ライフサイエンスコーパス: hydroquinone

1 erivatives such as catechol, resorcinol, and hydroquinone.

2 formation of two tautomeric forms of emodin hydroquinone.

3 f GS-trichloro-p-hydroquinone to trichloro-p-hydroquinone.

4 ore rigid semiquinone and to the much looser hydroquinone.

5 the inhibition of tube formation induced by hydroquinone.

6 uction, yielding a glutathiylated conjugated hydroquinone.

7 se effects induced by the benzene metabolite hydroquinone.

8 (red)), and (iii) two-electron fully reduced hydroquinone.

9 and the conversion of vitamin K to vitamin K hydroquinone.

10 g group from an appropriately functionalized hydroquinone.

11 and the conversion of vitamin K to vitamin K hydroquinone.

12 elatively fast reaction of the reagent and p-hydroquinone.

13 lly oxidizing alkaline phosphatase-generated hydroquinone.

14 oth an unstable neutral blue semiquinone and hydroquinone.

15 ster electron transfer affords the colorless hydroquinone.

16 none with a more negative potential than the hydroquinone.

17 o(salophen)-catalyzed aerobic oxidation of p-hydroquinone.

18 intermediate oxidizes a second equivalent of hydroquinone.

19 gher than those observed in solutions of 1,4-hydroquinone.

20 f magnitude greater than in solutions of 1,4-hydroquinone.

21 in solutions of simple quinones such as 1,4-hydroquinone.

22 m the oxidized quinone to the doubly reduced hydroquinone.

23 athionyl-hydroquinones (GS-hydroquinones) to hydroquinones.

24 ition, and GS-HQRs convert the conjugates to hydroquinones.

25 clusively upon reaction with monosubstituted hydroquinones.

26 sence of GS-HQRs suggests they use common GS-hydroquinones.

27 ydroquinones and then enzymatic reduction to hydroquinones.

28 ed the GS-hydroquinones to the corresponding hydroquinones.

29 se in the oxidizability of these chlorinated hydroquinones.

30 duction of quinones and quinoid compounds to hydroquinones.

31 etic mechanism, generating the corresponding hydroquinones.

32 PcpA (2,6-dichloro-p-hydroquinone 1,2-dioxygenase) from Sphingobium chlorophe

33 e propose a general reaction mechanism for p-hydroquinone 1,2-dioxygenases.

34 ts, is a member of the recently discovered p-hydroquinone 1,2-dioxygenases.

35 xposure to the benzene metabolites catechol, hydroquinone, 1,2,4-benzenetriol, and p-benzoquinone.

36 olites of benzene, namely, phenol, catechol, hydroquinone, 1,2,4-trihydroxybenzene (trihydroxybenzene

37 uinone unexpectedly gave a monoalkylated 1,4-hydroquinone/1,4-benzoquinone electron donor-acceptor co

38 hich is chemically reduced to 2,5-dichloro-p-hydroquinone (2,5-DiCHQ).

39 This study focused on 2,6-dimethoxy hydroquinone (2,6-DMHQ), an analogue to a common fungal

40 ns to generate tert-butanol and 1-acetyl-1,4-hydroquinone, 8, apparently by an SN1 mechanism.

41 oxygeldanamycin (17AAG) to the corresponding hydroquinone, a more potent 90-kDa heat shock protein (H

42 an overall 2 e-, 2 H+ reduction to make the hydroquinone, a much better description of the overall r

43 A tight hydrophobic pocket provides p-hydroquinones access to the Fe(II) centre.

44 alternative peroxidase substrates, such as p-hydroquinone, acetaminophen, anticancer mitoxantrone, an

45 Hydroquinone also caused an increase in Ten Eleven Trans

46 ulation by reducing vitamin K epoxide to the hydroquinone, an essential cofactor for the gamma-glutam

47 en appears to be first-order each in ionized hydroquinone and dioxygen, yielding hydrogen peroxide st

48 proton across the hydrogen bond between the hydroquinone and His154 at the cytochrome b site.

49 atechol), their 1,4-dihydroxyphenyl isomers (hydroquinone and homogentisic acid) are able to modify a

50 ansforms p-nitrophenol stoichiometrically to hydroquinone and hydroxyquinol.

51 e capable of cleaving the aromatic ring of p-hydroquinone and its substituted variants, is a member o

52 A novel hydroquinone and NADH oxidase with protein disulfide-thi

53 otential of approximately 110 mV between the hydroquinone and semiquinone state.

54 ct, and a hydrogen-bonded adduct between the hydroquinone and the Co(III)-O2 species.

55 nterface by the reduction of benzoquinone to hydroquinone and the resulting interfacial pH change is

56 e that could accommodate various substituted hydroquinones and a hydrogen network of three Tyr residu

57 Our findings suggest that hydroquinones and benzoquinones, which are interchangeab

58 y laccase-catalyzed domino reactions between hydroquinones and cyclic 1,3-dicarbonyls using aerial ox

59 This study showed that reactions between hydroquinones and iron oxides could produce favorable co

60 l roles: channeling GS-hydroquinones back to hydroquinones and reducing benzoquinones via spontaneous

61 d the structural aspects of stabilization of hydroquinones and their ability to generate reactive oxy

62 enzoquinones via spontaneous formation of GS-hydroquinones and then enzymatic reduction to hydroquino

63 phenols and solvent water adducts (catechol, hydroquinone) and ammonium ion.

64 FMN N5, the anionic ionization state of the hydroquinone, and for a change in the hybridization stat

65 Hydroquinone, and its mercapturic acid pathway metabolit

66 ate derivative 5,6-isopropylidine ascorbate, hydroquinone, and the hydroxylamine TEMPOH all rapidly a

67 he electronic structure of the fully reduced hydroquinone anionic state, FlH(-), however, has been th

68 ner, demonstrating increased efficacy of the hydroquinone ansamycin relative to its parent quinone.

69 uinone ansamycins are metabolized by NQO1 to hydroquinone ansamycins and that Hsp90-mediated trans-ci

70 hat both trans- and cis-amide isomers of the hydroquinone ansamycins exhibited increased binding affi

71 recombinant human NQO1 to the corresponding hydroquinone ansamycins was monitored by high-performanc

72 QO1, was used, and HPLC analysis showed that hydroquinone ansamycins were formed by the MDA468/NQ16 c

73 sm-based inhibitor of NQO1, showing that the hydroquinone ansamycins were more potent Hsp90 inhibitor

74 samycins by NQO1 generates the corresponding hydroquinone ansamycins, which exhibit enhanced Hsp90 in

75 itional contacts between yeast Hsp90 and the hydroquinone ansamycins, which translated to greater int

76 ard a selective desymmetrization of the meso-hydroquinone are also reported.

77 Hydroquinones are important mediators of electron transf

78 being that the ortho adducts (o-aminophenol, hydroquinone) are formed in a higher ratio in comparison

79 e reactant and product, 1,4-benzoquinone and hydroquinone, are separated during the assay by differen

80 spray solvent flow rate and the addition of hydroquinone as a redox buffer to the spray solvent were

81 istic experiments support the role of flavin hydroquinone as a single electron reductant, flavin semi

82 ith a scan rate of 100 mV in the presence of hydroquinone as electron mediator and 0.1M phosphate buf

83 These conditions employing biorenewable hydroquinone as reagent were developed from initial expe

84 nto the electrode surface in the presence of hydroquinone as the redox mediator.

85 With unsubstituted hydroquinone as the substrate 2,3-disubstituted p-benzoq

86 hiol-hydroquinones, for example, S-cysteinyl-hydroquinone, as substrates.

87 In turn, benzoquinone is electroreduced into hydroquinone at the electrode.

88 potential physiological roles: channeling GS-hydroquinones back to hydroquinones and reducing benzoqu

89 c measurements confirm the formation of beta-hydroquinone (beta-HQ) clathrate with molecular hydrogen

90 FLEEL and vitamin K hydroquinone both stabilized the propeptide-carboxylase

91 ne with recombinant UBIAD1 revealed that the hydroquinone, but not the quinone form of menadione, was

92 a quinone with a thiol to form a substituted hydroquinone by reductive 1,4-Michael addition.

93 din, which is then converted to chrysophanol hydroquinone by the reductase ClaC and the dehydratase C

94 Selective reduction of quinones to hydroquinones by addition of small molar excesses of dit

95 inert but can be converted into photolabile hydroquinones by mild reduction in situ.

96 ive yield along with well-defined, colorless hydroquinone byproducts.

97 GS-hydroquinones can be spontaneously formed from benzoquin

98 iciency site (with 2,6-dimethoxyphenol and p-hydroquinone catalytic efficiencies of approximately 70

99 Hydroquinone caused reactivation of a methylated reporte

100 s treated with the Nrf2 activator tert-butyl hydroquinone, chromatin immunoprecipitation with Nrf2 an

101 A 2-coordinate Pd(0)-hydroquinone complex (4-H) was prepared using a one-pot

102 role in PCP degradation and converted the GS-hydroquinone conjugates back to the intermediates of the

103 te, likely due to the accumulation of the GS-hydroquinone conjugates inside the cell.

104 The fate and effect of GS-hydroquinone conjugates were unknown.

105 he elevated potential of the FMN semiquinone/hydroquinone couple (-172 mV) is also an adaptation that

106 semiquinone couple = +70 mV, FMN semiquinone/hydroquinone couple = -180 mV, and heme = -180 mV) indic

107 The potentials of the semiquinone/hydroquinone couple of both FMN and FAD are altered to a

108 ive carbon electrodes, combining the quinone/hydroquinone couple with the Br2/Br(-) redox couple, yie

109 is much higher than that of the semiquinone/hydroquinone couple.

110 The pK(a) of parent hydroquinone decreased by 1 unit as the degree of chlori

111 iron sulfur protein and an integral membrane hydroquinone dehydrogenase, respectively.

112 -type cytochrome that functions as the major hydroquinone dehydrogenase.

113 The hydroquinone detected in vitro is a dead-end product mos

114 -hydroquinone (TriCH) and then to dichloro-p-hydroquinone (DiCH) in the PCP degradation pathway.

115 esults provide mechanistic insights into the hydroquinone dioxygenases.

116 chol monophosphate, ascorbic 2-phosphate and hydroquinone diphosphate) that could previously not be u

117 5-hydroxymethylcytosine formation following hydroquinone exposure as well as the induction of glutam

118 This work demonstrates that hydroquinone exposure leads to active and functional DNA

119 tage of cells accumulating in G2+M following hydroquinone exposure, indicating that it may have a rol

120 tation the anionic semiquinone (FAD(*-)) and hydroquinone (FADH(-)) have longer lifetimes that are co

121 ur ET dynamics dictate that only the anionic hydroquinone flavin can be the functional state in photo

122 Azotobacter vinelandii flavodoxin hydroquinone (FldHQ) is a physiological reductant to nit

123 he target of warfarin and provides vitamin K hydroquinone for the carboxylation of select glutamic ac

124 potential of the flavin mononucleotide (FMN) hydroquinones for one-electron reduction in the Desulfov

125 (nonenzymatic) reduction of emodin to emodin hydroquinone, for example with sodium dithionite, is obl

126 did not use GS-benzoquinones or other thiol-hydroquinones, for example, S-cysteinyl-hydroquinone, as

127 ted pentad molecules, AQ is converted to its hydroquinone form (AQH2) via reversible intramolecular e

128 using the crystal structure of the relevant hydroquinone form and compared to the results of the Clo

129 odeling supported increased stability of the hydroquinone form of 19-phenyl-DMAG in the active site o

130 onalized surface is toggled from the reduced hydroquinone form to the oxidized benzoquinone form by t

131 is non-emissive, while upon reduction to the hydroquinone form, B-VKQH2, BODIPY fluorescence is resto

132 the monoanion, HQ(-), the kinetically active hydroquinone form, reducing Cu(II) with an intrinsic rat

133 thought to convert the alkoxide-epoxide to a hydroquinone form.

134 uently dihydroxylated, yielding the reduced (hydroquinone) form of sorgoleone.

135 current associated with the oxidation of the hydroquinone formed.

136 mical shifts of the BPP34C10, its internal p-hydroquinone forms pi-pi-stacking interactions with only

137 of the generation of a redox-labile cyclized hydroquinone, further demonstrating the lack of involvem

138 ir of redox-active substituents, quinone and hydroquinone groups, which allow the enantiomerization t

139 ne (GSH)-dependent reduction of glutathionyl-hydroquinones (GS-hydroquinones) to hydroquinones.

140 kinetics of reduction of benzoquinone (Q) to hydroquinone (H(2)Q) by the Os(IV) hydrazido (trans-[Os(

141 this end, a liquid battery is designed using hydroquinone (H2BQ) aqueous solution as catholyte and gr

142 es, the amperometric signal using the system hydroquinone/H2O2 was related to the levels of p53-autoa

143 (I) by O2 and the reduction of Cu(II) by 1,4-hydroquinone (H2Q) in the presence of O2 in 0.7 M NaCl s

144 cular cobalt complex, Co(salophen), and para-hydroquinone (H2Q) serve as effective cocatalysts for th

145 orresponding (two-electron) reduction to the hydroquinone (H2Q) via the Pauling bond-length/bond-orde

146 e, HAT= 1,4,5,8,9,12-hexaazatriphenylene) by hydroquinone (H2Q), N-acetyl-tyrosine (N-Ac-Tyr) or guan

147 article contact, the presence of the aqueous hydroquinone (H2Q)/benzoquinone (Q) couple in a flowing

148 Benzene and its metabolite hydroquinone have been shown to lead to decreased levels

149 rs sought to determine whether cytokines and hydroquinone (HQ) affect mCRP expression in cultured hum

150 hiafulvalene (BPTTF) and weaker aryl donors, hydroquinone (HQ) and 1,5-dioxynaphthalene (DNP), respec

151 es is studied using model phenolic compounds hydroquinone (HQ) and its oxidized counterpart benzoquin

152 the addition of H(2)O(2) in the presence of hydroquinone (HQ) as mediator was used as transduction s

153 upon the addition of H2O2 in the presence of hydroquinone (HQ) as mediator were used to monitor the e

154 rent arising upon addition of H2O2 and using hydroquinone (HQ) as redox mediator in solution.

155 upon the addition of H2O2 in the presence of hydroquinone (HQ) as redox mediator was used as the tran

156 abeled with horseradish peroxidase (HRP) and hydroquinone (HQ) as the redox mediator.

157 Hydroquinone (HQ) is one of the most frequently used and

158 the effect of nonlethal oxidant injury with hydroquinone (HQ) on MMP-2 activity.

159 ns only in the presence of additives such as hydroquinone (HQ) or tetrathiafulvalene (TTF).

160 Transient kinetics of hydroquinone (HQ) oxidation by wt-TcDyP showed that conv

161 ction was realized by spiking 10 microL of a hydroquinone (HQ) solution into 40 microL of buffer solu

162 tor and amperometric detection with the H2O2/hydroquinone (HQ) system at dual screen-printed carbon e

163 ion at -0.20V was carried out using the H2O2/hydroquinone (HQ) system.

164 We previously reported that hydroquinone (HQ), a major pro-oxidant in cigarette smok

165 ared from the reaction of bisphenol A (BPA), hydroquinone (HQ), and resorcinol (RS) in different solv

166 The gold standard for treatment is hydroquinone (HQ), which reduces pigmentation through it

167 none (sq), or the two-electron fully reduced hydroquinone (hq).

168 ins high concentrations of a potent oxidant, hydroquinone (HQ).

169 ectrode for amperometric detection using the hydroquinone (HQ)/H2O2 system.

170 synthesis and evaluation of a highly soluble hydroquinone hydrochloride derivative of 17-AAG, 1a (IPI

171 IPI-504, the highly soluble hydroquinone hydrochloride derivative of 17-AAG, was syn

172 adical and fully reduced neutral and anionic hydroquinones in solution.

173 itions, Aer cannot be further reduced to the hydroquinone, in contrast to the proposed Aer signaling

174 nones, and neutral and anionic fully reduced hydroquinones--in solution and in inert protein environm

175 tetramers consisting of four monomer units (hydroquinone, indolequinone, and its two tautomers), in

176 ent with anti-ChM-I siRNA markedly abrogated hydroquinone-induced inhibition of tube formation in TrH

177 ChM-I in TrHB-MECs and protected cells from hydroquinone-induced inhibition of tube formation.

178 d tube formation in TrHBMECs; NQO1 inhibited hydroquinone-induced up-regulation of ChM-I in TrHB-MECs

179 Hydroquinone inhibited TrHBMEC tube formation at concent

180 entrin A and herbindole B from a common meso-hydroquinone intermediate prepared by a ruthenium-cataly

181 d]C5(OH)[double bond]C4(OH)[bond] of PQQH(2) hydroquinone is assisted by general acid protonatation o

182 cleophilic attack in Michael-type additions, hydroquinone is not and acts as a passivating agent.

183 Hydroquinone is oxidized into benzoquinone by the HRP/H2

184 Since GS-trichloro-p-hydroquinone is uncommon in nature, the extensive presen

185 The carboxylase uses vitamin K hydroquinone (KH(2)) epoxidation to drive Glu carboxylat

186 farin-resistant enzyme reducing vitamin K to hydroquinone (KH(2)) is probably not NQO1, (3) there app

187 ch depends upon the oxygenation of vitamin K hydroquinone (KH2).

188 ertiary butyl phenol and monobenzyl ether of hydroquinone, known triggers of vitiligo.

189 istent with that expected of semiquinone and hydroquinone-like moieties respectively.

190 quinone group, such as 1,4-benzoquinone and hydroquinone, likely contributed to the transit toxicity

191 derivatives consisting of 1,4-disubstituted hydroquinones linked by methylene bridges in the 2,5-pos

192 chlorophenolicum made the mutant lose the GS-hydroquinone lyase activities in the cell extracts.

193 The GS-hydroquinone lyase reactions catalyzed by PcpF are rathe

194 Monobenzyl ether of hydroquinone (MBEH) is a Food and Drug Administration ap

195 ulture model to investigate the mechanism of hydroquinone-mediated changes in DNA methylation.

196 arbon counter electrodes, was facilitated by hydroquinone-mediated electron transfer.

197 e substrate (hydrogen peroxide) along with a hydroquinone mediator then allowed an enzymatic reaction

198 The quinone/semiquinone and semiquinone/hydroquinone midpoint potentials (E(q/sq) and E(sq/hq))

199 ic solutions and support the hypothesis that hydroquinone moieties can reduce Fe(III) in natural wate

200 on of covalent bonds by oxidizing unreactive hydroquinone moieties in LHA to reactive, electrophilic

201 ng electrons from electron-rich phenolic and hydroquinone moieties in the DOM, while O3 reacted via e

202 ne ligand was synthesized with a noninnocent hydroquinone moiety as the central arene (1-H).

203 rufin-derived phenoxyl radical by the drugs' hydroquinone moiety back to resorufin.

204 tion rather than direct redox cycling of the hydroquinone moiety is a source of adaphostin-induced RO

205 s an inner sphere Pd-based process where the hydroquinone moiety only subsequently participates in th

206 teraction of a paraquat beta-proton with the hydroquinone moiety; this is the first time this interac

207 nthesized on controlled pore glass using the hydroquinone-O,O'-diacetic acid linker.

208 of the cavitand by forming H-bonds with the hydroquinone OH groups.

209 ight into the content and impact of quinones/hydroquinones on the optical properties of HS and CDOM.

210 ely, the results show how Co(salophen) and p-hydroquinone operate synergistically to mediate O2 reduc

211 ted cell surface proteins that catalyze both hydroquinone or NADH oxidation and protein disulfide-thi

212 ants show little or no detectable effects on hydroquinone or quinone exchange and binding at the Q(o)

213 Using flavodoxin hydroquinone or reduced ferredoxin obtained by electron

214 rocenylmethyl)trimethylammonium cation and p-hydroquinone) or catechols (dopamine, epinephrine, norep

215 lamine, but a smaller population reacts with hydroquinone over the course of a 24 h exposure to the r

216 two catalytically active domains, termed the hydroquinone oxidation (Q(o)) and quinone reduction (Q(i

217 can also be widely varied and still support hydroquinone oxidation illustrate the considerable resil

218 physical and chemical environment supporting hydroquinone oxidation rates comparable to those seen in

219 osynthetic Rhodobacter capsulatus results in hydroquinone oxidation rates that are between 5 and 50-f

220 te between the arylamine N-hydroxylation and hydroquinone oxidation reactions.

221 by measuring the current required to reduce hydroquinone oxidized during the regeneration of the HRP

222 Quinone/hydroquinone PCET redox couples were used to produce a p

223 pted intact (hydroxylamine) and salt-washed (hydroquinone) photosystem II.

224 -catalyzed deoxyenation of the corresponding hydroquinone precursors is described.

225 tive hydroxyphenyl ester to an electroactive hydroquinone, providing an electrical activity that can

226 to varying degrees, ranging from changes in hydroquinone (QH(2)) oxidation and cyt c(1) reduction ki

227 idase (Q(o)) site in this complex oxidizes a hydroquinone (quinol), reducing two one-electron carrier

228 ) site catalysis of two-electron, two-proton hydroquinone-quinone oxidation-reduction.

229 pending on the degree of the chlorination of hydroquinone/quinone and the presence or absence of GSH.

230 The benzoquinone and hydroquinone redox couple was examined as a representati

231 The FMN semiquinone/hydroquinone redox couple was found to be similar in bot

232 Cyclic voltammetry (CV) of the quinone/hydroquinone redox couple was used to monitor the nucleo

233 Benzoquinone/hydroquinone redox interconversion by the reversible Os(

234 This slurry allows the 2e(-)/2H(+) quinone/hydroquinone redox reactions while suppressing proton re

235 anges in the stabilities of the oxidized and hydroquinone redox states of the FMN, none of the replac

236 ine, beta-naphthoflavone, and tertiary butyl hydroquinone reduced MIOX expression.

237 Our results show that 1,4-hydroquinone reduces Fe(III) in acidic conditions, gener

238 Glutathionyl-hydroquinone reductases (GS- HQRs) are a newly identifie

239 S-Glutathionyl-hydroquinone reductases (GS-HQRs) are a new class of glu

240 rmediates and by-products, including several hydroquinone, resorcinol and catechol derivatives, eithe

241 0 s(-1) mM(-1) for 2,6-dimethoxyphenol and p-hydroquinone, respectively) was localized at the main he

242 r reduction of benzoquinone and oxidation of hydroquinone, respectively.

243 ne steps, including the hydroxylation of the hydroquinone ring by CLK-1 and two O-methylation steps m

244 pillar[5]arene, a macrocycle containing five hydroquinone rings linked through their para positions b

245 ing compounds, including phenol, resorcinol, hydroquinone, serotonin, and ascorbic acid, had minimal

246 In contrast, monosubstituted hydroquinones show a limited amount of ring cleavage but

247 An excess of the hydroxylamine TEMPOH or of hydroquinone similarly reduces Fe(III)~CO(2), and TEMPO

248 , the semiquinone radicals generated in pure hydroquinone solution are rapidly oxidized by dioxygen,

249 proton-coupled electron transfer from a semi-hydroquinone species and a Co(III)-hydroperoxide interme

250 ne; in contrast, the anionic semiquinone and hydroquinone species were observed with the wild type an

251 tabilizing the vase form only in the reduced hydroquinone state of the cavitand by forming H-bonds wi

252 ction to either the ASQ or the fully reduced hydroquinone state produces the same conformational resp

253 ase form and are only present in the reduced hydroquinone state.

254 whereas the excited anionic semiquinone and hydroquinone states donate an electron to the adenine mo

255 at a higher rate in both the semiquinone and hydroquinone states.

256 eutral, and anionic semiquinone, and anionic hydroquinone states.

257 Two merotriterpenoid hydroquinone sulfates designated adociasulfate-13 (1) an

258 ing chemical reaction as demonstrated by the hydroquinone tagging of an initially disulfide-linked pe

259 as developed for determination of tert-butyl-hydroquinone (TBHQ) in edible vegetable oils, based on C

260 ane fusion and virus infectivity, tert-butyl hydroquinone (TBHQ), shows that the inhibitor binds in a

261 utylated hydroxytoluene (BHT) and tert-butyl hydroquinone (TBHQ), were determined in different edible

262 of a photocatalyst (2,3,5,6-tetrachloro-1,4-hydroquinone, TCHQ) via reaction of CA with the solvent.

263 One of the enzymes is tetrachloro-p-hydroquinone (TeCH) reductive dehalogenase (PcpC), which

264 K 2,3-epoxide to vitamin K and to vitamin K hydroquinone, the latter required by the enzyme gamma-ca

265 termined the effect of substrates: vitamin K hydroquinone, the pentapeptide FLEEL, and NaHCO(3), on t

266 Reduction of quinones to hydroquinones, the major redox active moieties in NOM, i

267 he O(2)-dependent, two-electron oxidation of hydroquinones, the protein was reprogrammed to catalyse

268 The asymmetric reduction of emodin hydroquinone to (R)-3,8,9,10-tetrahydroxy-6-methyl-3,4-d

269 eir corresponding radicals ArO(*) and TEMPO, hydroquinone to benzoquinone, and dihydroanthracene to a

270 pressure-corrected diffusion coefficient of hydroquinone to demonstrate the effect of this phenomeno

271 gulation of ChM-I may explain the ability of hydroquinone to inhibit TrHB-MEC tube formation.

272 ructural basis of electron transfer from FMN-hydroquinone to its partners, three deletion mutants in

273 e of formal potential on pH and oxidation of hydroquinone to quinone by O(2) at basic pH.

274 ation, cytochrome bc(1) efficiently oxidizes hydroquinone to quinone, but how it performs this reacti

275 epoxide reductase (VKOR) generates vitamin K hydroquinone to sustain gamma-carboxylation of many bloo

276 (GSH)-dependent reduction of GS-trichloro-p-hydroquinone to trichloro-p-hydroquinone.

277 n shown to catalyze the oxidation of various hydroquinones to benzoquinones in the presence of t-BuOO

278 e to air, consistent with the reoxidation of hydroquinones to quinones.

279 -HQRs from yeast and bacteria reduced the GS-hydroquinones to the corresponding hydroquinones.

280 reduction of glutathionyl-hydroquinones (GS-hydroquinones) to hydroquinones.

281 In summary, hydroquinone treatment up-regulated ChM-I and inhibited

282 -dependent conversion of TeCH to trichloro-p-hydroquinone (TriCH) and then to dichloro-p-hydroquinone

283 onors such as NADH, pinacyanol chloride, and hydroquinone undergo the TAML-catalyzed oxidation by O2.

284 ch is composed of a crown ether containing a hydroquinone unit and a 1,5-diaminonaphthalene unit, int

285 le diprotonated derivative in which only the hydroquinone unit resides inside the cavity of the tetra

286 ol; 0.14 muM for pyrogallol and 0.21 muM for hydroquinone, using square-wave voltammetry (SWV).

287 es spontaneously reacted with GSH to form GS-hydroquinones via Michael addition, and four GS-HQRs fro

288 Hydroquinone was found to up-regulate chondromodulin-I (

289 0.2V vs. Ag|AgCl while a mixture of H2O2 and hydroquinone was injected into the microfluidic device.

290 lectrochemical oxidation of enzyme-generated hydroquinone was measured.

291 Hydroxylamine and hydroquinone were used to probe the oxidation states of

292 When 2,3-disubstituted hydroquinones were employed as starting materials the 2,

293 In addition, two chlorinated o-hydroquinones were prepared, 6-chloro-biphenyl-3,4-diol

294 s a reduction of a quinone intermediate to a hydroquinone, which goes hand-in-hand with a stabilizing

295 site is assumed to be a polycyclic aromatic hydroquinone whose oxidation to the corresponding conjug

296 nyl compounds using catalytic amounts of 1,4-hydroquinone with a copper nanoparticle electron transfe

297 Interactions of 1,4-hydroquinone with soluble iron species over a pH range o

298 ons led to the reduction of benzoquinones to hydroquinones with the concomitant oxidation of GSH to o

299 H466D resulted in the formation of a neutral hydroquinone, with no stabilization of the flavin semiqu

300 substrate specificity for 2,6-disubstituted hydroquinones, with halogens greatly preferred at those