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1 ng against human papillomavirus and low-risk human papillomavirus.
2 dy, we assessed the importance of the CTD of human papillomavirus 11 (HPV11) E1 in vivo, using a cell
4 ed tetracycline-inducible vector system, and human papillomavirus 16 (HPV 16) E6 and E7 gene-immortal
7 In agreement with previous studies, we found human papillomavirus 16 (HPV16) and HPV18 in oropharynge
11 rect cleavage of capsid-associated L2 during human papillomavirus 16 (HPV16) infection remains poorly
16 ed a novel interaction between Rint1 and the human papillomavirus 16 (HPV16) transcription and replic
17 s, including bovine papillomavirus (BPV) and human papillomavirus 16 (HPV16), associate with the cell
19 iority 1 month after the last dose of 2-dose human papillomavirus 16/18 AS04-adjuvanted (AS04-HPV-16/
23 ecipitated with the BPV-1 E2 protein, as did human papillomavirus 31 (HPV-31) E2, which also colocali
25 n Australia, high uptake of the quadrivalent human papillomavirus (4vHPV) vaccine has led to reductio
27 a phosphorylated peptide motif derived from human papillomavirus 8 (HPV-8), the E2 hinge region incl
28 -26 years showed efficacy of the nine-valent human papillomavirus (9vHPV; HPV 6, 11, 16, 18, 31, 33,
30 und for male circumcision protecting against human papillomavirus and low-risk human papillomavirus.
33 CC with known p16 tumor status (surrogate of human papillomavirus) and cigarette smoking history (pac
35 lief with the recently described epidemic of human papillomavirus-associated head and neck squamous c
36 r T cell responses in complete regression of human papillomavirus-associated metastatic cervical canc
37 regarding the association between beta-genus human papillomavirus (beta-HPV) and cutaneous squamous c
38 evaluated the association between beta genus human papillomaviruses (betaPV) and keratinocyte carcino
41 , Escherichia coli, influenza virus, cancer, human papillomavirus, dopamine, glutamic acid, IgG, IgE,
43 ility of certain viruses (hepatitis B and C, human papillomavirus, etc) to cause cancer has been time
47 itive for the p16 protein, an indicator that human papillomavirus had a role in the causation of the
49 e.g., atherosclerosis, cancer), viral (e.g., human papillomavirus, herpes virus) and bacterial (e.g.,
51 awareness of the tremendous global impact of human papillomavirus (HPV) -caused cancers, refocus the
52 compared regimens including 2 doses (2D) of human papillomavirus (HPV) 16/18 AS04-adjuvanted vaccine
53 igated the natural history of high-risk anal human papillomavirus (HPV) among a multinational group o
54 transmitted diseases treatment guidelines on human papillomavirus (HPV) and anogenital warts (AGWs),
55 n antiretroviral therapy (ART) and high-risk human papillomavirus (HPV) and cervical lesions in women
56 of the DNA tumor viruses, including E7 from human papillomavirus (HPV) and E1A from adenovirus, as p
57 ave been mixed on whether naturally acquired human papillomavirus (HPV) antibodies may protect agains
66 ithin the endosome triggers uncoating of the human papillomavirus (HPV) capsid, whereupon host cyclop
70 the prevalence and risk factors for genital human papillomavirus (HPV) detection among men who deny
71 norectal swabs for cytologic examination and human papillomavirus (HPV) detection and genotyping.
72 amine the temporal relationship between oral human papillomavirus (HPV) detection and risk of head an
73 -CoV), Mycobacterium tuberculosis (MTB), and human papillomavirus (HPV) DNA based on a colorimetric p
77 iltrating T cells selected when possible for human papillomavirus (HPV) E6 and E7 reactivity (HPV-TIL
79 cell differentiation-dependent regulation of human papillomavirus (HPV) gene expression is required f
81 and minor (L2) capsid proteins of oncogenic human papillomavirus (HPV) genotype 31 (HPV31) to determ
82 e has led to reductions in the prevalence of human papillomavirus (HPV) genotypes 6, 11, 16, and 18 i
86 ly representative prevalence data on genital human papillomavirus (HPV) in males in the United States
87 9-valent viruslike particle vaccine against human papillomavirus (HPV) includes the HPV types in the
90 obiota (VM) may influence risk of persistent Human Papillomavirus (HPV) infection and cervical carcin
91 to analyze the associations between cervical human papillomavirus (HPV) infection and human immunodef
92 luated the risk factors associated with oral human papillomavirus (HPV) infection and oral lesions in
93 n about the type-specific prevalence of anal human papillomavirus (HPV) infection and risk factors fo
95 s the risk of sequential acquisition of anal human papillomavirus (HPV) infection following a type-sp
96 Lesbians and bisexual women are at risk for human papillomavirus (HPV) infection from female and mal
98 however, its association with cervicovaginal human papillomavirus (HPV) infection has not been studie
99 ciation between vaginal douching and genital human papillomavirus (HPV) infection have found contrary
100 thesized to explain the excess in detectable human papillomavirus (HPV) infection in Latin America, v
107 investigated bereavement in association with human papillomavirus (HPV) infection, both HPV16 and oth
111 have sex with men (MSM) are at high risk for human papillomavirus (HPV) infection; vaccination is rec
114 examined the concordance of penile and oral human papillomavirus (HPV) infections in the United Stat
126 The two licensed bivalent and quadrivalent human papillomavirus (HPV) L1 (the major papillomavirus
127 The presence of neutralizing epitopes in human papillomavirus (HPV) L1 virus-like particles (VLPs
132 Naturally induced serum antibodies against human papillomavirus (HPV) may affect risks of subsequen
133 cularly those in immunocompromised patients, human papillomavirus (HPV) may be an important factor.
134 Recent birth cohorts vaccinated against human papillomavirus (HPV) may be protected against up t
142 cervical cancer in England, UK, will change: human papillomavirus (HPV) screening will be the primary
143 Importance: Pathology-based measures of human papillomavirus (HPV) status are routinely obtained
145 aryngeal cases with information available on human papillomavirus (HPV) status indicated that this as
154 e influence on biopsy and treatment rates of human papillomavirus (HPV) triage of cytology showing at
156 8, Scotland launched routine vaccination for human papillomavirus (HPV) types 16 and 18, targeted at
157 ta on the relative carcinogenic potential of human papillomavirus (HPV) types among women infected wi
160 Case reports have suggested a link between human papillomavirus (HPV) vaccination and development o
162 ling studies have been widely used to inform human papillomavirus (HPV) vaccination policy decisions;
168 ctiveness of providing 3-doses of nonavalent human papillomavirus (HPV) vaccine (9vHPV) to females ag
170 ation of antibody response following reduced human papillomavirus (HPV) vaccine doses has not been de
181 ted partial cross-protection by the bivalent human papillomavirus (HPV) vaccine, which targets HPV-16
185 First generation bivalent and quadrivalent human papillomavirus (HPV) vaccines have been introduced
192 control group, GUWE treated DCs pulsed with human papillomavirus (HPV)-16 E6/E7 peptides significant
193 neoplastic transformation of human cells, so human papillomavirus (HPV)-associated cancers will also
196 with men (MSM) bear a substantial burden of human papillomavirus (HPV)-associated disease, prospecti
198 However, the role of gammadelta T cells in human papillomavirus (HPV)-associated uterine cervical S
201 sustained in CIN3, consistent with high-risk human papillomavirus (HPV)-induced tumor suppressor inac
202 ion levels were significantly upregulated in human papillomavirus (HPV)-infected cell lines and tissu
209 ancerization of the skin from which multiple human papillomavirus (HPV)-positive squamous cell carcin
212 Peptide-based therapeutic vaccines against human papillomavirus (HPV)-related cancers are usually d
213 experienced an increase in the incidence of human papillomavirus (HPV)-related cancers that are not
214 m of this study was to determine the risk of human papillomavirus (HPV)-related carcinomas and premal
216 ed patients develop persistent, stigmatizing human papillomavirus (HPV)-related cutaneous and genital
218 urpose Cancer survivors are at high risk for human papillomavirus (HPV)-related morbidities; we estim
221 ol (AJCC/UICC) staging system, developed for human papillomavirus (HPV)-unrelated disease, discrimina
227 ng of E6AP by the E6 oncoprotein of distinct human papillomaviruses (HPV) contributes to the developm
231 increased expression of the major high-risk human-papillomavirus (HPV) oncogenes E6 and E7 in basal
233 eplication (human cytomegalovirus [HCMV] and human papillomavirus [HPV]) and transcription (HSV-1, HC
235 sitivity and limit of detection of high-risk human papillomavirus (HPV16), compared to the standard s
248 Despite preventive vaccines for oncogenic human papillomaviruses (HPVs), cervical intraepithelial
250 Persistent infection by specific oncogenic human papillomaviruses (HPVs), including HPV58, has been
254 Carolina, to compare clearance of high-risk human papillomavirus (HR-HPV) infection between ethnicit
255 The risk of anal cancer due to high-risk human papillomavirus (HR-HPV) is higher in women living
256 18/45 genotype assay (AHPV GT) for high-risk human papillomavirus (hrHPV) detection, clinical perform
257 cervical squamous cell carcinomas, high-risk human papillomavirus (HRHPV) DNA is usually integrated i
260 PV-related disease.IMPORTANCE High-risk-type human papillomaviruses (hrHPVs) cause 5% of all cancer c
264 on; 33.9% (95% CI: 24.3-43.5%) for high-risk human papillomavirus infection from cervical samples and
266 aced on the discoveries made in the study of human papillomaviruses, Merkel cell carcinoma-associated
268 a normal Pap test are positive for oncogenic human papillomavirus (oncHPV) types is important for set
271 of C57BL/6 mice with a replication-defective human papillomavirus pseudovirus (HPV PsV) expressing HS
273 protocols on patient-derived xenografts from human papillomavirus-related (HPV(+)) head and neck squa
275 C was also associated with increased risk of human papillomavirus-related cancers, including anal can
279 se; therefore, stratification of patients by human papillomavirus status in clinical trials is indica
280 or molecular characteristics, including age, human papillomavirus status, tumor grade and TP53 mutati
284 blished a cell-free in vitro system to study human papillomavirus type 16 (HPV16) assembly, a poorly
285 r to identify cellular factors that regulate human papillomavirus type 16 (HPV16) gene expression, ce
290 polycomb-mediated epigenetic methylations in human papillomavirus type 16 E7 expressing cells, and in
293 red for gene transduction by three oncogenic human papillomavirus types, HPV16, HPV18, and HPV31.
296 ne adjuvanted with aluminum hydroxide, and a human papillomavirus vaccine adjuvanted with aluminum hy
297 toxoid, and reduced acellular pertussis; and human papillomavirus vaccines) in addition to time postt
298 ANCE After internalization, the nonenveloped human papillomavirus virion uncoats in the endosome, whe
299 A recent clinical trial using the 9-valent human papillomavirus virus (HPV) vaccine has shown that
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