ライフサイエンスコーパス: hydrogen peroxide

1 , 2-methylpropionamidine dihydrochloride and hydrogen peroxide).

2 ating superoxide anion radical to oxygen and hydrogen peroxide.

3 action describes the reaction of Fe(II) with hydrogen peroxide.

4 y with O-phenylenediamine in the presence of hydrogen peroxide.

5 ection and imaging of beta-galactosidase and hydrogen peroxide.

6 fusion flux and determine the diffusivity of hydrogen peroxide.

7 dizes these enzymes at rates comparable with hydrogen peroxide.

8 understanding of the possible mechanisms of hydrogen peroxide.

9 cy is tuned by changing the concentration of hydrogen peroxide.

10 including peroxiredoxin PrxA, in response to hydrogen peroxide.

11 ived from dioxygen and its congeners such as hydrogen peroxide.

12 NADPH oxidase, superoxide radical anion, and hydrogen peroxide.

13 utions containing horseradish peroxidase and hydrogen peroxide.

14 s in the presence of exogenous or endogenous hydrogen peroxide.

15 ation caused by both urate hydroperoxide and hydrogen peroxide.

16 hyde while simultaneously reducing oxygen to hydrogen peroxide.

17 observed in the presence of dioxygen and/or hydrogen peroxide.

18 evelopment of other energetic materials with hydrogen peroxide.

19 s), including superoxide, peroxynitrite, and hydrogen peroxide.

20 , as well as in cells treated with exogenous hydrogen peroxide.

21 MDA receptor antagonism or the break down of hydrogen peroxide.

22 at blocks the oxidation of solution phase of hydrogen peroxide.

23 nglet oxygen, superoxide anion radicals, and hydrogen peroxide.

24 recombinant VEGF and exposed to hyperoxia or hydrogen peroxide.

25 ibly inhibited by the drug disulfiram and by hydrogen peroxide.

26 into 40 microL of buffer solution containing hydrogen peroxide.

27 3) dispersion of MoS2 in aqueous solution of hydrogen peroxide.

28 ed cells that were stressed with arsenite or hydrogen peroxide.

29 e stress, as measured based on resistance to hydrogen peroxide.

30 exposure of both to 70 muM Cu(2+) and 10 mM hydrogen peroxide.

31 e solution of red blood cells was exposed to hydrogen peroxide.

32 ch could be normalized by the application of hydrogen peroxide.

33 in the presence of varying concentrations of hydrogen peroxide.

34 s accompanied by increased susceptibility to hydrogen peroxide.

35 g human primary fibroblasts to a low dose of hydrogen peroxide.

36 om intact erythrocytes to the same extent as hydrogen peroxide.

37 s capable of measuring 8 muL samples of both hydrogen peroxide (0-5 mM, 2.72 x 10(-6) A.mM(-1)) and t

38 Exposure to hydrogen peroxide (1%) decreased the biofilm biomass and

39 p procedure ((1) oxidation of glutathione by hydrogen peroxide; (2) decomposition of the hydrogen per

40 tions including formaldehyde (0.074g.L(-1)), hydrogen peroxide (21.0g.L(-1)), bicarbonate (4.0g.L(-1)

41 nd then digested in a mixture of nitric acid/hydrogen peroxide (3:1).

42 ss conditions including an oxidative stress (hydrogen peroxide), a heavy metal stress (arsenite) and

43 ents of the Martian surface, iron oxides and hydrogen peroxide, act in synergy with irradiated perchl

44 Using this approach, we find that hydrogen peroxide addition increases S-sulfination of hu

45 chemical reactions with sodium borohydride, hydrogen peroxide, alpha-methoxy-alpha-(trifluoromethyl)

46 llent peroxidease-like catalytic activity in hydrogen peroxide-Amplex red (AR) system (AR is oxidized

47 e also developed a GO enzyme assay using the hydrogen peroxide-Amplex red reporter system.

48 xhaled hydrogen ions, nitric oxide products, hydrogen peroxide and 8-isoprostanes were generally elev

49 ion of important biomarkers such as glucose, hydrogen peroxide and cancer biomarkers and highlight th

50 response of multi-valent CNPs in presence of hydrogen peroxide and demonstrate an enzyme-free CNP-bas

51 luminescence reaction occurred with luminol, hydrogen peroxide and HRP enzyme, and the emission of li

52 ed c-di-AMP levels, increased sensitivity to hydrogen peroxide and increased production of extracellu

53 ABA induces stomatal closure through hydrogen peroxide and nitric oxide (NO) along with subse

54 from NADPH oxidase, as well as the amount of hydrogen peroxide and nitrotyrosine.

55 otype made resistant cells more sensitive to hydrogen peroxide and nutrient starvation.

56 otein synthesis: oxidative stress induced by hydrogen peroxide and nutrient stresses caused by amino

57 atalytic activity of red blood cells towards hydrogen peroxide and on surface-induced haemolysis.

58 of superoxide, which acts as a precursor of hydrogen peroxide and other reactive oxygen species that

59 yme that converts superoxide into less toxic hydrogen peroxide and oxygen, functions in the gustatory

60 requirement for Spatzle ligand downstream of hydrogen peroxide and protease function, both of which a

61 the corresponding spirodioxyselenuranes with hydrogen peroxide and subsequent reduction back to the o

62 including thiol depletion and generation of hydrogen peroxide and superoxide, but none of them can f

63 PP3-KEAP1 interaction is strongly induced by hydrogen peroxide and that DPP3 is required for timely N

64 single device capable of the measurement of hydrogen peroxide and total cholesterol.

65 t additives (e.g., hypochlorite, persulfate, hydrogen peroxide) and subsequently with diverse dissolv

66 endly slurry that consists of mainly silica, hydrogen peroxide, and citric acid.

67 R do form a disulfide bond when treated with hydrogen peroxide, and formation of this Cys(201)-Cys(20

68 drial poison rotenone, amyloid beta-peptide, hydrogen peroxide, and high levels of glutamate.

69 t for luminol CL reaction in the presence of hydrogen peroxide, and thus the luminol CL response is l

70 butions of singlet oxygen, hydroxyl radical, hydrogen peroxide, and triplet dissolved organic matter

71 peroxidase 1 macrophages and that restoring hydrogen peroxide antioxidant defenses suppressed this e

72 ion, reactive oxygen species (superoxide and hydrogen peroxide) appeared.

73 odels were treated with potassium cyanide or hydrogen peroxide as controls, and epidermal growth fact

74 perometric measurements were performed using hydrogen peroxide as substrate.

75 efore adsorption and after regeneration with hydrogen peroxide at 20 degrees C.

76 current transients produced by oxidation of hydrogen peroxide at one IrOx NP provided information ab

77 tion showed strong evidence pointing towards hydrogen peroxide being the primary current source, conf

78 sensitive to Mn during anaerobiosis or when hydrogen peroxide biogenesis is significantly reduced.

79 10, resembling the conditions of industrial hydrogen peroxide bleaching (P stage).

80 MALDI-TOF mass spectrometry indicates that hydrogen peroxide blocks labeling of cysteine 600, which

81 nt differential generation of superoxide and hydrogen peroxide by AAO4 and the induction of AAO4 expr

82 x) and biodegradation of toxic superoxide to hydrogen peroxide by superoxide dismutase.

83 lectivity of pnGFP toward peroxynitrite over hydrogen peroxide by using site-directed mutagenesis, X-

84 For example, hydrogen peroxide can be decomposed to oxygen by 0.44 U

85 tention assay, we show that within a minute, hydrogen peroxide causes accumulation of Pol II near pro

86 considered unadulterated and formaldehyde-, hydrogen peroxide-, citrate-, hydroxide- and starch-adul

87 ystem was successfully used to determine the hydrogen peroxide concentration in real-time analyses.

88 ditionally, this detector was used to detect hydrogen peroxide concentrations in commercial hair dye

89 The hydrogen peroxide content and antimicrobial activity of

90 , sugar content, antimicrobial activity, and hydrogen peroxide content were selected in current work

91 ssociated gastritis, where its production of hydrogen peroxide contributes to DNA damage and subseque

92 ndrial superoxide which, after conversion to hydrogen peroxide, contributes to cellular membrane depo

93 The hydrogen peroxide detection limit of the flexible MSM ph

94 Comparisons between current hydrogen peroxide diffusion studies and previously publi

95 t of CAFs but not prostate cancer cells with hydrogen peroxide directly inhibited mmp-3 promoter acti

96 Hydrogen peroxide dominates the passivating process duri

97 acid, easily generated from acetic acid and hydrogen peroxide, dramatically increases the effectiven

98 enase cytochrome P450 OleTJE (CYP152L1) is a hydrogen peroxide-driven oxidase that catalyzes oxidativ

99 and reliable quantification of intracellular hydrogen peroxide during cancer therapy constitutes an u

100 employed printed electrochemical sensors for hydrogen peroxide electroreduction integrated with print

101 cyanide, and oxidative phosphorylation using hydrogen peroxide, employing the phasor approach require

102 dium hypochlorite, 70% isopropyl alcohol, 3% hydrogen peroxide, ethyl alcohol, water immersion, ultra

103 been knocked down for GIMAP6, treatment with hydrogen peroxide, FasL, or okadaic acid significantly i

104 n-catalyzed hydroxyl radical production from hydrogen peroxide (Fenton's reaction) and subsequent aqu

105 Upon the addition of hydrogen peroxide for 1 min to cells preloaded with a bi

106 and the subsequent mediators superoxide and hydrogen peroxide for pulmonary oxygen sensing and signa

107 Hydrogen peroxide formation is dramatically suppressed,

108 In addition, hydrogen-peroxide formation is identified as a competing

109 using a slight excess (1.5 equiv) of aqueous hydrogen peroxide, from the oxidation of a broad range o

110 Evidence for a feedback mechanism where hydrogen peroxide generated during the oxidation of asco

111 Hydrogen peroxide generation was greatly diminished at a

112 HUVEC oxygen consumption and superoxide and hydrogen peroxide generation were measured in 69 infants

113 ndocytic compartments resulting in endosomal hydrogen peroxide generation, which suppresses antiviral

114 different stresses, subsequently maintaining hydrogen peroxide (H2 O2 ) homeostasis in Arabidopsis.

115 Reducing the levels of hydrogen peroxide (H2 O2 ) in myb36-5 significantly resc

116 urons with HRP and subsequent treatment with hydrogen peroxide (H2 O2 ) modified the properties of ne

117 release H2 S until triggered by ROS, such as hydrogen peroxide (H2 O2 ), superoxide (O2(-) ), and per

118 L), an enzyme required for the generation of hydrogen peroxide (H2 O2 ).

119 tection of DNA oxidation [in the presence of hydrogen peroxide (H2O2) and an H2O2/iron (III) chloride

120 zed composites were tested for biosensing of hydrogen peroxide (H2O2) and as supercapacitor electrode

121 rates both superoxide (O2(-*) or HOO(*)) and hydrogen peroxide (H2O2) and leads to the same set of pr

122 producing intermediate by-products, such as hydrogen peroxide (H2O2) and other reactive oxygen speci

123 hilus led to a significant increase in plant hydrogen peroxide (H2O2) and salicylic acid levels.

124 atalytic oxidation of styrenes using aqueous hydrogen peroxide (H2O2) and the cationic palladium(II)

125 abolism also generates superoxide (O2()) and hydrogen peroxide (H2O2) as bona fide products in reacti

126 ct of chitosan (CH), salicylic acid (SA) and hydrogen peroxide (H2O2) at different concentrations on

127 imary sludge (PS) at the anode and producing hydrogen peroxide (H2O2) at the cathode.

128 anism underlying oxidative stress induced by hydrogen peroxide (H2O2) between murine corneal epitheli

129 Hydrogen peroxide (H2O2) can act as a signaling molecule

130 Hydrogen peroxide (H2O2) elicits ROS that induces skin a

131 of the rates of oxygen consumption (VO2) and hydrogen peroxide (H2O2) emission as a function of PCoA

132 ased on the oxidation of cysteine (CSH) with hydrogen peroxide (H2O2) enzymatically generated by alco

133 consisting of a cathodic cell that produced hydrogen peroxide (H2O2) followed by an ultraviolet (UV)

134 The direct synthesis of hydrogen peroxide (H2O2) from H2 and O2 represents a pot

135 Electrochemical production of hydrogen peroxide (H2O2) from water oxidation could prov

136 We conceived that extracellular hydrogen peroxide (H2O2) generated by Duox diffuses thro

137 ce (MR) method for noninvasively quantifying hydrogen peroxide (H2O2) in aqueous solutions based on c

138 production of the ROS superoxide (O2(-)) and hydrogen peroxide (H2O2) in brackish and freshwater pond

139 To investigate the role of apoplastic hydrogen peroxide (H2O2) in gymnosperm phenolic metaboli

140 was observed using Hydro Quininone (HQ) and Hydrogen peroxide (H2O2) in PB (Phosphate Buffer) electr

141 ed in significantly higher concentrations of hydrogen peroxide (H2O2) in plant tissues at the vegetat

142 mine (AgNPs-OA) is reported for detection of hydrogen peroxide (H2O2) in wastewater samples.

143 Hydrogen peroxide (H2O2) is a highly relevant metabolite

144 Hydrogen peroxide (H2O2) is an endogenous molecule that

145 Hydrogen peroxide (H2O2) is an important signaling molec

146 Hydrogen peroxide (H2O2) is known as a key molecule in a

147 Hydrogen peroxide (H2O2) is then reduced by avidin-HRP i

148 Hydrogen peroxide (H2O2) is usually considered to be an

149 Rapid and dynamic change in hydrogen peroxide (H2O2) levels can serve as an importan

150 We found that under Fe deficiency, hydrogen peroxide (H2O2) levels were enhanced in a FIT-d

151 Nonradical species such as hydrogen peroxide (H2O2) or singlet molecular oxygen, ra

152 Hydrogen peroxide (H2O2) plays an important role in redo

153 One proposed signal is hydrogen peroxide (H2O2) produced by chloroplasts in a l

154 palmitoyl ascorbate (PA) as a prooxidant for hydrogen peroxide (H2O2) production in tumor tissue is s

155 Glucose has been quantified by measuring hydrogen peroxide (H2O2) reduction by chronoamperometry

156 RATIONALE: Hydrogen peroxide (H2O2) regulates vascular tone in the

157 Short-term application of hydrogen peroxide (H2O2) rescues (from depleted ROS) and

158 e yields of cellular superoxide (O2(*-)) and hydrogen peroxide (H2O2) ROS products, indicating cohere

159 lectrode in order to prepare an amperometric hydrogen peroxide (H2O2) sensor.

160 nversion to NO2(-) and NO3(-) in an alkaline hydrogen peroxide (H2O2) solution.

161 al sensor was developed for the detection of hydrogen peroxide (H2O2) using a reduced graphene oxide-

162 A potent, diffusible messenger is hydrogen peroxide (H2O2), but its consequences on extrac

163 zyme, which catalyzes the reduction of O2 to hydrogen peroxide (H2O2), has been implicated in the car

164 nstrated that CS-generated ROS, particularly hydrogen peroxide (H2O2), impaired adenosine stimulated

165 from the photolysis of nitrous acid (HONO), hydrogen peroxide (H2O2), ozone (O3), formaldehyde (HCHO

166 in astrocytes exposed to oxidative stress by hydrogen peroxide (H2O2), p38 activation did not inhibit

167 erties are essential for ROS, in the form of hydrogen peroxide (H2O2), to induce cell death.

168 Hydrogen peroxide (H2O2), used here as a model oxidant u

169 oxygen (O2) as an oxidant in the presence of hydrogen peroxide (H2O2), we demonstrated that the resul

170 lonic epithelial cells produce extracellular hydrogen peroxide (H2O2), which acts as a potent signali

171 ated that high glucose induced production of hydrogen peroxide (H2O2), which down regulated silent in

172 efficiently despite an apparent decrease of hydrogen peroxide (H2O2), which was previously shown to

173 on human intestinal Caco-2 cells exposed to hydrogen peroxide (H2O2)-induced oxidative stress.

174 leoredoxin 1 (NRX1) targets enzymes of major hydrogen peroxide (H2O2)-scavenging pathways, including

175 g period with 3,3-diaminobenzidine (DAB) and hydrogen peroxide (H2O2).

176 the activation of a trigger by activation by hydrogen peroxide (H2O2).

177 g, which was exacerbated upon treatment with hydrogen peroxide (H2O2).

178 all domains of life, dissipates the oxidant hydrogen peroxide (H2O2).

179 um, has constitutive activity, and generates hydrogen peroxide (H2O2).

180 ol for sensitively and selectively measuring hydrogen peroxide (H2O2).

181 s unreactive to millimolar concentrations of hydrogen peroxide (H2O2).

182 n (Fe(2+)) and inducing the decomposition of hydrogen peroxide (H2O2).

183 talytically active while the P450s generated hydrogen peroxide (H2O2).

184 of sterilization processes applying gaseous hydrogen peroxide (H2O2).

185 ved antibacterial components, defensin-1 and hydrogen peroxide (H2O2).

186 highly sensitive and selective detection of hydrogen peroxide (H2O2).

187 species (ROS) [hydroxyl radicals ((*)OH) and hydrogen peroxide (H2O2)] catalyzed by ambient particula

188 Hydrogen-peroxide (H2O2)-induced growth of small-sized g

189 However, measuring ROS (hydrogen peroxide, H2O2) content in vivo is now possible

190 by singlet oxygen ((1)O2) or superoxide and hydrogen peroxide have been attributed to either cell de

191 ecreased oxidative stress (Nox2, HIF-1alpha, hydrogen peroxide, hydroxynonenal), and fibrogenesis (al

192 n of toxic substances, such as formaldehyde, hydrogen peroxide, hypochlorite, dichromate, salicylic a

193 demonstrated to be a significant degrader of hydrogen peroxide in anoxic Escherichia coli Intriguingl

194 triggered by light (405 nm) as well as with hydrogen peroxide in aqueous phosphate buffer.

195 are related to an increased concentration of hydrogen peroxide in bacterially infected glutathione pe

196 me selective quantification of intracellular hydrogen peroxide in Cisplatin-treated human renal HK-2

197 s, indicating a role of NOX2/p47phox-derived hydrogen peroxide in mediating the basal activity of NO

198 (R = H, Me, CF3CO, MeSO2, CF3SO2) react with hydrogen peroxide in MeOH, THF, MeCN or AcOH to form the

199 Our findings also demonstrate a role for hydrogen peroxide in the mechanisms tightly regulating N

200 rial monoamine oxidases MAO-A/B and produces hydrogen peroxide in the nucleus as a byproduct of demet

201 in system that increase levels of endogenous hydrogen peroxide in the yeast Saccharomyces cerevisiae

202 In this study, copper-catalysed hydrogen peroxide induced oxidation of C-hordein caused

203 ed a cell viability of 82.90+/-0.78% against hydrogen peroxide-induced oxidative stress on EA.hy926,

204 mote angiogenesis and to protect ECs against hydrogen peroxide-induced oxidative stress.

205 at FGF2 priming protected cultured DPCs from hydrogen-peroxide-induced cell death and increased the n

206 be utilized as a redox switch to monitor the hydrogen-peroxide-induced oxidative stress in living SW4

207 The coregulator hydrogen peroxide-inducible clone 5 (Hic-5) is required

208 dize reduced FADH2 and thus does not produce hydrogen peroxide; instead, it uses a cytochrome b-like

209 d an increase in cellular protection against hydrogen peroxide insult in a pentose phosphate pathway

210 models predicting that the rapidly diffusing hydrogen peroxide intermediate is well mixed.

211 rescence units; P < 0.05), and released more hydrogen peroxide into the supernatant after hyperoxia e

212 ew analytical methods to accurately quantify hydrogen peroxide is of great interest.

213 Mitochondrial production of superoxide and hydrogen peroxide is potentially important in cell signa

214 ctive Cys residues (Cys(53) and Cys(397)) by hydrogen peroxide (k = 17.3 +/- 1.3 m(-1) s(-1) at pH 7.

215 ork, ascorbate-glutathione cycle components, hydrogen peroxide levels and the proteomic profile of ca

216 mediator from nitric oxide to mitochondrial hydrogen peroxide (% max diameter at 100 cm H2O: vehicle

217 icularly important in coupling intracellular hydrogen peroxide metabolism to glutathione oxidation, a

218 rescence and color properties of HA and that hydrogen peroxide might whiten HA-AAAs by oxidizing the

219 irradiation of anilines in the presence of a hydrogen peroxide/O2 system by wavelengths that overlap

220 s nanomolar concentrations of superoxide and hydrogen peroxide on irradiation with simulated sunlight

221 GOx) on gold to catalyze glucose, and detect hydrogen peroxide on Pt sites.

222 droxyquinones (HQ), peroxynitrites (PN), and hydrogen peroxide, on their ability to induce unfolded p

223 tion (to water) to a 2e(-)/2H(+) process (to hydrogen peroxide) only by increasing the temperature fr

224 howed greater induction of IL-6 secretion by hydrogen peroxide or benzo[a]pyrene diolepoxide treatmen

225 foxides or sulfones by treatment with dilute hydrogen peroxide or mCPBA, respectively.

226 levels of survival when exposed to exogenous hydrogen peroxide or murine macrophages.

227 eas PEG-catalase scavenging of intracellular hydrogen peroxide or RNAi of NOX2 produced similar resul

228 peroxiredoxin (AhpC) to detoxify ROS such as hydrogen peroxide, organic hydroperoxide, and peroxynitr

229 pH 6.8 and 52% in the presence of Ca(2+) and hydrogen peroxide over unmodified proteins.

230 w is discussed, including the use of oxygen, hydrogen peroxide, ozone and other oxidants in flow.

231 izing agents, including sodium hypochlorite, hydrogen peroxide, ozone and sodium periodate, are descr

232 Hydrogen peroxide produced by KDM1A catalysis reduces th

233 the biological effects of XO were due to the hydrogen peroxide produced by the enzyme.

234 theoretically and experimentally investigate hydrogen peroxide production activity trends for a range

235 xamined the effects of a variant in DUOX2 on hydrogen peroxide production in HEK293 cells.

236 k to emerging clean energy reactions such as hydrogen peroxide production, carbon dioxide reduction,

237 ugh an age-related increase in mitochondrial hydrogen peroxide production.

238 ation to the mitochondria, where it promotes hydrogen peroxide production.

239 enones and cyclohexene ketones with aqueous hydrogen peroxide, providing the corresponding epoxides

240 ommercial powdered infant formulas generated hydrogen peroxide, ranging from 10.46 to 11.62 muM, when

241 Hydrogen peroxide reduced thrombospondin 2 (an MMP-3 sup

242 from the oxygen reduction reaction (ORR) or hydrogen peroxide reduction on platinum, has been invest

243 When analyzed using 2.5mM of hydrogen peroxide reduction reaction using cyclic voltam

244 O4@GO particles, which subsequently catalyze hydrogen peroxide reduction to detect PSA and PSMA.

245 s an important role in the detoxification of hydrogen peroxide released either during photorespiratio

246 hydrogen peroxide; (2) decomposition of the hydrogen peroxide residue by catalase).

247 We examine the genetic basis of hydrogen peroxide resistance in three related yeast cros

248 spectrum of cellular processes contribute to hydrogen peroxide resistance, including DNA repair, scav

249 h the complex genetic and molecular basis of hydrogen peroxide resistance, we show two examples where

250 protein kinases OPEN STOMATA1 and GUARD CELL HYDROGEN PEROXIDE-RESISTANT1 (GHR1) in Xenopus laevis oo

251 ee- and four-fold more hydroxyl radicals and hydrogen peroxide, respectively, than TiO2 at 160 mmHg.

252 production and susceptibility to killing by hydrogen peroxide, respectively.

253 Activated HSC are also important sources of hydrogen peroxide resulting from the activation of NADPH

254 rgeted H2O2 scavenger, mitochondria-targeted hydrogen peroxide scavenger ebselen, reduced Sirt3 S-glu

255 polyethylene glycol-catalase (PEG-catalase; hydrogen peroxide scavenger) had no effect.

256 whereas, increase in DPPH (48.32-59.62%) and hydrogen peroxide scavenging activities (35.44-63.07mM-T

257 current system through chemiluminescence for hydrogen peroxide sensing.

258 Here the authors use a fluorescent hydrogen peroxide sensor to provide evidence that H2O2 i

259 ary collecting duct cells in the presence of hydrogen peroxide showed increased activity of p53 at Se

260 AAO4 and the induction of AAO4 expression by hydrogen peroxide shown here suggest a self-amplificatio

261 rements were carried out at -0.20V by adding hydrogen peroxide solution onto the electrode surface in

262 urrent density among silica, citric acid and hydrogen peroxide solution.

263 Two polymorphic hydrogen peroxide solvates of 2,4,6,8,10,12-hexanitro-2,

264 dom34 and ski7 hbs1 mutants are sensitive to hydrogen peroxide stress and accumulate an NSD substrate

265 melanogaster females but not males adapt to hydrogen peroxide stress, whereas males but not females

266 er untreated conditions as well as following hydrogen peroxide stress.

267 ntification of oxidation in the Fc region of hydrogen peroxide-stressed Rituximab, using a single, co

268 gic states or during oxidative stress (e.g., hydrogen peroxide, superoxide and hydroxyl radicals, nit

269 val following treatment with antibiotics and hydrogen peroxide, supporting the importance of the prot

270 ell's redox state: oxidative signals such as hydrogen peroxide suppress MGL activity in a reversible

271 The flux of hydrogen peroxide through 1-mm discs of bovine enamel wa

272 g by increasing superoxide radical anion and hydrogen peroxide through a mechanism that promotes acti

273 adicals are produced from the ORR byproduct, hydrogen peroxide, through a 1 electron reduction pathwa

274 ith O2 and its derived intermediates such as hydrogen peroxide to afford a variety of metal-oxygen in

275 nical treatments N-acetylcysteine, EDTA, and hydrogen peroxide to disrupt in vitro biofilms and kill

276 nanoparticles in catalytic decomposition of hydrogen peroxide to oxygen and water.

277 hrough ketone diboration, react rapidly with hydrogen peroxide to release alcohols, aldehydes, and ke

278 ility of elutriate fractions was tested with hydrogen peroxide to track photoprotein and/or protein-b

279 n the presented study, DHNQ was treated with hydrogen peroxide under alkaline conditions at pH 10, re

280 xidine bathing, decolonization for MRSA, and hydrogen peroxide vaporizer for MDR-AB) were used less c

281 y of about 98% for H2O2 production.Producing hydrogen peroxide via electrochemical oxidation of water

282 t the main source of increased production of hydrogen peroxide was a block in respiratory chain and d

283 Hydrogen peroxide was also formed in 10% yield consisten

284 reductase, a primary scavenger of endogenous hydrogen peroxide was also identified.

285 Furthermore, hydrogen peroxide was characterized as the central modul

286 We observed that hydrogen peroxide was generated during thermal treatment

287 utral pH, electrochemical water oxidation to hydrogen peroxide was observed, albeit with low (15%) Fa

288 pectral features from an experiment in which hydrogen peroxide was used to induce oxidative stress in

289 reening-compatible assays for superoxide and hydrogen peroxide, we identified potential inhibitors of

290 ascorbic acid, maximum levels of measurable hydrogen peroxide were achieved with an initial concentr

291 Here we show that the ROS superoxide and hydrogen peroxide were present in dark waters at compara

292 d in vivo in E. coli survival in response to hydrogen peroxide, where mutation of W52 leads to decrea

293 The superoxide dismutates to hydrogen peroxide, which is used by myeloperoxidase to g

294 of reactive oxygen species (ROS) induced by hydrogen peroxide, which led to increased levels of an o

295 the dying cells stimulates the production of hydrogen peroxide, which orchestrates tissue repair via

296 In addition, cytokinin and hydrogen peroxide, which promote root differentiation, i

297 nduced killing depended on the production of hydrogen peroxide, which required increased supply of NA

298 The hydrogen peroxide will be further reduced to water by ho

299 We replaced hydrogen peroxide with t-butyl hydroperoxide and found t

300 Treatment of the reaction products with hydrogen peroxide yields the corresponding syn-configure