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1 nologic conditions, and various forms of the hypereosinophilic syndrome.
2 atory diseases including allergic asthma and hypereosinophilic syndrome.
3 ive as a corticosteroid-sparing agent in the hypereosinophilic syndrome.
4 with no increase in clinical activity of the hypereosinophilic syndrome.
5 antibody, mepolizumab, in patients with the hypereosinophilic syndrome.
6 ents negative for FIP1L1-PDGFRA who have the hypereosinophilic syndrome.
7 d thrombotic diathesis characteristic of the hypereosinophilic syndrome.
8 eosinophilia-myalgia syndrome and idiopathic hypereosinophilic syndrome.
9 n eosinophilic leukemia and the 'idiopathic' hypereosinophilic syndromes.
10 eosinophilic leukemia and in the idiopathic hypereosinophilic syndrome also have been further define
11 ively controls eosinophilia in patients with hypereosinophilic syndrome and normal interleukin-5 conc
12 eceptor alpha (PDGFRalpha) as a cause of the hypereosinophilic syndrome and of chronic eosinophilic l
16 reported to be effective in the treatment of hypereosinophilic syndrome (HES) and a rare eosinophilia
17 ity in identifying a subset of patients with hypereosinophilic syndrome (HES) and an underlying myelo
25 cytic leukemia (CMML), 101 with MDS, 11 with hypereosinophilic syndrome (HES), 8 with systemic mastoc
26 ene has been identified as a cause of clonal hypereosinophilic syndrome (HES), called F/P-positive ch
39 esulting in the development of an idiopathic hypereosinophilic syndrome (IHES) with eosinophilic derm
41 asthma, allergic and parasitic diseases, and hypereosinophilic syndromes, in addition to more recentl
42 tail the diagnosis and management of various hypereosinophilic syndromes including the clonal eosinop
48 very of the FIP1L1-PDGFRA fusion gene in the hypereosinophilic syndrome is an example of the power of
49 or patients in whom no underlying disease or hypereosinophilic syndrome is found, the term hypereosin
51 pression was decided in four patients due to hypereosinophilic syndrome, parvovirus infection, aspira
54 Ulcerated lesions from patients with the hypereosinophilic syndrome showed ECP and EDN deposition
56 humans and dysregulated in individuals with hypereosinophilic syndrome, this long non-coding RNA may
58 ssful empiric treatment of patients with the hypereosinophilic syndrome with the selective tyrosine k
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