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1 erexpressed in approximately 24% of analyzed human mammary carcinomas.
2 activated to anti-BgCDE in human cells, the human mammary carcinoma cell line MCF-7 was treated with
6 gnals required for MDA-MB-231 and MDA-MB-435 human mammary carcinoma cell spreading on vitronectin or
7 ndividual cells of two different cell lines, human mammary carcinoma cells and rat liver epithelial c
8 in cultured Chinese hamster ovary cells and human mammary carcinoma cells and reduced intracellular
9 owth) ranging from 160 nM in sensitive MCF-7 human mammary carcinoma cells to 17 microM in relatively
11 TD-truncated CXCR4 (CXCR4-DeltaCTD) in MCF-7 human mammary carcinoma cells to determine whether the C
12 estrogen receptor binding activity in MCF-7 human mammary carcinoma cells, and 4-(hydroxymethyl)estr
13 g a search for cell cycle-regulated genes in human mammary carcinoma cells, we identified HSIX1, a re
24 L-60 cell line) or cytochrome P450 activity (human mammary carcinoma MCF-7 cell line), cultures were
25 tem for nontoxic and low-dose coexposures of human mammary carcinoma MCF-7 cells against polycyclic a
31 e found that increased expression of Tid1 in human mammary carcinomas overexpressing ErbB-2 suppresse
33 ata find correlation with lung metastases of human mammary carcinomas that are associated with myeloi
36 vitro antitumor activity against MDA-MB-435 human mammary carcinoma was also determined for natural
38 we use proteomics to investigate the ECM of human mammary carcinoma xenografts and show that primary
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