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1 each other and stimulated by dehydration and hyperosmolarity.
2 elial cell function is adversely affected by hyperosmolarity.
3 esulted from the sustained hyperglycemia and hyperosmolarity.
4 stress and was maintained through 360 min of hyperosmolarity.
5 d that the autophagic flux was unaffected by hyperosmolarity.
6 mass spectrometry, increased in response to hyperosmolarity.
7 exposing confluent endothelial monolayers to hyperosmolarity.
8 xpressing wild-type ASGP-Rs was inhibited by hyperosmolarity.
9 there was normal inhibition by forskolin and hyperosmolarity.
10 pathways mediate the responses to touch and hyperosmolarity.
11 nic and, to an even greater degree, by acute hyperosmolarity.
12 NODM), comprising ketoacidosis (334, 8.1%), hyperosmolarity (131, 3.2%), renal complications (1,286,
15 l conditions encountered in the lung cavity (hyperosmolarity, acidic pH, and low oxygen tension, amon
24 h conditions found in human tissues, such as hyperosmolarity and presence of aminoglycoside antibioti
25 n kinase (ERK) was diminished in response to hyperosmolarity and serum factors in MEKK1(-/-) cells.
26 These experiments provide a link between hyperosmolarity and tear instability, suggesting that hy
30 ose biosynthesis, hydrophilins, responses to hyperosmolarity, and hypersalinity, reactive oxygen spec
32 ctivation, which can be induced by oxidants, hyperosmolarity, and proinflammatory cytokines, leading
36 tivated by UV-C irradiation, heat shock, and hyperosmolarity as well as by tumor necrosis factor alph
37 hough BetT activity increased in response to hyperosmolarity, BetT mediated significant uptake under
38 utations in osm-10 eliminate the response to hyperosmolarity but have no effect on responses to nose
39 fically by methylmethane sulfonate (MMS) and hyperosmolarity but not by ultraviolet radiation, ionizi
42 ast, the activation of the stress kinases by hyperosmolarity, by the DNA-cross-linking agent diepoxyb
44 guanylyl cyclase assays indicated that acute hyperosmolarity decreased NPR-B activity in a reversible
47 We conclude that in lung venular capillaries hyperosmolarity deteriorates barrier properties, possibl
50 ophagy in NP cells was not TonEBP-dependent; hyperosmolarity did not upregulate autophagy as previous
56 osmosensor involved in the regulation of the hyperosmolarity glycerol mitogen-activated protein kinas
57 orylated on Ser-38 and Ser-63 in response to hyperosmolarity, heat shock, and arsenite treatment but
58 n of neurotransmitter release was induced by hyperosmolarity, high potassium, or action potential fir
60 tant role in cell resistance and adaption to hyperosmolarity in many tissues like kidney and liver.
65 uroursodeoxycholate (TUDC) and cAMP reversed hyperosmolarity-induced Fyn activation and triggered re-
68 ause p38 and p42/44 inhibition prevented the hyperosmolarity-induced increase in IL-8 production.
69 ficient mutant of focal adhesion kinase, the hyperosmolarity-induced increases in activity of focal a
70 monstrate that PS protects human cornea from hyperosmolarity-induced inflammation and oxidative stres
72 diated ocular surface disease, inhibited the hyperosmolarity-induced MMP production and JNK activatio
76 pal slices activates p38 and JNK and whether hyperosmolarity is a potential apoptotic stimulus in thi
80 of 0 mm/5' in both eyes, accompanied by tear hyperosmolarity, mild meibomian gland dysfunction, reduc
84 However little is known about the effects of hyperosmolarity on short term regulation of the Na(+)-ta
88 physiological derivatives (such as oxidants, hyperosmolarity, or glycation products) on tissues direc
89 r dysfunctions by multiple factors including hyperosmolarity, oxidant formation, and protein kinase C
91 n culture study supported that extracellular hyperosmolarity plays no role in promoting autophagy in
94 We conclude that a brief period of vascular hyperosmolarity protects against acid-induced ALI when t
95 hese studies provide the first evidence that hyperosmolarity regulates TauT activity and expression i
96 e to our knowledge that in lung capillaries, hyperosmolarity remodels the endothelial barrier and the
97 the combination of low O2 concentration and hyperosmolarity resulted in an approximate 10- to 15-fol
98 was designed to evaluate the effect of donor hyperosmolarity secondary to diabetes insipidus, an almo
99 rophil induced either by stress stimuli (UV, hyperosmolarity, sphingosine) or by anti-Fas antibody or
100 factor, nuclear factor (NF)-kappaB, because hyperosmolarity stimulated both NF-kappaB DNA binding an
101 protein kinases, which effect contributed to hyperosmolarity-stimulated IL-8 production, because p38
104 instability produce transient shifts in tear hyperosmolarity that lead to chronic epithelial stress,
107 nts with normal osmolarity (<312 mOsm/L) and hyperosmolarity values (>/=312 mOsm/L) had respective OS
109 In addition, the proinflammatory effects of hyperosmolarity were, in a large part, mediated by activ
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