ライフサイエンスコーパス: hydrogen sulfide

1 e (FEPO) for use in in vivo visualization of hydrogen sulfide.

2 injury that was prevented in the presence of hydrogen sulfide.

3 nsoluble and unreactive until it reacts with hydrogen sulfide.

4 ation of l-cysteine from O-acetyl serine and hydrogen sulfide.

5 n response to hypoxia and is not specific to hydrogen sulfide.

6 s with the safe removal of one equivalent of hydrogen sulfide.

7 ty of these membranes to resist poisoning by hydrogen sulfide.

8 proposed synchronization mechanism involving hydrogen sulfide.

9 model in vitro in the absence or presence of hydrogen sulfide.

10 isolates also lacked the capacity to produce hydrogen sulfide.

11 e of the enzyme, without the intervention of hydrogen sulfide.

12 on monoxide and, as described more recently, hydrogen sulfide.

13 he gas phase were 8.7 x 10(-4) mug/L air for hydrogen sulfide, 1 x 10(-4) mug/L air for methyl mercap

14 Hydrogen sulfide, a signaling gas, affects several cell

15 Because the clinical significance of hydrogen sulfide administration in rodents may not be ap

16 concrete in sewers is known to be caused by hydrogen sulfide, although the role of wastewater in reg

17 These results suggest that hydrogen sulfide ameliorates sFlt1-induced hypertension,

18 4Fe-4S] enzyme that decomposes L-cysteine to hydrogen sulfide, ammonia, and pyruvate has been isolate

19 te, ascorbic acid, acetaminophen, uric acid, hydrogen sulfide, ammonium, ammonia, and both protonated

20 Hydrogen sulfide, an endogenous signaling molecule, play

21 r enzymes that can catalyze the formation of hydrogen sulfide, an important gaseous signaling molecul

22 ganic matter, Ti with hydroxide, and Ag with hydrogen sulfide and ammonia; less than 1% of dissolved

23 st that for some bacterial metabolites, like hydrogen sulfide and butyrate, the extent of their oxida

24 Fe(III/II)-IL/DMF can remove hydrogen sulfide and can be regenerated through an elect

25 er material selection and surface treatment, hydrogen sulfide and carbonyl sulfide standard mixtures

26 are based on the long-standing paradigm that hydrogen sulfide and chloride are the ligands responsibl

27 Hydrogen sulfide and methyl mercaptan were derivatized u

28 mated analysis based on the reaction between hydrogen sulfide and N,N-dimethyl-p-phenylenediamine in

29 cted the ability of the wine to release back hydrogen sulfide and other mercaptans during AR-aging.

30 hat permit the sequestration and delivery of hydrogen sulfide and oxygen to chemoautotrophic bacteria

31 product generated by enzymatic oxidation of hydrogen sulfide and polysulfides.

32 Among the VSCs, hydrogen sulfide and sulfur dioxide are considered most

33 be protective by intermediate conversion to hydrogen sulfide and thiosulfate.

34 essure carbon dioxide and impurities such as hydrogen sulfide and water are not available.

35 ng and inhibit gas extraction, produce toxic hydrogen sulfide, and induce corrosion leading to downho

36 dehyde, acetone, acetic acid, hexanoic acid, hydrogen sulfide, and methanol were increased in the can

37 dehyde, acetone, acetic acid, hexanoic acid, hydrogen sulfide, and methanol) to discriminate cancer p

38 mitter H2S in its aqueous form (bisulfide or hydrogen sulfide anion) based on the alteration of Forst

39 tably carbon monoxide, cyanide, cyanate, and hydrogen sulfide, are potent inhibitors of Ni-containing

40 topril, bovine and human serum albumins, and hydrogen sulfide, are reported.

41 This study identifies hydrogen sulfide as a novel cellular mediator that can m

42 tudies are warranted to evaluate the role of hydrogen sulfide as a novel therapeutic agent for vascul

43 ned biosensor could detect concentrations of hydrogen sulfide as low as 0.5 ppm.

44 of dissolved monomethylmercury (CH3Hg) with hydrogen sulfide as possible bacterial mediated or abiot

45 n steps are driven by ultraviolet light, use hydrogen sulfide as the reductant and can be accelerated

46 ncreased sulfide concentrations by oxidizing hydrogen sulfide before its diffusion to oxygenated surf

47 aggerated ISR and greater loss of endogenous hydrogen sulfide but normal inhibition of mTORC1 and mai

48 ated by changes to nuclear factor-kappa B or hydrogen sulfide but that occurred together with a modul

49 roducts from food residues such as genotoxic hydrogen sulfide by sulfur-reducing bacteria in response

50 We report that hydrogen sulfide can induce a suspended animation-like s

51 Headspace gas concentrations (hydrogen sulfide, carbon dioxide, and methane), pore wat

52 prising methane, ethane, other hydrocarbons, hydrogen sulfide, carbon dioxide, nitrogen, and water.

53 Gaseous molecules including nitric oxide, hydrogen sulfide, carbon monoxide and oxygen mediate num

54 et al. (2015) show that the gasotransmitter hydrogen sulfide co-operates with growth factor TGF-beta

55 cell concentration, immobilization bed type, hydrogen sulfide concentration, and geometrical shape of

56 In the harmful organic-rich sediment, hydrogen sulfide concentrations were low initially but i

57 Hydrogen sulfide-dependent effects were mainly observed

58 more pronouncedly responding SO2 is used for hydrogen sulfide detection.

59 articularly short-chain fatty acids, but not hydrogen sulfide, directly modulate circadian clock gene

60 Treatment with a hydrogen sulfide donor, diallyl trisulfide, prevented th

61 ts of hydrogen sulfide, we proposed that the hydrogen sulfide donor, sodium sulfide (Na2S), would att

62 xpressing animals intraperitoneally with the hydrogen sulfide-donor sodium hydrosulfide (NaHS) (50 mi

63 This vascular remodeling is reversible, and hydrogen sulfide donors are likely to improve pregnancy

64 This implies that hydrogen sulfide donors may be therapeutic.

65 versial data are available on the effects of hydrogen sulfide during hemorrhage.

66 Maintaining physiological levels of hydrogen sulfide during ischemia is necessary to limit i

67 orm for the in situ determination of gaseous hydrogen sulfide, employing a 470 nm light emitting diod

68 factor representation of the system based on hydrogen sulfide, Fe(II) and Fe(III).

69 Subsequently, the hydrogen sulfide formed by bacterial decomposition of co

70 l response is mediated by CA and may involve hydrogen sulfide formed in the reaction of COS and water

71 dosymbionts of these progenitors made use of hydrogen sulfide from biogenic sources (e.g., decaying w

72 e-Claus process, are commonly used to remove hydrogen sulfide from hydrocarbon gas streams such as la

73 ponse is not known, we find that addition of hydrogen sulfide gas to growing cells recapitulates all

74 lanogaster and mice from lethal exposures of hydrogen sulfide gas.

75 beta-synthase and cystathionine gamma-lyase, hydrogen sulfide-generating enzymes, was significantly r

76 neutrophil infiltration, cysteamine, mucin, hydrogen sulfide, ghrelin, adiponectin and the influence

77 yceophosphocholine, cystathionine, cysteine, hydrogen sulfide, glutathione disulfide, and glutathione

78 was most severe in sites with high levels of hydrogen sulfide (>100 ppm) and carbon dioxide (>1%) gas

79 a growing recognition of the significance of hydrogen sulfide (H(2)S) as a biological signaling molec

80 types of ancient bacteria and archea rely on hydrogen sulfide (H(2)S) for their energy production, eu

81 The rHCYase produces hydrogen sulfide (H(2)S) from tHCY without interference

82 Although the toxicity of hydrogen sulfide (H(2)S) has been substantiated for almo

83 Hydrogen sulfide (H(2)S) has emerged as a new and import

84 Hydrogen sulfide (H(2)S) has emerged as an important sig

85 es was to investigate the role of changes in hydrogen sulfide (H(2)S) homeostasis in the pathogenesis

86 Many prokaryotic species generate hydrogen sulfide (H(2)S) in their natural environments.

87 Hydrogen sulfide (H(2)S) is a gaseous neuromodulator tha

88 Hydrogen sulfide (H(2)S) is a gaseous vasodilator produc

89 Hydrogen sulfide (H(2)S) is a prominent EDRF, since mice

90 Hydrogen sulfide (H(2)S) is a recently described endogen

91 Hydrogen sulfide (H(2)S) is a unique gasotransmitter, wi

92 Hydrogen sulfide (H(2)S) is a volatile gas of considerab

93 Hydrogen sulfide (H(2)S) is an endogenous signaling mole

94 The recent discovery that hydrogen sulfide (H(2)S) is an endogenously produced gas

95 Hydrogen sulfide (H(2)S) is emerging as an important med

96 Hydrogen sulfide (H(2)S) is known to play a physiologica

97 Hydrogen sulfide (H(2)S) is naturally produced in animal

98 We sought to evaluate the impact of hydrogen sulfide (H(2)S) on the outcome after CA and car

99 ck of reliable methods to accurately measure hydrogen sulfide (H(2)S) produced in vitro has impeded r

100 rdioprotection with tadalafil is mediated by hydrogen sulfide (H(2)S) signaling in a PKG-dependent fa

101 ablished that exposure of mammalian cells to hydrogen sulfide (H(2)S) suppresses mitochondrial functi

102 optotic actions of NF-kappaB are mediated by hydrogen sulfide (H(2)S) synthesized by cystathionine ga

103 ler electric discharge experiment containing hydrogen sulfide (H(2)S) were recently discovered and an

104 This study examined the role of hydrogen sulfide (H(2)S), a novel gasotransmitter, in th

105 Hydrogen sulfide (H(2)S), a recently described endotheli

106 The detection of hydrogen sulfide (H(2)S), a toxic gas and an important b

107 ert garlic-derived organic polysulfides into hydrogen sulfide (H(2)S), an endogenous cardioprotective

108 acid derivative (ACS67) that donates the gas hydrogen sulfide (H(2)S).

109 ts are often found to contain high levels of hydrogen sulfide (H(2)S).

110 (4) Hydrogen sulfide (H(2)S/HS(-)) inhibits CO oxidation but

111 Hydrogen sulfide (H2 S) and hydrogen polysulfides (H2 Sn

112 Hydrogen sulfide (H2 S) exhibits promising protective ef

113 Hydrogen sulfide (H2 S) has emerged as a crucial biomole

114 Although hydrogen sulfide (H2 S) is perhaps best known as a toxic

115 Here we propose that hydrogen sulfide (H2 S), as a glial-released inflammator

116 The endogenously produced gasotransmitter, hydrogen sulfide (H2 S), is a novel molecule that mitiga

117 a the reaction of the gaseous biotransmitter hydrogen sulfide (H2S) and disulfides (RSSR) and/or sulf

118 evidence suggests that the gaseous molecules hydrogen sulfide (H2S) and nitric oxide (NO) enhances pl

119 thionine-beta-synthase (CBS) and its product hydrogen sulfide (H2S) are aberrantly upregulated in col

120 Gaseous signaling molecules such as hydrogen sulfide (H2S) are produced endogenously and med

121 We identified the gaseous molecule hydrogen sulfide (H2S) as the major effector molecule dr

122 , are believed to be chiefly responsible for hydrogen sulfide (H2S) biogenesis.

123 to a significant down-regulation of cellular hydrogen sulfide (H2S) by 50% and of glutathione (GSH) b

124 acrophage inducible NOS was inhibited by the hydrogen sulfide (H2S) donor Na2S with IC50 values of ap

125 Hydrogen sulfide (H2S) elicits pleiotropic physiological

126 Hydrogen sulfide (H2S) emissions were determined from an

127 Hydrogen sulfide (H2S) exhibits beneficial effects in th

128 a new biological process for the removal of hydrogen sulfide (H2S) from low pressure biogas, the nee

129 A new sensor for the detection of hydrogen sulfide (H2S) gas has been developed to replace

130 provide strong evidence for the role of the hydrogen sulfide (H2S) gas in the protective effects of

131 Hydrogen sulfide (H2S) has been increasingly recognized

132 Hydrogen sulfide (H2S) has been observed in relatively h

133 Hydrogen sulfide (H2S) has been recognized as a signalli

134 Hydrogen sulfide (H2S) has emerged as the most important

135 Hydrogen sulfide (H2S) has long been known as a toxic ga

136 rapeutic manipulation of the gasotransmitter hydrogen sulfide (H2S) has recently been proposed as a n

137 Hydrogen sulfide (H2S) has recently been recognized as a

138 Hydrogen sulfide (H2S) impairs mitochondrial respiration

139 (CSE) is one of the major enzymes producing hydrogen sulfide (H2S) in lungs, participating in the re

140 We sought to investigate the role of hydrogen sulfide (H2S) in regulating vasomotor tone in t

141 Cystathionine gamma-lyase (CSE) produces hydrogen sulfide (H2S) in the cardiovascular system.

142 is study was designed to examine the role of hydrogen sulfide (H2S) in the generation of oxidized low

143 The physiological functions of hydrogen sulfide (H2S) include vasorelaxation, stimulati

144 Because hydrogen sulfide (H2S) inhibits high glucose-induced mat

145 We have previously shown that hydrogen sulfide (H2S) inhibits high glucose-induced pro

146 Hydrogen sulfide (H2S) is a biologically important small

147 Hydrogen sulfide (H2S) is a critical gaseous signaling m

148 Hydrogen sulfide (H2S) is a gaseous mediator that promot

149 Hydrogen sulfide (H2S) is a hazardous gas, which not onl

150 We now show that hydrogen sulfide (H2S) is a physiologic gasotransmitter

151 Hydrogen sulfide (H2S) is a reactive small molecule gene

152 Hydrogen sulfide (H2S) is a signaling molecule that is t

153 Hydrogen sulfide (H2S) is an attractive agent for myocar

154 Hydrogen sulfide (H2S) is an endogenous gaseous mediator

155 Hydrogen sulfide (H2S) is an endogenous gasotransmitter

156 Hydrogen sulfide (H2S) is an endogenously produced gaseo

157 Hydrogen sulfide (H2S) is an essential biological signal

158 Hydrogen sulfide (H2S) is an important biological mediat

159 Hydrogen sulfide (H2S) is an important biological signal

160 Hydrogen sulfide (H2S) is an important biological signal

161 The gasotransmitter hydrogen sulfide (H2S) is an important tuner of the card

162 Hydrogen sulfide (H2S) is an integral signaling molecule

163 Hydrogen sulfide (H2S) is increasingly recognized to mod

164 Hydrogen sulfide (H2S) is now recognized as a physiologi

165 Hydrogen sulfide (H2S) is now recognized as an important

166 Hydrogen sulfide (H2S) is produced by yeast during winem

167 Hydrogen sulfide (H2S) is produced endogenously in vivo

168 cent experimental studies have revealed that hydrogen sulfide (H2S) is produced enzymatically in all

169 The gasotransmitter, hydrogen sulfide (H2S) is recognized as an important med

170 Hydrogen sulfide (H2S) is the most abundant gas-phase sp

171 Hydrogen sulfide (H2S) is the most recently accepted end

172 Hydrogen sulfide (H2S) is the third biological gasotrans

173 Hydrogen sulfide (H2S) plays important roles in the ente

174 enocarcinoma tissue expresses high levels of hydrogen sulfide (H2S) producing enzymes, namely, cystat

175 ne gamma-lyase (CGL), resulting in increased hydrogen sulfide (H2S) production and protection from he

176 we tested the hypothesis that restoration of hydrogen sulfide (H2S) production in diabetic BMCs impro

177 Previous studies have demonstrated that hydrogen sulfide (H2S) protects against multiple cardiov

178 Hydrogen sulfide (H2S) regulates various physiological p

179 Endogenous hydrogen sulfide (H2S) renders bacteria highly resistant

180 O2 sensing by carbon monoxide (CO)-sensitive hydrogen sulfide (H2S) signaling contribute to reflex va

181 ed memory impairment in brain is mediated by hydrogen sulfide (H2S) synthesized by cystathionine beta

182 Carbon monoxide (CO) and hydrogen sulfide (H2S) used to be thought of simply as l

183 Hydrogen sulfide (H2S) was discovered as a third gasotra

184 Here we found that hydrogen sulfide (H2S) was required for Foxp3(+) Treg ce

185 ido-aza-BODIPY 3a selectively interacts with hydrogen sulfide (H2S) when compared to other molecules.

186 e hydrolisis of sodium thiophosphate (TP) to hydrogen sulfide (H2S) which in the presence Cd(2+) ions

187 Hydrogen sulfide (H2S), a cardioprotective gas, is endog

188 Hydrogen sulfide (H2S), a gaseous messenger produced mai

189 Hydrogen sulfide (H2S), a gaseous species produced by bo

190 Hydrogen sulfide (H2S), a novel gasotransmitter, is endo

191 Hydrogen sulfide (H2S), a small signaling gas molecule i

192 Hydrogen sulfide (H2S), an endogenously produced small m

193 ns emit odorant chemicals including ammonia, hydrogen sulfide (H2S), and volatile organic compounds.

194 ulfur dioxide (SO2), nitrogen dioxide (NO2), hydrogen sulfide (H2S), formaldehyde (HCHO), carbon mono

195 fur network generate the signaling molecule, hydrogen sulfide (H2S), from the amino acids cysteine an

196 chemical approach to the direct detection of hydrogen sulfide (H2S), in aqueous solutions, covering a

197 ling molecule and the subject of this study, hydrogen sulfide (H2S), is much less known.

198 Precursors to hydrogen sulfide (H2S), methanethiol (MeSH), ethanethiol

199 with lower wine pH associated with decreased hydrogen sulfide (H2S), methanethiol, dimethyl sulfide,

200 The relationship between hydrogen sulfide (H2S), microRNAs (miRs), matrix metallo

201 fering gas species, including ammonia (NH3), hydrogen sulfide (H2S), nitric oxide (NO), carbon mono-o

202 rine acidification dynamics based on oxygen, hydrogen sulfide (H2S), pH, dissolved inorganic carbon a

203 l-containing compounds, including thiols and hydrogen sulfide (H2S), play important but differential

204 t signaling molecules, nitric oxide (NO) and hydrogen sulfide (H2S), proceeds via elusive mechanism(s

205 Additionally, we reveal that hydrogen sulfide (H2S), rather than sulfide, was likely

206 Buildup of hydrogen sulfide (H2S), which functions as a signaling m

207 a higher number of sulfane sulfur atoms than hydrogen sulfide (H2S), which has various physiological

208 ftenberg isolates, including 17 atypical non-hydrogen sulfide (H2S)-producing isolates, were detected

209 the actual signaling molecules derived from hydrogen sulfide (H2S).

210 nitric oxide (NO), carbon monoxide (CO), and hydrogen sulfide (H2S).

211 Recently, hydrogen sulfide has been acknowledged as a gasotransmit

212 We prepared microbubbles carrying hydrogen sulfide (hs-MB) with different H2S/C3F8 ratios

213 erapeutic effect and mechanisms of action of hydrogen sulfide in an animal model of sFlt1-induced hyp

214 l for tracking the variation of bisulfide or hydrogen sulfide in extracellular fluids.

215 e in our understanding of the role played by hydrogen sulfide in life processes.

216 c heme group and catalyzes the production of hydrogen sulfide in mammalian cells.

217 previous findings, demonstrates the role of hydrogen sulfide in regulating autophagy.

218 sess the direct anti-inflammatory effects of hydrogen sulfide in vivo, we used a peritonitis model by

219 Hydrogen sulfide is a cardioprotective signaling molecul

220 Hydrogen sulfide is a critical signaling molecule, but h

221 Hydrogen sulfide is a highly reactive molecule that is c

222 Hydrogen sulfide is a highly toxic gas-second only to ca

223 Hydrogen sulfide is a newly identified modulator of prot

224 Hydrogen sulfide is a vasorelaxant and proangiogenic gas

225 Hydrogen sulfide is an essential catabolite that interve

226 Hydrogen sulfide is an essential gasotransmitter associa

227 Hydrogen sulfide is an important biological signaling mo

228 Hydrogen sulfide is an important endogenous mediator tha

229 Hydrogen sulfide is detected by UV-assisted quantitative

230 Hydrogen sulfide is found in many environments including

231 Accordingly, no direct metabolic effect of hydrogen sulfide is observed in large mammals.

232 , and ethanolamine, while in the presence of hydrogen sulfide, L-serine yielded L-cysteine.

233 ching between low steady-state intracellular hydrogen sulfide levels and the higher concentrations wh

234 Hydrogen sulfide maintains differentiated smooth muscle

235 ntly argued that a moderate level of inhaled hydrogen sulfide may have a potential benefit in patient

236 e have examined the underlying mechanisms of hydrogen sulfide-mediated lung protection and determined

237 activated receptor gamma inhibitor abolished hydrogen sulfide-mediated protection in ventilated anima

238 Hydrogen sulfide-mediated protection was associated with

239 man (patho)physiology with a central role in hydrogen sulfide metabolism.

240 th anaerobic biostimulation processes (e.g., hydrogen sulfide, methane production) during full-scale

241 The five examined thiols were hydrogen sulfide, methanethiol, trifluoromethanethiol, t

242 dehyde, acetone, acetic acid, hexanoic acid, hydrogen sulfide, methanol, and phenol-were found to be

243 A novel hydrogen sulfide microbial biosensor was developed based

244 In plants, hydrogen sulfide mitigates stress and regulates importan

245 We hypothesized that hydrogen sulfide modulates high glucose (30 mm) stimulat

246 rough the production of acetate, methane, or hydrogen sulfide modulates the efficiency of energy extr

247 ith LEVC, the potency and efficacy of sodium hydrogen sulfide (NaHS) (10-1000 muM) on K(ATP) currents

248 re able to detect H(2)S produced from sodium hydrogen sulfide (NaHS) added at concentrations as low a

249 between sulfur oxides, SOn (n = 1, 2, 3) and hydrogen sulfide (nH2S), resulting in the efficient form

250 s at mid-depths where acids are produced via hydrogen sulfide oxidation in waters mixed upward from a

251 directly deliver a single species, including hydrogen sulfide, perthiol, and COS, and will be very us

252 We assert that hydrogen sulfide plays an important role in regulating m

253 ised by the development of novel fluorescent hydrogen sulfide probes.

254 ls independently of Atf4 expression, whereas hydrogen sulfide production is promoted via GCN2-ATF4 pa

255 sfunction reflected by a failure to maintain hydrogen sulfide production or apolipoprotein B100 (ApoB

256 other oral surfaces, and stool; hydrogen and hydrogen sulfide production were also differentially dis

257 hereas those encoding for methanogenesis and hydrogen sulfide production were higher in native Africa

258 nd the rate of COS uptake (or, by inference, hydrogen sulfide production).

259 duction of placental CSE activity, decreased hydrogen sulfide production, and smooth muscle cell dedi

260 The deletion strains have lost hydrogen sulfide production, nitrate reductase activity,

261 e stimulation, desulfoviridin, motility, and hydrogen sulfide production, plus susceptibility to anti

262 ccumulation of CBS in mitochondria increased hydrogen sulfide production, which prevented Ca(2+)-medi

263 Hydrogen sulfide reacts rapidly and quantitatively at pp

264 Hydrogen sulfide reduces ventilator-induced lung injury

265 from post-translational processes involving hydrogen sulfide-related chemistry.

266 f Fe(II) in Fe(III)-IL is beneficial for the hydrogen sulfide removal and the electrochemical regener

267 To improve the hydrogen sulfide removal efficiency with the application

268 Hydrogen sulfide removal is a long-standing economic and

269 bacterial gasotransmitters, nitric oxide and hydrogen sulfide represent.

270 ne to form cystathionine and either water or hydrogen sulfide, respectively.

271 Buzzing with activity: A hydrogen sulfide selective fluorogenic probe, 7-azido-4-

272 al blood flow via oxidative stress, reducing hydrogen sulfide signaling.

273 ons and volatile organic compounds, ammonia, hydrogen sulfide, sulfur dioxide and hydrogen are then d

274 report offers an insight into the profile of hydrogen sulfide synthesis during inflammation and highl

275 animal imaging, to monitor the regulation of hydrogen sulfide synthesis in vivo during dynamic inflam

276 granulocyte infiltration negatively affects hydrogen sulfide synthesis.

277 two distinct methods of measuring leukocyte hydrogen sulfide synthesis; methylene blue formation fol

278 designed to examine roles of the endogenous hydrogen sulfide synthesizing enzyme cystathionine beta-

279 e that physiologic modification of parkin by hydrogen sulfide, termed sulfhydration, enhances its cat

280 en conducted on the physiological effects of hydrogen sulfide, the underlying mechanisms are poorly u

281 in oil reservoirs produces toxic, corrosive hydrogen sulfide through microbial sulfate reduction, of

282 The gut normally converts ambient hydrogen sulfide to thiosulfate, which it then oxidizes

283 zable sulfur while mitigating the effects of hydrogen sulfide toxicity is poorly understood.

284 One hydrogen sulfide-triggered mechanism in the protection a

285 coupons were exposed to different levels of hydrogen sulfide under well-controlled conditions in lab

286 Prodrugs that release hydrogen sulfide upon esterase-mediated cleavage of an e

287 When hydrogen sulfide was added after 12 h of anoxia and low

288 Hydrogen sulfide was found to be strongly inhibitory to

289 The subsequent addition of hydrogen sulfide, water, methanol, hydroxylamine, cyanam

290 Because of the anti-inflammatory effects of hydrogen sulfide, we proposed that the hydrogen sulfide

291 uch as thioglycosides, thioacids, and sodium hydrogen sulfide were also alkynylated successfully to l

292 The effects of hydrogen sulfide were analyzed in a mouse ventilator-ind

293 rs (SMBs) nitric oxide, carbon monoxide, and hydrogen sulfide were initially viewed as environmental

294 d, likely due to the microbial production of hydrogen sulfide which favors stabilization of metals in

295 (MGL) catalyzes the decomposition of Hcy to hydrogen sulfide which forms fluorescent CdS nanoparticl

296 ulfate reducing microorganisms (SRM) produce hydrogen sulfide which is toxic, explosive, and corrosiv

297 cean, often accompanied by the production of hydrogen sulfide, which is toxic to higher organisms.

298 dontal pathogen Treponema denticola produces hydrogen sulfide, which may play a role in the host tiss

299 erial to parts-per-billion concentrations of hydrogen sulfide with potential applications in industri

300 Such prodrugs directly release hydrogen sulfide without the involvement of perthiol spe