1 l lead to conditions such as osteoporosis or hyperostosis.

2 ion, and the combined outcome is generalized hyperostosis.

3 xyvitamin D, soft tissue calcifications, and hyperostosis.

4 lies that features generalized craniotubular hyperostosis.

5 lastogenesis contributes at least in part to hyperostosis.

6 last activity in diffuse idiopathic skeletal hyperostosis and bone loss in diabetics.

7                           Infantile cortical hyperostosis (Caffey disease) is characterized by sponta

8 e, osteoporosis, diffuse idiopathic skeletal hyperostosis, crystalline arthropathy, neuropathic arthr

9 ly affected with diffuse idiopathic skeletal hyperostosis (DISH) and/or chondrocalcinosis, were ident

10 uchem (VB) disease, two generalized skeletal hyperostosis disorders that result from hyperactive Wnt

11                We have encountered a case of hyperostosis frontalis interna in which the uptake of la

12                                              Hyperostosis frontalis interna is the term used to descr

13  mineralization (familial tumoral calcinosis/hyperostosis hyperphosphatemia syndrome, hypophosphatemi

14 CMD) is a rare genetic disorder encompassing hyperostosis of craniofacial bones and metaphyseal widen

15 plasia (CMD) is a rare genetic disorder with hyperostosis of craniofacial bones and widened metaphyse

16  that the incisor phenotype is likely due to hyperostosis of mandibles, which distinguishes Ank (KI/K

17 s with renal osteodystrophy developed marked hyperostosis of the facial and cranial bones.

18  they had experienced an episode of cortical hyperostosis or not, had joint hyperlaxity, hyperextensi

19 SAPHO syndrome (synovitis, acne, pustulosis, hyperostosis, osteitis) is an inflammatory disorder of t

20 in response was established and the onset of hyperostosis that can be suppressed with a chemical chap