ライフサイエンスコーパス: hunger

1 amic sites to drive eating in the absence of hunger.

2 the impacts of the price increases on global hunger.

3 nctional exponent of ARC(AgRP) neuron-driven hunger.

4 re downstream of the metabolic regulation of hunger.

5 , buy their medications, and alleviate their hunger.

6 la melanogaster to identify neurons encoding hunger.

7 likely play a more general role in encoding hunger.

8 sitive to shifts in sucrose concentration or hunger.

9 tivated, they rapidly (within minutes) drive hunger.

10 dictive cues elicit eating in the absence of hunger.

11 mplicit bargain to ensure each other against hunger.

12 egulated in an opposite fashion and decrease hunger.

13 e hypothalamus are crucial to the control of hunger.

14 hich can be modified by internal states like hunger.

15 regulating hormones to transiently suppress hunger.

16 ective measures of whole-day food intake and hunger.

17 and could represent a physiologic signal for hunger.

18 ly interrupt progress toward a world without hunger.

19 reward-driven snack intake in the absence of hunger.

20 for reverse-engineering neural circuits for hunger.

21 g are affected by hypothalamic regulators of hunger.

22 i syndrome, a condition involving insatiable hunger.

23 ods after a standard meal designed to reduce hunger.

24 to drive food consumption during periods of hunger.

25 olutions in mice, independently of thirst or hunger.

26 sing neurons in the arcuate nucleus regulate hunger.

27 s sense the fed or fasted state and regulate hunger.

28 al work to induce a drive-like state such as hunger?

29 general discomfort (18% vs. 42%; P = 0.010), hunger (44% vs. 67%; P = 0.024), and weakness (22% vs. 4

30 , which aim to eradicate extreme poverty and hunger, achieve universal primary education, and promote

31 ts with high RMRs showed increased levels of hunger across the day (P < 0.0001) and greater food inta

32 these afferent neurons capable of triggering hunger advances understanding of how this intense motiva

33 al ingestion, and the return of appetite and hunger after fasting.

34 cents consumed more energy and reported less hunger after the large-array meal than after the standar

35 When infants signaled hunger again, they were fed a second meal of CMF.

36 cantly increased gastric volumes and reduced hunger (all P < 0.001).

37 2030 for a baseline scenario that would meet hunger alleviation targets for over 9 billion people.

38 We determined that hunger alone, induced by a ghrelin agonist, reduces AD p

39 Hunger also modulates behaviors related to food seeking

40 nce of CR, and the neuroendocrine aspects of hunger also prevent age-related cognitive decline.

41 Assistance Program (SNAP) aims to alleviate hunger among its beneficiaries by providing benefits to

42 standard meal may not ensure the absence of hunger among participants of all weight strata.

43 s of cognitive restraint, disinhibition, and hunger and 1-y changes in these eating behaviors predict

44 s of sleep restriction on the CNS control of hunger and appetite may have important implications for

45 HGI postexercise meal suppresses feelings of hunger and augments postprandial fullness sensations mor

46 ings, fructose versus glucose led to greater hunger and desire for food and a greater willingness to

47 After each block, participants rated hunger and desire for food.

48 r eating rate (P = 0.020), with postprandial hunger and desire to eat being lower (P = 0.019 and P<0.

49 lower energy expenditure coupled with higher hunger and disinhibition and/or delayed satiation compar

50 , and mandible opening were affected by both hunger and ethanol intoxication.

51 Participants rated their hunger and fullness before and after meals.

52 pite the decreased energy intake, ratings of hunger and fullness did not significantly differ across

53 d to monitor gastrointestinal complaints and hunger and fullness scores.

54 s (bitter, sweet, and umami) on food intake, hunger and fullness, gastrointestinal symptoms, and gast

55 intake, but the high-egg group reported less hunger and greater satiety postbreakfast.

56 Lower hunger and higher fullness ratings were seen premeal and

57 te to improved eating behavior (e.g. reduced hunger and improved satiety) following these bariatric p

58 tive therapy for obesity and rapidly reduces hunger and improves satiety through unknown mechanisms.

59 Moreover, DB decreases hunger and increases satiety ratings after a meal and sh

60 tinal physiology that contribute to reducing hunger and increasing sensations of satiation.

61 food can stimulate feeding in the absence of hunger and lead to maladaptive overeating behavior.

62 iquid preloads elicited greater postprandial hunger and lower fullness sensations, more rapid gastric

63 ny comprehensive strategy to tackle poverty, hunger and malnutrition in developing countries and thus

64 aste and smell are believed to be anhedonic, hunger and pain are muted, and body-image distortion obs

65 ould harness the power of business to reduce hunger and poverty for millions of families, contribute

66 orestation and land degradation overlap with hunger and poverty.

67 r 1a (GHSR1a), in the hypothalamus to signal hunger and promote food intake.

68 Hunger and prospective consumption dose-dependently decr

69 nvestigated brain response to rewards during hunger and satiated states to examine whether diminished

70 No differences were observed in hunger and satiation curves between morbidly obese and n

71 loss, serum obesity-related hormone levels, hunger and satiety assessments, and quality of life (QOL

72 Instead, hunger and satiety homeostatically modulate the microstr

73 47 kg (+/-SD) weight gain despite changes in hunger and satiety hormones ghrelin and leptin, and pept

74 egulating hormones and subjective ratings of hunger and satiety in 10 healthy volunteers.

75 Many molecular signals that represent hunger and satiety in the body have been identified, but

76 gnitive neurocircuitry did not differ during hunger and satiety in the RAN group.

77 , motilin and ghrelin plasma concentrations, hunger and satiety ratings, and food intake in healthy v

78 Accordingly, hunger and satiety regulate adult neurogenesis by modula

79 imilar energy intake, the SD group had lower hunger and satiety scores compared with the bvFTD group.

80 In contrast, hunger and satiety scores did not differ between the bvF

81 Subjective ratings of hunger and satiety were evaluated by visual analog scale

82 ir interaction with regulatory mechanisms of hunger and satiety, relevant to clinical issues.

83 Key factors driving eating behavior are hunger and satiety, which are controlled by a complex in

84 All participants completed surveys on hunger and satiety.

85 pha-MSH, provides insight into regulation of hunger and satiety.

86 ied by intrinsic and extrinsic cues, such as hunger and sensory stimuli.

87 timing did not affect rhythms of subjective hunger and sleepiness, master clock markers (plasma mela

88 Hunger and snack food intake were reduced only after a l

89 lly stimulated, they potently induce intense hunger and subsequent food intake.

90 The patients had a sustainable reduction in hunger and substantial weight loss (51.0 kg after 42 wee

91 vagal tone (heart rate variability (HRV)) on hunger and the postprandial response to GL.

92 ompared with the sham group, VAS ratings for hunger and the urge to eat declined significantly more (

93 Hunger and thirst are ancient homeostatic drives for foo

94 tly being uncovered, less is known about how hunger and thirst interact.

95 ar abundance signals and oppositely regulate hunger and thirst.

96 exture of food in the mouth independently of hunger and thus of reward value and pleasantness.

97 insecurity in areas currently vulnerable to hunger and undernutrition.

98 satiety, while the ghrelin receptor promotes hunger and weight gain.

99 rol of appetitive behavior by interoceptive "hunger" and "satiety" signals.

100 r hearing; people's bodily sensations (e.g., hunger); and control stories without people.

101 n substrate utilization, energy expenditure, hunger, and 24-hour metabolic hormone concentrations.

102 adaptation to caloric restriction, increased hunger, and a shift in relative substrate utilization to

103 ved in feeding behavior, glucose metabolism, hunger, and appetite.

104 eptions of dietary restraint, disinhibition, hunger, and control of eating.

105 e major circulating metabolic fuels regulate hunger, and each is affected by dietary composition.

106 tor urges, intrusive thoughts, sensations of hunger, and preoccupation with body shape and weight to

107 ed forebrain areas known to mediate feeding, hunger, and satiation while minimally affecting brainste

108 -GI meal decreased plasma glucose, increased hunger, and selectively stimulated brain regions associa

109 king energy state to the motivational drive, hunger, and, finally, limbic and cognitive processes tha

110 Hunger/appetite scores decreased in the first 2 weeks af

111 sible mechanism for how various responses to hunger are coordinated.

112 mechanism(s) underlying voluntary control of hunger are not well understood.

113 The neural mechanisms underlying hunger are poorly understood.

114 mines how the energy deficits that result in hunger are represented in the brain and promote feeding

115 ttenuates the rewarding effects of food- and hunger-associated brain stimulation reward.

116 an independent role of oleoylethanolamide in hunger-associated interoceptive signaling.

117 orted greater decreases in disinhibition and hunger at 1- and 10-y follow-ups (all P < 0.001 in both

118 significant relation between eating rate and hunger at the end of the meal or up to 3.5 h later.

119 inated impulsive action in DRL engendered by hunger, at a dose (1 mug) that significantly affected ne

120 8 min, respectively; P < 0.05), although the hunger AUC was not statistically different.

121 wer levels of 6-mo and 1-y disinhibition and hunger (beta = 0.13-0.29, P < 0.01 in men; beta = 0.11-0

122 obesity agents by lowering the set-point for hunger between meals.

123 nts, intake that is not only associated with hunger but also the incentive value of food.

124 is not affected by sucrose concentration or hunger but is altered by fluid viscosity.

125 BG was related to hunger but only in low disinhibited eaters.

126 combination of tastants inhibits feelings of hunger, but only the latter also reduces food intake.

127 circuitry to make foods more 'liked' during hunger by acting through VP.

128 food preference driven by nutrient-specific hunger can be essential for survival, yet little is know

129 In addition, hunger can promote the expression of food-associated mem

130 The consumption of breakfast reduced daily hunger compared with BS with no differences between meal

131 Greater hunger correlated with higher appeal ratings of "fatteni

132 regulated behavior that integrates taste and hunger cues to balance food intake with metabolic needs.

133 tion in mice with caloric-deficiency-induced hunger decreases feeding.

134 tain a cellular-level understanding of these hunger-dependent cortical response biases, we performed

135 Poverty, drought, and hunger devastate people on Africa's rangelands.

136 ns, typically via food shortages, consequent hunger, disease, and unrest.

137 ly activated by starvation and evoke intense hunger-display electrical and biochemical hyperactivity

138 sting these cells may supply the fundamental hunger drive that motivates feeding.

139 Hunger, driven by negative energy balance, elicits the s

140 A microanalysis of hunger-driven and palatability-driven feeding was carrie

141 ssion of SLC5A11 is sufficient for promoting hunger-driven behaviors and enhancing the excitability o

142 ssion in SLC5A11-expressing neurons produces hunger-driven behaviors even in fed flies, mimicking the

143 neurons is sufficient to promote feeding and hunger-driven behaviors; silencing these neurons has the

144 another OA receptor previously implicated in hunger-driven exuberant sugar intake.

145 Energy expenditure and hunger-driven food intake as well as olfactory function

146 eurons in the subesophageal ganglion blocked hunger-driven increases in the feeding response.

147 cultural expectations, psychological stress, hunger, dyspepsia, micronutrient deficiencies (Fe, Zn, a

148 height and weight, eating in the absence of hunger (EAH) at 5 y, and inhibitory control (a measure o

149 ories and increased eating in the absence of hunger (EAH) has been associated with increased energy i

150 Eating in the absence of hunger (EAH) is typically assessed by measuring youths'

151 Hunger elevates neural and behavioral responses to food

152 Hunger enhances sensitivity to reward, yet individuals w

153 an be modulated by behavioral states such as hunger, fear, stress, or a change in environmental conte

154 ulating eating behavior as well as decreased hunger feelings and diminished incentive value of food.

155 olved in this association between subjective hunger feelings and gastrointestinal motility during the

156 tilin-induced phase III contractions induced hunger feelings through a cholinergic pathway.

157 ically cause hypertension, but it promotes a hunger for salt and a transition from salt resistance to

158 sential prerequisites for ending poverty and hunger, for improving institutions and participation in

159 After implementation of the Healthy Hunger-Free Kids Act, change was associated with signifi

160 5 months after implementation of the Healthy Hunger-Free Kids Act.

161 dless of whether youth eat in the absence of hunger from a very large-array meal or from a standardiz

162 Hunger, fullness, and desire to eat (all P > 0.62) simil

163 s in change for BMI, household wealth index, hunger, general health perception score, or adherence to

164 In contrast, during hunger, gustatory inputs enter the hypothalamus and driv

165 manipulating central receptors for the gut "hunger" hormone ghrelin.

166 ir similarity for the meal-induced change in hunger (ICC: 0.41; P = 0.03), fullness (ICC: 0.39; P = 0

167 Behavioral tests demonstrated that perceived hunger, importance of eating, eating frequencies, and li

168 ritional value hold the promise to alleviate hunger in Africa.

169 pamine circuit that encodes protein-specific hunger in Drosophila The activity of these neurons incre

170 ional processes that are thought to underlie hunger in order to outline considerations for bridging t

171 ent progress into neural circuit analysis of hunger in the mouse by focusing on a starvation-sensitiv

172 t is not known why certain people experience hunger in the presence of large amounts of body energy.

173 ional regulation of key neurons that control hunger in vivo.

174 ood and implicates lower ability to suppress hunger in women as a contributing factor to gender diffe

175 re insufficient to produce the effect, and a hunger-induced feeding drive is insufficient to reveal t

176 (1 mug) that significantly affected neither hunger-induced PR enhancement nor hyperactivity.

177 show that a metabolic challenge by cold and hunger induces diurnality in otherwise nocturnal mice.

178 Thus, a hunger-inducing drug attenuates AD pathology, in the abs

179 Hunger is a complex behavioural state that elicits inten

180 Hunger is a complex motivational state that drives multi

181 Hunger is a hard-wired motivational state essential for

182 Hunger is a powerful drive that stimulates food intake.

183 The sensation of hunger is caused by an imbalance between energy intake a

184 Hunger is controlled by specialized neural circuits that

185 One immediate response to hunger is increased food consumption.

186 value in that they respond to food only when hunger is present and in that activations correlate line

187 Although progress is being made, how hunger is represented in the brain and how it coordinate

188 o have two core components-experience (e.g., hunger, joy) and agency (e.g., planning, self-control).

189 fullness levels were significantly lower and hunger levels were significantly higher after 25 chews t

190 mals also increased dry licking according to hunger levels, and developed conditioned preferences for

191 , these pathways are recruited at increasing hunger levels, such that low-risk changes (higher sugar

192 de (AgRP)) bypasses these signals to restore hunger-like response patterns in insular cortex.

193 OMC) neurons are extremely slow in affecting hunger (many hours).

194 sed sensitivity to the motivational drive of hunger may explain the ability of individuals with AN to

195 bic and cognitive processes that bring about hunger-mediated increases in reward value and perception

196 ty of the sGPNs in the AMMC, suggesting that hunger modulates the responsiveness of the secondary swe

197 Root et al. report that hunger modulates the sensitivity of specific olfactory s

198 teracts with visual processing, by examining hunger modulation of food-associated visual responses in

199 These findings reveal that hunger neurons drive feeding by transmitting a positive

200 ex, chemogenetic activation of hypothalamic 'hunger neurons' (expressing agouti-related peptide (AgRP

201 not made adequate progress toward overcoming hunger or achieving sustainable livelihoods.

202 ess information in different states, such as hunger or arousal.

203 doses of ethanol to examine how a change in hunger or inebriation state affected the social behaviou

204 ce and was not explained simply by increased hunger or lower satiety.

205 othalamus that respond to hormones signaling hunger or satiety.

206 ays that modulate the physiological state of hunger or satiety.

207 of weight gain, increases in food intake and hunger, or psychiatric disease, suggesting that AAPs exe

208 Hunger (P = 0.001) and desire to eat (P = 0.001) ratings

209 intragastric administration of DB decreased hunger (P = 0.008) and increased satiety ratings (P = 0.

210 tion, BNP decreased the subjective rating of hunger (P = 0.009) and increased the feeling of satiety

211 ot differ significantly, yet fluctuations in hunger (P = 0.019) and desire to eat (P = 0.026) over th

212 ANOVA confirmed that subjectively rated hunger (P = 0.569; SED: 3.8), fullness (P = 0.404; SED:

213 signals originating inside the body, such as hunger, pain and heart rate.

214 the frequency and length of their strides if hunger pangs compete with their desire to see cherry blo

215 ions were also significantly associated with hunger peaks (P < 0.05), and this association involved a

216 phase III contractions on the occurrence of hunger peaks and the involvement of a cholinergic pathwa

217 e sought to 1) investigate the occurrence of hunger peaks and their relation to phase III contraction

218 The occurrence of hunger peaks and their significant association with phas

219 An algorithm was developed to detect hunger peaks.

220 eptin but was unaffected by other satiety or hunger peptides.

221 trient infusion rapidly and durably inhibits hunger-promoting AgRP neurons in awake, behaving mice.

222 gnaling hormones (e.g., higher levels of the hunger-promoting hormone ghrelin) or hedonic brain respo

223 e from the satiety hormone leptin toward the hunger-promoting hormone ghrelin.

224 sting GLP-1, fasting PYY, PYY responses, and hunger-rating responses to the test drink (P < 0.05).

225 ch to the duodenum (P = 0.001) and decreased hunger ratings (P = 0.04).

226 Despite reporting normal decreases in hunger ratings after being fed, persons with HD have an

227 istration decreases both antral motility and hunger ratings during the fasting state, possibly becaus

228 In the morning and evening, hunger ratings increased (both P = 0.02), whereas rating

229 ect on fasting gastrointestinal motility and hunger ratings, motilin and ghrelin plasma concentration

230 ence concurrent energy intake and subjective hunger ratings.

231 nsulin, glucagon, cortisol, epinephrine, and hunger ratings.

232 ds for quality of life, wealth accumulation, hunger reduction, and risk management.

233 nt, we analyzed the hierarchical position of hunger related to rival drive states.

234 The relation between hunger, satiation, and integrated gastrointestinal motil

235 assess the effects of sleeve gastrectomy on hunger, satiation, gastric and gallbladder motility, and

236 Fasting and postprandial hunger, satiation, hormone concentrations, and gastric a

237 Although physiological state (e.g., hunger, satiety) and associated hormones are known to af

238 Interactions of Group (RAN, CW) x Visit (hunger, satiety) revealed that, for the CW group, hunger

239 Hunger, satiety, and desire to eat were measured after c

240 mg pure capsaicin) in healthy volunteers on hunger, satiety, and gastrointestinal symptoms and the r

241 ual analog scale scores were used to measure hunger, satiety, and gastrointestinal symptoms.

242 re not observed to have different effects on hunger, satiety, lipid profiles, or other inflammatory a

243 sive behavioral testing assessing changes in hunger/satiety regulation were performed during the firs

244 Satiety, hunger, satisfaction with the diet, and attendance at gr

245 combination of tastants and umami decreased hunger scores compared with placebo.

246 strate oxidation, core temperature, cold and hunger scores, and plasma parameters were repeated every

247 strate oxidation, core temperature, cold and hunger scores, or plasma parameters were found, nor was

248 than after the LGI meal (P < 0.001), whereas hunger sensations were approximately 9% lower after the

249 utcome) and alter body temperature, cold and hunger sensations, plasma metabolic parameters, and ener

250 brain extends the role of a small number of hunger-sensing neurons to include food-odor value repres

251 Ghrelin is the only known hunger signal derived from the peripheral tissues.

252 were excited by food deprivation and the gut hunger signal ghrelin.

253 egans senses specific amino acids as an anti-hunger signal.

254 e III contractions have been identified as a hunger signal.

255 ) pro-opiomelanocortin (POMC)-expressing and hunger signaling (orexigenic) agouti-related peptide (Ag

256 Ghrelin, a hunger signalling peptide derived from the peripheral ti

257 because of a failure to accurately recognize hunger signals, whereas overeating in bulimia nervosa co

258 could represent an exaggerated perception of hunger signals.

259 sensory context (P-interaction < 0.05), with hunger significantly being lower after consumption of th

260 r, satiety) revealed that, for the CW group, hunger significantly increased activation in reward sali

261 battery of behavioral assays, we found that hunger significantly increases its capacity to suppress

262 t consumption of sucrose in a novel elevated-hunger state (a positive incentive learning opportunity)

263 s reveal a new neural circuit that regulates hunger state and pathways associated with overeating dis

264 ally precise ingestion that was regulated by hunger state and sucrose concentration.

265 stronger food cue biases and sensitivity to hunger state and trial history.

266 circuit that integrates gustatory input and hunger state to modulate food ingestion.

267 experiencing the sucrose in a novel reduced-hunger state.

268 ssing dKCNQ channels, thereby conferring the hunger state.

269 sm by which these neurons rapidly coordinate hunger states with glucose homeostasis.

270 es of foods promote and guide consumption in hunger states, whereas satiation should dampen the senso

271 n the insular cortex of behaving mice across hunger states.

272 Reductions in levels of the hunger-stimulating hormone ghrelin have been proposed to

273 Why choose suicide bombing rather than a hunger strike or self-immolation?

274 es is constrained by satiety and promoted by hunger, suggesting an influence of motivational state.

275 r arms had equal access to an Action Against Hunger-supported programme.

276 calories from snack foods in the absence of hunger than did children in a control group.

277 emotions ate more cookies in the absence of hunger than did children whose mothers used this feeding

278 4% more calories (93 kcal) in the absence of hunger than did normal-weight siblings (P = 0.01).

279 more sweet palatable foods in the absence of hunger than do children of mothers who use this feeding

280 nderstanding how motivated behaviors such as hunger, thirst and sexual behaviors arise.

281 inhibitor that not only could satisfy 'drug hunger' through its slow-onset long-lasting DAT inhibito

282 ations, reveal that the well-known effect of hunger to enhance behavioral sensitivity to sugar is med

283 he income disparities that permit widespread hunger to persist despite aggregate economic advancement

284 However, the molecular details of how hunger triggers changes in the activity of neural circui

285 impacts of higher food prices on poverty and hunger under this scenario would likely have dwarfed the

286 gression models, severe food insecurity with hunger was associated with a reduced odds of consistent

287 Severe food insecurity with hunger was associated with reduced odds of condom use an

288 5.3 +/- 0.16 mmol/L; P = 0.005) and reported hunger was greater (P = 0.04) 4 h after the high- than t

289 consumption of snack foods in the absence of hunger was measured.

290 Hunger was rated and blood samples were taken before eac

291 Hunger was rated before each measurement.

292 RMR, daily energy intake, meal size, and hunger were assessed within the same day and across each

293 Ratings of hunger were decreased during nightshift days despite dec

294 Blood glucose (BG) and hunger were measured 30 and 150 min after consumption of

295 haviors (i.e., eating even in the absence of hunger), which were predictive of having a high body mas

296 associated with decreases in self-reports of hunger, which corroborates the involvement of this regio

297 -/perinatal famine exposure during the Dutch Hunger Winter of 1944-1945 and mortality through age 63

298 eptional exposure to famine during the Dutch Hunger Winter.

299 improved gastrointestinal symptoms and less hunger with GMP-MFs.

300 including alertness, social recognition, and hunger, yet, their mechanism of action is poorly underst