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1 erogeneous, but the lesion was predominantly hyperintense.
2 nsity that was isointense, heterogeneous, or hyperintense.
3  areas of regeneration appeared as hypo- and hyperintense.
4  had hyperintense rim; others were uniformly hyperintense.
5 hite matter on nonenhanced images and judged hyperintense.
6 8), T1-hypointense (17 of 18), and diffusion-hyperintense (15 of 15) lesions, with a sharp border tow
7 ), T2 or fluid-attenuated inversion recovery hyperintense (18 of 18), T1-hypointense (17 of 18), and
8                                     However, hyperintense (18)F-FDG uptake in the tumor, compared wit
9 congruent distribution of the two tracers or hyperintense activity on the leukocyte study, as compare
10                         It becomes enhanced (hyperintense) after contrast administration.
11 1-weighted MR images (n = 23), 18 cysts were hyperintense and five were isointense to cerebrospinal f
12  was compared with the number of alternating hyperintense and hypointense lines depicted.
13 uantitative (contrast-to-noise ratio between hyperintense and hypointense liver regions, coefficient
14  close correspondence between contours of T2-hyperintense and infarcted regions, and the transmural-e
15 ense; subgrade B, inhomogeneous; subgrade C, hyperintense; and subgrade D, hyperintense with swelling
16                                  T2-weighted hyperintense areas and microhemorrhages did not collocat
17 a high incidence of white matter T2-weighted hyperintense areas and pituitary abnormalities, with a l
18       A pelvic MRI revealed a mass including hyperintense areas on T1-weighted images and hypointense
19     The presence of white matter T2-weighted hyperintense areas was the most common pathologic findin
20 ltiple-layered appearances, with a prominent hyperintense band at the external surface of the cortex,
21    Magnetic resonance imaging studies showed hyperintense basal ganglia in 80% of patients with posts
22          Magnetic resonance imaging revealed hyperintense bilateral lesions in the dorsal brain stem
23 y neuroimaging outcome of having T2-weighted hyperintense brain lesions consistent with the 2010 McDo
24               The authors discussed a few T1-hyperintense brain lesions which did not include metasta
25 ere the infarcted muscles appeared diffusely hyperintense compared with adjacent muscles.
26 an diameter, 3.9 cm), including 38 benign T1 hyperintense cysts and 26 RCCs, were assessed.
27 ion with mixed-signal-intensity and multiple hyperintense droplets scattered through the cerebellar s
28 ic sources from catheter ablation can create hyperintense DWI punctate lesions in a canine model.
29 on MRI, i.e. as countless, tiny, slightly T1-hyperintense foci that did not enhance.
30 e could clearly differentiate viable glioma (hyperintense) from radiation necrosis (hypointense to is
31                    Retrospective analysis of hyperintense globus pallidus lesions in 16 children and
32       Significant ADC increases were seen in hyperintense globus pallidus lesions in the NF 1 group c
33                              Rate of iso- or hyperintense HCA on HPB phase gadoxetic acid-enhanced MR
34 were included to analyze the rate of iso- or hyperintense HCAs on HPB phase MR images.
35 , low-grade destruction of vertebral bodies, hyperintense/homogeneous signal from the vertebral bodie
36  (n = 7); hypointense, hyperintense (n = 2); hyperintense, hyperintense (n = 1).
37 ely: HCC, hyperintense, hypointense (n = 3); hyperintense, hyperintense (n = 1); hypointense, isointe
38 - and T2-weighted images, respectively: HCC, hyperintense, hypointense (n = 3); hyperintense, hyperin
39                                   Small HCC, hyperintense, hypointense (n = 7); hypointense, hyperint
40 Both dysplastic nodules with subfoci of HCC, hyperintense, hypointense.
41     All three high-grade dysplastic nodules, hyperintense, hypointense.
42 n nonsiderotic low-grade dysplastic nodules, hyperintense, hypointense.
43 cular pressure (IOP) elevation, CSS appeared hyperintense in both freshly prepared ovine eyes and liv
44 (particle-induced synovitis), lamellated and hyperintense (infection), and a homogeneous effusion wit
45                                          The hyperintense layer 1 closest to the vitreous likely cons
46 er and the inner and outer segments; and the hyperintense layer 3, the choroid.
47 magnetic resonance imaging detected a single hyperintense lesion in the left temporal lobe.
48 s or less from symptom onset, spinal cord T2-hyperintense lesion less than 3 vertebral segments, AQP4
49 ortion to the total cerebral T2-weighted MRI hyperintense lesion load.
50 trophy, disability, and advancing disease; a hyperintense lesion may be a clinically relevant biomark
51 ociation between miRNA and brain lesions (T2 hyperintense lesion volume [T2LV]), the ratio of T1 hypo
52 at baseline was correlated with enlarging T2-hyperintense lesion volumes over the subsequent year (rh
53                              At least one T1 hyperintense lesion was found in 113 patients (total, 34
54 he numbers of new or enlarging T(2)-weighted hyperintense lesions (all P<0.001) and new T(1)-weighted
55 daily BG-12), new or enlarging T(2)-weighted hyperintense lesions (both BG-12 doses), and new T(1)-we
56 =0.008, respectively) or new or enlarging T2 hyperintense lesions (each p<0.0001).
57 esions and of new or enlarging T(2)-weighted hyperintense lesions (P<0.001 for the comparison of each
58  In addition to gliomas and other tumors, T2 hyperintense lesions (unidentified bright objects or UBO
59 ed with MS, including perivenous T2-weighted hyperintense lesions and focal leptomeningeal enhancemen
60 cant ADC increases were measured both in the hyperintense lesions and in the normal-appearing areas o
61 n immunosuppressed patients, especially when hyperintense lesions are seen in the insular region and
62                                           T1 hyperintense lesions had lower ADCs compared with their
63 ity signal in globi pallidi in all patients; hyperintense lesions in midbrain were observed in three
64 ocations were performed, with and without T2-hyperintense lesions included.
65      Magnetic resonance imaging indicated T2-hyperintense lesions of her splenium.
66 ymphoma in the peripheral retina (n = 2) and hyperintense lesions on brain magnetic resonance imaging
67                  To retrospectively document hyperintense lesions on nonenhanced T1-weighted magnetic
68          The number of new or newly enlarged hyperintense lesions on T2-weighted magnetic resonance i
69      After adjustment for disease course, T1 hyperintense lesions remained associated with brain atro
70                                              Hyperintense lesions were observed on DWI (median maximu
71 hancing (Gd+) lesions or new or enlarging T2 hyperintense lesions) over 2 years in these trials.
72                            With exclusion of hyperintense lesions, significant ADC increases were mea
73                                We found that hyperintense (light) areas in MTC images were coextensiv
74 d computer simulations demonstrated that the hyperintense magnetic signal correlates with Abeta(1-42)
75 lso associated with increasing numbers of T2 hyperintense MRI lesions (OR = 2.36; 95% CI 1.21 to 4.59
76                                              Hyperintense MS plaques on T1-weighted MR images are com
77                          To test whether the hyperintense myocardium would exhibit partial functional
78  observed 2 days later, that the T2-weighted hyperintense myocardium would show partial functional re
79 intense, hyperintense (n = 2); hyperintense, hyperintense (n = 1).
80 rintense, hypointense (n = 3); hyperintense, hyperintense (n = 1); hypointense, isointense (n = 1).
81 erintense, hypointense (n = 7); hypointense, hyperintense (n = 2); hyperintense, hyperintense (n = 1)
82 ing: all lesions were hypointense on T2- and hyperintense (n=12) and isointense (n=6) on T1-weighted
83                                          T2w hyperintense nerve lesions were detectable in all MS pat
84              Inner retinal cell swelling was hyperintense on diffusion-weighted images at 3 hours and
85 1H images (tumors) or regions that were only hyperintense on fluid-attenuated inversion recovery (FLA
86                                Cast appeared hyperintense on nonenhanced T1-weighted images.
87   Gadolinium enhancement in lesions that are hyperintense on precontrast FLAIR images, such as intrap
88 e on T1-weighted images and iso- to slightly hyperintense on proton-density- and T2-weighted images.
89 showed exaggerated OC responses which became hyperintense on T(2)-weighted MRI at 24h.
90                      Melanotic melanomas are hyperintense on T1-weighted images because of paramagnet
91 on T2-weighted images and were predominantly hyperintense on T1-weighted images.
92                   Normal myocardium appeared hyperintense on T1-weighted inversion-recovery SE MR ima
93 es (from -4.87 +/- 6.1 to -1.79 +/- 5.7) and hyperintense on T2-weighted images (from 10.12 +/- 7.9 t
94 ges (from -5.77 +/- 5.9 to -7.8 +/- 6.8) and hyperintense on T2-weighted images (from 8.73 +/- 5.4 to
95 led multiple, disseminated lesions that were hyperintense on T2-weighted images and did not enhance a
96 mainly hypointense on T1-weighted images and hyperintense on T2-weighted images and significant restr
97 ghted images in all six patients and iso- to hyperintense on T2-weighted images in five patients.
98 atter immediately adjacent to the enhancing (hyperintense on T2-weighted images, but not enhancing on
99 ith the liver on T1-weighted images and were hyperintense on T2-weighted images.
100 the liver on T1-weighted images and slightly hyperintense on T2-weighted images.
101 t were hypointense on T1-weighted images and hyperintense on T2-weighted images.
102 rticularly in the setting of masses that are hyperintense on unenhanced MR images.
103 ed as benign at quantitative assessment were hyperintense on unenhanced MR images; all were diagnosed
104                  Fibroids were classified as hyperintense or hypointense relative to skeletal muscle
105 ncing fibroids selected for treatment had no hyperintense or hypointense signal intensity changes on
106 ly than have traditional measures such as T2 hyperintense or T1 hypointense lesion volumes.
107 rized with DW imaging than lesions that were hyperintense or well defined.
108 ism was denoted by the presence of multiple, hyperintense pleural spots on high-b-value DW images.
109 cclusion would be similar to the T2-weighted hyperintense region observed 2 days later, that the T2-w
110 Infarctlike lesion was defined as a nonmass, hyperintense region on spin-density- and T2-weighted ima
111 s consistent with meningioma and an adjacent hyperintense region on T2-weighted MR images were examin
112 LV) ischemic myocardium at risk (T2-weighted hyperintense region) early after myocardial infarction,
113 from isotropic DW images of enhancing tumor, hyperintense regions adjacent to enhancing tumor, normal
114 tionship between the transmural-extent of T2-hyperintense regions and that of the AAR (bright-blood-T
115 ultiregional (contrast-enhancing regions and hyperintense regions at nonenhanced fluid-attenuated inv
116 he difference in FA decreases in peritumoral hyperintense regions between these tumors approached but
117      Ablated areas of myocardium appeared as hyperintense regions directly adjacent to the catheter t
118 d the finding that the lateral borders of T2 hyperintense regions frequently extend far beyond that o
119 thology images were well correlated with the hyperintense regions measured on T1-weighted GRE images
120 s were also well correlated with the smaller hyperintense regions measured on those IR images with in
121  ADC maps, and (iii) visual determination of hyperintense regions on DWI.
122 ontralateral tissue, and 98 +/- 12 for FLAIR hyperintense regions surrounding tumors.
123                Mean FA values in peritumoral hyperintense regions were 0.178 (43% of normal WM value)
124                     Mean ADCs in peritumoral hyperintense regions were 1.309 x 10(-3) mm2/sec (mean p
125 lysis demonstrated a fingerprint match of T2-hyperintense regions with the intricate contour of infar
126 ncing tumor, normal-appearing WM adjacent to hyperintense regions, and analogous locations in the con
127 ancement ratio in the characterization of T1 hyperintense renal lesions, with both methods having low
128 ed in 41 patients with non-fat-containing T1 hyperintense renal lesions.
129                                            A hyperintense rim surrounding lesions on R2* maps was onl
130 : a central lesion with hypointense core and hyperintense rim with or without contrast enhancement; a
131                    Two-thirds of lesions had hyperintense rim; others were uniformly hyperintense.
132 On MRI there was mass showing both T1 and T2 hyperintense signal area suggestive of fat component.
133 plasty demonstrated an acellular zone with a hyperintense signal consistent with a mild interface opa
134 l CA3 [F(1,34) = 16.87, P < 0.0001], despite hyperintense signal evident in 5 of 18 patients on prese
135                       Diffusion restriction (hyperintense signal in DWI) was shown in the cortex of t
136 within the modular brain neuropil, revealing hyperintense signal in synapse-rich microdomains.
137 gra and globus pallidus, with a 'halo' of T1 hyperintense signal in the substantia nigra.
138           The T2 relaxometry demonstrated no hyperintense signal of the amygdala in any patient with
139                              An artificially hyperintense signal on FLAIR images can result from magn
140 , 74 cystic hemorrhagic adnexal lesions with hyperintense signal on T1-weighted images were identifie
141                                              Hyperintense signal on T1W MRI and comparison of axial T
142           Acute axonal nerve lesions cause a hyperintense signal on T2-weighted images at and distal
143         This was consistent with the lack of hyperintense signals in the anterior temporal pole and e
144            Most commonly, PRES presents with hyperintense signals on T2 and FLAIR sequences.
145    Thermal lesions appeared hypointense with hyperintense surrounding rims with all sequences in both
146 ries of patients, the presence of lamellated hyperintense synovitis at MR imaging of knee arthroplast
147                The sensitivity of lamellated hyperintense synovitis for infection was 0.86-0.92 (95%
148  radiologists for the presence of lamellated hyperintense synovitis.
149 hypointense band deeper in the cortex, and a hyperintense third band.
150 t test for both tumors and surrounding FLAIR hyperintense tissues versus GM, WM, CSF, and contralater
151                   All local recurrences were hyperintense to adjacent pelvic muscles on T2-weighted M
152 yma, 1 = isointense to brain parenchyma, 2 = hyperintense to brain parenchyma) by a pediatric neurora
153 images (n = 18), 17 cysts were isointense or hyperintense to cerebrospinal fluid.
154  to liver on T1-weighted images (n = 11) and hyperintense to liver on T2-weighted images (n = 10).
155 mages, the lesion was isointense or slightly hyperintense to muscle.
156                       On T1-weighted images, hyperintense tubal fluid was significantly correlated wi
157                          Neuroimaging showed hyperintense white matter abnormalities on T2 and fluid
158                                              Hyperintense white matter foci were seen on MR images in
159 assification into normal white matter and T2-hyperintense white matter hyperintensity volume was perf
160 nuated inversion recovery magnetic resonance hyperintense white matter voxels was performed using cer
161 s; subgrade C, hyperintense; and subgrade D, hyperintense with swelling.
162 bnormality (Modic changes), posterior anular hyperintense zone (HIZ), and facet arthropathy.
163  animals), was comparable to the size of the hyperintense zone on T2-weighted images 2 days later (43
164                      Edema, as detected by a hyperintense zone on T2-weighted images, resolved, and r

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