ライフサイエンスコーパス: hyperfunctioning

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1 for behavioral aberrations associated with a hyperfunctioning dopamine system.

2 her characteristic findings of MAS including hyperfunctioning endocrinopathies, polyostotic fibrous d

3 Surgery is an effective treatment for hyperfunctioning glands and benign and malignant tumours

4 ing parathyroid resection indicates multiple hyperfunctioning glands and to determine the appropriate

5 ocalize and confirm complete excision of all hyperfunctioning glands, was compared with group II, who

6 be indicative of the presence of additional hyperfunctioning glands.

7 y 50% and BIJ PTH successfully localized the hyperfunctioning glands.

8 30% of these cases are caused by one or two hyperfunctioning glands.

9 ity of these patients do not have additional hyperfunctioning glands.

10 and further exploration revealed additional hyperfunctioning glands.

11 whereas patients with OCD showed larger and hyperfunctioning insular-striatal regions that may be po

12 d by toxic multinodular goitre, and solitary hyperfunctioning nodules.

13 n a central hypernoradrenergic state and the hyperfunctioning of specific central CRH pathways that e

14 t could predict the likelihood of additional hyperfunctioning parathyroid glands and let the surgeon

15 500 mg, there is a 9% chance for additional hyperfunctioning parathyroid glands based on the WIN nom

16 mogram predicts the likelihood of additional hyperfunctioning parathyroid glands during parathyroidec

17 Radioguided resection of hyperfunctioning parathyroid glands has been shown to be

18 tiglandular disease, Met-PET/CT identified 2 hyperfunctioning parathyroid glands in 1 patient, 1 glan

19 Although operative failure can be due to hyperfunctioning parathyroid glands in ectopic locations

20 ately predicted the likelihood of additional hyperfunctioning parathyroid glands.

21 sitive predictive value for the detection of hyperfunctioning parathyroid tissue in patients after su

22 Preoperative localization of hyperfunctioning parathyroid tissue in patients with pri

23 receptor have been identified as a cause of hyperfunctioning thyroid adenomas, and germline mutation

24 ate localization and confirm excision of all hyperfunctioning tissue, the success rate of reoperative

25 em, together with dorsal and fronto-striatal hyperfunctioning, which may reflect poor affect reactivi

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