ライフサイエンスコーパス: human influenza A virus

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1 s only a very minor role in the evolution of human influenza A virus.

2 ," is virucidal for H1 hemagglutinin-bearing human influenza A viruses.

3 as one mechanism for the emergence of novel human influenza A viruses.

4 the relative virulence of selected avian and human influenza A viruses.

5 cid residues that are highly conserved among human influenza A viruses.

6 e and replaced the HA gene of the prevailing human influenza A viruses.

7 otoxicity was demonstrated against avian and human influenza A viruses.

8 /20/99) or an H3 serotype (A/Panama/2007/99) human influenza A virus and then used these constructs a

9 mples of such viruses include three pandemic human influenza A viruses and canine parvovirus in dogs.

10 The NS1A proteins of human influenza A viruses bind CPSF30, a cellular factor

11 An especially novel aspect of human influenza A virus binding is its ability to equiva

12 The NS1 protein of human influenza A viruses binds the 30-kDa subunit of th

13 The human influenza A virus continues to thrive even among p

14 ts of sequencing 209 complete genomes of the human influenza A virus, encompassing a total of 2,821,1

15 Although the surface proteins of human influenza A virus evolve rapidly and continually p

16 it of the RNA polymerase complex of seasonal human influenza A viruses has been shown to localize to

17 Human influenza A virus (IAV) vaccination is limited by

18 Here we investigated the replication of human influenza A virus in bat cell lines and the barrie

19 Our data suggest that replication of human influenza A viruses in a nonnative host drives the

20 801) elicit robust CTL responses against any human influenza A virus, including H7N9, whereas ethnici

21 its recognition by the immune system during human influenza A virus infection.

22 the possible roles of anti-M2 antibodies in human influenza A virus infection.

23 sing the pandemic risk posed by specific non-human influenza A viruses is an important goal in public

24 y reference strain derived from the earliest human influenza A virus isolate, WS/33.

25 this residue is not conserved in a number of human influenza A virus isolates.

26 n influenza A virus hemagglutinins (HAs) and human influenza A virus matrix (M) proteins M1 and M2, w

27 esults suggest that the currently prevailing human influenza A viruses might have lost their ability

28 that phosphorylation of the NS1 protein of a human influenza A virus occurs not only at the threonine

29 Human influenza A viruses preferentially bind alpha2,6-l

30 To determine the role of MxA in blocking human influenza A virus replication in primate cells, we

31 Deep sequencing of adapted human influenza A virus revealed a mutation in the PA po

32 was similar to that of a primary response to human influenza A viruses; serum neutralizing antibody w

33 ly, found in the NS1A proteins of almost all human influenza A virus strains.

34 ial cells, the primary cell type infected by human influenza A virus strains.

35 y phosphorylation of the NS1 protein of this human influenza A virus that regulates its replication i

36 We showed that human influenza A virus uses canonical sialic acid recep

37 ne the extent of homologous recombination in human influenza A virus, we assembled a data set of 13,8

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