ライフサイエンスコーパス: hypermetabolism

1 regions with significant hypometabolism and hypermetabolism.

2 mal liver function, lipid abnormalities, and hypermetabolism.

3 glucose metabolism and amyloid-beta-related hypermetabolism.

4 creased browning of white adipose tissue and hypermetabolism.

5 l of malignant hyperthermia and heat-induced hypermetabolism.

6 at Abeta deposition directly caused reactive hypermetabolism.

7 s the dependable reversal of skeletal muscle hypermetabolism.

8 heral chemoreceptor gain is augmented during hypermetabolism.

9 entilation, was doubled during the period of hypermetabolism.

10 urn long-term oxandrolone treatment improves hypermetabolism and body composition.

11 A severe burn injury leads to marked hypermetabolism and catabolism, which are associated wit

12 study was to investigate activation-induced hypermetabolism and hyperemia by using a multifrequency

13 as being potentially associated with REE or hypermetabolism and hypometabolism after LTx.

14 s in accordance with the notion that glucose hypermetabolism and hypometabolism reflect fundamentally

15 th functional neuroimaging studies reporting hypermetabolism and increased regional cerebral blood fl

16 Hypermetabolism and malnourishment are common in the int

17 Burn trauma triggers hypermetabolism and muscle wasting via increased cellula

18 us in schizophrenia is characterized by both hypermetabolism and reduced size.

19 ropranolol during hospitalization attenuates hypermetabolism and reverses muscle-protein catabolism.

20 the role of the inflammasome in burn-induced hypermetabolism and, potentially, developing novel thera

21 e and regional distribution of inflammation (hypermetabolism) and tissue failure, apoptosis, or atrop

22 Because anorexia, hypermetabolism, and elevated cytokine levels are freque

23 body surface area), its effects on postburn hypermetabolism, and the long-term cosmetic and function

24 FDG-PET demonstrated an area of intense hypermetabolism, and wide surgical resection was perform

25 of therapeutic strategies that aim to blunt hypermetabolism appears warranted.

26 Sepsis and excessive hypermetabolism are also associated with protein catabol

27 cellular protection mechanisms and systemic hypermetabolism are initiated and controlled.

28 Hypermetabolism associated with severe burns was thought

29 CA1 hypermetabolism at baseline predicted hippocampal atroph

30 Focal glucose hypermetabolism at the level of cervical spinal cord com

31 The identification of unexpected foci of hypermetabolism at whole-body FDG PET may signal the pre

32 h scan was evaluated for abnormal unexpected hypermetabolism based on unusual location (ie, foci that

33 ignificantly associated with the presence of hypermetabolism before LTx and the cumulative dose of pr

34 s regulate inflammatory cytokines that cause hypermetabolism/catabolism via acute phase response, lea

35 response contributes to multiorgan failure, hypermetabolism, complications, and death.

36 r, may contribute to multiple organ failure, hypermetabolism, complications, and death.

37 d resting energy expenditure, but if and how hypermetabolism contributes to disease pathology is unkn

38 nd/or secondary effects such as hyperphagia, hypermetabolism, disturbed glucose homeostasis, altered

39 It has been recently shown that hypermetabolism does not completely resolve after healin

40 luid resuscitation, sepsis, immune function, hypermetabolism, early excision, wound healing, scar for

41 m were concomitant with a loss of lean mass, hypermetabolism, hepatic steatosis, dyslipidemia, and be

42 Hypermetabolism/hypometabolism were low but present at t

43 CT scans of 13 tumors showed intense diffuse hypermetabolism in 12 and response to therapy in all 12

44 Our main finding was hypermetabolism in a cluster comprising the bed nucleus

45 Hypermetabolism in a ventral emotional neural system dur

46 ectly the link between neuroinflammation and hypermetabolism in aged mice.

47 th the controversial observation of cerebral hypermetabolism in aging WT mice.

48 r previous finding of relative basal ganglia hypermetabolism in AIDS dementia complex (ADC) and to de

49 The role of hypermetabolism in cancer cachexia remains unclear.We st

50 so shows that TSC patients with ASDs display hypermetabolism in deep cerebellar structures, compared

51 owed a nearly identical pattern of hypo- and hypermetabolism in groups 1 and 2.

52 flammation, consistent with prior reports of hypermetabolism in inflammatory disorders.

53 Higher HOMA-IR predicted hypermetabolism in MCI-progressors and hypometabolism in

54 tudies that have reported anterior cingulate hypermetabolism in the disorder.

55 Conversely, they showed relative hypermetabolism in the right inferior, middle, and super

56 We also observed hypermetabolism in the same cluster in rats expressing c

57 Diffuse hypermetabolism in the subcortical and deep white matter

58 post hoc analyses, depressed patients showed hypermetabolism in these areas during both waking and NR

59 Age-dependent cortical hypermetabolism in WT mice relative to young animals age

60 y; OR: 1.48 (95% CI: 1.01, 2.17); P = 0.044].Hypermetabolism is correlated with clinical and biologic

61 Conversely, hypermetabolism is likely compensatory in regions where

62 Studying the role of regional hypermetabolism is needed to better understand its inter

63 with decreased proinflammatory mediators and hypermetabolism, leading to a significant shorter ICU st

64 These findings show that hippocampal hypermetabolism leads to atrophy in psychotic disorder a

65 This hypermetabolism may be an adaptive response to an inabil

66 Hypermetabolism might be detrimental in other structures

67 suggest that the hyperpnoea observed during hypermetabolism might be mediated by an increase in the

68 cytokine expression profile, organ function, hypermetabolism, muscle protein synthesis, incidence of

69 ess response to burn trauma, with a focus on hypermetabolism, muscle wasting, and stress-induced diab

70 he human brain, (18)F-FDG PET shows cerebral hypermetabolism of aged wild-type (WT) mice relative to

71 f PAS kinase is consistent with the reported hypermetabolism of PAS kinase-deficient mice, identifyin

72 skeletal muscle may account for the resting hypermetabolism of patients with HIV lipoatrophy.

73 mal metabolic brain network characterized by hypermetabolism of the basal ganglia, supplementary moto

74 We determined that hypermetabolism protected TAZ kd mice from weight gain.

75 While hypermetabolism remains an intriguing target for interve

76 process, but little is known about regional hypermetabolism, sometimes observed in the brain of pati

77 There were discrete areas of hypermetabolism suggestive of malignancy (positive) in 1

78 Surprisingly, despite the hypermetabolism, their body temperature is not elevated.

79 valuate the intracranial lesions for glucose hypermetabolism to suggest malignancy, mutiplicity of in

80 tic patients, compared with normometabolism, hypermetabolism was associated with a reduced median sur

81 poor response to escitalopram, while insula hypermetabolism was associated with remission to escital

82 In at-risk patients, hypermetabolism was found to begin in CA1 and spread to

83 Hypermetabolism was negatively correlated only with hypo

84 The presence of hypermetabolism was significantly associated with the pr

85 imary lesion, 58 abnormal unexpected foci of hypermetabolism were identified in 53 patients.

86 ve blood loss, significant fluid shifts, and hypermetabolism, which alter the pharmacokinetics of man

87 ine reproduced a similar regional pattern of hypermetabolism, while repeated exposure shifted the hip

88 association between brain hypometabolism and hypermetabolism with motor scores of patients with early