ライフサイエンスコーパス: human prostate cancer cell line

1 c acid, is constitutively expressed in LNCaP human prostate cancer cell line.

2 ance gene were transferred into cells from a human prostate cancer cell line.

3 scription factors, is occupied by FOXA1 in a human prostate cancer cell line.

4 aspase 3, and cleaved caspase 3 in the LNCaP human prostate cancer cell line.

5 the p53-positive, COX-2-negative MDA-PCa-2b human prostate cancer cell line.

6 o changes in histone lysine methylation in a human prostate cancer cell line.

7 of the androgen-unresponsive, p53-null, PC-3 human prostate cancer cell line.

8 and ARCaP) and nonproducing (PC-3 and DU145) human prostate cancer cell lines.

9 SV1, were present in 22Rv1, LNCaP, and VCaP human prostate cancer cell lines.

10 downstream mediator of MKK4, in six of eight human prostate cancer cell lines.

11 ensitive LNCaP and androgen-insensitive PC-3 human prostate cancer cell lines.

12 the expression of NuSAP in the LNCaP and PC3 human prostate cancer cell lines.

13 can stimulate secreted activity of MMP-9 in human prostate cancer cell lines.

14 ssion of HIF-1alpha was evaluated in rat and human prostate cancer cell lines.

15 PTEN status and PDGF isoforms were noted in human prostate cancer cell lines.

16 ive (LNCaP) and androgen-insensitive (DuPro) human prostate cancer cell lines.

17 teolytic and osteoblastic lesions induced by human prostate cancer cell lines.

18 apable of inducing cell cycle progression in human prostate cancer cell lines.

19 itutive and IL-6-induced STAT3 activation in human prostate cancer cell lines.

20 so regulated by p53 and induces apoptosis in human prostate cancer cell lines.

21 e, induces apoptosis in androgen-independent human prostate cancer cell lines.

22 e major cause of communication deficiency in human prostate cancer cell lines.

23 trated that DAB2IP is often downregulated in human prostate cancer cell lines.

24 n of CpG island sequences in EDNRB in 5 of 5 human prostate cancer cell lines, 15 of 21 primary prost

25 BCRP, as well as xenografts derived from the human prostate cancer cell line 22Rv1 that naturally exp

26 ree well-characterized, androgen-independent human prostate cancer cell lines: (a) PC3; (b) PC3M; and

27 y prostate cancer have principally relied on human prostate cancer cell lines, all of which were deri

28 In human prostate cancer cell lines, although not in the mo

29 urately estimates cell cycle peak times in a human prostate cancer cell line and it correctly identif

30 ired for PEITC-induced apoptosis in the PC-3 human prostate cancer cell line and that the PEITC-induc

31 Drebrin is also upregulated in human prostate cancer cell lines and co-localizes with a

32 with tumor-associated antigens expressed on human prostate cancer cell lines and patient-derived car

33 Several human prostate cancer cell lines and primary cultures of

34 ivation of one STAT family member, Stat3, in human prostate cancer cell lines and primary prostate tu

35 We studied established human prostate cancer cell lines and primary prostatic e

36 e human MUC18 (huMUC18) cDNA gene from three human prostate cancer cell lines and three human melanom

37 We analysed three well characterized human prostate cancer cell lines and three metastatic pr

38 in vitro by DU-145, PC-3, TSU-PR1, and LnCaP human prostate cancer cell lines and whether Linomide in

39 her levels of Akt activation are observed in human prostate cancer cell lines and xenografts lacking

40 these genes is found to be uniformly lost in human prostate cancer cell lines and xenografts, and pre

41 rolongs the survival of mice inoculated with human prostate cancer cell lines and xenografts.

42 Complement deposition on Du145 cells (human prostate cancer cell line) and anti-MUC1 mAb-media

43 , is not expressed by any of the established human prostate cancer cell lines, and ETB binding is dec

44 on, and epithelial-mesenchymal transition in human prostate cancer cell lines, and stable overexpress

45 receptor expression in the normal prostate, human prostate cancer cell lines, and tissue derived fro

46 nds were tested for cytotoxicity against two human prostate cancer cell lines, androgen-dependent LNC

47 prostate tumor cell line, androgen-repressed human prostate cancer cell line (ARCaP), either transduc

48 An androgen-repressed human prostate cancer cell line, ARCaP, was established

49 -induced expression of HIF-1alpha protein in human prostate cancer cell lines are associated with inc

50 We determined that, in human prostate cancer cell lines, ARv567es functioned as

51 In this study we have used three human prostate cancer cell lines but have focused on the

52 esses the growth and tumorigenic capacity of human prostate cancer cell lines, but enhances migratory

53 CXCR4 expression were determined for several human prostate cancer cell lines by reverse transcriptio

54 We show here that human prostate cancer cell lines can constitutively prod

55 a series of androgen receptor-positive (AR+) human prostate cancer cell lines, CD133+ cells are prese

56 ssion in the mac25/IGFBP-rP1-transfected M12 human prostate cancer cell line compared to M12 control

57 consistently overexpressed in this panel of human prostate cancer cell lines compared to normal huma

58 cell death of both the androgen-independent human prostate cancer cell line CWR22Rv and the androgen

59 We studied human prostate cancer cell lines CWR22Rv1 and CWR-R1, wh

60 Knockdown of PTK6 in the PC3 human prostate cancer cell line disrupts FAK and AKT act

61 AdRSVpHyde significantly inhibited growth of human prostate cancer cell lines DU145 and LNCaP in cult

62 rmined the inhibitory effects of Tkip on the human prostate cancer cell lines DU145 and LNCaP.

63 ptosis in cultures of late-stage, metastatic human prostate cancer cell lines DU145, PC3, and LNCaP.

64 Constitutive expression of MCM7 in a human prostate cancer cell line, DU145, resulted in mark

65 but have focused on the androgen-independent human prostate cancer cell line, DU145, to ask: (a) whet

66 e metastasis of a tumorigenic, nonmetastatic human prostate cancer cell line, DU145.

67 In this study, we use the human prostate cancer cell lines, DU145 and PC3, to inve

68 ith a role of IFI 16 in cellular senescence, human prostate cancer cell lines either did not express

69 In addition, all of the three human prostate cancer cell lines examined (DU145, PC3, a

70 he 44 and 33 kDa Pim-1, are expressed in all human prostate cancer cell lines examined.

71 this work, we studied gene transfection of a human prostate cancer cell line exposed to megahertz pul

72 the first documented case of an established human prostate cancer cell line from a primary tumor of

73 the first documented case of an established human prostate cancer cell line from primary tumor of a

74 Experiments with human prostate cancer cell lines have shown that forced

75 tic, osteolytic, and mixed lesions formed by human prostate cancer cell lines in a severe combined im

76 xin induce apoptosis in androgen-independent human prostate cancer cell lines in vitro.

77 he expression of IGFBP-rP1 can be induced in human prostate cancer cell lines in vivo on interaction

78 Analyses of Nkx3.1 knockout mice and human prostate cancer cell lines indicate that NKX3.1 re

79 over, overexpression of functional IFI 16 in human prostate cancer cell lines inhibited colony format

80 We have previously shown that in the human prostate cancer cell line LN-CaP, 1,25-(OH)2D3 med

81 GL significantly inhibited the viability of human prostate cancer cell line LNCaP (androgen-dependen

82 ergistic increase of cytotoxicity toward the human prostate cancer cell line LNCaP and a significant

83 induces apoptosis in the androgen-sensitive human prostate cancer cell line LNCaP through an androge

84 Similar results were obtained using the human prostate cancer cell line LNCaP, in which endogeno

85 ergistic increase of cytotoxicity toward the human prostate cancer cell line LNCaP, regardless of the

86 e proteomic profile of the poorly metastatic human prostate cancer cell line LNCaP, with its highly m

87 fect of raloxifene in the androgen-sensitive human prostate cancer cell line LNCaP.

88 tivities as antiandrogens were tested in the human prostate cancer cell line LNCaP.

89 phosphorylation, using mutated Stat3 in the human prostate cancer cell line LNCaP.

90 rthotopic model of prostate cancer using the human prostate cancer cell line LNCaP.

91 synergistically to inhibit the growth of the human prostate cancer cell line LNCaP.

92 We examined the biological properties of human prostate cancer cell lines LNCaP and PC-3, in whic

93 e genes expressed differentially between the human prostate cancer cell lines LNCaP and PC-3, which h

94 colon carcinoma cell lines DLD-1 and HCT116; human prostate cancer cell lines LNCaP and PC-3; human l

95 of reverse sialylation activity is noted in human prostate cancer cell lines LNCaP and PC3.

96 We have reported previously that among human prostate cancer cell lines LNCaP but not PC-3 cell

97 c protease (SENP1) in the androgen-sensitive human prostate cancer cell line (LNCaP).

98 aper we demonstrate that SHBG is produced in human prostate cancer cell lines (LNCaP, DU 145, and PC

99 Four cultured human prostate cancer cell lines (LnCap, DU145, DuPro, a

100 entamines to inhibit growth in four cultured human prostate cancer cell lines (LnCap, DU145, PC-3, an

101 Stat3 was observed in human prostate cancer, human prostate cancer cell lines (LNCaP, DU145, PC3, and

102 When the human prostate cancer cell line, LNCaP 104-S, the growth

103 In the current study, we use a human prostate cancer cell line, LNCaP as a model to per

104 Additionally, the human prostate cancer cell line, LNCaP, also formed nucl

105 This possibility was tested by using the AS human prostate cancer cell lines, LNCaP, CWR22Rv1, and L

106 In this investigation, we studied four human prostate cancer cell lines, LNCaP, DU145, LAPC4, a

107 The androgen-independent human prostate cancer cell lines, LNCaP-104R1, ALVA31 an

108 n mixture, protein expression profiling in a human prostate cancer cell line model, and identificatio

109 clones derived from a primary tumour-derived human prostate cancer cell line (OPCT-1), and its use to

110 The human prostate cancer cell line PC-3 was used to determi

111 i-proliferative effects of adaphostin in the human prostate cancer cell line PC-3.

112 enzyme forms of caspase-1, -3, and -9 in the human prostate cancer cell lines PC-3, DU-145, TSU-Pr1m

113 ), human biphenotypic leukemic cells (SP), a human prostate cancer cell line (PC-3), murine melanoma

114 (HMG) proteins HMGI(Y) when tested in three human prostate cancer cell lines (PC-3 > DU-145 > LNCaP)

115 In the human prostate cancer cell line, PC-3, delivery of exoge

116 adrenoceptor antagonists was examined in two human prostate cancer cell lines, PC-3 and DU-145, and a

117 al androgen receptor antagonists against two human prostate cancer cell lines, PC-3 and DU-145, in th

118 of the compound in two androgen-independent human prostate cancer cell lines, PC-3 and DU-145.

119 ibited growth inhibitory effects against the human prostate cancer cell line PC3 at submicromolar con

120 t, the highly undifferentiated, bone-derived human prostate cancer cell line PC3 did not produce an o

121 In a human prostate cancer cell lines PC3 (p53(null)), curcum

122 expression of ITGA7 induced apoptosis in the human prostate cancer cell lines PC3 and DU145.

123 enocopied by partial knockdown of STAT1 in a human prostate cancer cell line (PC3), suggesting that t

124 minant-negative BMP-RII (BMP-RIIDN) into the human prostate cancer cell line, PC3M.

125 Human prostate cancer cell lines PPC-1, which has an ina

126 ssing cells, PrLZ was detected in all of the human prostate cancer cell lines, regardless of androgen

127 NA-mediated myosin VI knockdown in the LNCaP human prostate cancer cell line resulted in impaired in

128 Recent biochemical data suggest that human prostate cancer cell lines show a redox imbalance

129 2M), purified from the conditioned medium of human prostate cancer cell lines, stimulated growth and

130 tes could be demonstrated in any established human prostate cancer cell line tested, and ETB mRNA, de

131 ore differentiated androgen-responsive LNCaP human prostate cancer cell line that is poorly tumorigen

132 Our results demonstrated that, in different human prostate cancer cell lines, the phosphotyrosine (T

133 evels of RNase L by severalfold in the DU145 human prostate cancer cell line through the stable expre

134 nal response of LNCaP, an androgen-sensitive human prostate cancer cell line, to MSA by using high-de

135 n and cell-cycle regulators were observed in human prostate cancer cell lines, transiently transfecte

136 Transfection of the E-cadherin gene into the human prostate cancer cell line TSU.Pr-1 induced cell-ce

137 s were assessed using the BB2r-positive PC-3 human prostate cancer cell line under hypoxic and normox

138 Genetic modulation of PPARD in human prostate cancer cell lines validated the tumor sup

139 Using human prostate cancer cell lines, we have found that TGF

140 Using rapidly dividing human prostate cancer cell lines, we identified mitotica

141 Using xenografts propagated from human prostate cancer cell lines, we reveal that nuclear

142 Using a series of experiments in human prostate cancer cell lines, we validate the highes

143 Human prostate cancer cell lines were used as models, as

144 DU-145 and PC-3 human prostate cancer cell lines were used to test the e

145 Androgen-dependent and androgen-independent human prostate cancer cell lines were used to test the e

146 ociated mutant BRCA1 transgenes in DU-145, a human prostate cancer cell line with low endogenous expr

147 s to C4-2B cell line, a variant of the LNCaP human prostate cancer cell line with propensity for bone

148 cells and orthotopic transplants of various human prostate cancer cell lines with AR over-expression

149 Further we established human prostate cancer cell lines with the mycER construc

150 RNA profiling of human prostate cancer cell lines with various degrees of