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1 s been found previously to interact with the human immunodeficiency virus, type 1 accessory protein V
2 ssociated with BV, such as preterm birth and human immunodeficiency virus type 1 acquisition, are ass
3 41 subunit of the envelope glycoprotein from human immunodeficiency virus type 1, an essential subuni
8 ribe the mechanical responses of vancomycin, human immunodeficiency virus type 1 antigens and coagula
10 CD4(+) T cells was observed in subjects with human immunodeficiency virus type 1 coinfection without
11 opoietic stem/progenitor cells (HSPCs) using human immunodeficiency virus, type 1-derived lentiviral
14 lex virus type 2 (HSV-2; herpes) exacerbates human immunodeficiency virus type 1 (HIV) by unclear mec
15 ) alfa plus ribavirin decreases the proviral human immunodeficiency virus type 1 (HIV) DNA level.
18 DSC) expansion and T-cell dysfunction during human immunodeficiency virus type 1 (HIV) infection are
20 Progressive T cell depletion during chronic human immunodeficiency virus type 1 (HIV) infection is a
23 esses may contribute to morbidity in treated human immunodeficiency virus type 1 (HIV) infection.
24 ormed a cohort study involving patients with human immunodeficiency virus type 1 (HIV) infection.
27 nt and understudied topic of cell-associated human immunodeficiency virus Type 1 (HIV) mucosal transm
28 whether distinct populations have differing human immunodeficiency virus type 1 (HIV) neutralizing a
29 cable comorbidities are more prevalent among human immunodeficiency virus type 1 (HIV)-infected indiv
31 s remains a significant cause of death among human immunodeficiency virus type 1 (HIV)-infected perso
34 diated cytotoxicity (ADCC), in prevention of human immunodeficiency virus type 1 (HIV-1) acquisition
35 been shown to be preferentially targeted by human immunodeficiency virus type 1 (HIV-1) and are impl
36 ned elusive for important pathogens, such as human immunodeficiency virus type 1 (HIV-1) and herpesvi
37 ion is an efficient mechanism to disseminate human immunodeficiency virus type 1 (HIV-1) and human T
38 reporter genes delivered by vectors based on human immunodeficiency virus type 1 (HIV-1) and Mason-Pf
40 lpha (AgmTRIM5alpha) potently restricts both human immunodeficiency virus type 1 (HIV-1) and simian i
41 ts the infectivity of retroviruses including human immunodeficiency virus type 1 (HIV-1) and the mobi
42 most therapeutic and vaccine candidates for human immunodeficiency virus type 1 (HIV-1) are evaluate
45 s of this regimen on cellular reservoirs for human immunodeficiency virus type 1 (HIV-1) are unknown.
46 2.0), a real-time polymerase chain reaction human immunodeficiency virus type 1 (HIV-1) assay with h
48 ng proteins incorporated into virions during human immunodeficiency virus type 1 (HIV-1) assembly pro
50 generally suppresses the replication of the human immunodeficiency virus type 1 (HIV-1) but does not
51 its the release of virions of the retrovirus human immunodeficiency virus type 1 (HIV-1) by tethering
52 restriction activity, each was fused to the human immunodeficiency virus type 1 (HIV-1) CA binding p
57 of measles and associations with outcome and human immunodeficiency virus type 1 (HIV-1) coinfection,
58 BEC3F (A3F) was highly localized into mature human immunodeficiency virus type 1 (HIV-1) cores and id
60 of preventing perinatal transmission (PT) of human immunodeficiency virus type 1 (HIV-1) depends on b
61 ia A who were exposed to but uninfected with human immunodeficiency virus type 1 (HIV-1) did not reve
66 a number of years that integration sites of human immunodeficiency virus type 1 (HIV-1) DNA show a p
71 inical trial to evaluate the safety and anti-human immunodeficiency virus type 1 (HIV-1) efficacy of
73 ing conserved CD4-induced (CD4i) epitopes on human immunodeficiency virus type 1 (HIV-1) Env and able
74 reactivity (ER) describes the propensity of human immunodeficiency virus type 1 (HIV-1) Env to chang
76 the gp120 subunits of a soluble recombinant human immunodeficiency virus type 1 (HIV-1) envelope (En
78 y simian immunodeficiency virus (SIV) versus human immunodeficiency virus type 1 (HIV-1) envelope ant
83 s between the gp120 and gp41 subunits of the human immunodeficiency virus type 1 (HIV-1) envelope gly
86 used that target nonoverlapping sites on the human immunodeficiency virus type 1 (HIV-1) envelope.
88 eservoir represents a critical challenge for human immunodeficiency virus type 1 (HIV-1) eradication
89 s maintained in a quiescent state.IMPORTANCE Human immunodeficiency virus type 1 (HIV-1) establishes
92 es maintain the ability to interact with the human immunodeficiency virus type 1 (HIV-1) Gag precurso
93 an T-cell leukemia virus type 1 (HTLV-1) and human immunodeficiency virus type 1 (HIV-1) Gag proteins
97 velope glycoprotein (Env) trimers of various human immunodeficiency virus type 1 (HIV-1) genotypes ar
98 (Ab) responses against V1V2 epitopes of the human immunodeficiency virus type 1 (HIV-1) gp120 envelo
99 placing the single VSV glycoprotein (G) with human immunodeficiency virus type 1 (HIV-1) gp160 to cre
100 virtide) and other peptides derived from the human immunodeficiency virus type 1 (HIV-1) gp41 C-termi
106 motif-containing protein 5 (TRIM5) restricts human immunodeficiency virus type 1 (HIV-1) in a species
107 al components of vaccine-induced immunity to human immunodeficiency virus type 1 (HIV-1) in humans an
108 % of genetically linked infections caused by human immunodeficiency virus type 1 (HIV-1) in serodisco
109 iously shown to decrease the accumulation of human immunodeficiency virus type 1 (HIV-1) in the super
110 About 10% of new diagnoses of subtype B human immunodeficiency virus type 1 (HIV-1) in the Unite
111 In the absence of therapy, CXCR4 (X4)-tropic human immunodeficiency virus type 1 (HIV-1) increases ov
112 s chronic infection with RNA viruses such as human immunodeficiency virus type 1 (HIV-1) induces prof
113 ed antiretroviral therapy (cART) in semen of human immunodeficiency virus type 1 (HIV-1) infected men
114 usceptible tuberculosis in a setting of high human immunodeficiency virus type 1 (HIV-1) infection an
115 n are recognized as important in controlling human immunodeficiency virus type 1 (HIV-1) infection an
116 rs in early childhood and is associated with human immunodeficiency virus type 1 (HIV-1) infection an
117 al therapy (cART) administered shortly after human immunodeficiency virus type 1 (HIV-1) infection ca
119 iescent proviral genomes that persist during human immunodeficiency virus type 1 (HIV-1) infection de
121 hylaxis has been shown to reduce the risk of human immunodeficiency virus type 1 (HIV-1) infection in
133 ciency virus (SIV) infection and humans with human immunodeficiency virus type 1 (HIV-1) infection th
134 identified as a late postentry suppressor of human immunodeficiency virus type 1 (HIV-1) infection, i
135 potent anti-retroviral drugs in controlling human immunodeficiency virus type 1 (HIV-1) infection, l
136 rial of preexposure chemoprophylaxis against human immunodeficiency virus type 1 (HIV-1) infection, t
137 al contraceptives regulate susceptibility to human immunodeficiency virus type 1 (HIV-1) infection, t
138 e HCV infection in participants with chronic human immunodeficiency virus type 1 (HIV-1) infection.
139 ophylaxis (PrEP) candidate for prevention of human immunodeficiency virus type 1 (HIV-1) infection.
140 cell-based gene modification therapy against human immunodeficiency virus type 1 (HIV-1) infection.
141 source of type I IFN (IFN-I) in response to human immunodeficiency virus type 1 (HIV-1) infection.
142 ired T-cell function was investigated during human immunodeficiency virus type 1 (HIV-1) infection.
143 interventions to prevent the acquisition of human immunodeficiency virus type 1 (HIV-1) infection.
144 ete roles and functions of DRFs during early human immunodeficiency virus type 1 (HIV-1) infection.
145 of CD4+ T lymphocytes, a major reservoir for human immunodeficiency virus type 1 (HIV-1) infection.
146 ed as prophylaxis can prevent acquisition of human immunodeficiency virus type 1 (HIV-1) infection.
149 timulated genes (ISGs) and potently suppress Human immunodeficiency virus type 1 (HIV-1) infectivity
150 OBEC3G (A3G) is a human enzyme that inhibits human immunodeficiency virus type 1 (HIV-1) infectivity,
153 ointestinal mucosa is the primary site where human immunodeficiency virus type 1 (HIV-1) invades, amp
155 nhibiting infection with diverse variants of human immunodeficiency virus type 1 (HIV-1) is a key, as
173 impact of CD4+ regulatory T cells (Tregs) on human immunodeficiency virus type 1 (HIV-1) pathogenesis
175 plication to clinically undetectable levels, human immunodeficiency virus type 1 (HIV-1) persists in
176 otyping success rate was evaluated in 12 828 human immunodeficiency virus type 1 (HIV-1) plasma sampl
177 ic visual display, we examine the changes in human immunodeficiency virus type 1 (HIV-1) plasma viral
178 The gp120 portion of the envelope spike on human immunodeficiency virus type 1 (HIV-1) plays a crit
182 Local Alignment Program (LAP) using 115,118 human immunodeficiency virus type 1 (HIV-1) protease, re
183 ic guide RNAs (gRNAs), can excise integrated human immunodeficiency virus type 1 (HIV-1) provirus fro
187 (HCV) infection in patients coinfected with human immunodeficiency virus type 1 (HIV-1) remains a me
189 is C virus (HCV) in patients coinfected with human immunodeficiency virus type 1 (HIV-1) remains an u
190 (ART) limits proviral reservoirs, a goal for human immunodeficiency virus type 1 (HIV-1) remission st
191 tissues (LTs) are the principal sites where human immunodeficiency virus type 1 (HIV-1) replicates a
193 main-containing protein 1 (SAMHD1) restricts human immunodeficiency virus type 1 (HIV-1) replication
195 ymphoid tissues (LTs) are principal sites of human immunodeficiency virus type 1 (HIV-1) replication,
196 Tregs) suppress T-cell immune activation and human immunodeficiency virus type 1 (HIV-1) replication,
198 ciations of CCR5-Delta32 heterozygosity with human immunodeficiency virus type 1 (HIV-1) reservoir si
201 t the ribonuclease (RNase) H activity of the human immunodeficiency virus type 1 (HIV-1) reverse tran
206 thout condom use among 911 African HSV-2 and human immunodeficiency virus type 1 (HIV-1) serodiscorda
208 ve reduced effectiveness in the treatment of human immunodeficiency virus type 1 (HIV-1) subtype C in
210 vir plus 2 nucleos(t)ides for maintenance of human immunodeficiency virus type 1 (HIV-1) suppression.
211 this study was to investigate the effect of human immunodeficiency virus type 1 (HIV-1) Tat on the P
212 s an interferon (IFN)-inducible inhibitor of human immunodeficiency virus type 1 (HIV-1) that acts at
213 ith latency reversing agents (LRAs) enhances human immunodeficiency virus type 1 (HIV-1) transcriptio
215 atory state has been hypothesized to enhance human immunodeficiency virus type 1 (HIV-1) transmission
216 affect efavirenz pharmacokinetics, maternal human immunodeficiency virus type 1 (HIV-1) treatment ou
218 functional antibodies remains a challenge in human immunodeficiency virus type 1 (HIV-1) vaccine deve
219 design are being developed as immunogens in human immunodeficiency virus type 1 (HIV-1) vaccine deve
223 to be an important component of a protective human immunodeficiency virus type 1 (HIV-1) vaccine.
224 mune responses and inflammation to candidate human immunodeficiency virus type 1 (HIV-1) vaccines rep
227 Recent studies have evaluated cumulative human immunodeficiency virus type 1 (HIV-1) viral load (
230 important role in regulating the assembly of human immunodeficiency virus type 1 (HIV-1) virus partic
232 ave shown that infection of cells containing human immunodeficiency virus type 1 (HIV-1) with KSHV le
233 of single-stranded DNA (ssDNA) derived from human immunodeficiency virus type 1 (HIV-1), activated t
234 -cell lymphotropic virus type 1 (HTLV-1) and human immunodeficiency virus type 1 (HIV-1), but arises
235 ergistically induce the expression of latent human immunodeficiency virus type 1 (HIV-1), but studies
238 The surface envelope glycoprotein (SU) of Human immunodeficiency virus type 1 (HIV-1), gp120(SU) p
239 e strong dependence of retroviruses, such as human immunodeficiency virus type 1 (HIV-1), on host cel
240 nd development of three major model systems [human immunodeficiency virus type 1 (HIV-1), Rous sarcom
241 rs based on the env genes of two isolates of human immunodeficiency virus type 1 (HIV-1), specificall
242 ing delayed progression of disease caused by human immunodeficiency virus type 1 (HIV-1), yet a ligan
243 ess to AIDS, in stark contrast to pathogenic human immunodeficiency virus type 1 (HIV-1)-human and SI
244 lated with rotavirus vaccine responses in 68 human immunodeficiency virus type 1 (HIV-1)-infected (an
245 013 that reported virological outcomes among human immunodeficiency virus type 1 (HIV-1)-infected adu
246 a, has long been known to be elevated in the human immunodeficiency virus type 1 (HIV-1)-infected bra
247 ance cytotoxic T lymphocyte (CTL) evasion in human immunodeficiency virus type 1 (HIV-1)-infected CD4
250 cy virus (SHIV)-infected rhesus macaques and human immunodeficiency virus type 1 (HIV-1)-infected hum
251 alleles (e.g., HLA-B*27) are enriched among human immunodeficiency virus type 1 (HIV-1)-infected ind
253 ssion in the central nervous system (CNS) of human immunodeficiency virus type 1 (HIV-1)-infected ind
254 combination antiretroviral therapy (cART) to human immunodeficiency virus type 1 (HIV-1)-infected inf
258 ion of combination antiretroviral therapy to human immunodeficiency virus type 1 (HIV-1)-infected pre
259 -blind, placebo-controlled clinical trial on human immunodeficiency virus type 1 (HIV-1)-infected sub
261 are reported in uninfected children born to human immunodeficiency virus type 1 (HIV-1)-infected wom
262 at women on PrEP had IgA with higher average human immunodeficiency virus type 1 (HIV-1)-neutralizing
263 ed infectious virions, non-bNAbs and mucosal human immunodeficiency virus type 1 (HIV-1)-positive IgG
264 onal regulatory T cells (Tregs) can suppress human immunodeficiency virus type 1 (HIV-1)-specific imm
265 tion with an anti-PD-L1 antibody may improve human immunodeficiency virus type 1 (HIV-1)-specific imm
275 ck-and-kill" therapeutic approach to reverse human immunodeficiency virus type-1 (HIV) latency from C
277 sease (GUD) in a cohort of women living with human immunodeficiency virus type-1 (HIV-1) in Burkina F
280 a wide range of enveloped viruses, including human immunodeficiency virus, type 1 (HIV-1) by directly
281 -1beta is associated with the progression of human immunodeficiency virus, type 1 (HIV-1) disease or
282 brane-proximal external region (MPER) of the human immunodeficiency virus, type 1 (HIV-1) envelope gl
283 e supposed to form a natural barrier against human immunodeficiency virus, type 1 (HIV-1) infection.
284 y contribute to racial disparities in plasma human immunodeficiency virus type 1 : HIV-1) RNA levels
285 e replication of primate lentiviruses (e.g., human immunodeficiency virus type 1 [HIV-1] and simian i
286 or tenofovir/emtricitabine (TDF/FTC) in 1857 human immunodeficiency virus type 1-infected, treatment-
288 be fruitful for this scaffold, since strong human immunodeficiency virus type 1 integrase (HIV-1 IN)
289 virus, reovirus, vesicular stomatitis virus, human immunodeficiency virus type 1, or herpes simplex v
290 96-1997, 1998-1999, 2000-2001], and AIDS and human immunodeficiency virus type 1 RNA at baseline) mor
292 The role of the adenosine (ADO) pathway in human immunodeficiency virus type 1/simian immunodeficie
293 ized Rous sarcoma virus (RSV) intasomes with human immunodeficiency virus type 1 strand transfer inhi
294 transduction domain of the tat protein from human immunodeficiency virus type 1 (tat-cyclotraxin-B).
299 at this position and either co-expression of human immunodeficiency virus type 1 vpu or siRNA-mediate
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