ライフサイエンスコーパス: hyaline

1 brosis, ballooning degeneration, and Mallory hyaline).

2 ly wounded dogs did not form the superficial hyaline acellular lamina found in 92% of dogs with SCCED

3 A prominent superficial stromal hyaline acellular zone composed of collagen fibrils in t

4 All engineered grafts contained a mixed hyaline and fibrous cartilage matrix.

5 flammation, ballooning degeneration, Mallory hyaline, and fibrosis.

6 fibrosis, ballooned hepatocytes, and Mallory hyaline, and two patients had cryptogenic cirrhosis thou

7 colemmal accumulations of myosin that have a hyaline appearance.

8 hypercellularity of the visceral epithelium, hyaline arteriolosclerosis, and interstitial fibrosis we

9 ated by differential centrifugation of adult hyaline articular cartilage collagenase digests, mineral

10 Calcium crystals are universally present in hyaline articular cartilage, as well as the meniscus of

11 ; the highest specific activity was found in hyaline articular cartilage, the lowest in brain.

12 eleased from old and young human and porcine hyaline articular cartilage.

13 teral corner syndrome, and in evaluating the hyaline articular cartilage.

14 nd that they were phenotypically unique from hyaline articular chondrocytes isolated from the knee jo

15 s), hippocampal sclerosis, lacunar infarcts, hyaline atherosclerosis, siderocalcinosis, and Lewy body

16 osis with or without the presence of Mallory hyaline bodies.

17 using hereditary inclusion body myopathy and hyaline body myopathy and the identification of mutation

18 Both hyaline cartilage and fibrocartilage can calcify in a va

19 -bearing unossified epiphyseal and articular hyaline cartilage and for the distal femoral and proxima

20 the long alpha1(IX) transcript expressed in hyaline cartilage and matched the predicted sequence of

21 Three-dimensional hyaline cartilage can be engineered using BMSCs from pat

22 ar (82 [25%] vs 21 [19%], P = .25); isolated hyaline cartilage defect (77 [23%] vs 20 [18%], P = .29)

23 key signaling events required for postnatal hyaline cartilage formation.

24 T2 spatial variation of patellar hyaline cartilage in children is similar to that of pate

25 , pigmentation was widespread throughout the hyaline cartilage in either granular composition or as b

26 The graft consisted of hyaline cartilage in four, mixed fibrohyaline cartilage

27 cartilaginous condylar cartilage, similar to hyaline cartilage in long bones, directly transform into

28 disc and cartilage is different from that of hyaline cartilage in other diarthrodial joints, and litt

29 dely expressed, but high levels are found in hyaline cartilage in the adult, bone tissue in newborn m

30 infused bioscaffolds were fully covered with hyaline cartilage in the articular surface.

31 The prevalence of hyaline cartilage lesions was particularly high (86% at

32 maturation of articular chondrocytes in the hyaline cartilage of both developing and degenerated joi

33 ed to TMJ fibrocartilage and not seen in the hyaline cartilage of the knee.

34 intensity and in most cases attached to the hyaline cartilage of the lunate and scaphoid.

35 The trilaminar appearance depicted within hyaline cartilage on MR images obtained with this sequen

36 the hypertrophic differentiation of cultured hyaline cartilage particles.

37 over expression of type X collagen which, in hyaline cartilage structure is not characteristic of the

38 Once hyaline cartilage was extensively pigmented, there was a

39 cruciate), elastic cartilage, meniscus, and hyaline cartilage were analyzed for NTTPHase activity (t

40 jority of vertebrate species have a layer of hyaline cartilage within the fibrous sclera giving an ex

41 al type II, IX, and XI collagen phenotype of hyaline cartilage, but the fibrils appear abnormally thi

42 vitro under hypoxia (2.5% O2) produced more hyaline cartilage, which expressed typical articular car

43 ble of alleviating necrosis at the centre of hyaline cartilage.

44 gical development of these distinct types of hyaline cartilage.

45 rogenesis into either permanent or transient hyaline cartilage.

46 nt enabled the maintenance of functional and hyaline cartilage.

47 has a similar collagen phenotype to that of hyaline cartilage.

48 tion of type II collagen is observed in both hyaline cartilages and is secondary to proteoglycan loss

49 a constitutively active human MMP-13 to the hyaline cartilages and joints of transgenic mice.

50 The collagen framework of hyaline cartilages, including articular cartilage, consi

51 edge of the BP to ramify extensively in the hyaline cell area.

52 longitudinal network in the border cell and hyaline cell region.

53 like anomaly phenotype), and accumulation of hyaline cells in vitreous.

54 types of SCs, homogene cells, border cells, hyaline cells, ganglion cells, and connective tissue cel

55 ic margin, while others were granular with a hyaline core.

56 by its more severe phenotype, which includes hyaline deposits in multiple organs, recurrent infection

57 Muscle fibers with hyaline desmin-containing cytoplasmic inclusions in comb

58 bind to all three major types of cartilage (hyaline, elastic, and fibrocartilage) and perform equall

59 Juvenile hyaline fibromatosis (JHF) and infantile systemic hyalin

60 Juvenile hyaline fibromatosis (JHF) is an autosomal recessive con

61 ns linked to the genetic disorders, juvenile hyaline fibromatosis, and infantile systemic hyalinosis.

62 The identification of hyaline fibrous tissue, with numerous crystalline basoph

63 Of the 80 dematiaceous and 154 hyaline fungi sequenced, 65 and 51.2%, respectively, gav

64 ophilic protein isolated from characteristic hyaline gastric lesions was identified as Ym2, a member

65 logically, there is widespread deposition of hyaline (glycoprotein) material and disruption/reduplica

66 Histologic analysis revealed invasive hyaline hyphae and some darkly pigmented structures that

67 ctures of the joints, and an accumulation of hyaline in the dermis.

68 hological diagnosis of neuronal intranuclear hyaline inclusion disease may also have polyglutamine re

69 Rosenthal fibers (RF), intra-astrocytic hyaline inclusions, accumulate in various pathological c

70 rimentally wounded dogs is distinct from the hyaline lamina observed in dogs with SCCED.

71 brane involved the thickening of the central hyaline layer and a reduction in the epithelial cells on

72 s contributes to the ordered assembly of the hyaline layer and elevation of the fertilization envelop

73 y mAb183 to show that cell attachment to the hyaline layer is necessary for bottle cell formation and

74 The major constituent of the hyaline layer is the protein hyalin, a fibrillar glycopr

75 The hyaline layer of echinoderm embryos is an extraembryonic

76 n that is deposited to the inner side of the hyaline layer.

77 ote then becomes covered by a newly secreted hyaline layer.

78 or a fibronectin-like substrate, and for the hyaline layer.

79 the mechanisms of cell interactions with the hyaline layer.

80 Hyaline-like cartilage matrix production was observed in

81 myofibrillar disarray and degeneration with hyaline-like inclusions.

82 tivity in micromass culture, and generated a hyaline-like translucent cartilage particle in serum-fre

83 Ultrastructurally, the hyaline masses are composed of intermediate-density amor

84 The hyaline masses are intensely congophilic; react strongly

85 all vacuoles, and nemaline rods flanking the hyaline masses or congregated under the sarcolemma.

86 g in aggregation of the degraded residues in hyaline masses, and causes replication of Z disks, resul

87 titin, alpha-actinin, and slow myosin in the hyaline masses, signal nonlysosomal protein degradation.

88 e, irregularly polygonal, and mostly central hyaline masses, small vacuoles, and nemaline rods flanki

89 The hyaline material is positive with periodic acid-Schiff a

90 subpopulation of smaller cells that surround hyaline material.

91 f a residual mass, which revealed a shrunken hyaline matrix with preserved collagenous architecture.

92 HFOV had less pulmonary inflammation in the hyaline membrane disease (HMD) recovery phase.

93 use alveolar damage with pulmonary edema and hyaline membrane formation associated with accumulation

94 e clinical disease, diffuse alveolar damage, hyaline membrane formation, alveolitis, and death were n

95 tory cells, interstitial and alveolar edema, hyaline membrane formation, and ultimately fibrosis.

96 n pulmonary edema and acute lung injury with hyaline membrane formation, leading to decreased oxygena

97 aracterized by acute alveolar cell death and hyaline membrane formation, sustained up-regulation of h

98 characterized by diffuse alveolar damage and hyaline membrane formation.

99 The presence of hyaline membranes (HM) and airway fibrin (AF) was signif

100 turation, fibrosis, peripheral inflammation, hyaline membranes, and foamy alveolar macrophages, a phe

101 e in preterm infants, including atelectasis, hyaline membranes, and the lack of pulmonary surfactant

102 respiratory distress such as atelectasis and hyaline membranes.

103 aphs corresponded to intraalveolar edema and hyaline membranes.

104 results were categorized into those showing hyaline, mixed fibrohyaline cartilage, fibrocartilage, a

105 ies show ballooning degeneration and Mallory hyaline or fibrosis.

106 presence of hepatocyte ballooning, Mallory's hyaline, or fibrosis, which are important features in th

107 Microscopically, hyphae were hyaline, septate, and branched and remained totally devo

108 of biopsy tissue from lungs and brain showed hyaline, septate, branched hyphae with clamp connections

109 's methenamine silver stains showed numerous hyaline, septate, fungal hyphae of various lengths, many

110 The hyaline structures are strongly congophilic.

111 ghly electron-dense material, resembling the hyaline structures of myofibrillar myopathy.

112 arbouring amorphous, granular or pleomorphic hyaline structures, and vacuoles containing membranous m

113 ncluding lipid and proteinaceous substances (hyaline substances).

114 pathological signature with numerous round, hyaline, TAR DNA-binding protein 43 (TDP-43)-positive in

115 l fibrosis (CI), tubular atrophy, arteriolar hyaline thickening, fibrous intimal thickening (CV), and

116 hritis, highlighted by widespread glomerular hyaline thrombi, being common among NZM.Baff(-/-) mice b

117 The fundamental pathologic lesion is a hyaline thrombus composed of platelets and some fibrin a

118 foci in the bone marrow and soft tissues and hyaline tubular casts in the kidneys.

119 tric vs multicentric), histopathologic type (hyaline vascular [HV] vs plasma cell [PC]), anatomical l

120 Three histologic variants (hyaline vascular, plasma-cell, and mixed) and two clinic

121 her subdivided according to whether they had hyaline vascular, plasma-cell, or mixed disease.

122 Castleman disease (n = 24) typically had the hyaline-vascular type (n = 23), were asymptomatic (n = 1