ライフサイエンスコーパス: hydroxyindoleacetic acid

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1 mpulse control, alcohol abuse, and low CSF 5-hydroxyindoleacetic acid.

2 e test and/or measurement of CSF levels of 5-hydroxyindoleacetic acid.

3 Interestingly, cerebrospinal fluid (CSF) 5-hydroxyindoleacetic acid (5-HIAA) and fenfluramine-induc

4 he relationship between aggression and CSF 5-hydroxyindoleacetic acid (5-HIAA) concentration with tha

5 separation after birth; their behavior and 5-hydroxyindoleacetic acid (5-HIAA) concentrations in CSF

6 an increase in homovanillic acid (HVA) or 5-hydroxyindoleacetic acid (5-HIAA) concentrations in the

7 Low concentrations of CSF 5-hydroxyindoleacetic acid (5-HIAA) have been consistently

8 liability for antisocial behavior and CSF 5-hydroxyindoleacetic acid (5-HIAA) in newborns was explor

9 owed reduced levels of the 5-HT metabolite 5-hydroxyindoleacetic acid (5-HIAA) in several regions inc

10 (DA), serotonin (5-HT) and its metabolite 5-hydroxyindoleacetic acid (5-HIAA) in the PVN.

11 47 vs 122.0 +/- 50.7 pmol/mL; P<.01), and 5-hydroxyindoleacetic acid (5-HIAA) levels were significan

12 5-hydroxyindoleacetic acid (5-HIAA), a biomarker of pharma

13 scans and changes in the level of urinary 5-hydroxyindoleacetic acid (5-HIAA), a metabolite of serot

14 CSF homovanillic acid (HVA), 5-hydroxyindoleacetic acid (5-HIAA), and 3-methoxy-4-hydro

15 d duodenal mucosal concentrations of 5-HT, 5-hydroxyindoleacetic acid (5-HIAA), and kynurenic acid (K

16 s in tissue levels of 5-HT, its metabolite 5-hydroxyindoleacetic acid (5-HIAA), and norepinephrine in

17 tic acid (DOPAC), homovanillic acid (HVA), 5-hydroxyindoleacetic acid (5-HIAA), and serotonin (5-HT)

18 doses and the serotonin (5-HT) metabolite, 5-hydroxyindoleacetic acid (5-HIAA), at the highest dose.

19 f tryptophan and the serotonin catabolite, 5-hydroxyindoleacetic acid (5-HIAA), but not that of the n

20 in the brain content of serotonin (5-HT), 5-hydroxyindoleacetic acid (5-HIAA), dopamine (DA), norepi

21 by reduced brainstem serotonin (5-HT) and 5-hydroxyindoleacetic acid (5-HIAA), fewer 5-HT(1A) autore

22 fer from controls in CSF concentrations of 5-hydroxyindoleacetic acid (5-HIAA), homovanillic acid (HV

23 Cerebrospinal fluid was assayed for 5-hydroxyindoleacetic acid (5-HIAA), norepinephrine, 3-met

24 Brain levels of 5-HT and its metabolite, 5-hydroxyindoleacetic acid (5-HIAA), were determined using

25 hromogranin A (CgA), serotonin, or urinary 5-hydroxyindoleacetic acid (5-HIAA), while topographic loc

26 y serotonin and the metabolite interferant 5-hydroxyindoleacetic acid (5-HIAA).

27 analyze levels of 5-HT and its metabolite, 5-hydroxyindoleacetic acid (5-HIAA); levels of TPH2; and 5

28 els of the major metabolites of serotonin (5-hydroxyindoleacetic acid [5-HIAA]), dopamine (homovanill

29 Thyroid function was normal, as were 5-hydroxyindoleacetic acid and chromogranin A levels.

30 The CSF 5-hydroxyindoleacetic acid and homovanillic acid levels di

31 We used CSF (AVP), CSF 5-hydroxyindoleacetic acid, and the prolactin response to

32 Tumor markers (chromogranin A and 5-hydroxyindoleacetic acid) before and after treatment wer

33 oholism and lower mean cerebrospinal fluid 5-hydroxyindoleacetic acid concentration than late-onset a

34 f alcoholism and lower cerebrospinal fluid 5-hydroxyindoleacetic acid concentration.

35 ly predictive of lower cerebrospinal fluid 5-hydroxyindoleacetic acid concentrations in controls but

36 5-HT and 5-hydroxyindoleacetic acid content of limbic nuclei were d

37 rebral glucose utilization, while 5-HT and 5-hydroxyindoleacetic acid content were quantified in limb

38 dopamine, and norepinephrine metabolites (5-hydroxyindoleacetic acid, homovanillic acid, and 3-metho

39 particularly low mean cerebrospinal fluid 5-hydroxyindoleacetic acid, homovanillic acid, and tryptop

40 t changes were observed in CSF measures of 5-hydroxyindoleacetic acid, homovanillic acid, dehydroepia

41 had raised ratios of homovanillic acid to 5-hydroxyindoleacetic acid in cerebrospinal fluid, of rang

42 Tail pinch had no effect on 5-hydroxyindoleacetic acid in either brain region.

43 nding of low cerebrospinal fluid levels of 5-hydroxyindoleacetic acid in higher-lethality suicide att

44 concentrations of the serotonin metabolite 5-hydroxyindoleacetic acid in the CSF of suicide cases ver

45 noradrenaline and the serotonin metabolite 5-hydroxyindoleacetic acid in the hippocampus.

46 s on extracellular 5-hydroxytryptamine and 5-hydroxyindoleacetic acid in the striatum and hippocampus

47 ve determination of tryptophan, serotonin, 5-hydroxyindoleacetic acid, kynurenine, and kynurenic acid

48 lamine increased, and serotonin metabolite 5-hydroxyindoleacetic acid levels decreased, to a much gre

49 ancreatic polypeptide, and 24-hour urinary 5-hydroxyindoleacetic acid levels in all patients.

50 angiographic findings, chromogranin A and 5-hydroxyindoleacetic acid levels were measured and were 7

51 tered significantly by forced swimming but 5-hydroxyindoleacetic acid levels were reduced to 60% of b

52 nockout mice had normal levels of 5-HT and 5-hydroxyindoleacetic acid, possibly because of an up-regu

53 ge, region of origin, and urinary level of 5-hydroxyindoleacetic acid predicted survival by univariat

54 a mediating phenotype, cerebrospinal fluid 5-hydroxyindoleacetic acid, provides preliminary evidence

55 mine to 90% above basal levels and reduced 5-hydroxyindoleacetic acid to 45% of basal levels in the s

56 resected was 33 cm, and mean 24-hour urine 5-hydroxyindoleacetic acid was 120 mg.

57 d DA and higher levels of MAO A metabolite 5-hydroxyindoleacetic acid were found in the forebrain reg

58 yphenylacetic acid, homovanillic acid, and 5-hydroxyindoleacetic acid were measured in the brainstem,

59 ith PRL[d-FEN] responses (but not with CSF 5-hydroxyindoleacetic acid), which in turn was correlated

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