ライフサイエンスコーパス: hydronephrosis

1 fluid-filled due to the severe polyuria and hydronephrosis.

2 obstruction of urine outflow and death from hydronephrosis.

3 strate the predominant role of cPLA2alpha in hydronephrosis.

4 ching or in distal ureter maturation, and no hydronephrosis.

5 with associated hydroureter, megaureter and hydronephrosis.

6 n between the expression of these miRNAs and hydronephrosis.

7 genes in mutant ureters before the onset of hydronephrosis.

8 e cystic kidney disease, renal fibrosis, and hydronephrosis.

9 indicated in infants with isolated antenatal hydronephrosis.

10 he ureter and this is followed by congenital hydronephrosis.

11 ued ectopic Ptch2 expression and obstructive hydronephrosis.

12 ary tract was affected by diverticulosis and hydronephrosis.

13 logy increases with the severity of prenatal hydronephrosis.

14 s of alkaline phosphatase, urolithiasis, and hydronephrosis.

15 ation of the child with prenatally diagnosed hydronephrosis.

16 of bladder, and the development of end-stage hydronephrosis.

17 h wild-type mice, despite similar degrees of hydronephrosis.

18 elvic junction obstruction, a common form of hydronephrosis.

19 been most often applied to the evaluation of hydronephrosis.

20 ations may contribute to hereditary forms of hydronephrosis.

21 onal knockout mice have renal hypoplasia and hydronephrosis.

22 ruction, poor renal function and significant hydronephrosis.

23 structed, thereby protecting the kidney from hydronephrosis.

24 ereas non-surviving null mice exhibit marked hydronephrosis.

25 tomotic stenosis (6.8% vs. 0.4%, P=0.02) and hydronephrosis (12.9% vs. 5.3%, P=0.02) in the SKT cohor

26 e from the kidney to the bladder can lead to hydronephrosis, a common birth defect associated with ob

27 cess can result in malpositioned ureters and hydronephrosis, a common cause of renal disease in child

28 e Pax3-Cre transgene) resulted in congenital hydronephrosis accompanied by reduced branching, abnorma

29 uplex and multiplex kidneys, renal agenesis, hydronephrosis and cystic kidney disease.

30 Analysis of Ret mutant mice at birth reveals hydronephrosis and defective ureter maturation, abnormal

31 esoderm-derived renal progenitors results in hydronephrosis and failure to develop a patent pelvic-ur

32 gressive renal hemosiderosis concurrent with hydronephrosis and glomerulonephritis.

33 can of the chest, abdomen, and pelvis showed hydronephrosis and hydroureter with marked cortical thin

34 ult of obstruction without manifestations of hydronephrosis and it can be confused with leakage from

35 occur in humans, including renal hypoplasia, hydronephrosis and mega-ureter, abnormalities also seen

36 aneous recessive mutation that causes severe hydronephrosis and obstructive nephropathy in affected m

37 Three patients had mild hydronephrosis and one had distal hydroureter; these con

38 efective pacemaker cell differentiation with hydronephrosis and provide a cellular basis for one of t

39 s most commonly applied to the evaluation of hydronephrosis and provides valuable insight into a wide

40 nically relevant abnormality (eg, unilateral hydronephrosis and/or urolithiasis in patients with an o

41 ficient mice exhibit a dilated renal pelvis (hydronephrosis) and a small papilla.

42 rifices resulting in the back flow of urine, hydronephrosis, and altered renal function indicators.

43 ular peritoneal thickening, lymphadenopathy, hydronephrosis, and bowel obstruction were less common a

44 s sharply reduced, apparently as a result of hydronephrosis, and fractional excretion of electrolytes

45 sents early with intestinal hypoperistalsis, hydronephrosis, and hydroureters.

46 -fold increased sensitivity to cleft palate, hydronephrosis, and lethality.

47 truncation, vertebral segmentation defects, hydronephrosis, and limb hypoplasia, resembling humans w

48 ing late gestation expansion of the bladder, hydronephrosis, and rapid demise after parturition.

49 sent with enuresis, urinary-tract infection, hydronephrosis, and voiding dysfunctions as a result of

50 treating children with a history of prenatal hydronephrosis are included in this review.

51 The underlying causes of functional hydronephrosis are not well understood.

52 aluation of the CT studies by using proximal hydronephrosis as a criterion for minimal invasion impro

53 sensitive to dioxin-induced cleft palate and hydronephrosis as compared with embryos from an Ahr(+/+)

54 genesis and function causing hydroureter and hydronephrosis at birth.

55 at, when abnormal, can cause obstruction and hydronephrosis at birth; whether ND defects underlie sim

56 caused bilateral UPJ obstruction and severe hydronephrosis beginning at embryonic day 17.5.

57 arge tumor suppressor, results in congenital hydronephrosis characterized by urinary tract abnormalit

58 miR-143/145-deficient mice developed hydronephrosis, characterized by severe papillary atroph

59 Congenital progressive hydronephrosis (cph) is a spontaneous recessive mutation

60 onounced glomerulosclerosis was evident, and hydronephrosis developed with low penetrance.

61 o lack of mature germ cells, and 50% develop hydronephrosis due to ureteral hyperplasia.

62 rvals to detect complications like calculus, hydronephrosis, etc.

63 did not enable discrimination of obstructive hydronephrosis from unobstructive hydronephrosis in chil

64 more commonplace, the management of neonatal hydronephrosis has become one of the most interesting an

65 cell-mediated ureteritis, leading to kidney hydronephrosis (hereafter called acetate-induced renal d

66 Dlg1-null mice exhibit hydronephrosis, hydroureter, and occasionally hypoplasti

67 om bladder epithelial cells causing prenatal hydronephrosis, hydroureter, and vesicoureteric reflux.

68 CBA) dams led to cleft palate in only 8% and hydronephrosis in 69% of embryos.

69 examination revealed the presence of severe hydronephrosis in almost all animals, affecting lean as

70 Kidney hydronephrosis in C2RD was caused by ureteral obstructio

71 bstructive hydronephrosis from unobstructive hydronephrosis in children.

72 influence the incidence of cleft palate and hydronephrosis in developing mice exposed to the polluta

73 ound examination revealed a hilar mass, with hydronephrosis in five and stenosis of renal vessels in

74 ing of the molecular basis of nonobstructive hydronephrosis in humans.

75 rodibenzo-p-dioxin) induces cleft palate and hydronephrosis in mice, when exposed in utero; these eff

76 urinary concentrating ability, polyuria, and hydronephrosis in mice.

77 benzo-p-dioxin (TCDD) induces nonobstructive hydronephrosis in mouse neonates through upregulation of

78 yonic day 9 (E9) led to palatal clefting and hydronephrosis in nearly 100% of embryos by E17.

79 areful observation of patients with moderate hydronephrosis in well functioning kidneys.

80 The diagnosis of prenatal hydronephrosis, indications for and timing of prenatal i

81 Hydronephrosis is a common disease characterized by dila

82 Prenatal hydronephrosis is diagnosed with an incidence of 1:100 t

83 The natural history of prenatal hydronephrosis is difficult to determine, and therefore

84 nts with a history of any degree of prenatal hydronephrosis is routine, but ascertaining which patien

85 Fetal hydronephrosis is the most common anomaly detected on an

86 hibited severe polyuria (10 ml/day), extreme hydronephrosis, low plasma potassium, high blood pH, hyp

87 ction must be treated, it is also clear that hydronephrosis may well exist without significant obstru

88 de VUR (n = 2), renal dysplasia (n = 2), and hydronephrosis (n = 1).

89 dings were cholelithiasis (n=3), obstructive hydronephrosis (n=1), small-bowel dilatation (n=1), and

90 , high plasma potassium, metabolic acidosis, hydronephrosis of varying severity, and high plasma reni

91 as defined as stable serum creatinine and no hydronephrosis on follow-up.

92 ts between kidneys with increasing degree of hydronephrosis (P > .5).

93 l reflux (P=0.02) and four of four developed hydronephrosis (P=0.002).

94 CT findings (tissue rim sign, hydronephrosis, perinephric fat stranding, perinephric f

95 culus increases the risk of hematuria and/or hydronephrosis, presenting with colicky pain as in the p

96 sitional cell carcinoma of the renal pelvis, hydronephrosis proximal to the tumor may cause overstagi

97 The postnatal management of prenatal hydronephrosis remains controversial.

98 knockouts experienced vesicoureteral reflux, hydronephrosis, renal dysfunction, and, in the offspring

99 esence of a tissue rim sign and the grade of hydronephrosis, renal fascial thickening, and renal pare

100 cedure-related major complications occurred (hydronephrosis requiring nephrostomy due to gross hematu

101 machinery in the mutants before the onset of hydronephrosis suggest that the congenital obstructive n

102 One patient had mild hydronephrosis that resolved.

103 have no renin mRNA expression in the kidney, hydronephrosis, thickening of renal arterial walls, and

104 , were hypertension (13 [7%] vs eight [4%]), hydronephrosis (three [2%] vs seven [4%]), back pain (fi

105 reening has changed from simple detection of hydronephrosis to selection for specific diagnosis-based

106 ation of the pathway leading to the onset of hydronephrosis using the TCDD-exposed mouse model will d

107 indomethacin-rescued -/- adults except that hydronephrosis was mild.

108 The incidence of cleft palate and hydronephrosis was not significantly different in fetuse

109 Proximal hydronephrosis was present in 80% of overstaged cases (e

110 The incidences of cleft palate and hydronephrosis were assessed and genomic DNA from embryo

111 Narrowed ureters with hydronephrosis were found only in the Tl1a transgenic mi

112 dred twenty-six children suspected of having hydronephrosis were hydrated prior to undergoing both co

113 The impaired bladder function and subsequent hydronephrosis were secondary to involvement of the cent

114 Young null mutants had hydronephrosis, were severely dehydrated, and approximat

115 l morphology with no evidence of significant hydronephrosis, whereas non-surviving null mice exhibit

116 ry tract abnormalities, including congenital hydronephrosis, which is the leading cause of renal fail

117 renal/ureteral duplication, hydroureter, and hydronephrosis, which were visible prenatally.

118 n of all children with a history of prenatal hydronephrosis with a voiding cystourethrogram may reduc

119 ren with a history of any degree of prenatal hydronephrosis with an ultrasound and voiding cystoureth

120 red with degree of obstruction and degree of hydronephrosis with analysis of variance.

121 canning 5 weeks posttransplant revealed mild hydronephrosis with several parenchymal cystic areas mea

122 ent date, age, carcinoma in situ status, and hydronephrosis) with propensity scores to patients who u

123 of Six1 in the ureter led to hydroureter and hydronephrosis without anatomical obstruction when kidne

124 ction is the most common cause of congenital hydronephrosis, yet the underlying pathogenesis is undef