ライフサイエンスコーパス: hypercoagulable

1 A striking feature of hypercoagulable conditions is the focal nature of the re

2 nd intestinal function tests and no signs of hypercoagulable disease.

3 thrombocytopenia (HIT) is an immune-mediated hypercoagulable disorder caused by antibodies to platele

4 aspirin; 5 received warfarin; and 3 (2 whose hypercoagulable disorder was corrected and 1 with sarcoi

5 analyzing the association between inherited hypercoagulable disorders and thrombosis have shown conf

6 Acquired hypercoagulable disorders such as heparin-induced thromb

7 ous thrombosis and an increased incidence of hypercoagulable disorders.

8 t is suggested that alpha-profibrin may be a hypercoagulable fibrin precursor because it is converted

9 rillation (AF), suggesting the presence of a hypercoagulable or prothrombotic state.

10 95, on Holter monitoring in 2 of 192; and on hypercoagulable panel in 30 of 189 patients.

11 The hypercoagulable phenotype can be induced in younger chim

12 d Protein S deficiency demonstrates that the hypercoagulable phenotype may develop in the recipient o

13 mean age, 27 [16-46] years) had an abnormal hypercoagulable profile.

14 actors, including immobilization (P < .001), hypercoagulable state (P = .003), excess estrogen state

15 diagnosed as having Prinzmetal angina and a hypercoagulable state 3 years ago after an ST-elevation

16 ients with severe HIV-tuberculosis display a hypercoagulable state and activation of the endothelium,

17 Surgery precipitates a hypercoagulable state and increases the formation of can

18 nd birth, but invariably succumb to a severe hypercoagulable state and massive thrombosis after 3 wee

19 The mechanisms underlying this hypercoagulable state are complex.

20 urse was complicated by the development of a hypercoagulable state associated with circulating antica

21 ence for the existence of a prothrombotic or hypercoagulable state associated with this disease.

22 through 1997 revealed 215 patients who had a hypercoagulable state attributed to primary or secondary

23 Pregnancy represents a hypercoagulable state characterized by increased thrombi

24 For patients with a hypercoagulable state corrected by OLT, antithrombotic t

25 IA on platelets and monocytes to propagate a hypercoagulable state culminating in life-threatening th

26 chanisms that contribute to the genesis of a hypercoagulable state during intestinal inflammation rem

27 imulation is pivotal to understanding of the hypercoagulable state found in most cancer patients.

28 hemorheological indices may contribute to a hypercoagulable state in CHF, especially in female patie

29 Several risk factors predispose to the hypercoagulable state in human AF, but whether acute ons

30 In parallel, mechanistic studies of the hypercoagulable state in humans and mouse models implica

31 In vitro coagulation assays demonstrated a hypercoagulable state in obese/T2D mice that was compara

32 y which PF4/heparin antibodies may promote a hypercoagulable state in patients with HITT.

33 ogenesis, and accord with a prothrombotic or hypercoagulable state in this arrhythmia.

34 celerate platelet aggregation resulting in a hypercoagulable state in which the platelet surface beco

35 of infected patients, and it occurs due to a hypercoagulable state induced by the associated inflamma

36 O size, degree of shunting, and a coexisting hypercoagulable state may be additional risk factors.

37 rinogen gamma, a protein associated with the hypercoagulable state of pancreatic cancer, discriminate

38 pression of clfA that is associated with the hypercoagulable state of the host and increased virulenc

39 suggest the preponderant role of a systemic hypercoagulable state over an intrathoracic venous compr

40 Records in 23 patients with a hypercoagulable state related to APC resistance (18 wome

41 addition, PS exposure can play a role in the hypercoagulable state reported to exist in severe beta-t

42 icoagulation or other therapy to control the hypercoagulable state should be given.

43 lieve that severe beta-thalassemia induces a hypercoagulable state that could be partially caused by

44 deficiency has been hypothesized to cause a hypercoagulable state that could induce thrombosis in sm

45 Obesity promotes a chronic inflammatory and hypercoagulable state that drives cardiovascular disease

46 Activation of ECs by aPL may create a hypercoagulable state that precedes and contributes to t

47 wall or blood flow must be associated with a hypercoagulable state to produce thrombosis.

48 VO by 44% (HR, 1.44; 95% CI, 1.23-1.68), and hypercoagulable state was associated with a 145% increas

49 rombosis was documented in 9 patients, and a hypercoagulable state was identified in 10.

50 Stroke risk factors or other causes of a hypercoagulable state were found in 12 patients.

51 ce of antiphospholipid syndrome and no other hypercoagulable state were included if the observed thro

52 liver cirrhosis (LC) behaves as an acquired hypercoagulable state with increased thrombotic risk.

53 nt cell arteritis, carotid stenosis, stroke, hypercoagulable state, and DM with ophthalmic complicati

54 ssessed included immobilization, malignancy, hypercoagulable state, excess estrogen state, a history

55 onary embolism, peripheral vascular disease, hypercoagulable state, myocardial infarction) or anticoa

56 To investigate the etiology of this hypercoagulable state, we have used the Russell's viper

57 lity of band 3 null mice may be related to a hypercoagulable state, which appears to originate from c

58 normalities contribute to a prothrombotic or hypercoagulable state, which increases the risk of throm

59 lation is associated with a prothrombotic or hypercoagulable state, which may contribute to an increa

60 G-CSF induces a hypercoagulable state, which may predispose certain dono

61 EG demonstrated that both developed a marked hypercoagulable state, which was treated with low-molecu

62 otein synthesis subsequently contribute to a hypercoagulable state.

63 of other risk factors, may contribute to the hypercoagulable state.

64 and that a defective clearance can induce a hypercoagulable state.

65 atic and clinical factors contribute to this hypercoagulable state.

66 s have an increased risk of VTE because of a hypercoagulable state.

67 sociated with the generation of an intrinsic hypercoagulable state.

68 A subset of patients was evaluated for hypercoagulable state.

69 lling the requirement for a prothrombotic or hypercoagulable state.

70 Pregnancy is associated with a hypercoagulable state.

71 r disease risk factors and other causes of a hypercoagulable state.

72 activated protein C and leads to a relative hypercoagulable state.

73 se nature of the atherosclerotic disease and hypercoagulable state.

74 is believed to be the cause of the patient's hypercoagulable state.

75 Are ESRD patients with hypercoagulable states (HCS) (antithrombin III deficienc

76 ertension (HR = 1.62; 95% CI: 1.26-2.07) and hypercoagulable states (HR = 2.46; 95% CI: 1.51-4.00).

77 Congenital and acquired hypercoagulable states are reviewed here, with an emphas

78 Congenital and acquired hypercoagulable states arise from an imbalance between p

79 ce has implicated the two most common venous hypercoagulable states in ischemic heart disease.

80 Noncirrhotic patients with hypercoagulable states tend to develop PB when PVT exten

81 Hypercoagulable states were demonstrated in most episode

82 Hypercoagulable states were found upon reevaluating five

83 Hypercoagulable states were prospectively detected in 10

84 e apply the model to two clinically relevant hypercoagulable states, caused by deficiency of either a

85 ests for a variety of inherited and acquired hypercoagulable states, were carried out.

86 ension, hemoconcentration, hemodilution, and hypercoagulable states.

87 was observed in anticoagulated patients with hypercoagulable states.

88 ctically anticoagulating study patients with hypercoagulable states.

89 is used, clinicians should be aware of this hypercoagulable syndrome.

90 Antithrombin III deficiencies and hypercoagulable TEG parameters were prevalent among pati

91 mbin III and anti-Factor Xa deficiencies and hypercoagulable TEG parameters, including elevated coagu

92 ADAMTS-13 was an independent predictor of a hypercoagulable TEG profile and platelet count, endotoxi

93 elieve younger women with SVT should undergo hypercoagulable testing to identify the need for long-te

94 s; mean age, 32 [16-61] years) had undergone hypercoagulable testing.

95 who received 34 mg/m(2) were treated after a hypercoagulable work-up to exclude potential risk factor

96 ipient (four of seven patients with positive hypercoagulable workup).