ライフサイエンスコーパス: hyperactivity

1 ice displayed reduced motor coordination and hyperactivity.

2 h the hyperflexible phenotype, and posterior hyperactivity.

3 ive (SH) rats, which are known to exhibit CB hyperactivity.

4 used PNEC hyperinnervation and nodose neuron hyperactivity.

5 romic defects, exhibit inner ear defects and hyperactivity.

6 n, highlighting a rigid, set mode of sensory hyperactivity.

7 by increased azoxystrobin uptake induced by hyperactivity.

8 rcuit dysfunction that is caused by cortical hyperactivity.

9 ase and thyroid stimulating hormone receptor hyperactivity.

10 In ClockDelta19 mice, AUT1 reversed hyperactivity.

11 state, and attenuated OBX-induced locomotor hyperactivity.

12 duced adult body mass, social avoidance, and hyperactivity.

13 disease of TSC1/TSC2 inactivation and mTORC1 hyperactivity.

14 imbic seizures can be supported by GABAergic hyperactivity.

15 s and risk of offspring conduct disorder and hyperactivity.

16 depending on the level and timing of mTORC1 hyperactivity.

17 [14%] of 610 siblings, p=0.010), inattention-hyperactivity (15 [19%] vs 86 [14%], p<0.0001), social w

18 ue to either Tbx1 or Slc12a2 mutations cause hyperactivity; (2) it is vestibular dysfunction, which f

19 lso necessary for inner ear function, causes hyperactivity; (2) vestibular rather than auditory failu

20 ciations with vision problems, microcephaly, hyperactivity, a tendency to obesity, and feeding diffic

21 renatal stress and both conduct disorder and hyperactivity, after adjusting for sex, parental educati

22 t rs12765063 is associated with impulsivity, hyperactivity and addiction-related phenotypes.

23 rather than socio-environmental reasons why hyperactivity and anxiety disorders occur at higher rate

24 rather than auditory dysfunction that causes hyperactivity and anxiety-related symptoms; and (3) the

25 e therapeutic strategy to reduce hippocampal hyperactivity and attenuate behavioral deficits in schiz

26 responses that would otherwise limit B cell hyperactivity and autoantibody production.

27 ior in two mouse models: amphetamine-induced hyperactivity and ClockDelta19 mutants.

28 arly loss of entorhinal input on hippocampal hyperactivity and cognitive deficits characterizing earl

29 nistration of retigabine normalized neuronal hyperactivity and depressive behaviours.

30 rms of spreading neuronal toxicity, synaptic hyperactivity and direct glutamate excitotoxicity.

31 cortex-amygdala circuitry: intrinsic sensory hyperactivity and disinhibition give rise to frontal ove

32 ion of mGluR5 results in the exacerbation of hyperactivity and epileptic phenotypes.

33 11 years to concurrent anxiety, depression, hyperactivity and impulsivity, inattention, and conduct

34 6 years of age included anxiety, depression, hyperactivity and impulsivity, inattention, conduct prob

35 ed synaptic plasticity, reduced sociability, hyperactivity and increased anxiety.

36 cific behavioural phenotype characterised by hyperactivity and increased anxiety.

37 oc2a+/- fam57ba+/- double heterozygotes show hyperactivity and increased seizure susceptibility relat

38 the neuropeptide neuromedin U (Nmu) promotes hyperactivity and inhibits sleep in zebrafish larvae, wh

39 n the existing circuitry, reduce hippocampal hyperactivity and normalize aberrant dopamine neuron act

40 induced hypothalamic-pituitary-adrenal axis hyperactivity and reduced fear- and anxiety-related beha

41 in-expressing cells reduced pyramidal neuron hyperactivity and reversed mechanical allodynia.

42 characterized by a combination of functional hyperactivity and smaller gray matter volume compared wi

43 mission and plasticity in vivo, resulting in hyperactivity and spatial memory impairment.

44 lymphoid organs manifest similar COX-2/PGE2 hyperactivity and T cell suppression.

45 en after 20 d and thus block cocaine-induced hyperactivity and toxicity for a long period.

46 PRC2 hyperactivity and/or hypertrimethylation of H3K27 are as

47 ults suggest an association between MeHg and hyperactivity, and imply that fish chronically exposed t

48 ities, including deficits in motor learning, hyperactivity, and increased risk-taking and self-injuri

49 ng self-injurious and risk-taking behaviors, hyperactivity, and learning and memory defects.

50 (Mn) exposure with inattention, impulsivity, hyperactivity, and oppositional behaviors, but causal in

51 ) dose-dependently ameliorated PPI deficits, hyperactivity, and risk-taking behaviors, in a fashion a

52 conditions including sleep disorders, motor hyperactivity, and seizures.

53 cterized by autism, intellectual disability, hyperactivity, and seizures.

54 stibular rather than auditory failure causes hyperactivity; and (3) the severity rather than the age

55 teraction, compulsive behaviors, aggression, hyperactivity, anxiety, depression, and somatosensory ga

56 t NOTCH2 signaling and consequent osteoclast hyperactivity are closely associated with the bone-relat

57 thoughts is also associated with hippocampal hyperactivity, arising from dysfunctional GABAergic inte

58 enhanced phasic dopamine release, behavioral hyperactivity, associative learning deficits, and a para

59 o cholerae was sufficient to block locomotor hyperactivity at 10 dpf.

60 negative feedback loop that blocks neuronal hyperactivity at least partly through the inhibition of

61 to belong to the high symptom trajectory for hyperactivity (B = 0.46, p < .05) and conduct disorder (

62 ion-deficit/hyperactivity disorder symptoms (hyperactivity, but not inattention) on the basis of indi

63 we investigated to what extent blocking this hyperactivity can improve optogenetic retinal prosthetic

64 At 10 dpf, axenic zebrafish exhibited hyperactivity compared to conventionalized and conventio

65 nmental exposures and children's inattention/hyperactivity, conduct problems, and educational achieve

66 By affording means of monitoring CDK4 hyperactivity consequent to cancer-associated molecular

67 mice, but it remains unclear whether kinase hyperactivity contributes to the behavioral phenotypes.

68 f the ClockDelta19 mouse is characterized by hyperactivity, decreased anxiety-like behavior, decrease

69 n the forebrain (Plcg1(f/f); CaMKII) exhibit hyperactivity, decreased anxiety-like behavior, reduced

70 r (94.2 [1.69]; P = .004), attention-deficit/hyperactivity disorder (96.3 [0.91]; P = .002), oppositi

71 Attention-deficit/hyperactivity disorder (ADHD) affects 39 million people

72 Attention-deficit/hyperactivity disorder (ADHD) and anxiety-related disord

73 PBDEs and intelligence or Attention Deficit/Hyperactivity Disorder (ADHD) and attention-related beha

74 opmental disorders such as attention deficit hyperactivity disorder (ADHD) and autism spectrum disord

75 d between extraversion and attention-deficit-hyperactivity disorder (ADHD) and between openness and s

76 Attention-deficit/hyperactivity disorder (ADHD) and bipolar disorder (BPD)

77 been effective in treating attention-deficit/hyperactivity disorder (ADHD) and is currently the first

78 Patients with attention-deficit/hyperactivity disorder (ADHD) and obsessive-compulsive d

79 s used clinically to treat attention-deficit/hyperactivity disorder (ADHD) and off-label as a perform

80 m spectrum disorder (ASD), attention deficit hyperactivity disorder (ADHD) and schizophrenia (SCZ).

81 tric conditions, including attention deficit hyperactivity disorder (ADHD) and substance use disorder

82 n the relationship between attention-deficit hyperactivity disorder (ADHD) and younger relative age i

83 Patients with attention-deficit/hyperactivity disorder (ADHD) are at an increased risk o

84 Individuals with attention-deficit/hyperactivity disorder (ADHD) are at greater risk for ac

85 ated that individuals with attention-deficit/hyperactivity disorder (ADHD) are more likely to experie

86 Epilepsy and attention-deficit/hyperactivity disorder (ADHD) are strongly associated; h

87 adult outcome of childhood attention deficit hyperactivity disorder (ADHD) could guide novel interven

88 Attention-deficit/hyperactivity disorder (ADHD) diagnosis is based on repo

89 ectrum disorders (ASD) and attention-deficit/hyperactivity disorder (ADHD) frequently co-occur.

90 uestioned the stability of attention-deficit hyperactivity disorder (ADHD) from childhood to adulthoo

91 Attention-deficit/hyperactivity disorder (ADHD) has an uncertain etiology,

92 Neuroimaging studies of attention-deficit/hyperactivity disorder (ADHD) have most commonly reporte

93 ng (fMRI) studies of adult attention-deficit/hyperactivity disorder (ADHD) have revealed various ADHD

94 ences were associated with attention-deficit/hyperactivity disorder (ADHD) in a recent multi-site, me

95 medications used to treat attention-deficit/hyperactivity disorder (ADHD) increase sympathetic tone

96 Attention-deficit hyperactivity disorder (ADHD) is a common childhood beha

97 Importance: Attention-deficit/hyperactivity disorder (ADHD) is a heritable neurodevelo

98 Attention-deficit hyperactivity disorder (ADHD) is a neurodevelopmental di

99 Attention-deficit hyperactivity disorder (ADHD) is a prevalent and highly

100 Attention-deficit/hyperactivity disorder (ADHD) is a prevalent development

101 Attention-deficit/hyperactivity disorder (ADHD) is associated with high ra

102 Attention-deficit/hyperactivity disorder (ADHD) is associated with structu

103 Recognition that adult attention-deficit/hyperactivity disorder (ADHD) is common, seriously impai

104 Although attention-deficit hyperactivity disorder (ADHD) is highly heritable, and l

105 pectrum disorder (ASD) and attention-deficit/hyperactivity disorder (ADHD) is increasingly appreciate

106 Attention-deficit/hyperactivity disorder (ADHD) is now recognized to occur

107 Attention-deficit/hyperactivity disorder (ADHD) is the most common childho

108 ldren and adolescents with attention deficit hyperactivity disorder (ADHD) is unclear.

109 Attention-deficit/hyperactivity disorder (ADHD) often persists into adoles

110 g adults without childhood attention deficit hyperactivity disorder (ADHD) often present to clinics s

111 Attention-deficit/hyperactivity disorder (ADHD) shows substantial heritabi

112 Attention-deficit/hyperactivity disorder (ADHD) symptoms are most commonly

113 d the effects of childhood attention deficit hyperactivity disorder (ADHD) symptoms, both inattention

114 rder, major depression and attention deficit hyperactivity disorder (ADHD) using genomic data from 15

115 For many years, attention-deficit hyperactivity disorder (ADHD) was thought to be a childh

116 g intellectual disability, attention deficit/hyperactivity disorder (ADHD), and autism spectrum disor

117 genetic susceptibility of Attention-Deficit/Hyperactivity Disorder (ADHD), but without much success.

118 oride for the treatment of attention-deficit/hyperactivity disorder (ADHD), little is known about age

119 Attention-deficit hyperactivity disorder (ADHD), one of the most common ps

120 ms of depression, anxiety, attention/deficit hyperactivity disorder (ADHD), oppositional defiant diso

121 ted behavioral hallmark of attention-deficit/hyperactivity disorder (ADHD), strongly influences addic

122 stand the underpinnings of attention-deficit/hyperactivity disorder (ADHD), we targeted the relations

123 ition is a core deficit of attention deficit hyperactivity disorder (ADHD), which is a common childho

124 ity among individuals with attention deficit hyperactivity disorder (ADHD), yet associations between

125 on of which is a factor in attention-deficit/hyperactivity disorder (ADHD).

126 S), and characteristics of attention-deficit/hyperactivity disorder (ADHD).

127 and could be relevant for attention-deficit/hyperactivity disorder (ADHD).

128 mal model for the study of attention deficit hyperactivity disorder (ADHD).

129 ychopathologies, including attention deficit hyperactivity disorder (ADHD).

130 iatric disorders including attention deficit hyperactivity disorder (ADHD).

131 predispose individuals to attention-deficit/hyperactivity disorder (ADHD).

132 pectrum disorder (ASD) and attention-deficit hyperactivity disorder (ADHD).

133 atopic dermatitis (AD) and attention-deficit/hyperactivity disorder (ADHD).

134 ttention problems, such as attention deficit/hyperactivity disorder (ADHD).

135 s have been identified for attention-deficit/hyperactivity disorder (ADHD).

136 ulsive disorder (OCD), and attention deficit hyperactivity disorder (ADHD).

137 n children and adults with attention deficit hyperactivity disorder (ADHD).

138 variant has been linked to attention-deficit/hyperactivity disorder (ADHD); however, the underlying m

139 a Exchange (ABIDE) and the Attention-Deficit Hyperactivity Disorder (ADHD-200) databases.

140 3 [95% CI, 0.62-1.13]), or attention-deficit/hyperactivity disorder (HR, 0.99 [95% CI, 0.79-1.25]).

141 =3540), autism (N=16 146), attention-deficit/hyperactivity disorder (N=18 726) and affective disorder

142 Attention-deficit/hyperactivity disorder according to DSM-IV diagnostic cr

143 tween the Pro33 allele and attention-deficit hyperactivity disorder and ASDs.

144 ng infantile parkinsonism, attention-deficit/hyperactivity disorder and autism spectrum disorder.

145 ), behavioral disturbance (attention deficit hyperactivity disorder and conduct disorder), psychosis-

146 atric disorders, including attention-deficit hyperactivity disorder and drug addiction.

147 Some conditions, such as attention-deficit/hyperactivity disorder and inpatient well-newborn care,

148 schizophrenia, depression, attention-deficit hyperactivity disorder and substance abuse disorders.

149 Attention-deficit/hyperactivity disorder and well-dental care (including d

150 xposed were diagnosed with attention-deficit/hyperactivity disorder by age 15 years.

151 gotic twins discordant for attention deficit hyperactivity disorder can elucidate mechanisms that con

152 of why many children with attention deficit hyperactivity disorder do not outgrow the disorder by ad

153 tensive drugs, sleep aids, attention-deficit/hyperactivity disorder drugs, and antidepressant drugs)

154 of insomnia [sleep aids], attention-deficit/hyperactivity disorder drugs, antidepressant drugs, and

155 The adult form of attention-deficit/hyperactivity disorder has a prevalence of up to 5% and

156 2.02 [95% CI, 1.80-2.26]; attention-deficit/hyperactivity disorder HR, 2.21 [95% CI, 2.04-2.39]).

157 schizophrenia, autism, and attention-deficit/hyperactivity disorder in humans.

158 tism spectrum disorder and attention-deficit/hyperactivity disorder in offspring.

159 mitter dysregulation, e.g. attention deficit hyperactivity disorder or Alzheimer's disease.

160 : 1.4, 7.7) higher odds of attention deficit/hyperactivity disorder problems compared with children w

161 itude) relates to specific attention-deficit/hyperactivity disorder symptoms (hyperactivity, but not

162 sorder, schizophrenia, and attention-deficit/hyperactivity disorder).

163 patient well-newborn care, attention-deficit/hyperactivity disorder, and asthma among all conditions.

164 , including schizophrenia, attention deficit/hyperactivity disorder, and autism spectrum disorders.

165 sorders, including autism, attention-deficit/hyperactivity disorder, and schizophrenia.

166 athology, such as anxiety, attention deficit hyperactivity disorder, and substance use.

167 ric diagnosis of interest (attention-deficit/hyperactivity disorder, autism spectrum disorder, schizo

168 rs in offspring, including attention deficit/hyperactivity disorder, autism, and schizophrenia.

169 r clinical development for attention deficit hyperactivity disorder, binge eating disorder, cocaine a

170 moking with drug and AUDs, attention-deficit hyperactivity disorder, bipolar disorder and antisocial

171 diabetes, waist-hip ratio, attention deficit hyperactivity disorder, bipolar disorder, major depressi

172 ty in patients with autism/attention deficit hyperactivity disorder, compared with respective control

173 schizophrenia, depression, attention-deficit/hyperactivity disorder, eating disorders, autism spectru

174 ssociated with addiction, attention-deficit/ hyperactivity disorder, schizophrenia, and Parkinson's d

175 ture of DD overlapped with attention-deficit/hyperactivity disorder, schizophrenia, major depression,

176 autism spectrum disorders, attention deficit/hyperactivity disorder, severe learning disability, cere

177 of attention--symptoms of attention deficit hyperactivity disorder--from resting-state connectivity

178 s, particularly autism and attention-deficit hyperactivity disorder.

179 order, autism, anxiety and attention deficit/hyperactivity disorder.

180 tism spectrum disorder, or attention-deficit/hyperactivity disorder.

181 syndrome, autism, and attention deficit and hyperactivity disorder.

182 pathological gambling, and attention deficit hyperactivity disorder.

183 25 (4.8%) met criteria for attention-deficit/hyperactivity disorder.

184 or depressive disorder, or attention-deficit/hyperactivity disorder.

185 riability in patients with attention deficit hyperactivity disorder.

186 the exclusion of comorbid attention-deficit/hyperactivity disorder.

187 chiatric disorders such as attention-deficit hyperactivity disorder.

188 isorder, schizophrenia and attention deficit-hyperactivity disorder.

189 nia, bipolar disorder, and attention-deficit/hyperactivity disorder.

190 between schizophrenia and attention deficit hyperactivity disorder/autism.

191 mood dysregulation, and/or attention-deficit/hyperactivity disorders and 22 healthy youths.

192 to a novel compound phenotype consisting of hyperactivity, disrupted circadian behavior patterns, an

193 driving glutamate dysregulation and neuronal hyperactivity during AD.SIGNIFICANCE STATEMENT Neuronal

194 The results suggest that left amygdala hyperactivity during negative autobiographical recall is

195 ent levels predicted amygdala, but not BNST, hyperactivity during shock anticipation.

196 and also provides a fertile ground of motor hyperactivity for tic learning.

197 Aberrant neural hyperactivity has been observed in early stages of Alzhe

198 Paradoxically, TGF-beta hyperactivity has been postulated to be the primary driv

199 e kinase inhibitor (TKI) to counteract FGFR3 hyperactivity has yet to be evaluated.

200 s with human studies, we discuss the circuit hyperactivity hypothesis for OCD, a potential circuitry

201 tting of lupus-like CD4 T cell-driven B cell hyperactivity, IL-21 signaling on Ag-specific donor CD8

202 nd found that losing this complex results in hyperactivity, impaired learning and memory, and abnorma

203 ibition of BACE activity can rescue neuronal hyperactivity, impaired long-range circuit function, and

204 iency in the Treg lineage resulted in immune hyperactivity, implicating Notch activity in Treg homeos

205 Hyperactivity improvements at 3 months were associated w

206 in female adolescents may be causal, whereas hyperactivity-impulsivity appears to act indirectly, thr

207 sorder (ADHD) symptoms, both inattention and hyperactivity-impulsivity, on the development of smoking

208 Hyperactivity-impulsivity, while also more strongly rela

209 More symptoms of hyperactivity/impulsivity (t = -2.63; P = .008) and inat

210 UT1 completely prevented amphetamine-induced hyperactivity in a dose-dependent manner, similar to the

211 NRG2 KOs exhibit hyperactivity in a novelty-induced open field, deficits

212 le of both cholinergic sprouting and dentate hyperactivity in AD symptomatogenesis should be consider

213 The subsequent reconstitution of SC mTORC1 hyperactivity in adult animals resulted in focal hypermy

214 SK4 or Poly(I:C) was not sufficient to block hyperactivity in axenic larvae.

215 t colonization using antibiotics also caused hyperactivity in conventionally colonized larvae.

216 chronic pain in a rat SCI model depends upon hyperactivity in dorsal root ganglia (DRG) neurons.

217 luding pain, that are promoted by persistent hyperactivity in DRG neurons.

218 nd that partial EC lesion generates abnormal hyperactivity in EC/dentate networks.

219 eration, but reduced depression and promoted hyperactivity in F1 offspring.

220 The medial caudate nucleus also shows hyperactivity in humans lacking a functional FOXP2 allel

221 Our results also suggest that dentate hyperactivity in MCI patients may be directly related to

222 T1 was unable to prevent amphetamine-induced hyperactivity in mice lacking Kv3.1 channels.

223 s accruing for broad, threat-neutral sensory hyperactivity in post-traumatic stress disorder.

224 Given the observations of neuronal hyperactivity in prodromal AD, we propose that this APOE

225 ion, and hypothalamic-pituitary-adrenal axis hyperactivity in stress vulnerability.

226 Hyperactivity in the CSTC pathway is involved in obsessi

227 More specifically, hyperactivity in the hippocampus may be detrimental and

228 t that the development of psychosis involves hyperactivity in the hippocampus that drives increased a

229 Compared with NTC, PTSD patients showed hyperactivity in the right anterior insula and bilateral

230 ects of different levels of sustained mTORC1 hyperactivity in the SC lineage, we disrupted negative r

231 ortex (mPFC); compared with TEC, PTSD showed hyperactivity in the ventral mPFC.

232 Symptoms of conduct disorder and hyperactivity in their offspring were measured at 6, 9,

233 onfidence interval (CI): 1.03, 1.20] and the hyperactivity-inattention subscale scores at 5 y (IRR: 1

234 , adolescents with high levels of conduct or hyperactivity/inattention symptoms who had also experien

235 la-precuneus connectivity is associated with hyperactivity/inattention symptoms.

236 ress in individuals with greater conduct and hyperactivity/inattention symptoms.

237 The authors sought to explore how conduct, hyperactivity/inattention, and emotional symptoms are as

238 ficiency in asthmatic airways promotes Orai1 hyperactivity, increased ASM contraction and airway hype

239 mals displayed behavioural anomalies such as hyperactivity, increased time in the open arms of the el

240 Neuronal hyperactivity is an early phenotype in both AD mouse mod

241 es pathological sleepiness [1-4], whereas OH hyperactivity is associated with stress and anxiety [5-1

242 Further analysis revealed that this neuronal hyperactivity is driven by decreased background inhibiti

243 Surprisingly, the resulting mTORC1 hyperactivity led to markedly delayed onset of both deve

244 However, HA instillation ameliorated bladder hyperactivity, lessened bladder mucosa damage, and decre

245 ffecting brain and body, including seizures, hyperactivity, macrocephaly, and obesity.

246 dromal AD, we propose that this APOE4-driven hyperactivity may be a causative factor driving increase

247 sensitivity achieved by blocking spontaneous hyperactivity may extend the dynamic range of optogeneti

248 In an amphetamine-induced hyperactivity model, compound (+)-19 showed significant

249 gy, and metabolomics-with the most prominent hyperactivity occurring in the entorhinal cortex.

250 The hyperactivity of cry1L407F is not related to a higher FA

251 ein, we demonstrated that overactivation and hyperactivity of FRS2alpha, as well as overexpression of

252 Hyperactivity of LHb is found in both rodent models of d

253 pancreatic cancer development by sustaining hyperactivity of multiple oncogenic signaling pathways,

254 An ASOX-mediated hyperactivity of SN dopamine neurons, which was caused b

255 eater number of stressful life events showed hyperactivity of the amygdala and several regions across

256 in DA neuron population activity, driven by hyperactivity of the BLA.

257 on of local ATP microgradients caused by the hyperactivity of the hippocampal network, at least in th

258 Overexpression and hyperactivity of this signaling pathway is observed in v

259 se hunger-display electrical and biochemical hyperactivity on exposure to dietary doses of ethanol in

260 estibular dysfunction differentiates whether hyperactivity or anxiety co-occurs with inner ear dysfun

261 f vestibular dysfunction can predict whether hyperactivity or anxiety coexist with inner ear dysfunct

262 ally inappropriate levels of inattention and hyperactivity or impulsivity.

263 resulted in behaviors that recapitulate the hyperactivity phenotype caused by absence of maternal re

264 iety-like behavior, but resulted in a robust hyperactivity phenotype in novel and home cage environme

265 Here, we have identified an APOE4-associated hyperactivity phenotype in the brains of aged APOE mice

266 lted in more pronounced social avoidance and hyperactivity, phenotypes not abrogated by cross-fosteri

267 suggested that the pathological spontaneous hyperactivity present in dystrophic retinas may contribu

268 Left stellate ganglion (LSG) hyperactivity promotes ischemia induced ventricular arrh

269 term for the syndrome-paroxysmal sympathetic hyperactivity (PSH)-and clear diagnostic criteria define

270 function results in consistent microcephaly, hyperactivity, reduced anxiety, and deficient spatial le

271 (2)) conferred increased risk of inattention-hyperactivity (relative risk [RR] 1.53, 95% CI 1.13-2.08

272 at a cloned alpha-like BiOctR also induced a hyperactivity response in Balanus cyprids mediated by th

273 The change in parent-rated inattention and hyperactivity scores over the first 3 months of MPH medi

274 monstrate that inhibition of mGluR5 corrects hyperactivity, seizures, and elevated de novo synaptic p

275 ay aberrant behavioral phenotypes, including hyperactivity, social interaction deficits, and increase

276 their activity in favor of pyramidal neuron hyperactivity: somatostatin-expressing and parvalbumin-e

277 on of cortical GABAergic neurons may lead to hyperactivity states such as seizures or contribute to t

278 main body cell fate determinant Eya, and Wnt hyperactivity strongly biases cells towards polar and st

279 1 years of age using a screening instrument (hyperactivity subscale of the Strength and Difficulties

280 odromal Syndromes, and for attention-deficit/hyperactivity, substance-related, and mood disorders.

281 rder (n = 25) demonstrated intrinsic sensory hyperactivity (suppressed posterior alpha power, source-

282 MPH-mediated improvements in inattention and hyperactivity symptom scores.

283 isk ratio [RR], 1.42; 95% CI, 1.25-1.62) and hyperactivity symptoms (RR, 1.31; 95% CI, 1.16-1.49), wh

284 sociated changes in ADHD inattentiveness and hyperactivity symptoms at 3 months.

285 nd to be common to both conduct problems and hyperactivity symptoms.

286 brain injury develop a state of sympathetic hyperactivity that can persist for weeks or months, cons

287 nitus may play a role in auditory and limbic hyperactivity, the non-auditory effects of blast and pot

288 m trajectories of 1) conduct disorder and 2) hyperactivity throughout childhood, identified using lat

289 episodes of dramatic behavioral changes from hyperactivity to "depression-like" behavior, suggestive

290 rect evidence linking locus coeruleus system hyperactivity to PTSD hyperresponsiveness is sparse.

291 These findings verify that NCC hyperactivity underlies FHHt but also reveal that NCC-de

292 ese were improved after blocking spontaneous hyperactivity using meclofenamic acid, a gap junction bl

293 failed to inhibit conditioned avoidance and hyperactivity using pharmacological and transgenic model

294 on (1.34 ng/bee) impaired locomotion, caused hyperactivity (velocity: +109%; time moving: +44%) short

295 emory, executive function, and attention and hyperactivity, were assessed at baseline and postinterve

296 on-free samples were characterized by limbic hyperactivity, whereas no such group differences were fo

297 gher in visual cortex of MAM rats (posterior hyperactivity), which might parallel perceptual problems

298 n Shank3 (Shank3B(-/-)) shows early cortical hyperactivity, which triggers increased SPN excitatory s

299 e-treated rats exhibited significant bladder hyperactivity with an increase in micturition frequency

300 from immunocomplex deposition due to B cell hyperactivity with increased risk for B cell lymphoma de