ライフサイエンスコーパス: hyperlipidaemia

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1 hand osteoarthritis (HOA) could be linked to hyperlipidaemia.

2 ducing the population burden associated with hyperlipidaemia.

3 tentiate the adverse effects of postprandial hyperlipidaemia.

4 verweight, insulin resistance, diabetes, and hyperlipidaemia.

5 uch as type-2 diabetes and familial combined hyperlipidaemia.

6 te cytoplasmic neutral lipid vesicles during hyperlipidaemia.

7 L receptor, a major determinant of inherited hyperlipidaemias.

8 Among controls 13% had untreated hyperlipidaemia, 11% were prescribed statins, 7% other L

9 hey may determine the degree of postprandial hyperlipidaemia, a known risk factor for the development

10 l1(-/-)Apoe(-/-) and Apoe(-/-)mice decreases hyperlipidaemia and atherosclerosis.

11 yndromes, type 2 diabetes, obesity, combined hyperlipidaemia and essential hypertension, are complex

12 oxidized lipids is an important link between hyperlipidaemia and fatty streak formation.

13 IV-1 have been linked with increased risk of hyperlipidaemia and hyperglycaemia.

14 sis and are widely used for the treatment of hyperlipidaemia and ischaemic heart disease.

15 a powerful therapeutic avenue to ameliorate hyperlipidaemia and protect from atherosclerosis.

16 idence for gross phenotypes such as obesity, hyperlipidaemia and type 2 diabetes.

17 proaches to understanding diabetes mellitus, hyperlipidaemias and heart disease in the general popula

18 ty-acid metabolism, with insulin resistance, hyperlipidaemia, and atherosclerosis as target diseases.

19 ed glucose tolerance and new-onset diabetes, hyperlipidaemia, and cardiovascular disease.

20 el-adjusted for smoking, sex, age, diabetes, hyperlipidaemia, and hypertension-hazard ratio 6.5, 0.9-

21 haemic heart disease, obesity, hypertension, hyperlipidaemia, and non-insulin-dependent diabetes mell

22 duals with a clinical diagnosis of untreated hyperlipidaemia; and a randomly selected group of other

23 plain the genetic basis of familial combined hyperlipidaemia, another familial lipid disorder in whic

24 of triglyceride-rich lipoproteins, and their hyperlipidaemia becomes progressively more severe with a

25 conventional risk factors (eg, hypertension, hyperlipidaemia, excessive alcohol intake, smoking, obes

26 Familial combined hyperlipidaemia (FCHL) is a common, multifactorial disor

27 We mapped the hyperlipidaemia gene (Hyplip1) to the distal portion of

28 such as obesity, polycystic ovarian disease, hyperlipidaemia, hypertension and atherosclerosis.

29 entional cardiovascular risk factors such as hyperlipidaemia, hypertension, and diabetes are common i

30 latter effect being of possible relevance to hyperlipidaemia in type 2 diabetes mellitus.

31 dy mass index for individuals with untreated hyperlipidaemia (odds ratio 0.72 [95% CI 0.45-1.14]), or

32 aim was to examine the functional effect of hyperlipidaemia on blood monocytes.

33 These data demonstrate distinct effects of hyperlipidaemia on the chemotaxis and cytoskeletal regul

34 compared with people who had no diagnosis of hyperlipidaemia or exposure to other lipid-lowering drug

35 ribed statins, 7% other LLAs, and 69% had no hyperlipidaemia or LLA exposure.

36 the underlying metabolic defects in primary hyperlipidaemia or to define the impact of diseases such

37 dent of the presence or absence of untreated hyperlipidaemia, or exposure to nonstatin LLAs.

38 c effects (hypersensitivity to asparaginase, hyperlipidaemia, osteonecrosis, asparaginase-associated

39 ely to have hypertension, diabetes mellitus, hyperlipidaemia, prior stroke, lacunes, deep microbleeds

40 n E, lipid peroxidation and liver enzymes in hyperlipidaemia rabbits were investigated.

41 It is likely that the hyperlipidaemia results from a mutation of a novel gene

42 artery disease, blood pressure, diabetes, or hyperlipidaemia, suggesting that this locus is specific

43 ption, uncontrolled high blood pressure, and hyperlipidaemia--that can be effectively addressed for i

44 Although hyperlipidaemia was not reduced in treated animals, end-

45 is is reproduced by either hyperglycaemia or hyperlipidaemia, which also increase tissue levels of UD

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