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1 hyaline fibromatosis, and infantile systemic hyalinosis.
2 t damage index parameters and for arteriolar hyalinosis.
3 rteriopathy (TA, n=233, 56%); (2) arteriolar hyalinosis (AH, n=89, 21%); and (3) no characteristic va
4 phrotoxic serum showed persistent glomerular hyalinosis and albuminuria 96 hours after injection, whe
5 significant relationship between arteriolar hyalinosis and global glomerulosclerosis (r=0.61, P<0.00
6 s a progressive increase in renal arteriolar hyalinosis and in the percentage of glomeruli demonstrat
9 l matrix increase, 32.0% abnormal arteriolar hyalinosis, and 32.9% abnormal vascular intimal thickeni
10 h, glomerulosclerosis, afferent and efferent hyalinosis, and tubulointerstitial inflammation and fibr
11 esangiolysis, microaneurysms, and arteriolar hyalinosis associated with massive proteinuria and renal
15 hese aging mice, we found high incidences of hyalinosis (eosinophilic cytoplasmic change) in the glan
18 e exhibited dramatic albuminuria, arteriolar hyalinosis, increased glomerular basement membrane thick
19 ne fibromatosis (JHF) and infantile systemic hyalinosis (ISH) are autosomal recessive syndromes of un
20 ria, and renal insufficiency with arteriolar hyalinosis, mesangial matrix expansion, mesangiolysis wi
21 odular glomerulosclerosis), focal arteriolar hyalinosis, mesangiolysis, and focal mild interstitial f
23 s to a decreased glomerular filtration rate, hyalinosis of afferent arterioles, and striped cortical
25 developed mild hyperuricemia with arteriolar hyalinosis, tubular injury and striped interstitial fibr
27 , and 10-year prevalence moderate arteriolar hyalinosis was similar: CSA was 5.4%, 38.4%, and 79.1%;
28 acrolimus nephrotoxicity (nodular arteriolar hyalinosis) was found in 21% (15 of 69) of biopsies in 3
29 ar atrophy, arteriosclerosis, and arteriolar hyalinosis were graded from 0 to 3+, using definitions s
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