ライフサイエンスコーパス: hyperkinetic

1 , the majority of which are intermittent and hyperkinetic.

2 inesia (n=5), parkinsonism (n=4), or a mixed hyperkinetic and hypokinetic movement disorder (n=2).

3 The presence of both hyperkinetic and hypokinetic movement disorders such as

4 Dyskinesias are hyperkinetic and involuntary movements that may result f

5 homologues of two HERG-interacting proteins, Hyperkinetic and K channel regulator 1 (KCR1), modify un

6 nesia (PNKD) is characterized by spontaneous hyperkinetic attacks that are precipitated by alcohol, c

7 in DNAH11, the only PCD gene known to cause hyperkinetic beat.

8 Behavioral symptoms included hyperkinetic behavior (100%), and a majority exhibited h

9 attenuated prepulse inhibition (males); and hyperkinetic behavior.

10 The sheep developed a hyperkinetic cardiovascular response concomitant with a

11 failure, there is a sympathetically mediated hyperkinetic cardiovascular response to exercise that li

12 Another with hyperkinetic ciliary beat had 2 mutations in DNAH11, the

13 sterone activation, and inflammation lead to hyperkinetic circulation, volume overload, and vascular

14 on deficit hyperactivity disorder (ADHD) and hyperkinetic disorder (HKD) are made on the basis of phe

15 genic mice raising the possibility that this hyperkinetic disorder has a unique pharmacogenetic profi

16 ion-deficit hyperactivity disorder (ADHD) or hyperkinetic disorder is currently unknown.

17 ars, who met diagnostic criteria for ADHD or hyperkinetic disorder, but not schizophrenia and autism,

18 attention-deficit/hyperactivity disorder or hyperkinetic disorder.

19 y (odds ratio, 2.71 [95% CI, 1.29-5.70]) and hyperkinetic disorders (2.19 [1.13-4.25]) were higher am

20 or of VMAT2 that is used in the treatment of hyperkinetic disorders associated with Huntington diseas

21 We also identified two patients with hyperkinetic disorders carrying the smallest SHANK3-span

22 se changes have been detected in humans with hyperkinetic disorders either via direct recordings from

23 ptor ligands for treatment of both hypo- and hyperkinetic disorders of basal ganglia origin, such as

24 ical and preclinical studies have shown that hyperkinetic disorders such as Huntington's disease, dys

25 increased risk for substance use disorders, hyperkinetic disorders, and mental retardation.

26 relative paucity of comprehensive reviews of hyperkinetic disorders, even though they are equally or

27 eedback-controlled neurostimulation to treat hyperkinetic disorders.

28 resently in use for symptomatic treatment of hyperkinetic disorders.

29 in the GPi is important in the mechanism of hyperkinetic disorders.

30 antibodies should be tested in patients with hyperkinetic encephalitis and neuropsychiatric prodrome

31 Previous studies have demonstrated hyperkinetic endocardial motion of noninfarcted myocardi

32 w that the Drosophila beta subunit homologue Hyperkinetic (Hk) associates with members of the ether g

33 Drosophila Hyperkinetic (Hk) mutations alter a gene encoding a homo

34 Mutants of the Shaker (Sh), Hyperkinetic (Hk), and ether a go-go (eag) genes, which

35 tassium (K(+)) channel beta-subunit (Kvbeta) Hyperkinetic (Hk).

36 tiated by a beta modulatory subunit coded by Hyperkinetic (Hk).

37 s will receive l-DOPA and eventually develop hyperkinetic involuntary movements termed dyskinesia.

38 neurodegenerative disorder characterized by hyperkinetic involuntary movements, including motor rest

39 thesized that dlBST could play a role in LID hyperkinetic manifestations.

40 These results are consistent with the hyperkinetic model, which suggests that hemiballismus re

41 Early-onset torsion dystonia is a hyperkinetic movement disorder caused by a deletion of o

42 lonus dystonia syndrome is a childhood onset hyperkinetic movement disorder characterized by predomin

43 individuals from two families affected by a hyperkinetic movement disorder due to homozygous mutatio

44 A common form of the hyperkinetic movement disorder dystonia is caused by mut

45 The paroxysmal hyperkinetic movement disorder phenocopies a form of dys

46 Chorea is a hyperkinetic movement disorder resulting from dysfunctio

47 et torsion dystonia is an autosomal dominant hyperkinetic movement disorder that has recently been li

48 sion dystonia (ITD) is an autosomal dominant hyperkinetic movement disorder with incomplete penetranc

49 ormal, but developed ataxia and a paroxysmal hyperkinetic movement disorder.

50 ologically dystonia most closely resembles a hyperkinetic movement disorder.

51 spose some individuals to the development of hyperkinetic movement disorders and seizures.

52 at endogenous mu agonists may play a role in hyperkinetic movement disorders by inducing sustained ac

53 The dystonias are a heterogeneous group of hyperkinetic movement disorders characterised by involun

54 s are still lacking, effective management of hyperkinetic movement disorders demands that physicians

55 therapy for the treatment of hypokinetic and hyperkinetic movement disorders has, however, led to an

56 Hyperkinetic movement disorders include tremors, dystoni

57 manifest in parkinsonism or a wide range of hyperkinetic movement disorders including chorea, ballis

58 nt for medication-refractory hypokinetic and hyperkinetic movement disorders, and it is being explore

59 on studies in animal models and humans with hyperkinetic movement disorders, it is postulated that d

60 treat medically intractable hypokinetic and hyperkinetic movement disorders, the course of the palli

61 ed to reverse these changes in patients with hyperkinetic movement disorders.

62 een shown to contribute to both epilepsy and hyperkinetic movement disorders.

63 ovel therapeutic target for the treatment of hyperkinetic movement disorders.

64 lly targeted treatments for chorea and other hyperkinetic movement disorders.

65 , has been extended to the treatment of many hyperkinetic movement disorders.

66 e receptor blocker, is used to treat several hyperkinetic movement disorders.

67 n the subthalamic nucleus (STN) results in a hyperkinetic movement syndrome, similar to the HD phenot

68 orphism, progressive psychomotor disability, hyperkinetic movements, and axial hypotonia with variabl

69 opathy, seizures, autonomic instability, and hyperkinetic movements.

70 indicate that SHANK3 overexpression causes a hyperkinetic neuropsychiatric disorder.

71 The hyperkinetic phase causes a drop in slo gene expression

72 Women had a greater hyperkinetic response than men in the noninfarct zone (S

73 test, and 2 subscores were extracted: (1) a hyperkinetic score, combining dystonia and chorea, and (

74 temperature and using hyperactivity mutants (Hyperkinetic, Shaker) demonstrates that the rate of this

75 ells also requires the K(+) channel subunits Hyperkinetic, Shaker, and ether-a-go-go.

76 Hyperkinetic states are common in human movement disorde

77 nt such as Parkinson's disease as well as to hyperkinetic states such as those induced by stimulant d

78 patients (18%) were classified as "mismatch-hyperkinetic." The mean LVEF for these patients signific