ライフサイエンスコーパス: hypermobility

1 , using a cutoff of >/=6 for the presence of hypermobility.

2 t have the orthopedic manifestation of joint hypermobility.

3 sess associations of specific variables with hypermobility.

4 e encompassed by the seemingly trivial term, hypermobility.

5 y account for some subgroups of benign joint hypermobility.

6 dition, a positive stress test, and urethral hypermobility.

7 sex, hand pain, chondrocalcinosis, and hand hypermobility.

8 ective tissue abnormalities, including joint hypermobility.

9 s also evaluated to assess lesser degrees of hypermobility.

10 up of the hand in association with articular hypermobility.

11 rome, are characterized by generalized joint hypermobility.

12 Hypermobility, a largely unacknowledged though epidemiol

13 Hypermobility, also termed ligamentous laxity, may prese

14 Hypermobility among children in the cohort (mean age 13.

15 ificant negative association between present hypermobility and age was observed.

16 o previous studies showing an association of hypermobility and CMC OA, in this cohort there was no ev

17 der larvae prolonged feeding, and suppressed hypermobility and cooperative burrowing behaviors.

18 The association of hypermobility and DIP OA was not evaluated, because evid

19 e of these procedures in women with urethral hypermobility and genuine stress incontinence seems clea

20 hat there is an inverse relationship between hypermobility and hand and knee OA, and that hypermobili

21 Hypermobility and hypermobility syndrome are common cond

22 We assessed hypermobility and its relationship to radiographic hand

23 haracterized by an association between joint hypermobility and musculoskeletal pains, the latter occu

24 ts (OR 1.57) (P=0.037 for the interaction of hypermobility and obesity).

25 evaluated the association between articular hypermobility and primary OA.

26 The association of hypermobility and radiographic OA of the PIP, CMC, and m

27 have evaluated the association of articular hypermobility and radiographic osteoarthritis (OA) in hu

28 , chest wall deformity, dural ectasia, joint hypermobility, and acetabular protrusion.

29 rs of older larvae, including food aversion, hypermobility, and cooperative burrowing.

30 is leading to premature loss of teeth, joint hypermobility, and mild skin findings.

31 A self-report questionnaire on joint hypermobility as well as data on age, height, weight, es

32 intrinsic sphincter deficiency and urethral hypermobility, assessing symptom severity and predicting

33 They may observe the hypermobility but fail to appreciate its significance in

34 ide hope that more common varieties of joint hypermobility can be understood and that effective thera

35 Recognition of hypermobility combined with a thorough patient evaluatio

36 tissue/myopathy overlap disorders with joint hypermobility, contractures, mild skeletal dysplasia and

37 levels and extensive joint radiographic and hypermobility data were also available for the GOGO coho

38 6,022 children evaluated, the prevalence of hypermobility (defined as a Beighton score of >/=4 [i.e.

39 we performed hand and knee examinations and hypermobility evaluations (Beighton criteria) and obtain

40 dy demonstrated a joint-protective effect of hypermobility for radiographic OA of PIP joints.

41 compelling new knowledge is the finding that hypermobility, if sought, is the most common finding amo

42 describe the point prevalence and pattern of hypermobility in 14-year-old children from a population-

43 as Morquio syndrome or achondroplasia, have hypermobility in a more limited distribution.

44 erum HA levels did not differ in relation to hypermobility in either cohort.

45 suggestion of a positive association between hypermobility in girls and variables including physical

46 ased; however, the mechanisms underlying Ty1 hypermobility in most rtt mutants are poorly characteriz

47 othesis that COMP levels are associated with hypermobility in patients with OA and individuals withou

48 ors have a substantial contribution to joint hypermobility in the adult female population.

49 or the past was based on recall of childhood hypermobility in the first 2 decades of life.

50 ither checkpoint pathway is required for Ty1 hypermobility in two rtt mutants that are competent for

51 We have shown that the prevalence of hypermobility in UK children is high, possibly suggestin

52 DMT induced hypermobility in wild-type mice but not in sigma-1 recep

53 criteria for generalized ligamentous laxity (hypermobility) in children are widely used, their validi

54 ie classic Ehlers-Danlos syndrome, and joint hypermobility is an important clinical manifestation.

55 hypermobility and hand and knee OA, and that hypermobility is associated with lower serum COMP levels

56 In rtt101delta mutants, hypermobility is stimulated through the DNA damage pathw

57 Joint hypermobility is widely prevalent in all communities yet

58 ive tissue, manifesting as early-onset joint hypermobility, joint contractures, muscle weakness and b

59 Extensive joint hypermobility, lower serum cartilage oligomeric matrix p

60 ogenesis and how a history of cervical spine hypermobility may be a needed predisposing physical char

61 In addition, hypermobility may be associated with collagen disorders

62 A clinical examination revealed hypermobility of the upper lip and absence of generalize

63 ence of slight vertical maxillary excess and hypermobility of the upper lip.

64 ssociated with vertical maxillary excess and hypermobility of the upper lip.

65 Heightened awareness of subtle hypermobility or symptomatic joint laxity on physical ex

66 an abnormal joint phenotype, similar to the hypermobility phenotype in classic Ehlers-Danlos syndrom

67 atologists to accept the challenges posed by hypermobility-related disorders, which have, in the past

68 Many conditions that cause joint hypermobility resolve with nonoperative management, but

69 nherited form of EDS (characterized by joint hypermobility, skin hyperextensibility, and cardiac valv

70 Joint hypermobility syndrome (JHS) is characterized by an asso

71 Hypermobility and hypermobility syndrome are common conditions with univer

72 Benign joint hypermobility syndrome, for example, shares similar auto

73 management of joint problems associated with hypermobility syndrome.

74 families, one of which diagnosed with joint hypermobility syndrome/Ehlers-Danlos syndrome hypermobil

75 In persons with hypermobility, the odds of OA in PIP joints was lower (f

76 ords of patients with Ehlers-Danlos Syndrome hypermobility type (HEDS), including demographic informa

77 ypermobility syndrome/Ehlers-Danlos syndrome hypermobility type (JHS/EDS-HT), characterized by idiopa

78 excluded, and for at least some cases of the hypermobility type of EDS, a condition marked by gross j

79 The prevalence of hypermobility was 13% in the CARRIAGE family and 5% in t

80 The overall prevalence of hypermobility was 19.5% in MZ twins and 22.1% in DZ twin

81 GO subsets without radiographic OA, in which hypermobility was also associated with a significantly r

82 Tooth hypermobility was also present.

83 In the CARRIAGE family, hypermobility was associated with a significantly reduce

84 Hypermobility was defined as a Beighton score of >/=4, a

85 dy mass index-adjusted heritability of joint hypermobility was estimated to be 70% (95% confidence in

86 Whereas large-joint hypermobility was more common, lens dislocation and reti

87 Significantly greater concordance for joint hypermobility was observed in the MZ twins when compared

88 The Beighton criteria for assessment of hypermobility were recorded on a 0-9-point scale.

89 The lower odds of PIP OA with hypermobility were significant after adjusting for sex a

90 sion was used to examine the relationship of hypermobility with radiographic OA in each joint group,