ライフサイエンスコーパス: hyper-

1 lative to the control at 51, 66, and 81 mM K(hyper).

2 tion diminishing with further increases in K(hyper).

3 onally limited pathogen followed by defense (hyper) activation during reproduction, reveals a subtle

4 at 21 mM K(+) in all three groups (K(iso), K(hyper), and K(suc)).

5 smography to assess endothelium-dependent (% Hyper) and endothelium-independent vasodilatation (% Nit

6 hase, had no effect on the response of the K(hyper) and K(suc) groups at 21 mM but significantly enha

7 With the K(hyper) and K(suc) groups, the magnitude of the dilation

8 ng a new conformational grid search program, HYPER, and used together with longer-range constraints a

9 categorized into 7,537 differentially (46.6% hyper- and 53.4% hypo-) methylated regions.

10 rom strawberry exerted significantly greater hyper- and bathochromic spectral shifts than their acyla

11 arin; continued advances in the treatment of hyper- and dyslipidemias, new roles for beta-blockade in

12 oach identified 76 markers, of which 68 were hyper- and eight hypomethylated (LME, P < 0.05).

13 we provide evidence that genome-wide cancer hyper- and hypo- DNA methylation patterns are independen

14 ssible patterns of statistically significant hyper- and hypo- methylation in comparisons involving an

15 , denatonium, detergents, heavy metals, both hyper- and hypo-osmotic shock and nose touch.

16 the disulfide-reducing agent dithiothreitol, hyper- and hypo-osmotic shock, amino acid starvation, ni

17 resulting in a shift in the balance between hyper- and hypo-phosphorylated 4E-BP1 and translational

18 yeast pro-prion protein Ure2p binds to both hyper- and hypo-phosphorylated Gln3p.

19 ctional supercoiling assays reveal that both hyper- and hypo-phosphorylated histones can be efficient

20 nent, KaiC, undergoes regular cycles between hyper- and hypo-phosphorylated states with a period of c

21 d in vivo using (2)H(2)O labeling in normal, hyper- and hypo-proliferative conditions.

22 reported a similar pattern of both increased hyper- and hypo-sensitivities across multiple senses.

23 me remodeling by human Swi-Snf is similar on hyper- and hypoacetylated MMTV arrays.

24 We find that both hyper- and hypoacetylation of individual lysines are ass

25 s to be regulated by phosphorylation in both hyper- and hypoactive striatal DA neurons; in the latter

26 acterized by concentric rings of alternating hyper- and hypoautofluorescence, and foveal sparing of p

27 PET,CO2 and CFV were computed separately for hyper- and hypocapnia during the LBNP and no-LBNP condit

28 0 mmHg, on cerebrovascular responsiveness to hyper- and hypocapnia in healthy humans.

29 overall cerebrovascular reactivity to CO(2) (hyper- and hypocapnia) was lower in patients with apnea

30 intenance of the cerebrovascular response to hyper- and hypocapnia.

31 e associated with dominant forms of familial hyper- and hypocholesterolemia.

32 2R partial agonists to simultaneously target hyper- and hypodopaminergia.

33 perechoic mass in 13 tumors and random mixed hyper- and hypoechogenicity with associated architectura

34 e is severely disrupted and characterized by hyper- and hypoediting of different genes, such as hyper

35 Hyper- and hypoenhanced regions were also smaller (eg, 2

36 In addition, for a series of hyper- and hypofusogenic F mutants, we quantified F-trig

37 Hyper- and hypofusogenicity can each be manifested throu

38 Hyper- and hypoglycemia were associated with poor neurol

39 pring of immune-challenged mothers displayed hyper- and hypomethylated CpGs at numerous loci and at d

40 Individual hyper- and hypomethylated promoters allowed discriminati

41 ings demonstrate a surprisingly high rate of hyper- and hypomethylation as a function of age in norma

42 Hyper- and hypomethylation at the IGF2-H19 imprinting co

43 monstrate that maternal asthma leads to both hyper- and hypomethylation in neonatal DCs, and that bot

44 inct subgroup characterized by both aberrant hyper- and hypomethylation.

45 Thus, the response to both hyper- and hypoosmolar stimuli is impaired in trpv4-/- m

46 Furthermore, in response to hyper- and hypoosmotic shock, E. coli cells resumed thei

47 verify whether ictal perfusion changes, both hyper- and hypoperfusion, correspond to electrically con

48 ating, well-defined regions of portal venous hyper- and hypoperfusion; it likely has a multifactorial

49 progression; upregulated pRb displaying both hyper- and hypophosphorylated forms; increased levels of

50 The added recombinant CTD was quickly hyper- and hypophosphorylated in extract, became associa

51 We also provide evidence that both hyper- and hypophosphorylation inhibit SR protein splici

52 rmation required to interpret the changes in hyper- and hyporeflexive bands in pediatric spectral-dom

53 that genetic variations between FcgammaRIIA hyper- and hyporesponders regulate FcgammaRIIA-mediated

54 eterogeneity but did not distinguish between hyper- and hyposignal within an otherwise uniform activi

55 be inhibited with different selectivities by hyper- and hypotensive regions of the PAG.

56 ion was assessed by applying rapid pulses of hyper- and hypotensive stimuli to the neck via a customi

57 their linear bending elastic regime in both hyper- and hypotonic conditions.

58 s without CF, we investigated the effects of hyper- and hypotonicity on ion transport processes.

59 crystalline complex was consistent with the hyper- and hypsochromically shifted absorption spectra o

60 polar (DeltarcrR-NP) rcrR mutants, which are hyper- and nontransformable, respectively, to dissect th

61 Despite similarities in symptoms, hyper- and normosensitive patients with IBS differ in ce

62 Both the hyper- and suppressed-activity subtypes of MDD patients

63 The adenopathy at US was hyper- and/or isoechoic relative to the liver and thyroi

64 lysis revealed an extensive influence of DNA hyper- as well as hypomethylation on miRNA regulation.

65 characterization are well-defined synthetic (hyper)-branched polymers.

66 catechin-3-gallate blocked expression of the hyper-, but not hypophosphorylated Bcl-X(L) in mitochond

67 At week 1, %S was depressed in HYPER, COMB, and HYPO (9+/-8%, 7+/-6%, and 5+/-4%, respe

68 ning (%S) was measured in regions displaying HYPER, COMB, or HYPO contrast patterns and in remote reg

69 o significant effect on dilations at lower K(hyper) concentrations but constricted the arteries relat

70 The estimated MGF were (hyper) disc-like, such that each neuron's firing modulat

71 aggregates from three hamster prion strains (Hyper, Drowsy, SSLOW) subjected to minimal manipulations

72 of sites by ignoring reads with excessive ('hyper') editing that do not easily align to the genome.

73 ign called high-yield-pileup-event-recovery (HYPER) electronics for processing the detector signal in

74 PrP(Sc) formation for the hyper (HY) and drowsy (DY) strains of the transmissible

75 amsters in the sciatic nerve with either the hyper (HY) or drowsy (DY) strain of the transmissible mi

76 (TME) agent prior to superinfection with the hyper (HY) strain of TME can completely block HY TME fro

77 erinfection with the short-incubation-period hyper (HY) strain of transmissible mink encephalopathy (

78 Binding of the hyper (HY) strain of transmissible mink encephalopathy (

79 in hamsters resulted in the selection of the hyper (HY) TME PrP(Sc) strain-dependent conformation and

80 rom the brains of hamsters infected with the hyper (HY), drowsy (DY), and 263K TSE strains yielded si

81 The stages of hyper-, hypo-, and hyper-methylation patterns of the CDX

82 representation of these TFBS was observed in hyper-/hypo-methylated sequences where signi.cant change

83 These hyperadditive effects of CB hyper-/hypocapnia agree with previous findings using CB

84 iscovery of familial mutations implicated in hyper-/hypocholesterolemia by linkage studies and single

85 In four CB-denervated dogs, absence of hyper-/hypoventilatory responses to CB perfusion with PC

86 capnia agree with previous findings using CB hyper-/hypoxia.We propose that hyperaddition is the domi

87 endothelium plays a permissive role in the K(hyper)-induced response.

88 Areas of cortical lobar hypo (hyper)-metabolism in the cerebrum that were 2 SDx from t

89 enous infusion of either 0.9% (ISO) or 3.0% (HYPER) NaCl saline, 12 subjects were passively heated un

90 Mice conditionally lacking UPF3A exhibit "hyper" NMD and display defects in embryogenesis and game

91 ined as COMB (first pass hypoenhancement) or HYPER (normal first pass signal enhancement).

92 a) a direct effect of the K(+)/osmolality (K(hyper)) on the endothelium or (b) a 'permissive' role of

93 d by hyperenhanced signal on delayed images (HYPER), or (3) both absence of normal first-pass enhance

94 tes with RNA polymerase II that has either a hyper- or a hypophosphorylated CTD.

95 ication during virion maturation by inducing hyper- or aberrant IN multimerization but are largely in

96 he blood glucose level is essential to avoid hyper- or hypo-glycemic shocks associated with many meta

97 e precipitated by aggressive correction of a hyper- or hypo-osmolar state and until recently has been

98 us research has not clarified whether neural hyper- or hypo-responsiveness to anticipated rewards pro

99 esults obtained with hypoacetylated MMTV and hyper- or hypoacetylated 5S rDNA arrays.

100 Finally, our data show that hyper- or hypoactivity of PP5 mutants increases Hsp90 bi

101 ions, gender, gestational age, pneumothorax, hyper- or hypocarbia, severity of illness, and cranial i

102 Naturally occurring mutations can lead to hyper- or hypocholesterolemia in human.

103 -blocking drug, in combination with either a hyper- or hypocholesterolemic diet, to show that elevate

104 oclonal antibody binding data indicated that hyper- or hypofusogenic mutations in the KKR motif affec

105 Sustained dysregulation of blood glucose (hyper- or hypoglycemia) associated with type 1 diabetes

106 None of the transplant recipients have had a hyper- or hypoglycemic episode since PIT and no complica

107 nal intensity average to identify regions of hyper- or hypointensity.

108 ue genes were identified as overlapping with hyper- or hypomethylated regions, respectively.

109 ely determines which CGIs will be eventually hyper- or hypomethylated.

110 metabolic diseases may result in findings of hyper- or hypomobility, or carpal tunnel syndrome.

111 E613R allele of CD45 does not function as a hyper- or hypomorphic allele but rather alters the subst

112 rreflectivity over drusen, drusen cores, and hyper- or hyporeflectivity of drusen were also associate

113 lude irradiation, hyperbaric oxygen therapy, hyper- or hypothermic therapy, and photodynamic therapy.

114 n semidominant mutation, which led to either hyper- or neofunctionalization of a redundant homoeologo

115 ng, low blood pressure, normal magnesium and hyper- or normocalciuria; they define a distinct subset

116 this response must be tightly regulated, as hyper- or suboptimal responses can be detrimental to the

117 GFP)-based fluorescent indicators roGFP2 and HyPer, respectively.

118 However, in HYPER, %S improved at week 7 from 9+/-8% to 18+/-5% (P<0

119 How hyper(-)sc emerges in the middle of a positively superco

120 The essentially additive "hyper" shift due to lead chelation brought the Soret ban

121 The lead(II) chelate 19d gave an additional "hyper" shift that brought the Soret band to 604 nm.

122 transformation was also observed for strain Hyper, suggesting that this phenomenon was not limited t

123 e damage (SNN plus infarction) was larger in hyper- than in normoglycemic animals at 2 and 4 h of rec

124 ysiologic significance of the shift from MTL hyper- to hypometabolism associated with IR.

125 ngly, when hamster-adapted strains (263K and Hyper) were subjected to dgPMCAb, their proteinase K dig

126 y decreases in Na(+), (2) hypertonic K(+) (K(hyper)) where K(+) was increased without a concomitant a