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1 the epidermis was markedly hyperplastic and hyperkeratotic.
2 ary school-aged female patient with bands of hyperkeratotic-affected skin on the upper and lower extr
5 sorder typified by dystrophic nails, painful hyperkeratotic calluses in glabrous skin, and lesions in
6 ntly causes oral lesions as well as PPK-like hyperkeratotic calluses on Krt16(-/-) front and hind paw
7 ated protein-1 (Slurp1), associated with the hyperkeratotic disorder mal de Meleda, is abundantly exp
9 Unmodified XLI keratinocytes regenerated a hyperkeratotic epidermis lacking STS expression with def
10 e produced that exhibited a hyperplastic and hyperkeratotic epithelium overlying a large dermal fibro
11 reeminent with UV-A-dependent phototoxicity, hyperkeratotic folliculitis, hand-foot skin reaction, ha
13 -93) years underwent gentle curettage of the hyperkeratotic lesions followed by aminolevulinic acid p
14 lesions, whereas 0 of 17 normal tissues or 2 hyperkeratotic lesions had detectable telomerase activit
15 tate PPKs are characterized by circumscribed hyperkeratotic lesions on the palms and soles with consi
16 th bullous ichthyosis in early childhood and hyperkeratotic lichenified plaques in the flexural areas
18 rhesus LCV infection in these proliferative, hyperkeratotic, or parakeratotic epithelial cell lesions
20 d indocyanine green (ICG) readily penetrated hyperkeratotic PC skin and normal callused regions compa
21 On day 4, ML.myc2 transgenic pups develop a hyperkeratotic phenotype, which progressively worsens un
22 rinatal lethality occurred due to the severe hyperkeratotic phenotype, which restricted movement.
23 focal and diffuse palmoplantar keratoderma, hyperkeratotic plaques on the trunk and limbs, varying d
24 diffuse, nonepidermolytic PPK and verrucous hyperkeratotic plaques over the joints and in flexures a
26 atal phenotype that consists of skin that is hyperkeratotic, scaly, and essentially devoid of fur.
27 ll and at 4-5 days of age develop patches of hyperkeratotic skin where no hair grows, resulting in a
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