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1 erexpressed in approximately 24% of analyzed human mammary carcinomas.
2  activated to anti-BgCDE in human cells, the human mammary carcinoma cell line MCF-7 was treated with
3                          We also treated the human mammary carcinoma cell line MCF-7 with DB[a,l]P an
4 GA1 and BRCA1 mRNA and protein expression in human mammary carcinoma cell lines and tissues.
5  anchorage-independent growth of a series of human mammary carcinoma cell lines.
6 gnals required for MDA-MB-231 and MDA-MB-435 human mammary carcinoma cell spreading on vitronectin or
7 ndividual cells of two different cell lines, human mammary carcinoma cells and rat liver epithelial c
8  in cultured Chinese hamster ovary cells and human mammary carcinoma cells and reduced intracellular
9 owth) ranging from 160 nM in sensitive MCF-7 human mammary carcinoma cells to 17 microM in relatively
10                       Exposure of MDA-MB-231 human mammary carcinoma cells to an ionizing radiation d
11 TD-truncated CXCR4 (CXCR4-DeltaCTD) in MCF-7 human mammary carcinoma cells to determine whether the C
12  estrogen receptor binding activity in MCF-7 human mammary carcinoma cells, and 4-(hydroxymethyl)estr
13 g a search for cell cycle-regulated genes in human mammary carcinoma cells, we identified HSIX1, a re
14     Using Sdc1 deletion mutants expressed in human mammary carcinoma cells, we identified the active
15 roduction causes increased growth in vivo of human mammary carcinoma cells.
16 response to epidermal growth factor (EGF) in human mammary carcinoma cells.
17  to suppress anchorage-independent growth in human mammary carcinoma cells.
18 tion of growth arrest and apoptosis of MCF-7 human mammary carcinoma cells.
19 ring the G1 phase of the cell cycle of MCF-7 human mammary carcinoma cells.
20 iated with the TGF-beta signaling pathway in human mammary carcinoma cells.
21 vels, motility and the invasive potential of human mammary carcinoma cells.
22  a Tcfap2c target gene in murine, as well as human, mammary carcinoma cells.
23                                           In human mammary carcinoma MCF-7 and mouse hepatoma Hepa-1
24 L-60 cell line) or cytochrome P450 activity (human mammary carcinoma MCF-7 cell line), cultures were
25 tem for nontoxic and low-dose coexposures of human mammary carcinoma MCF-7 cells against polycyclic a
26 ryl hydrocarbon dimethylbenz[a]anthracene in human mammary carcinoma MCF-7 cells.
27 found to reversibly inhibit the N-SMase from human mammary carcinoma MCF7 cells.
28 gland NMuMG, human normal bladder HCV29, and human mammary carcinoma MCF7 cells.
29                       Adhesion of metastatic human mammary carcinoma MDA-MB-435 cells to the basement
30 cifically protected by nuclear proteins from human mammary carcinoma MDA468 cells.
31 e found that increased expression of Tid1 in human mammary carcinomas overexpressing ErbB-2 suppresse
32                            Immunolabeling of human mammary carcinoma showed that WNT7B immunoreactivi
33 ata find correlation with lung metastases of human mammary carcinomas that are associated with myeloi
34      Our results show that in usual types of human mammary carcinomas, the Id1 protein is expressed e
35  both systemic P388 murine leukemia and MX-1 human mammary carcinoma transplanted in nude mice.
36  vitro antitumor activity against MDA-MB-435 human mammary carcinoma was also determined for natural
37                       In nude mice bearing a human mammary carcinoma xenograft (MX-1), marked tumor r
38  we use proteomics to investigate the ECM of human mammary carcinoma xenografts and show that primary
39 l mucin expressed in most breast cancers) on human mammary carcinoma xenografts in nude mice.

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