ライフサイエンスコーパス: hyperabsorption

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1 derlie the pathophysiology of the intestinal hyperabsorption.

2 CTSB to activate ENaC, contributing to Na(+) hyperabsorption and depletion of CF ASL volume.

3 fective in CF cultures with prolonged volume hyperabsorption and mucus accumulation.

4 Diminished cerebrospinal fluid production, hyperabsorption, and leak are postulated mechanisms of s

5 ation of CF airway cultures prevented CF ASL hyperabsorption, and this effect was abolished when SPLU

6 cesses unopposed, which results in net fluid hyperabsorption, dehydration of intestinal contents, and

7 speculated that airway surface liquid (ASL) hyperabsorption generates a concentrated airway mucus th

8 ficit in hepcidin results in intestinal iron hyperabsorption; however, the local effectors mediating

9 ive disorder characterized by (a) intestinal hyperabsorption of all sterols, including cholesterol an

10 aracellular sodium transport to paracellular hyperabsorption of calcium and magnesium.

11 uria may be attributable to gastrointestinal hyperabsorption of calcium rather than a renal calcium l

12 tations in either ABCG5 or G8 proteins, with hyperabsorption of dietary sterols and decreased hepatic

13 of mucus from the glands could be blocked by hyperabsorption of fluid in the gland ducts.

14 is C and alcoholic liver disease, leading to hyperabsorption of iron and its accumulation in the live

15 the human hemachromatosis protein (HFE) and hyperabsorption of iron by the duodenal enterocyte.

16 These data indicate that hyperabsorption of iron in trf (hpx) mice is not solely

17 In this state, hyperabsorption of iron is also observed and can signifi

18 not require blood transfusion for survival, hyperabsorption of iron is the leading cause of iron ove

19 iting transferrin deficiency, marked anemia, hyperabsorption of iron, and elevated hepatic iron store

20 a role in two CF-associated characteristics: hyperabsorption of sodium and susceptibility to bacteria

21 ow in CF, but surprisingly, there was no net hyperabsorption of sodium or water during perfusion of a

22 c potential in CF airways by reducing sodium hyperabsorption, restoring lung epithelial surface fluid

23 -)/HCO3 (-) hyposecretion and triggers Na(+) hyperabsorption through the epithelial Na(+) channel (EN

24 These data link airway surface liquid hyperabsorption to the high incidence of P. aeruginosa b

25 g (GHS) rats is accompanied by intestinal Ca hyperabsorption with normal serum 1,25-dihydroxyvitamin

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