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1 ion (cytochrome c) or cholesterol synthesis (hydroxymethylglutaryl-CoA reductase).
2 tein maturation and promoting degradation of hydroxymethylglutaryl-CoA reductase.
3 of gamma-tocotrienol, indicating the role of hydroxymethylglutaryl-CoA reductase.
4 -binding protein-2 and reduced expression of hydroxymethylglutaryl-CoA reductase.
5 ding protein cleavage-activating protein and hydroxymethylglutaryl-CoA reductase.
6 (AMPK), phosphorylates and regulates in vivo hydroxymethylglutaryl-CoA reductase and acetyl-CoA carbo
8 protein pathway and increases expression of hydroxymethylglutaryl-CoA reductase and low density lipo
9 nhibited by both lovastatin, an inhibitor of hydroxymethylglutaryl CoA reductase, and alendronate.
10 carboxykinase, and type I deiodinase but not hydroxymethylglutaryl-CoA reductase, cytochrome c, and a
11 ed in the regulated degradation of wild-type hydroxymethylglutaryl-CoA reductase in the ER membrane.
12 or the hepatic uptake of this liver-specific hydroxymethylglutaryl-CoA reductase inhibitor in rat and
13 hamster ovary cell lines with lovastatin (a hydroxymethylglutaryl-CoA reductase inhibitor) and meval
14 sulfate, and thyroid hormone, as well as the hydroxymethylglutaryl-CoA reductase inhibitor, pravastat
16 d that "high-risk" patients are treated with hydroxymethylglutaryl CoA reductase inhibitors to reduce
17 dence suggests that long term treatment with hydroxymethylglutaryl-CoA reductase inhibitors, or stati
18 ord "surgery" and the MeSH term for statins "hydroxymethylglutaryl-CoA reductase inhibitors." Studies
19 e stimulated the rapid inactivation of their hydroxymethylglutaryl-CoA reductase, presumably through
21 REBP binding to three specific target genes: hydroxymethylglutaryl-CoA reductase (Red), fatty acid sy
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