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1 sed 77 twin pregnancies, comprising complete hydatidiform mole (CHM) and healthy co-twin, to ascertai
6 , NLRP7 and NLRP2, cause familial biparental hydatidiform mole and multilocus imprinting disturbance,
7 e genome-wide restriction maps of a complete hydatidiform mole and three lymphoblast-derived cell lin
8 haliana, Drosophila melanogaster and a human hydatidiform mole cell line (CHM1) from SMRT sequencing.
10 development of the conceptus into a complete hydatidiform mole in which extraembryonic trophoblastic
13 esent evidence of homozygosity of a complete hydatidiform mole using 20 diallelic markers distributed
14 emalignant disorders of complete and partial hydatidiform mole, and the malignant disorders of invasi
17 istinguishable from an androgenetic complete hydatidiform mole, in which abnormal extra-embryonic tis
23 niparental tissues of germline origin, i.e., hydatidiform moles (paternal origin) and complete ovaria
25 sentation of the paternal genome in sporadic hydatidiform moles (purely androgenetic in complete hyda
26 iform moles (purely androgenetic in complete hydatidiform moles and diandric triploid in partial hyda
28 diagnosis, routine morphologic assessment of hydatidiform moles continues to suffer from interobserve
30 eotide polymorphism markers in most complete hydatidiform moles indicating that these tumors are not
32 iform moles and diandric triploid in partial hydatidiform moles) is a fundamental genetic event leadi
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