ライフサイエンスコーパス: hyperperfusion

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1 ing patients vulnerable to cerebral hypo- or hyperperfusion.

2 xic edema, partial contrast enhancement, and hyperperfusion.

3 e interval 0.4% to 2.6%]) patients developed hyperperfusion.

4 and thalamus without cortical or cerebellar hyperperfusion.

5 n after donor nephrectomy is attributable to hyperperfusion and hypertrophy of the remaining glomerul

6 nce, etiology, and prevention strategies for hyperperfusion and ICH following CAS.

7 the ability of these endpoints to detect HCC hyperperfusion and, thereby, evaluated the suitability i

8 Although interictal hypoperfusion and ictal hyperperfusion are established localizing findings in pa

9 scular instability and cerebral ischemia and hyperperfusion are high, and anesthesia management shoul

10 Regions with ictal hyperperfusion are suggested to reflect seizure onset an

11 Identifying this pattern of hyperperfusion as typical for mesial temporal onset seiz

12 btractions were segmented to show regions of hyperperfusion at 1 SD above the mean.

13 ectomy (PH) in patients is leading to portal hyperperfusion but reduced hepatic arterial perfusion (H

14 the ictal studies, and three showed regional hyperperfusion corresponding to the hyperperfused region

15 usion" (CPP<CPPopt), severe disability with "hyperperfusion" (CPP>CPPopt), and favorable outcome was

16 Moreover, hyperperfusion did not correct the abnormal bioenergetic

17 ted as positive for osteomyelitis when focal hyperperfusion, focal hyperemia and focal bony uptake on

18 e same risk factors are involved in cerebral hyperperfusion following carotid endarterectomy.

19 Indeed, within 3 months after symptom onset, hyperperfusion had a positive predictive value of 88% fo

20 EG) monitored interictal SPECT (IISPECT) and hyperperfusion in immediate postictal or periictal SPECT

21 ly (P = 0.11) so; (b) the lower incidence of hyperperfusion in PISPECT in our series was due to the o

22 ess often concordant with the EEG focus than hyperperfusion in PISPECT, but not significantly (P = 0.

23 , AA showed increased grey matter volume and hyperperfusion in right posterior neocortical areas impl

24 ocalized MTLE most commonly show a region of hyperperfusion in the anterior temporal region, which of

25 uring typical gelastic seizures demonstrated hyperperfusion in the hamartomas, hypothalamic region, a

26 erfusion that corresponded to the regions of hyperperfusion in the ictal studies, and three showed re

27 emission computed tomography revealed focal hyperperfusion in the region of the cerebellar mass.

28 were measured under basal conditions, during hyperperfusion induced by pharmacological vasodilation w

29 sing HAP need to be evaluated to reverse the hyperperfusion-induced impairment of the spontaneous cou

30 Significant hyperperfusion is uncommon, even at a time when conventi

31 NF), air trapping/hypoperfusion (AT), normal/hyperperfusion (NOR), and bulla/cysts (BUL).

32 cardiac arrhythmic death and one death from hyperperfusion-related intracerebral hemorrhage.

33 Cerebral hyperperfusion syndrome (CHS) is an important complicati

34 Hyperperfusion syndrome and ICH can complicate carotid r

35 The study defined the incidence of cerebral hyperperfusion syndrome and intracranial hemorrhage (ICH

36 epartment to identify patients who developed hyperperfusion syndrome and/or ICH.

37 The hyperperfusion syndrome occurs infrequently following CA

38 attern of interictal hypoperfusion and ictal hyperperfusion that has been observed in subjects with e

39 ean rCBV, mean leakage coefficient K(2), and hyperperfusion volume (HPV), which is the fraction of th

40 The presence of hyperperfusion was inversely related to the occurrence o

41 Contralateral cerebellar hyperperfusion was observed in all cases.

42 This hyperperfusion was quantified by measuring the fraction

43 The most-contiguous area of hyperperfusion was the anterior temporal area extending

44 d quantitative FMT signal, denoting synovial hyperperfusion, was used to differentiate between synovi

45 We observed intense hyperperfusion within and at the edge of progressive mul

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