human immunodeficiency virus (HIV), yet only 51% of thos
133 PC) directs the localization and assembly of human immunodeficiency virus (HIV)-1 Gag polyprotein at
134 we report that the tetraspanin CD81 enhances human immunodeficiency virus (HIV)-1 reverse transcripti
136 phages may lead to improved understanding of human immunodeficiency virus (HIV)-associated cardiovasc
138 s among hospitalized patients diagnosed with human immunodeficiency virus (HIV)-associated tuberculos
139 ith drug-susceptible tuberculosis (including human immunodeficiency virus (HIV)-coinfected cases) and
142 ) is widely used in malaria-endemic areas in human immunodeficiency virus (HIV)-infected children and
144 tivation and inflammation remain elevated in human immunodeficiency virus (HIV)-infected individuals
147 e general population, but their frequency in human immunodeficiency virus (HIV)-infected individuals
148 scontinuation increases malaria incidence in human immunodeficiency virus (HIV)-infected individuals.
151 tted hepatitis C virus (HCV) infection among human immunodeficiency virus (HIV)-infected men who have
152 sarcoma (KS), one of the leading cancers in human immunodeficiency virus (HIV)-infected patients in
156 based on transient elastography (TE), among human immunodeficiency virus (HIV)-infected patients wit
157 ommunity-recruited prospective cohort of 961 human immunodeficiency virus (HIV)-infected people who i
158 uidelines recommend that all sexually active human immunodeficiency virus (HIV)-infected persons be t
159 n interventions for prevention of malaria in human immunodeficiency virus (HIV)-infected pregnant wom
161 with the increased morbimortality of chronic human immunodeficiency virus (HIV)-infected subjects.
167 n human papillomavirus (HPV) infection among human immunodeficiency virus (HIV)-negative or HIV-posit
169 sociation with increased suicidal risk among human immunodeficiency virus (HIV)-positive adults in Ug
171 Neurocognitive disorders remain common among human immunodeficiency virus (HIV)-positive adults, perh
172 are and antiretroviral (ARV) treatment among human immunodeficiency virus (HIV)-positive men who have
173 d effective diagnostic algorithms for use in human immunodeficiency virus (HIV)-positive patients are
174 We compared estimated costs of retesting human immunodeficiency virus (HIV)-positive persons befo
176 yces marneffei infection is a major cause of human immunodeficiency virus (HIV)-related death in Sout
177 linical trials evaluating IIV efficacy among human immunodeficiency virus (HIV)-uninfected and HIV-in
178 ti-Ethnic Study of Atherosclerosis (MESA), a Human Immunodeficiency Virus (HIV)-uninfected cohort, wh
180 e demonstrated improved birth outcomes among human immunodeficiency virus (HIV)-uninfected pregnant w
181 bine (FTC) preexposure prophylaxis (PrEP) in human immunodeficiency virus (HIV)-uninfected young men
193 owledge gaps remain about how the Ryan White human immunodeficiency virus (HIV)/AIDS Program (RW) con
196 NLR-NAR) events and mortality in a cohort of human immunodeficiency virus (HIV)/hepatitis C virus (HC
197 are accessory proteins encoded by different human immunodeficiency virus (HIV)/simian immunodeficien
199 ntravaginal practices may be associated with human immunodeficiency virus (HIV-1) infection risk; how
200 es have been reported in persons living with human immunodeficiency virus (HIV; PLWH) who are receivi
202 patitis C virus (HCV)-infected subjects (31% human immunodeficiency virus [HIV] positive) pre- and po
204 nfluenza virus, respiratory syncytial virus, human immunodeficiency virus, human T cell leukemia viru
207 ervational cohort study was conducted in 302 human immunodeficiency virus-infected patients who had a
208 AM lateral flow assay (LF-LAM) results among human immunodeficiency virus-infected patients with diss
212 om 13 European and North American cohorts of human immunodeficiency virus-infected, antiretroviral th
215 act lens wearers (P = 0.21) or patients with human immunodeficiency virus infection or AIDS (P = 0.60
222 se of morbidity and mortality in people with human immunodeficiency virus (PWHIV) on effective antire
223 s in this area; (3) lessons learned from the human immunodeficiency virus research field, where pregn
224 nation of M41L + L210W + T215rev showed full human immunodeficiency virus RNA suppression while recei
225 d, previously reported to be an inhibitor of human immunodeficiency virus RNase H, inhibited pUL89 en
227 potently inhibited in vivo HIV-1 and simian-human immunodeficiency virus (SHIV) infection in humaniz
228 administration of bNAbs in a macaque simian/human immunodeficiency virus (SHIV) model is associated
229 y incompletely neutralize the clade C simian-human immunodeficiency virus (SHIV) stock (SHIV-327c) at
230 solates in vitro and protects against simian-human immunodeficiency virus (SHIV) when delivered paren
231 tion data from macaques infected with simian/human immunodeficiency virus (SHIV), we observe a lower
232 lycan antibody PGT121, in chronically simian-human immunodeficiency virus (SHIV)-SF162P3-infected mac
233 ere, we infected rhesus macaques with simian-human immunodeficiency viruses (SHIV) and followed the d
234 for demographics, clinical characteristics, human immunodeficiency virus status, and period of treat
235 profile, low body mass index (<18.5 kg/m2), human immunodeficiency virus status, and study site, par
237 itis, and reactive arthritis and facilitates human immunodeficiency virus transmission in men is weak
239 marker, and early mortality in patients with human immunodeficiency virus/tuberculosis co-infection.
240 lex virus type 2 (HSV-2; herpes) exacerbates human immunodeficiency virus type 1 (HIV) by unclear mec
241 Progressive T cell depletion during chronic human immunodeficiency virus type 1 (HIV) infection is a
244 been shown to be preferentially targeted by human immunodeficiency virus type 1 (HIV-1) and are impl
245 ned elusive for important pathogens, such as human immunodeficiency virus type 1 (HIV-1) and herpesvi
246 reporter genes delivered by vectors based on human immunodeficiency virus type 1 (HIV-1) and Mason-Pf
251 of measles and associations with outcome and human immunodeficiency virus type 1 (HIV-1) coinfection,
255 ing conserved CD4-induced (CD4i) epitopes on human immunodeficiency virus type 1 (HIV-1) Env and able
256 s between the gp120 and gp41 subunits of the human immunodeficiency virus type 1 (HIV-1) envelope gly
257 s maintained in a quiescent state.IMPORTANCE Human immunodeficiency virus type 1 (HIV-1) establishes
258 es maintain the ability to interact with the human immunodeficiency virus type 1 (HIV-1) Gag precurso
259 velope glycoprotein (Env) trimers of various human immunodeficiency virus type 1 (HIV-1) genotypes ar
260 virtide) and other peptides derived from the human immunodeficiency virus type 1 (HIV-1) gp41 C-termi
262 al components of vaccine-induced immunity to human immunodeficiency virus type 1 (HIV-1) in humans an
263 In the absence of therapy, CXCR4 (X4)-tropic human immunodeficiency virus type 1 (HIV-1) increases ov
264 usceptible tuberculosis in a setting of high human immunodeficiency virus type 1 (HIV-1) infection an
265 iescent proviral genomes that persist during human immunodeficiency virus type 1 (HIV-1) infection de
269 e HCV infection in participants with chronic human immunodeficiency virus type 1 (HIV-1) infection.
270 ete roles and functions of DRFs during early human immunodeficiency virus type 1 (HIV-1) infection.
271 of CD4+ T lymphocytes, a major reservoir for human immunodeficiency virus type 1 (HIV-1) infection.
280 Local Alignment Program (LAP) using 115,118 human immunodeficiency virus type 1 (HIV-1) protease, re
282 (HCV) infection in patients coinfected with human immunodeficiency virus type 1 (HIV-1) remains a me
283 (ART) limits proviral reservoirs, a goal for human immunodeficiency virus type 1 (HIV-1) remission st
286 vir plus 2 nucleos(t)ides for maintenance of human immunodeficiency virus type 1 (HIV-1) suppression.
287 ith latency reversing agents (LRAs) enhances human immunodeficiency virus type 1 (HIV-1) transcriptio
288 design are being developed as immunogens in human immunodeficiency virus type 1 (HIV-1) vaccine deve
289 important role in regulating the assembly of human immunodeficiency virus type 1 (HIV-1) virus partic
291 e strong dependence of retroviruses, such as human immunodeficiency virus type 1 (HIV-1), on host cel
292 ess to AIDS, in stark contrast to pathogenic human immunodeficiency virus type 1 (HIV-1)-human and SI
293 lated with rotavirus vaccine responses in 68 human immunodeficiency virus type 1 (HIV-1)-infected (an
294 a, has long been known to be elevated in the human immunodeficiency virus type 1 (HIV-1)-infected bra
295 are reported in uninfected children born to human immunodeficiency virus type 1 (HIV-1)-infected wom
296 tion with an anti-PD-L1 antibody may improve human immunodeficiency virus type 1 (HIV-1)-specific imm
299 ized Rous sarcoma virus (RSV) intasomes with human immunodeficiency virus type 1 strand transfer inhi
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