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1 ng with acute respiratory tract illness, for human metapneumovirus.
2 nfluenza A, respiratory syncytial virus, and human metapneumovirus.
3 ditional methods, primarily rhinoviruses and human metapneumovirus.
4 syncytial virus (RSV), human bocavirus, and human metapneumovirus.
7 arainfluenza viruses 1-4, influenza A and B, human metapneumovirus, adenovirus, and human rhinoviruse
8 ruses; parainfluenza viruses 1, 2, 3, and 4; human metapneumovirus; adenovirus; enterovirus-rhinoviru
9 uses 1-3, influenza viruses AH1, AH3, and B, human metapneumovirus, adenoviruses, and bocavirus) and
10 rus, as well as respiratory syncytial virus, human metapneumovirus, adenoviruses, picornaviruses, and
11 influenza, respiratory syncytial virus, and human metapneumovirus among patients with CAP of all age
12 G proteins from other subtype C viruses and human metapneumovirus and more than 170 aa larger than t
13 i) respiratory syncytial viruses A and B and human metapneumovirus, and (iii) parainfluenza virus typ
15 of human rhinoviruses, human coronaviruses, human metapneumovirus, and human bocavirus, as well as t
17 luenza viruses, respiratory syncytial virus, human metapneumovirus, and the deadly zoonotic henipavir
20 family Paramyxoviridae includes two members, human metapneumovirus (hMPV) and avian metapneumovirus (
22 n severe bronchiolitis and dual infection by human metapneumovirus (hMPV) and human respiratory syncy
23 inflammatory chemokine production induced by human metapneumovirus (hMPV) and Nipah virus (NiV), sugg
24 coproteins derived from two human pathogens, human metapneumovirus (hMPV) and respiratory syncytial v
26 act infections caused by the paramyxoviruses human metapneumovirus (hMPV) and respiratory syncytial v
33 ngly, GFP-expressing AMPV and GFP-expressing human metapneumovirus (HMPV) could be recovered using th
40 de-PLx system resulted from the detection of human metapneumovirus (HMPV) in 9 specimens, human CoV (
41 den of respiratory syncytial virus (RSV) and human metapneumovirus (HMPV) in older adults in comparis
42 nts (ASRs) (bioMerieux) for the detection of human metapneumovirus (hMPV) in respiratory samples.
43 ntibody MAb-8 was evaluated for detection of human metapneumovirus (HMPV) in shell vial centrifugatio
44 n immunocompromised hosts, but the impact of human metapneumovirus (hMPV) in this setting was previou
74 e important clinical implications.IMPORTANCE Human metapneumovirus (HMPV) is a recently discovered pa
86 uman respiratory syncytial virus (hRSV), the human metapneumovirus (hMPV) is one of the leading cause
91 on by challenging B cell-deficient mice with human metapneumovirus (HMPV) several weeks after primary
92 Human respiratory syncytial virus (hRSV) and human metapneumovirus (hMPV) share virologic and epidemi
93 A microarray (Virochip) was used to detect a human metapneumovirus (hMPV) strain associated with a cr
94 he CAN98-75 (CAN75) and the CAN97-83 (CAN83) human metapneumovirus (HMPV) strains, which represent th
95 eavage activation of the fusion F protein of human metapneumovirus (HMPV) to replication and pathogen
96 kine production by BALB/c mice infected with human metapneumovirus (hMPV) was compared to respiratory
100 ed the capability of MS for the detection of human metapneumovirus (HMPV), a common cause of respirat
106 piratory syncytial virus (RSV), enterovirus, human metapneumovirus (hMPV), adenovirus (AdV), and rhin
108 es human respiratory syncytial virus (hRSV), human metapneumovirus (hMPV), and human parainfluenza vi
109 ivatives of the CAN97-83 clinical isolate of human metapneumovirus (HMPV), consensus nucleotide seque
113 ts: adenovirus, coronaviruses HKU1 and NL63, human metapneumovirus (hMPV), influenza A virus (to type
114 gnosis of respiratory syncytial virus (RSV), human metapneumovirus (HMPV), parainfluenza virus 1 to 3
116 u-A, Flu-B, PIV-1, PIV-2, PIV-3, PIV-4, RSV, human metapneumovirus (hMPV), rhinoviruses (RhVs), enter
117 (RSV), Human Parainfluenza Virus (HPIV), and Human Metapneumovirus (hMPV), we adopt a theoretical app
120 man respiratory syncytial viruses (HRSV) and human metapneumoviruses (HMPV) were involved in the etio
122 s such as human respiratory syncytial virus, human metapneumovirus, human parainfluenza virus type 3,
123 ral infections (respiratory syncytial virus, human metapneumovirus, human rhinovirus, and adenovirus)
131 compared the gene expression of TCD8 during human metapneumovirus infection to those in acute or chr
132 table by both commercial assays (adenovirus, human metapneumovirus, influenza A virus, influenza B vi
135 cture and activities of CR-VI+, a portion of human Metapneumovirus L consisting of CR-VI and the poor
136 ay epithelial cells or mice with recombinant human metapneumovirus lacking SH expression (rhMPV-Delta
138 us types 1, 2, and 3 (PIV1, PIV2, and PIV3), human metapneumovirus (MPV), and adenovirus (AdV) in 1,1
139 tribute to TCD8 impairment induced by either human metapneumovirus or influenza virus infection.
141 er viruses (ie, respiratory syncytial virus, human metapneumovirus, parainfluenza virus, and influenz
142 luenza viruses, coronavirus, rhinovirus, and human metapneumovirus, represent a considerable global h
143 nfluenza virus, coronaviruses, rhinoviruses, human metapneumovirus, respiratory syncytial virus, para
144 of respiratory syncytial virus, adenovirus, human metapneumovirus, rhinovirus, and influenza virus b
150 influenza, respiratory syncytial virus, and human metapneumovirus were substantially more common in
151 iratory tract illnesses were attributable to human metapneumovirus, which means that 12 percent of al
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