ライフサイエンスコーパス: hydroethidine

1 lated rat skeletal muscle mitochondria using hydroethidine.

2 ons, as monitored with the fluorescent probe hydroethidine.

3 hidium produced from superoxide oxidation of hydroethidine.

4 micro mol/L)-enhanced chemiluminescence and hydroethidine (2 micro mol/L)-based confocal microscopy,

5 eased in aorta (measured using lucigenin and hydroethidine) after LPS, and levels of superoxide were

6 By use of the oxidative fluorescent dye hydroethidine, an in situ assay indicated markedly incre

7 deomicroscopy in mesentery microvessels with hydroethidine, an oxidant-sensitive fluoroprobe, showed

8 cent products of the membrane-permeable dyes hydroethidine and 2',7'-dichlorofluorescin diacetate, re

9 were determined on arterial segments via the hydroethidine assay and on stimulated endothelial cell c

10 Immunocytochemistry and hydroethidine-based detection of intracellular superoxid

11 The oxidation of hydroethidine by superoxide forms the membrane-impermeab

12 to measure the rate of ROS generation using hydroethidine, dicarboxyfluorescein diacetate, or MitoSO

13 fluorescence after intravenous injection of hydroethidine due to superoxide radicals in photorecepto

14 sections were determined by dihydroethidium (hydroethidine) fluorescence staining.

15 teolysis), and dihydrotetramethylrosamine or hydroethidine (fluorescent upon oxidation).

16 the product formed from the reaction between hydroethidine (HE) and superoxide (O(2)(.-)).

17 The putative oxidation of hydroethidine (HE) has become a widely used fluorescent

18 ells via fluorescence microscopy with either hydroethidine (HE) or its mitochondrially targeted deriv

19 show that MPO activity can be assessed using hydroethidine (HE), a probe commonly employed for the de

20 ct of reaction of superoxide (O(2)(*-)) with hydroethidine (HE), namely 2-hydroxyethidium (2-OH-E(+))

21 aded with the O(2)(.-)-sensitive fluorophore hydroethidine (HE).

22 the spin trap and by histofluorescence using hydroethidine (HE, 5 micromol/L) and dichlorodihydrofluo

23 rformed by using the oxidation-sensitive dye hydroethidine (HEt) to determine whether the relatively

24 imaging microfluorimetry of the oxidation of hydroethidine (HEt) to ethidium can be used to monitor s

25 Using hydroethidine (HEt), a fluorescent probe for superoxide,

26 By using multiple probes (hydroethidine, hydropropidine, Amplex Red, and coumarin

27 s released as evidenced by the conversion of hydroethidine in the extracellular environment to ethidi

28 gen species (ROS) generation, assessed using hydroethidine, in motor neurons.

29 ; flow cytometric analysis with a vital dye (hydroethidine) indicated that 1.5 mM NO lysed the erythr

30 onship between MMP-9 expression and oxidized hydroethidine, indicating reactive oxygen species (ROS)

31 confirmed by fluorescence techniques, mainly hydroethidine oxidation and horseradish peroxidase-based

32 maging of superoxide radical distribution by hydroethidine oxidation in vulnerable neurons.

33 mitochondrial source of this ROS generation, hydroethidine oxidation was inhibited by the mitochondri

34 as investigated by measuring the products of hydroethidine oxidation.

35 rogenic probes, as follows: superoxide using hydroethidine, peroxynitrite using boronate-based probes

36 y specific oxidation of a fluorescent probe, hydroethidine, reflecting decreased activity of Mn-SOD.

37 r studies using the oxidation-sensitive dye, hydroethidine, revealed Zn2+-dependent reactive oxygen s

38 lular production of O(2)(.-), as measured by hydroethidine staining, and inhibited cell growth.

39 Superoxide anion formation was assayed by hydroethidine staining.

40 mployed the superoxide-mediated oxidation of hydroethidine to ethidium to dynamically and directly as

41 wells were treated with triphenylphosphonium hydroethidine (TPP-HE), which forms the superoxide speci

42 Hydroethidine was used to identify superoxide radicals a

43 nd signals from the mitochondrially targeted hydroethidine, was increased in neurons with both the co

44 levels, markers of one-electron oxidation of hydroethidine, were observed at cytotoxic concentrations

45 xide formation was detected by reaction with hydroethidine within the first hour following activation