ライフサイエンスコーパス: humor

1 tration and protein concentration in aqueous humor.

2 olite concentrations in aqueous and vitreous humor.

3 n D(3) were detected in aqueous and vitreous humor.

4 are difficult to determine in human vitreous humor.

5 lony-forming units per milliliter of aqueous humor.

6 LM are the peripheral retina and/or vitreous humor.

7 ansforming growth factor-beta in the aqueous humor.

8 echanical properties of the natural vitreous humor.

9 e critical for the production of the aqueous humor.

10 ated and wild-type myocilin into the aqueous humor.

11 and sufficient for up-regulation by vitreous humor.

12 age, including metaphors, idioms, and verbal humor.

13 at the concentration levels found in aqueous humor.

14 mutant MYOC is not secreted into the aqueous humor.

15 yocilin complex formation in porcine aqueous humor.

16 duced pressure-dependent drainage of aqueous humor.

17 ls that separate the cornea from the aqueous humor.

18 vobiocin (0.1 mM) was present in the aqueous humor.

19 drug delivery vehicles through the vitreous humor.

20 iably high ESBA105 concentrations in aqueous humor.

21 ance of nutritional support from the aqueous humor.

22 d leukocytes within the aqueous and vitreous humors.

23 (64 days), amniotic fluid (38 days), aqueous humor (101 days), cerebrospinal fluid (9 months), breast

24 eyes was not different for both the vitreous humor (13,600 vs. 15,000 ng/day/mL; P = 0.73) and retina

25 re ebolavirus (EBOV) was detected in aqueous humor 14 weeks after the onset of EVD and 9 weeks after

26 Hmgb1 is accumulated in the vitreous humor 24 hours after IR.

27 ocilin was preincubated with porcine aqueous humor (78% +/- 77% when preincubated in DMEM containing

28 isms were found to predict time to recovery: humor, acting out, emotional hypochondriasis, and projec

29 studies conducted with adults indicate that humor activates specific brain regions, including the te

30 ccumbens in younger participants, indicating humor activation intensity changes during development.

31 ion of regulatory T cells (Tregs) by aqueous humor (AH) and identified TGF-beta as a critical factor.

32 TGFbeta(2)) is often elevated in the aqueous humor (AH) and trabecular meshwork (TM) of patients with

33 sure (IOP) resulting from diminished aqueous humor (AH) drainage through the trabecular pathway.

34 concentration of ANGPTL7 protein in aqueous humor (AH) from patients with glaucoma and control patie

35 enzyme-linked immunosorbent assay in aqueous humor (AH) obtained from human donor eyes (POAG and norm

36 To investigate whether the aqueous humor (AH) of Rb eyes has sufficient tumor-derived DNA t

37 of MRP4 in human TM (HTM) cells and aqueous humor (AH) outflow pathway was determined by RT-PCR, imm

38 ular pressure (IOP) due to increased aqueous humor (AH) outflow resistance, which is associated with

39 arising from increased resistance to aqueous humor (AH) outflow through the trabecular meshwork is a

40 The effects of neuromedin U (NMU) on aqueous humor (AH) outflow were determined in enucleated porcine

41 s from the resistance to drainage of aqueous humor (AH) produced by the ciliary body in a process req

42 icarbonate transport plays a role in aqueous humor (AH) secretion.

43 Aqueous humor (all cohorts), vitreous humor (cohort IV only), an

44 ls detectable after 24 hours in the vitreous humor and 4 hours in the RC.

45 leukin (IL)-6 and IL-8 levels in the aqueous humor and a concomitant approximately twofold (P < 0.001

46 secretion of mutant myocilin in the aqueous humor and by decreasing intracellular accumulation of my

47 pears to antagonize the formation of aqueous humor and cause an inhibition of both F(c) and F(u), des

48 NF-alpha, CCL-2/MCP-1, and ICAM-1 in aqueous humor and CCL-2/MCP-1 and ICAM-1 levels in retina were s

49 n unclassified uveitis, aqueous and vitreous humor and cerebrospinal fluid were also analyzed.

50 te via soluble agents present in the aqueous humor and implicate Best2 as a critical mediator of that

51 dition, Gremlin was present in human aqueous humor and in the perfusate medium of perfusion-cultured

52 d on elevated levels in glaucomatous aqueous humor and its ability to induce extracellular matrix (EC

53 ular adhesion molecule-1 (ICAM-1) in aqueous humor and retina and nuclear translocation of nuclear fa

54 ncentrations were determined in the vitreous humor and retina for 31 days (on days 2, 8, 15, 22, and

55 and drug content quantified in the vitreous humor and retina-choroid tissue by high-performance liqu

56 Freshly secreted aqueous humor and the aqueous humor in the anterior chamber angl

57 ug levels were observed in both the vitreous humor and the retina-choroid even on day 21 after diclof

58 form (hDE-117) was maintained in the aqueous humor and the target tissue (iris-ciliary body) up to 24

59 ulate pressure-dependent drainage of aqueous humor and thus intraocular pressure (IOP) are unknown.

60 Analysis of the myocilin in the aqueous humor and TM revealed that PBA significantly improved th

61 and comparing myocilin levels in the aqueous humor and trabecular meshwork of Tg-MYOC(Y437H) mice.

62 te release with detectable levels in aqueous humor and vitreous for at least 105 days.

63 , a single dose of DPT-GFX sustained aqueous humor and vitreous humor drug levels during the 24-hour

64 re capable of protecting the cornea, aqueous humor, and crystalline lens of rabbits from UV-induced p

65 helium, iris, ciliary body, retina, vitreous humor, and optic nerve.

66 Blood, vitreous humor, and retina samples were collected at predetermine

67 roughout the eye, including cornea, vitreous humor, and retina, suggesting a coordinated protective r

68 liver, spleen, kidney, brain, lung, vitreous humor, and vena cava in comparison to untreated controls

69 munosuppressive molecules within the aqueous humor (AqH) are thought to preserve ocular immune privil

70 Aqueous humor (AqH) from m-EAU and r-EAU was collected and studi

71 ed when the disease was at its peak; aqueous humor (AqH) was collected from one eye, and the other ey

72 LPS injection, eyes were enucleated, aqueous humor (AqH) was collected, and the number of infiltratin

73 after EIU, eyes were enucleated and aqueous humor (AqH) was collected.

74 re killed, eyes were enucleated, and aqueous humor (AqH) was collected.

75 yes were enucleated immediately, and aqueous humor (AqH) was collected.

76 eation of eyes and collection of the aqueous humor (AqH).

77 in which secretion and absorption of aqueous humor are circumferential around and through the iridoco

78 A significant decrease in aqueous humor ascorbate was observed in the exposed lotrafilcon

79 VEGF concentrations in aqueous humor at baseline were higher in patients with type 3 NV

80 giogenic factors upregulated in PDR vitreous humor besides VEGF, thus inhibiting their biological act

81 ry had steep oxygen gradients in the aqueous humor between the cornea and lens.

82 scularized structure located in the vitreous humor between the posterior surface of the lens and neur

83 t myocilin was not secreted into the aqueous humor but accumulated in the ER of the trabecular meshwo

84 ed a significant reduction in CFU in aqueous humor by 1 hour PI (P </= 0.0044).

85 min and fibrin into the aqueous and vitreous humor by 8 hours after infection.

86 d therapeutic drug concentrations in aqueous humor can be achieved for long-term (i.e., 28 days) prot

87 Aqueous and vitreous humor cell counts and protein levels and histopathology

88 ter efficacy with significantly less aqueous humor cells and lower vitreous opacity scores (p<0.05).

89 oxygen levels were decreased in the aqueous humor closest to the pars plicata of the ciliary body an

90 Aqueous humor (all cohorts), vitreous humor (cohort IV only), and blood samples (all cohorts)

91 factor (VEGF) concentrations in the aqueous humor decreased (P = 0.0003), whereas the concentrations

92 It is widely accepted that vitreous humor-derived FGFs are required for the differentiation

93 er treatment with IFN-gamma, porcine aqueous humor, dexamethasone, or a calcium ionophore in cells pr

94 blood direction than in the blood-to-aqueous humor direction, and active.

95 cal diseases including muscle, membrane, and humor disorders; optic nerve damage; and eyelid affectio

96 e inflow) and NO release to increase aqueous humor drainage (increase outflow).

97 of ECM is crucial to maintain normal aqueous humor drainage and intraocular pressure (IOP).

98 in IOP maintenance via modulation of aqueous humor drainage from the eye.

99 the result of abnormal resistance to aqueous humor drainage is a major contributing, and the only pre

100 cular pressure (IOP) due to impaired aqueous humor drainage is a major risk factor for the developmen

101 nterior chamber angle containing the aqueous humor drainage tissue in situ were imaged by TPEF to loc

102 acterized by increased resistance to aqueous humor drainage, elevated IOP, optic nerve degeneration a

103 e angle of the eye, thereby limiting aqueous humor drainage.

104 ed extroversion to positively correlate with humor-driven blood oxygenation level-dependent signal in

105 DPT-GFX sustained aqueous humor and vitreous humor drug levels during the 24-hour study, with a t(1/2

106 -alpha is elevated in the cornea and aqueous humor during allograft rejection and anterior uveitis.

107 Nanotubes persisted in the vitreous humor during the 15 days study and pazopanib levels in t

108 The novel pharmacology and aqueous humor dynamic effects of this molecule suggest it may be

109 Positional changes in other aqueous humor dynamic parameters may contribute to the change in

110 Aqueous humor dynamics (aqueous flow, outflow facility, and uveo

111 he correlation between parameters of aqueous humor dynamics and the influence of CCT in healthy volun

112 bimatoprost's distinctive impact on aqueous humor dynamics are unclear.

113 ellular basis of normal and abnormal aqueous humor dynamics in humans.

114 c agents or genetic manipulations on aqueous humor dynamics in mice and other animal models.

115 the effects of topical 0.005% KL on aqueous humor dynamics in normal monkey eyes.

116 he tissues and cells specialized for aqueous humor dynamics in zebrafish show conservation with that

117 The individual parameters of aqueous humor dynamics may influence each other to maintain intr

118 near correlations between individual aqueous humor dynamics parameters and pachymetry were evaluated

119 The interplay between parameters of aqueous humor dynamics suggests possible autoregulatory mechanis

120 fluence the individual parameters of aqueous humor dynamics that maintain IOP.

121 molecular and cellular mechanisms of aqueous humor dynamics using this species, the authors have char

122 Aqueous humor dynamics were compared in Best2(+/+) and Best2(-/-

123 Aqueous humor dynamics were evaluated fluorophotometrically and

124 s having a mechanoregulatory role in aqueous humor dynamics, with eNOS induction at elevated IOPs lea

125 clude fibrillar degeneration of the vitreous humor, early-onset cataract, minute crystalline deposits

126 o humor, suggesting these regions may form a humor-essential neural network already present in childh

127 ide (NO) increases the rate at which aqueous humor exits the eye; however, the involvement of soluble

128 endings to sensory ganglia, as evidenced by HuMOR expression in neuronal cell bodies located in the

129 We induced human mu-opioid receptor (HuMOR) expression in arthritic joints of mice, using the

130 uring the nocturnal period, although aqueous humor flow decreases by 50% or more at night.

131 nd cellular mechanisms that regulate aqueous humor flow have remained elusive.

132 The results provide evidence that aqueous humor flow is similar between eyes, that flow variation

133 Aqueous humor flow rate, IOP, and outflow facility were measured

134 riod to compensate for the decreased aqueous humor flow rate.

135 w facility (C) and fluorophotometric aqueous humor flow rates (F) were measured in nine normal monkey

136 regulation of outflow resistance of aqueous humor flow through the trabecular meshwork (TM).

137 observations support the posit that aqueous humor flow, which is a factor that contributes to the im

138 lar meshwork (TM) cells might detect aqueous humor fluid shear stress via interaction of the extracel

139 eye, particularly in the drainage of aqueous humor fluid, which are believed to bring about changes i

140 ficantly reduced when incubated with aqueous humor for 30 minutes (P </= 0.0001).

141 DEX was detected in the retina and vitreous humor for 6 months, with peak concentrations during the

142 ration and neutralization of VEGF in aqueous humor for 8-12 weeks.

143 rstanding the mechanisms controlling aqueous humor formation and thereby intraocular pressure.

144 functions, such as sperm activation, aqueous humor formation, and metabolic regulation.

145 ithelium and is oriented to subserve aqueous humor formation.

146 Analysis of aqueous humor from patients with primary open-angle glaucoma (PO

147 ork (TM) tissue controls drainage of aqueous humor from the anterior chamber of the eye primarily by

148 intain fluid homeostasis by draining aqueous humor from the eye into the systemic circulation.

149 e in the eye that functions to drain aqueous humor from the intraocular chamber into systemic circula

150 tify the effect of dynamic motion of aqueous humor from the posterior to the anterior chamber, and (3

151 Human aqueous humor (hAH) provides nutrition and immunity within the a

152 This study demonstrates that aqueous humor has a potent host defense capability and that S. a

153 nvolved in the detection and appreciation of humor in childhood.

154 levels are increased in aqueous and vitreous humor in patients with glaucoma and animal models of gla

155 eshly secreted aqueous humor and the aqueous humor in the anterior chamber angle are relatively deple

156 in the primary drainage pathway for aqueous humor in the eye is responsible for ocular hypertension,

157 The principal outflow pathway for aqueous humor in the human eye is through the trabecular meshwor

158 udy to date has used neuroimaging to examine humor in typically developing children.

159 to migrate from the plasma into the aqueous humor, increasing intraocular pressure.

160 A single injection of FIV(HuMOR) into the temporomandibular joints of Col1-IL-1bet

161 n many areas of nonliteral language, such as humor, irony, and teasing.

162 Humor is a vital component of human well-being.

163 heir apical surface, in contact with aqueous humor is hexagonal, whereas their basal surface is irreg

164 Vectorial flow of zebrafish aqueous humor is in contrast to that in mammals in which secreti

165 owth factor-beta2 (TGF-beta2) in the aqueous humor is the main cause of fibrosis of TM in POAG patien

166 ncentrations in intraocular tissues (aqueous humor, lens, and iris) versus eyes not receiving Y-27632

167 ts with AMD had significantly higher aqueous humor levels of cadmium (median: 0.70 micromol/L, IQR: 0

168 ificant differences were observed in aqueous humor levels of manganese and selenium between patients

169 MMC to the endothelium and into the aqueous humor may lead surgeons to reassess appropriate dosing a

170 When cultured in media containing aqueous humor, MYOC-associated exosomes increased 514% over cont

171 underwent aqueous humor (n = 5) or vitreous humor (n = 2) analysis and cerebrospinal fluid analysis

172 ssified uveitis, 7 of whom underwent aqueous humor (n = 5) or vitreous humor (n = 2) analysis and cer

173 respiratory samples, biopsies, and vitreous humor) obtained longitudinally or from different anatomi

174 nic acid (KA) was injected into the vitreous humor of B6.Cg-Tg(Thy1-YFP)HJrs/J mice.

175 Here, we provide evidence in both vitreous humor of diabetic patients and in retina of a murine mod

176 g 16 clinical samples collected from aqueous humor of patients undergoing therapeutic anterior chambe

177 e the cytokine concentrations in the aqueous humor of patients with acute nonarteritic anterior ische

178 erations of trace elements levels in aqueous humor of patients with non-exsudative (dry) AMD.

179 ch is found in higher amounts in the aqueous humor of patients with POAG.

180 free metalloproteinases (MMP) in the aqueous humor of patients with primary open angle glaucoma (POAG

181 , lactacystin was injected into the vitreous humor of the developing chicken eye.

182 ological media such as blood or the vitreous humor of the eye in situ.

183 ed with the collagen network of the vitreous humor of the eye.

184 that the authors discovered in the vitreous humor of the eye.

185 boxyfluorescein elimination from the aqueous humor of the perfused eye was reduced 80% when novobioci

186 we assessed the cytokine pattern in aqueous humors of 10 affected patients.

187 that direct the movement of fluid (vitreous humor or cerebrospinal fluid) into and under the retina.

188 been proposed to underlie increased aqueous humor outflow (AHO) resistance, which leads to elevated

189 acterized by increased resistance to aqueous humor outflow and a stiffer human trabecular meshwork (H

190 sruptor that decreases resistance to aqueous humor outflow and decreases intraocular pressure.

191 e eye, plays a key role in promoting aqueous humor outflow and maintenance of normal intraocular pres

192 that offer the potential to improve aqueous humor outflow and protect RGCs simultaneously, and prese

193 th a reduction in available area for aqueous humor outflow and the confinement of outflow to the vici

194 Presumably as a consequence of aqueous humor outflow blockage, they rapidly developed multiple

195 cked both PEA-induced enhancement of aqueous humor outflow facility and PEA-induced phosphorylation o

196 the administration of AEA increases aqueous humor outflow facility and that this effect of AEA invol

197 mouse (110%, P < 0.001) and reduced aqueous humor outflow facility in the mouse.

198 30, or 300 nM of noladin ether, the aqueous humor outflow facility increased concentration dependent

199 Aqueous humor outflow facility measured with electronic Schiotz

200 The effects of noladin ether on aqueous humor outflow facility were measured in a porcine anteri

201 was to investigate the variation of aqueous humor outflow facility with body position changes.

202 gnificantly reduced IOP and improved aqueous humor outflow facility, which was sustained for at least

203 ar meshwork (TM) cells increases the aqueous humor outflow facility.

204 (4) receptor stimulation facilitated aqueous humor outflow facility.

205 EA caused a transient enhancement of aqueous humor outflow facility.

206 llular matrix turnover and increases aqueous humor outflow facility.

207 tive for the CB1 receptor, increases aqueous humor outflow facility.

208 to measure the effects of JWH015 on aqueous humor outflow facility.

209 icacious in both, increasing ex vivo aqueous humor outflow in porcine eyes and inhibiting myosin ligh

210 sure (IOP) caused by a resistance to aqueous humor outflow in the trabecular meshwork (TM).

211 or alterations in morphology of the aqueous humor outflow pathway were observed after treatment with

212 able targets within the conventional aqueous humor outflow pathway, which is thought to be regulated/

213 ich caused sclerosis and blockade of aqueous humor outflow pathways.

214 nization is required to maintain the aqueous humor outflow resistance and intraocular pressure homeos

215 cular meshwork (TM) provides most of aqueous humor outflow resistance in the eye, an in vitro bioengi

216 ersican's potential contributions to aqueous humor outflow resistance, its segmental distribution in

217 mediated IOP elevation and increased aqueous humor outflow resistance.

218 he TM tissue and consequently impede aqueous humor outflow resulting in elevated intraocular pressure

219 PCG, defects in the anterior chamber aqueous humor outflow structures of the eye result in elevated i

220 e performed 3D SD-OCT imaging of the aqueous humor outflow structures with 2 devices: The Cirrus HD-O

221 caused by developmental defects in 2 aqueous humor outflow structures, Schlemm's canal (SC) and the t

222 sults demonstrate that PEA increases aqueous humor outflow through the TM pathway and these effects a

223 ssure due to increased resistance of aqueous humor outflow through the trabecular meshwork (TM).

224 The effects of GGTI-DU40 on aqueous humor outflow were determined using organ-cultured, perf

225 The effects of AEA on aqueous humor outflow were measured using a porcine anterior seg

226 he aqueous outflow pathway increases aqueous humor outflow, possibly through altered cell adhesive in

227 nd pressure-dependent (conventional) aqueous humor outflow.

228 TM) cell volume increase the rate of aqueous humor outflow.

229 thereby modulates the resistance to aqueous humor outflow.

230 e changes and changes in the rate of aqueous humor outflow; agents that decrease trabecular meshwork

231 cells may also provide resistance to aqueous humor outflow; therefore, this study tests the involveme

232 Ranibizumab concentration in aqueous humor peaked the first day after injection (range, 36.9-

233 To study the value and safety of aqueous humor polymerase chain reaction (PCR) analysis for Herpe

234 nics drive angle opening rather than aqueous humor pressurization.

235 In addition, FIV(HuMOR) prevented the attendant sensitization of trigemin

236 ial link in studying the neurodevelopment of humor processing across the lifespan, our findings contr

237 bilateral TOPJ activation may be specific to humor processing and not part of a general constellation

238 onventional outflow facility (C(t)), aqueous humor production (F(a)), and anterior chamber volume (V(

239 mouse eye results in a reduction in aqueous humor production and complete loss of the vitreous chamb

240 stent with a major role of alpha2 in aqueous humor production and suggests that, potentially, alpha2-

241 However, aqueous humor production in the same mice appears to be normal b

242 onkeys appeared to have no effect on aqueous humor production or tonographic outflow facility and may

243 ibit the Na,K-ATPase, hence reducing aqueous humor production.

244 onic anhydrase (CA) enzyme to reduce aqueous humor production.

245 the enclosed spaces are filled with aqueous humor rather than circulating blood.

246 Addition of human aqueous humor rather than FBS to trabecular monolayer cell cultu

247 luorescence or ELISA in homogenized vitreous humor, reflecting the greater spatial resolution of in s

248 Males and females share an extensive humor-response strategy as indicated by recruitment of s

249 s study and pazopanib levels in the vitreous humor, retina, and choroid-RPE at the end of the study w

250 es were developed and tested in the vitreous humor, RPE cell homogenates and intact RPE cells.

251 An aqueous humor sample was obtained during cataract surgery betwee

252 After RLB, aqueous humor samples harvested from nontreated eyes but not fro

253 t protein 1 (MCP-1) was reported in vitreous humor samples of patients with RD and diabetic retinopat

254 ines, and correlate levels with IOP, aqueous humor samples were analyzed from 23 eyes with open angle

255 For this pilot study, aqueous humor samples were collected from patients undergoing ca

256 Aqueous humor samples were obtained in 10 patients with acute NA

257 Aqueous humor samples were taken at the time of IVB pretreatment

258 echanisms: CA inhibition to decrease aqueous humor secretion (reduce inflow) and NO release to increa

259 lar anterior segment responsible for aqueous humor secretion and absorption have been well characteri

260 d to identify and study the sites of aqueous humor secretion and absorption in adult zebrafish eyes.

261 is controlled by the balance between aqueous humor secretion from the ciliary body (CB) and its drain

262 Zebrafish eyes show aqueous humor secretion primarily from the dorsal ciliary region

263 l culture from blood, saliva, urine, aqueous humor, semen, and breast milk from infected or convalesc

264 or novobiocin (0.1 mM) added to the aqueous humor side of the tissue, or MK571 (5-(3-(2-(7-chloroqui

265 ion detected parasite DNA in 8/60 OT aqueous humor specimens but failed to identify Type II strain al

266 A total of 132 aqueous humor specimens were collected before intravitreal aflib

267 A total of 859 aqueous humor specimens were taken before each intravitreal rani

268 Aqueous humor specimens were taken before each intravitreal rani

269 , IL-1beta released by PMNs into the aqueous humor stimulates FGF-2 synthesis in corneal endothelium

270 and are now being investigated as a vitreous humor substitute.

271 alcium ionophore, and a component of aqueous humor, suggesting that TM cells respond to environmental

272 olimbic activation in children's response to humor, suggesting these regions may form a humor-essenti

273 Aqueous humor supplementation may maintain cultured trabecular c

274 appears to form a complex in porcine aqueous humor that enables it to bind specifically within the tr

275 was diminished in cells treated with aqueous humor that was first passed through a 3-kDa or a 30-kDa,

276 ntraocular environment (aqueous and vitreous humors), the capsular tissue, and the intraocular lens (

277 ian eye is responsible for secreting aqueous humor to maintain intraocular pressure, which is elevate

278 e Ussing chambers was greater in the aqueous humor-to-blood direction than in the blood-to-aqueous hu

279 Aqueous humor turnover was enhanced more than twofold in Best2(-

280 oscleral outflow (F(u)), and rate of aqueous humor turnover, were calculated from measured data.

281 Mean aqueous humor VEGF concentrations before treatment initiation we

282 incubated in DMEM containing porcine aqueous humor versus 13% +/- 15% when preincubated with DMEM alo

283 a recognition and quantification of vitreous humor (VH) cystine as well as provide the portability fo

284 90DeltatarO were cultured in bovine vitreous humor (VH) in vitro or inoculated into the vitreous cham

285 g velocity of the suspension in the vitreous humor was 3 cm/h.

286 mized versus vitrectomized eyes for vitreous humor was 791 ng/mL (day 22) versus 731 ng/mL (day 22),

287 pressure and the level of Hmgb1 in vitreous humor was analyzed 24 hours after reperfusion.

288 g, and IL-1beta concentration in the aqueous humor was elevated in a time-dependent manner after free

289 nd caspase-3 ELISA; ascorbate in the aqueous humor was evaluated by nuclear magnetic resonance spectr

290 eaction (PCR) testing of aqueous or vitreous humor was positive for herpes simplex virus (HSV) or var

291 n the iridocorneal angle tissues and aqueous humor was studied by immunohistochemistry and Western bl

292 Aqueous humor was tested whole or fractionated by size exclusion

293 0) and 47,200 ng . d/g, and for the vitreous humor were 213 ng/mL (day 60) and 11,300 ng . d/mL, resp

294 acility and total TGFbeta2 levels in aqueous humor were measured.

295 average, VEGF concentrations in the aqueous humor were suppressed below the lower limit of quantific

296 ibited 3.5-fold lower solubility in vitreous humor, when compared with its sodium salt.

297 nd meniscal tissue were also infected by FIV(HuMOR), which presumably exerted an antiinflammatory eff

298 ial (CE) cells and is present in the aqueous humor, which bathes CE cells in vivo.

299 easured in the central plane of the vitreous humor with an SLO.

300 jection of fluorescein targeted the vitreous humor with no significant selectivity for posterior vers