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1 ve been linked to psychosis-related dopamine hyperfunction.
2 us liver disease and correlates with splenic hyperfunction.
3 ompensate for damage both by hypertrophy and hyperfunction after renal injury or ablation, these chan
4 l photoreceptor development characterized by hyperfunction and excess of the minority S (short wavele
5 the GluN3A reactivation corrected the NMDAR hyperfunction and prevented the full range of HD signs a
7 y in the hippocampus and consequent dopamine hyperfunction; and fifth, deficits in corollary discharg
9 her characteristic findings of MAS including hyperfunctioning endocrinopathies, polyostotic fibrous d
11 ing parathyroid resection indicates multiple hyperfunctioning glands and to determine the appropriate
12 ocalize and confirm complete excision of all hyperfunctioning glands, was compared with group II, who
18 reatic beta-cell to establish a template for hyperfunction in early life and beta-cell failure with a
19 whereas patients with OCD showed larger and hyperfunctioning insular-striatal regions that may be po
22 an underlying cause and correcting hypo- or hyperfunction of an endocrine gland, however, can often
23 involved, and the earliest known deficit is hyperfunction of glutamate-type N-methyl-d-aspartate rec
24 ugs for the treatment of diseases related to hyperfunction of L-type channels, such as Torsades de Po
27 n a central hypernoradrenergic state and the hyperfunctioning of specific central CRH pathways that e
28 the impact of NMDA hypofunction and dopamine hyperfunction on OFC neurons, suggesting that these neur
29 t could predict the likelihood of additional hyperfunctioning parathyroid glands and let the surgeon
30 500 mg, there is a 9% chance for additional hyperfunctioning parathyroid glands based on the WIN nom
31 mogram predicts the likelihood of additional hyperfunctioning parathyroid glands during parathyroidec
33 tiglandular disease, Met-PET/CT identified 2 hyperfunctioning parathyroid glands in 1 patient, 1 glan
34 Although operative failure can be due to hyperfunctioning parathyroid glands in ectopic locations
36 sitive predictive value for the detection of hyperfunctioning parathyroid tissue in patients after su
39 o determine how constitutive NMDAR hypo- and hyperfunction, respectively, affect the glutamate recept
41 ntial treatments for diseases of parathyroid hyperfunction, such as primary and secondary hyperparath
44 rophysiology as a correlate for the dopamine hyperfunction thought to underlie psychosis in patients.
45 receptor have been identified as a cause of hyperfunctioning thyroid adenomas, and germline mutation
46 ate localization and confirm excision of all hyperfunctioning tissue, the success rate of reoperative
47 ulated, such as genetic factors, masticatory hyperfunction, trauma, and continued growth, but the und
48 hat they exhibited hippocampal NMDA receptor hyperfunction, which likely drives the enhanced LTP.
49 em, together with dorsal and fronto-striatal hyperfunctioning, which may reflect poor affect reactivi
50 pressed subchondral bone loss and osteoclast hyperfunction while beta-agonist (isoproterenol) exacerb
51 mechanisms that account for the compensatory hyperfunction with insulin resistance (so-called beta-ce
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