ライフサイエンスコーパス: hybrid sterility

1 hanisms (for example, hybrid inviability and hybrid sterility).

2 e complex is complicated by complete F1 male hybrid sterility.

3 s not seem by itself to contribute to equine hybrid sterility.

4 of a hybrid incompatibility underlying F(1) hybrid sterility.

5 terochromatin evolution affects the onset of hybrid sterility.

6 rtant mechanism contributing to two types of hybrid sterility.

7 t of the X chromosome in genetic analyses of hybrid sterility.

8 roclivity of certain genes to be involved in hybrid sterility.

9 of interacting genes that contribute to F(1) hybrid sterility.

10 mity to QTL for morphological differences or hybrid sterility.

11 of the interacting genes that contribute to hybrid sterility.

12 autosomes do not contribute markedly to male hybrid sterility.

13 as a disproportionately large effect on male hybrid sterility.

14 erent ploidy levels is often associated with hybrid sterility.

15 d a very strong sex bias in the evolution of hybrid sterility.

16 se chromosome 17 in the t complex within the Hybrid sterility 1 (Hst1) region.

17 segregation distorter gene is essential for hybrid sterility, a strong reproductive barrier between

18 species' genomes is not a major cause of F1 hybrid sterility, although it may contribute to reproduc

19 F1 hybrid sterility and "hybrid breakdown" of F2 and later

20 We have examined the genetic basis of hybrid sterility and female species preferences in Droso

21 ge map and F4 progeny testing to investigate hybrid sterility and hybrid breakdown in a cross between

22 ts implicate different genetic mechanisms in hybrid sterility and hybrid breakdown, respectively.

23 ute to the observed variation in patterns of hybrid sterility and in rates of polyploidization.

24 Hybrid sterility and inviability have been unimportant i

25 s of the postzygotic isolating mechanisms of hybrid sterility and inviability, little is known about

26 s of the postzygotic isolating mechanisms of hybrid sterility and inviability, little is known about

27 tion were integrated with genetic studies of hybrid sterility and inviability.

28 r different possibility-the genes that cause hybrid sterility and lethality often come to differ betw

29 the evolution and genetics of interspecific hybrid sterility and lethality were once also thought to

30 We dissected the basis of this hybrid sterility and reached four main conclusions.

31 The genetic bases of hybrid sterility and segregation distortion are at least

32 heles gambiae and An. arabiensis suffer from hybrid sterility, and inviability effects are sometimes

33 Hybrid sterility appeared to be due to recombination wit

34 polymorphism at the Odysseus (OdsH) locus of hybrid sterility between Drosophila mauritiana and Droso

35 Incomplete hybrid sterility between the two species generates selec

36 article we investigate the genetic basis of hybrid sterility between two closely related species of

37 Lee et al. now report that hybrid sterility between two yeast species is caused by

38 We also evaluated the role of Prdm9 in hybrid sterility by assessing allelic differences of ZF

39 The genetic basis of hybrid sterility can provide insight into the genetic an

40 However, the genetics of hybrid sterility differ between D. yakuba and D. santome

41 Fourth, hybrid sterility does not involve a maternal effect, des

42 w resolution studies suggested that a single hybrid sterility factor was associated with this region.

43 articularly for loci not tightly linked to a hybrid sterility gene, may have erased the original patt

44 e the phylogeny inferred from the density of hybrid sterility genes with that inferred from molecular

45 verse species to be especially dense in male hybrid sterility genes.

46 r aspects of reproductive isolation (such as hybrid sterility) have become evident.

47 The underlying mechanisms causing hybrid sterility, however, are less well known.

48 ome had a disproportionately large effect on hybrid sterility in both reciprocal backcross hybrids.

49 nger (ZF) domains, have been associated with hybrid sterility in male house mice via spermatogenic fa

50 The mule, a classic example of hybrid sterility in mammals also exhibits a similar sper

51 Hybrid sterility in the heterogametic sex is a common fe

52 We discuss the possibility that hybrid sterility in this paradigmatic case of incipient

53 de toward understanding the genetic basis of hybrid sterility in various taxa.

54 different levels of fertilization success or hybrid sterility/inviability, is very common.

55 I find that hybrid sterility involves a single hybrid incompatibilit

56 According to the Dobzhansky-Muller model, hybrid sterility is a consequence of the independent evo

57 or all the other species, the major cause of hybrid sterility is antirecombination-the inability of d

58 Third, hybrid sterility is caused mainly by X-autosomal incompa

59 roductive barriers that can isolate species, hybrid sterility is frequently due to dysfunctional inte

60 Hybrid sterility is one of the earliest postzygotic isol

61 uring speciation, yet the molecular basis of hybrid sterility is poorly understood.

62 For one pair of species, hybrid sterility is probably caused by chromosomal rearr

63 Two SNPs in this list map near known hybrid sterility loci on chromosome 17 and the X chromos

64 epresent a universal mechanistic basis of F1 hybrid sterility manifested by pachytene arrest.

65 teractions among relatively few genes, while hybrid sterility may involve many more loci.

66 Despite predictions of the classic, hybrid-sterility model of chromosomal speciation, some o

67 the recombination-suppression model over the hybrid-sterility model of chromosome speciation are the

68 his gradual pattern is inconsistent with the hybrid-sterility model which, due to association of majo

69 Hybrid sterility of the heterogametic sex is one of the

70 evolution of reproductive barriers, such as hybrid sterility or inviability between populations.

71 strong X chromosome bias in the evolution of hybrid sterility or inviability but do find a very stron

72 While individual genes that cause hybrid sterility or inviability have been identified in

73 olation between species or subspecies and on hybrid sterility or inviability rather than on ecologica

74 ne's rule, which states that in instances of hybrid sterility or inviability, the heterogametic sex t

75 e identification of several genes that cause hybrid sterility or inviability-many of which have evolv

76 cause reproductive incompatibilities such as hybrid sterility or lethality?

77 n hybrids may elucidate the genetic basis of hybrid sterility or other hybrid dysfunctions that contr

78 nt mechanism that mediates chromosomal-based hybrid sterility phenotypes involving gametes with non-h

79 al region (PAR) was strongly associated with hybrid sterility phenotypes when heterozygous.

80 orm the mechanistic basis of some gene-based hybrid sterility phenotypes.

81 pparently few in number, the factors causing hybrid sterility show a remarkably complex pattern of ep

82 parison, loci underlying traits unrelated to hybrid sterility show no evidence for an accelerating ra

83 nsposons thus escape silencing and trigger a hybrid sterility syndrome termed P-M hybrid dysgenesis.

84 wer genes are required for the appearance of hybrid sterility than implied by previous studies of old

85 Hybrids suffer no hybrid sterility until they carry the "right" allele (Bo

86 s description of D. melanogaster/D. simulans hybrid sterility, we have discovered a strain of D. simu

87 ng approach, regions that contribute to male hybrid sterility were also identified.

88 Drosophila genome, no fewer than six loci of hybrid sterility were identified between two sibling spe

89 HALDANE's rule indicates that hybrid sterility will generally evolve in males prior to